Microparticles as Potential Mediators of High Glucose-Induced Renal Cell Injury

scientific article published on 06 August 2019

Microparticles as Potential Mediators of High Glucose-Induced Renal Cell Injury is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.3390/BIOM9080348
P932PMC publication ID6723350
P698PubMed publication ID31390845

P50authorShankar MunusamyQ57323497
P2093author name stringAbdelali Agouni
Mazhar Pasha
Sreenithya Ravindran
P2860cites workEndoplasmic Reticulum Stress in the Diabetic Kidney, the Good, the Bad and the UglyQ26799567
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Molecular Mechanisms Underpinning Microparticle-Mediated Cellular Injury in Cardiovascular Complications Associated with DiabetesQ64237035
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Glycemic control and critical illness: is the kidney involved?Q80683171
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High glucose increases the formation and pro-oxidative activity of endothelial microparticlesQ38432562
Endoplasmic reticulum stress in kidney function and disease.Q38518567
Exosomes from high glucose-treated glomerular endothelial cells trigger the epithelial-mesenchymal transition and dysfunction of podocytesQ38609396
Quercetin inhibits the mTORC1/p70S6K signaling-mediated renal tubular epithelial-mesenchymal transition and renal fibrosis in diabetic nephropathyQ38855746
Effect of BMP7 on podocyte transdifferentiation and Smad7 expression induced by hyperglycemiaQ38860770
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Nephroprotective Effects of Metformin in Diabetic Nephropathy.Q38954169
Markers of endothelial damage in patients with chronic kidney disease on hemodialysis.Q39026344
miR-21-containing microvesicles from injured tubular epithelial cells promote tubular phenotype transition by targeting PTEN proteinQ39104766
The Role of Endoplasmic Reticulum Stress in Diabetic NephropathyQ39167559
AMP-activated protein kinase inhibits TGF-β-, angiotensin II-, aldosterone-, high glucose-, and albumin-induced epithelial-mesenchymal transitionQ39209583
Angiotensin AT1 receptor activation mediates high glucose-induced epithelial-mesenchymal transition in renal proximal tubular cells.Q39686680
Phosphatidylinositol 3-kinase and xanthine oxidase regulate nitric oxide and reactive oxygen species productions by apoptotic lymphocyte microparticles in endothelial cells.Q40000297
Diabetic Kidney Disease: Challenges, Progress, and PossibilitiesQ40206755
Connective tissue growth factor plays an important role in advanced glycation end product-induced tubular epithelial-to-mesenchymal transition: implications for diabetic renal disease.Q40242733
Distribution of glucose transporters in renal diseasesQ41570521
Alteration of circulatory platelet microparticles and endothelial microparticles in patients with chronic kidney diseaseQ43156601
Activation of tubular epithelial cells in diabetic nephropathyQ44230963
Temporal Cross Talk Between Endoplasmic Reticulum and Mitochondria Regulates Oxidative Stress and Mediates Microparticle-Induced Endothelial Dysfunction.Q46521138
A more tubulocentric view of diabetic kidney disease.Q47108081
The Emerging Roles of Microparticles in Diabetic NephropathyQ47112260
Metformin attenuates albumin-induced alterations in renal tubular cells in vitroQ48048212
Therapeutic application of extracellular vesicles in kidney disease: promises and challengesQ48105132
Berberine attenuates podocytes injury caused by exosomes derived from high glucose-induced mesangial cells through TGFβ1-PI3K/AKT pathwayQ49602918
Podocyte-derived microparticles promote proximal tubule fibrotic signaling via p38 MAPK and CD36.Q49805134
Circulating microparticles levels are increased in patients with diabetic kidney disease: A case-control researchQ50154686
P275copyright licenseCreative Commons Attribution 4.0 InternationalQ20007257
P6216copyright statuscopyrightedQ50423863
P433issue8
P577publication date2019-08-06
P1433published inBiomoleculesQ23929963
P1476titleMicroparticles as Potential Mediators of High Glucose-Induced Renal Cell Injury
P478volume9

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Q92535740Catalpol Ameliorates Podocyte Injury by Stabilizing Cytoskeleton and Enhancing Autophagy in Diabetic Nephropathycites workP2860

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