scholarly article | Q13442814 |
P2093 | author name string | Anthony J Davis | |
Huiming Lu | |||
Eric A Hendrickson | |||
Janapriya Saha | |||
Pauline J Beckmann | |||
P2860 | cites work | Postreplicative Joining of DNA Double-Strand Breaks Causes Genomic Instability in DNA-PKcs–Deficient Mouse Embryonic Fibroblasts | Q59018430 |
The ATM protein kinase: regulating the cellular response to genotoxic stress, and more | Q38088935 | ||
Chromatin remodeling at DNA double-strand breaks | Q38090114 | ||
The C-terminal conserved domain of DNA-PKcs, missing in the SCID mouse, is required for kinase activity | Q38314660 | ||
Endogenous DNA Damage as a Source of Genomic Instability in Cancer | Q39130435 | ||
ATM, ATR, and DNA-PK: The Trinity at the Heart of the DNA Damage Response | Q39378761 | ||
ATM-dependent and -independent dynamics of the nuclear phosphoproteome after DNA damage | Q39622746 | ||
Chromatin relaxation in response to DNA double-strand breaks is modulated by a novel ATM- and KAP-1 dependent pathway | Q40252252 | ||
DNA-PK is activated by nucleosomes and phosphorylates H2AX within the nucleosomes in an acetylation-dependent manner | Q40315391 | ||
Complex H2AX phosphorylation patterns by multiple kinases including ATM and DNA-PK in human cells exposed to ionizing radiation and treated with kinase inhibitors | Q40511617 | ||
ATM and DNA-PK function redundantly to phosphorylate H2AX after exposure to ionizing radiation | Q40570996 | ||
Unraveling the complexities of DNA-dependent protein kinase autophosphorylation | Q40603571 | ||
DNA-dependent protein kinase stimulates an independently active, nonhomologous, end-joining apparatus | Q40892156 | ||
Single-stranded DNA orchestrates an ATM-to-ATR switch at DNA breaks | Q42021074 | ||
Cell cycle-dependent phosphorylation regulates RECQL4 pathway choice and ubiquitination in DNA double-strand break repair | Q47094825 | ||
Meta-analysis of DNA double-strand break response kinetics | Q47154064 | ||
Super-resolution imaging identifies PARP1 and the Ku complex acting as DNA double-strand break sensors | Q49977725 | ||
Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair | Q21145054 | ||
DNA ligase III and DNA ligase IV carry out genetically distinct forms of end joining in human somatic cells | Q24297891 | ||
ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage | Q24306743 | ||
KAP1, a novel substrate for PIKK family members, colocalizes with numerous damage response factors at DNA lesions | Q24337449 | ||
XRCC3 promotes homology-directed repair of DNA damage in mammalian cells | Q24597771 | ||
The DNA-damage response in human biology and disease | Q24606586 | ||
Dynamics of DNA damage response proteins at DNA breaks: a focus on protein modifications | Q24608343 | ||
Essential role for DNA-PKcs in DNA double-strand break repair and apoptosis in ATM-deficient lymphocytes | Q24649590 | ||
Common mechanisms of PIKK regulation | Q24653672 | ||
DNA-PK: a dynamic enzyme in a versatile DSB repair pathway | Q26866928 | ||
The DNA Damage Response: Making It Safe to Play with Knives | Q27861055 | ||
The catalytic subunit of DNA-dependent protein kinase regulates proliferation, telomere length, and genomic stability in human somatic cells | Q28115800 | ||
Human LIGIV is synthetically lethal with the loss of Rad54B-dependent recombination and is required for certain chromosome fusion events induced by telomere dysfunction | Q28118848 | ||
DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139 | Q28131715 | ||
MDC1 maintains genomic stability by participating in the amplification of ATM-dependent DNA damage signals | Q28292900 | ||
Autophosphorylation at serine 1981 stabilizes ATM at DNA damage sites | Q33589782 | ||
Dynamics of the PI3K-like protein kinase members ATM and DNA-PKcs at DNA double strand breaks | Q33602897 | ||
Site-specific phosphorylation dynamics of the nuclear proteome during the DNA damage response | Q33883063 | ||
Requirement for the kinase activity of human DNA-dependent protein kinase catalytic subunit in DNA strand break rejoining | Q33958394 | ||
BRCA1 modulates the autophosphorylation status of DNA-PKcs in S phase of the cell cycle | Q34313012 | ||
The DNA damage response: implications for tumor responses to radiation and chemotherapy | Q34449481 | ||
Opposing ISWI- and CHD-class chromatin remodeling activities orchestrate heterochromatic DNA repair | Q34759375 | ||
Congenital bone marrow failure in DNA-PKcs mutant mice associated with deficiencies in DNA repair | Q34825234 | ||
The complexity of phosphorylated H2AX foci formation and DNA repair assembly at DNA double-strand breaks | Q34915507 | ||
Multiple facets of histone variant H2AX: a DNA double-strand-break marker with several biological functions | Q35171563 | ||
Differential phosphorylation of DNA-PKcs regulates the interplay between end-processing and end-ligation during nonhomologous end-joining | Q35555378 | ||
Phosphorylation of Ku dictates DNA double-strand break (DSB) repair pathway choice in S phase | Q36627940 | ||
Clustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PK | Q36963312 | ||
DNA double strand break repair via non-homologous end-joining | Q37135926 | ||
Distinct roles of ATR and DNA-PKcs in triggering DNA damage responses in ATM-deficient cells | Q37264809 | ||
Formation of dynamic gamma-H2AX domains along broken DNA strands is distinctly regulated by ATM and MDC1 and dependent upon H2AX densities in chromatin. | Q37347174 | ||
scid mutation in mice confers hypersensitivity to ionizing radiation and a deficiency in DNA double-strand break repair | Q37404182 | ||
Detection and repair of ionizing radiation-induced DNA double strand breaks: new developments in nonhomologous end joining | Q38083939 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 18 | |
P1104 | number of pages | 13 | |
P304 | page(s) | 9467-9479 | |
P577 | publication date | 2019-10-01 | |
P1433 | published in | Nucleic Acids Research | Q135122 |
P1476 | title | DNA-PKcs promotes chromatin decondensation to facilitate initiation of the DNA damage response | |
P478 | volume | 47 |
Q94474304 | DNA damage response signaling pathways and targets for radiotherapy sensitization in cancer | cites work | P2860 |
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