scholarly article | Q13442814 |
P2093 | author name string | Dongmei Ye | |
Wei Ma | |||
Qiang Zhao | |||
Zhiwei Zhang | |||
Junhui Zhu | |||
Yiyang Xiao | |||
Gaosheng Xu | |||
P2860 | cites work | Neuropilins: expression and roles in the epithelium | Q26998786 |
Mechanisms of Translocation of ER Chaperones to the Cell Surface and Immunomodulatory Roles in Cancer and Autoimmunity. | Q35025948 | ||
Neuropilin 1 is an entry factor that promotes EBV infection of nasopharyngeal epithelial cells | Q35119824 | ||
Neuropilin-1 is a receptor for transforming growth factor beta-1, activates its latent form, and promotes regulatory T cell activity | Q36825006 | ||
A new prognostic histopathologic classification of nasopharyngeal carcinoma | Q36871850 | ||
EBV latent membrane protein 1 effects on plakoglobin, cell growth, and migration. | Q36994005 | ||
Prognostic implication of neuropilin-1 upregulation in human nasopharyngeal carcinoma | Q37366290 | ||
Calreticulin signaling in health and disease. | Q37988994 | ||
Calreticulin Expression in Human Non-Small Cell Lung Cancers Correlates with Increased Accumulation of Antitumor Immune Cells and Favorable Prognosis | Q38796822 | ||
Construction and identification of small hairpin RNA plasmids targeting neuropilin-1 gene and their inhibitory effect on human nasopharyngeal carcinoma CNE-2Z cell proliferation in vitro and in vivo | Q38804698 | ||
Calreticulin promotes migration and invasion of esophageal cancer cells by upregulating neuropilin-1 expression via STAT5A. | Q38955515 | ||
Overexpression of calreticulin contributes to the development and progression of pancreatic cancer | Q39058690 | ||
Functional roles of calreticulin in cancer biology | Q39607843 | ||
Proteome analysis of the transformation potential of the Epstein-Barr virus-encoded latent membrane protein 1 in nasopharyngeal epithelial cells NP69. | Q39990167 | ||
Epstein-Barr virus infection and nasopharyngeal carcinoma | Q40046633 | ||
Calreticulin and Arginylated Calreticulin Have Different Susceptibilities to Proteasomal Degradation | Q42126098 | ||
Calreticulin Is Required for TGF-β-Induced Epithelial-to-Mesenchymal Transition during Cardiogenesis in Mouse Embryonic Stem Cells. | Q42278306 | ||
Calreticulin expression is reduced in high-grade ovarian serous carcinoma effusions compared with primary tumors and solid metastases | Q44261967 | ||
Modulation of the Tumor Microenvironment by Epstein-Barr virus Latent Membrane Protein-1 in Nasopharyngeal Carcinoma. | Q47286778 | ||
Reduced SMAD2/3 activation independently predicts increased depth of human cutaneous squamous cell carcinoma. | Q51734226 | ||
Blockade of TGF-β/Smad signaling by the small compound HPH-15 ameliorates experimental skin fibrosis. | Q52726337 | ||
Clinicopathological significance of calreticulin in breast invasive ductal carcinoma. | Q54411702 | ||
Activation of sterol regulatory element-binding protein 1 (SREBP1)-mediated lipogenesis by the Epstein-Barr virus-encoded latent membrane protein 1 (LMP1) promotes cell proliferation and progression of nasopharyngeal carcinoma | Q57782075 | ||
Cancer Exacerbates Ischemic Brain Injury Via Nrp1 (Neuropilin 1)-Mediated Accumulation of Regulatory T Cells Within the Tumor | Q57789122 | ||
Identification of genomic alterations in nasopharyngeal carcinoma and nasopharyngeal carcinoma-derived Epstein-Barr virus by whole genome sequencing | Q59354661 | ||
Epstein-Barr virus-encoded LMP1 regulated Pim1 kinase expression promotes nasopharyngeal carcinoma cells proliferation | Q64246447 | ||
Nasopharyngeal carcinoma | Q86035551 | ||
mTORC2-mediated PDHE1α nuclear translocation links EBV-LMP1 reprogrammed glucose metabolism to cancer metastasis in nasopharyngeal carcinoma | Q91468424 | ||
P433 | issue | 5 | |
P304 | page(s) | 1257-1269 | |
P577 | publication date | 2020-01-01 | |
P1433 | published in | Journal of Cancer | Q6294901 |
P1476 | title | LMP1 Up-regulates Calreticulin to Induce Epithelial-mesenchymal Transition via TGF-β/Smad3/NRP1 Pathway in Nasopharyngeal Carcinoma Cells | |
P478 | volume | 11 |
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