| scholarly article | Q13442814 |
| P50 | author | Melissa Herwig | Q110513310 |
| Sophie Van Linthout | Q56961031 | ||
| Carsten Tschöpe | Q90667359 | ||
| P2093 | author name string | Zoltán Papp | |
| Nazha Hamdani | |||
| Wolfgang A Linke | |||
| Andreas Mügge | |||
| Peter Sandner | |||
| Kornelia Jaquet | |||
| Mária Lódi | |||
| Detmar Kolijn | |||
| Árpád Kovács | |||
| Peter Haldenwang | |||
| Peter H Reusch | |||
| Abdulatif Alhaj | |||
| Ines Falcão-Pires | |||
| Marcel Sieme | |||
| P2860 | cites work | Crucial role for Ca2(+)/calmodulin-dependent protein kinase-II in regulating diastolic stress of normal and failing hearts via titin phosphorylation | Q24307553 |
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| A novel paradigm for heart failure with preserved ejection fraction: comorbidities drive myocardial dysfunction and remodeling through coronary microvascular endothelial inflammation | Q26850196 | ||
| The emperor's new clothes: PDE5 and the heart | Q28543897 | ||
| Protein S-sulfenylation is a fleeting molecular switch that regulates non-enzymatic oxidative folding | Q28829133 | ||
| S-glutathionylation of cryptic cysteines enhances titin elasticity by blocking protein folding | Q30576327 | ||
| NO-independent regulatory site on soluble guanylate cyclase | Q32165040 | ||
| Vericiguat in patients with worsening chronic heart failure and preserved ejection fraction: results of the SOluble guanylate Cyclase stimulatoR in heArT failurE patientS with PRESERVED EF (SOCRATES-PRESERVED) study | Q33589713 | ||
| Heart failure with preserved ejection fraction: pathophysiology, diagnosis, and treatment | Q34664894 | ||
| Mind the gap (junction): cGMP induced by nitric oxide in cardiac myocytes originates from cardiac fibroblasts | Q92686456 | ||
| Heart Failure Risk Stratification and Efficacy of Sodium-Glucose Cotransporter-2 Inhibitors in Patients With Type 2 Diabetes Mellitus | Q93041711 | ||
| Role of Non-Myocyte Gap Junctions and Connexin Hemichannels in Cardiovascular Health and Disease: Novel Therapeutic Targets? | Q52354401 | ||
| Nitric oxide-sensitive guanylyl cyclase stimulation improves experimental heart failure with preserved ejection fraction. | Q52375176 | ||
| Acute stimulation of the soluble guanylate cyclase does not impact on left ventricular capacitance in normal and hypertrophied porcine hearts in vivo. | Q52803309 | ||
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| Sitagliptin reduces inflammation, fibrosis and preserves diastolic function in a rat model of heart failure with preserved ejection fraction | Q88911120 | ||
| Posttranslational Arginylation Regulates Striated Muscle Function | Q89292658 | ||
| Correction to: Soluble Guanylate Cyclase Stimulators and Activators | Q91624145 | ||
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| Discovery of riociguat (BAY 63-2521): a potent, oral stimulator of soluble guanylate cyclase for the treatment of pulmonary hypertension | Q34957241 | ||
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| Soluble guanylate cyclase as an emerging therapeutic target in cardiopulmonary disease | Q35010064 | ||
| Biological selectivity and functional aspects of protein tyrosine nitration | Q35134291 | ||
| Relaxin does not improve Angiotensin II-induced target-organ damage. | Q35141812 | ||
| Emerging importance of oxidative stress in regulating striated muscle elasticity | Q35155257 | ||
| Myocardial stiffness in patients with heart failure and a preserved ejection fraction: contributions of collagen and titin | Q35328760 | ||
| A novel mechanism involving four-and-a-half LIM domain protein-1 and extracellular signal-regulated kinase-2 regulates titin phosphorylation and mechanics. | Q36215905 | ||
| Diastolic dysfunction in hypertensive hearts: roles of perivascular inflammation and reactive myocardial fibrosis | Q36289289 | ||
| Phosphorylation of titin modulates passive stiffness of cardiac muscle in a titin isoform-dependent manner | Q36445698 | ||
| Key connexin 43 phosphorylation events regulate the gap junction life cycle | Q37001308 | ||
| Nitric oxide, tetrahydrobiopterin, oxidative stress, and endothelial dysfunction in hypertension | Q37102515 | ||
| Proteins and mechanisms regulating gap-junction assembly, internalization, and degradation | Q37158482 | ||
| Modulation of titin-based stiffness by disulfide bonding in the cardiac titin N2-B unique sequence | Q37281915 | ||
| Effect of phosphodiesterase-5 inhibition on exercise capacity and clinical status in heart failure with preserved ejection fraction: a randomized clinical trial | Q37328507 | ||
| PKC phosphorylation of titin's PEVK element: a novel and conserved pathway for modulating myocardial stiffness | Q37394610 | ||
| Muscle-strengthening and conditioning activities and risk of type 2 diabetes: a prospective study in two cohorts of US women | Q37477754 | ||
| Specific Cx43 phosphorylation events regulate gap junction turnover in vivo | Q37707287 | ||
| Molecular and cellular basis for diastolic dysfunction | Q38038531 | ||
| Gigantic business: titin properties and function through thick and thin | Q38195698 | ||
| A change of heart: oxidative stress in governing muscle function? | Q39310989 | ||
| Interleukin-6 receptor inhibition modulates the immune reaction and restores titin phosphorylation in experimental myocarditis | Q41689291 | ||
| Sildenafil and B-type natriuretic peptide acutely phosphorylate titin and improve diastolic distensibility in vivo | Q42149439 | ||
| Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs. | Q42445262 | ||
| Nitric oxide-mediated regulation of connexin43 expression and gap junctional intercellular communication in mesangial cells | Q42470387 | ||
| Protein kinase-A phosphorylates titin in human heart muscle and reduces myofibrillar passive tension | Q42499448 | ||
| Protein kinase A phosphorylates titin's cardiac-specific N2B domain and reduces passive tension in rat cardiac myocytes | Q44028492 | ||
| Hypophosphorylation of the Stiff N2B titin isoform raises cardiomyocyte resting tension in failing human myocardium. | Q46140653 | ||
| Improvement in left ventricular remodeling by the endothelial nitric oxide synthase enhancer AVE9488 after experimental myocardial infarction | Q46446883 | ||
| Meeting report: cGMP matters | Q46719063 | ||
| Dietary n-3 polyunsaturated fatty acids and direct renin inhibition improve electrical remodeling in a model of high human renin hypertension | Q46833923 | ||
| The Nitric Oxide Pathway in Pulmonary Vascular Disease | Q47660819 | ||
| Disulfide isomerization reactions in titin immunoglobulin domains enable a mode of protein elasticity | Q48043256 | ||
| Developing New Treatments for Heart Failure: Focus on the Heart | Q48115739 | ||
| Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction. | Q48772970 | ||
| Cardioprotection by ischemic postconditioning and cGMP-elevating agents involves cardiomyocyte nitric oxide-sensitive guanylyl cyclase. | Q50041563 | ||
| P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
| P6216 | copyright status | copyrighted | Q50423863 |
| P921 | main subject | Diastolic function | Q30314111 |
| P304 | page(s) | 345 | |
| P577 | publication date | 2020-05-25 | |
| P1433 | published in | Frontiers in Physiology | Q2434141 |
| P1476 | title | Enhanced Cardiomyocyte Function in Hypertensive Rats With Diastolic Dysfunction and Human Heart Failure Patients After Acute Treatment With Soluble Guanylyl Cyclase (sGC) Activator | |
| P478 | volume | 11 |
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