scholarly article | Q13442814 |
P356 | DOI | 10.1523/JNEUROSCI.2083-05.2005 |
P8608 | Fatcat ID | release_bznfregnpre2vhastqpanc6pja |
P932 | PMC publication ID | 6724840 |
P698 | PubMed publication ID | 16049186 |
P2093 | author name string | Masato Okada | |
Shigeyuki Nada | |||
Kazuaki Yoshikawa | |||
Masashi Yamada | |||
Isao Nishimura | |||
Ken-ichiro Kuwako | |||
Taichi Uetsuki | |||
Akari Hosokawa | |||
P2860 | cites work | Necdin-related MAGE Proteins Differentially Interact with the E2F1 Transcription Factor and the p75 Neurotrophin Receptor | Q24298935 |
Sortilin is essential for proNGF-induced neuronal cell death | Q24312692 | ||
The human chromosomal gene for necdin, a neuronal growth suppressor, in the Prader-Willi syndrome deletion region | Q24336557 | ||
Regulation of cell survival by secreted proneurotrophins | Q27919620 | ||
Trk receptors: roles in neuronal signal transduction | Q27919636 | ||
NRAGE, a novel MAGE protein, interacts with the p75 neurotrophin receptor and facilitates nerve growth factor-dependent apoptosis | Q28138611 | ||
Physical and functional interactions of neuronal growth suppressor necdin with p53 | Q28144785 | ||
Cellular and subcellular localization of necdin in fetal and adult mouse brain | Q28144941 | ||
Requirement of nerve growth factor for development of substance P-containing sensory neurones | Q72621102 | ||
Neuronally-expressed necdin gene: an imprinted candidate gene in Prader-Willi syndrome | Q73920284 | ||
Necdin is required for terminal differentiation and survival of primary dorsal root ganglion neurons | Q74361128 | ||
Expression of necdin, an embryonal carcinoma-derived nuclear protein, in developing mouse brain | Q28189907 | ||
The p75 neurotrophin receptor interacts with multiple MAGE proteins | Q28213040 | ||
Ectopic expression of necdin induces differentiation of mouse neuroblastoma cells | Q28219184 | ||
The necdin gene is deleted in Prader-Willi syndrome and is imprinted in human and mouse | Q28249408 | ||
The human necdin gene, NDN, is maternally imprinted and located in the Prader-Willi syndrome chromosomal region | Q28253418 | ||
Necdin, a postmitotic neuron-specific growth suppressor, interacts with viral transforming proteins and cellular transcription factor E2F1 | Q28258359 | ||
Necdin interacts with the Msx2 homeodomain protein via MAGE-D1 to promote myogenic differentiation of C2C12 cells | Q28505040 | ||
Absence of Ndn, encoding the Prader-Willi syndrome-deleted gene necdin, results in congenital deficiency of central respiratory drive in neonatal mice | Q28505055 | ||
Essential role for the Prader-Willi syndrome protein necdin in axonal outgrowth | Q28508700 | ||
Dual roles of the retinoblastoma protein in cell cycle regulation and neuron differentiation | Q28513619 | ||
Nuclear translocation of the p75 neurotrophin receptor cytoplasmic domain in response to neurotrophin binding | Q28564163 | ||
Mice lacking nerve growth factor display perinatal loss of sensory and sympathetic neurons yet develop basal forebrain cholinergic neurons | Q28584797 | ||
Disruption of the mouse necdin gene results in early post-natal lethality | Q28587922 | ||
NSCL-1 and NSCL-2 synergistically determine the fate of GnRH-1 neurons and control necdin gene expression | Q28591595 | ||
Neurotrophins and their receptors: a convergence point for many signalling pathways | Q29616108 | ||
E2F: a link between the Rb tumor suppressor protein and viral oncoproteins | Q29618375 | ||
A novel brain-specific mRNA encoding nuclear protein (necdin) expressed in neurally differentiated embryonal carcinoma cells | Q33652807 | ||
Cell cycle regulators in neural stem cells and postmitotic neurons | Q33913344 | ||
Prader-Willi syndrome: consensus diagnostic criteria. | Q34061685 | ||
Neurotrophins: key regulators of cell fate and cell shape in the vertebrate nervous system | Q34103744 | ||
Severe sensory and sympathetic neuropathies in mice carrying a disrupted Trk/NGF receptor gene. | Q34338664 | ||
Constitutive activation of Src family kinases in mouse embryos that lack Csk. | Q34366689 | ||
BDNF regulates eating behavior and locomotor activity in mice | Q34667950 | ||
Neurotrophin signaling through the p75 neurotrophin receptor | Q34778209 | ||
The neurotrophin receptor p75(NTR): novel functions and implications for diseases of the nervous system | Q34982367 | ||
Neurogenin1 and neurogenin2 control two distinct waves of neurogenesis in developing dorsal root ganglia | Q35200638 | ||
Brain-derived neurotrophic factor-deficient mice develop aggressiveness and hyperphagia in conjunction with brain serotonergic abnormalities | Q36784651 | ||
Structure and expression of the mouse necdin gene. Identification of a postmitotic neuron-restrictive core promoter | Q36790661 | ||
Cell death during development of the nervous system | Q37716922 | ||
Arrest of Cell Growth by Necdin, a Nuclear-Protein Expressed in Postmitotic Neurons | Q42825285 | ||
Impaired peripheral somatosensory function in children with Prader-Willi syndrome | Q47304792 | ||
Degeneration in vitro of post-mitotic neurons overexpressing the Alzheimer amyloid protein precursor | Q48432892 | ||
Degeneration in vivo of rat hippocampal neurons by wild-type Alzheimer amyloid precursor protein overexpressed by adenovirus-mediated gene transfer. | Q48493406 | ||
Disruption of the mouse Necdin gene results in hypothalamic and behavioral alterations reminiscent of the human Prader-Willi syndrome. | Q49155367 | ||
Expression of the Prader-Willi gene Necdin during mouse nervous system development correlates with neuronal differentiation and p75NTR expression. | Q52097061 | ||
Neurotrophin receptor genes are expressed in distinct patterns in developing dorsal root ganglia. | Q52223036 | ||
Activation of neuronal caspase-3 by intracellular accumulation of wild-type Alzheimer amyloid precursor protein. | Q53229787 | ||
P433 | issue | 30 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Neurotrophic tyrosine kinase, receptor, type 1 | Q14901156 |
Necdin, MAGE family member | Q21984208 | ||
Tumor necrosis factor receptor superfamily member 16 | Q21988151 | ||
P304 | page(s) | 7090-7099 | |
P577 | publication date | 2005-07-01 | |
P1433 | published in | Journal of Neuroscience | Q1709864 |
P1476 | title | Disruption of the paternal necdin gene diminishes TrkA signaling for sensory neuron survival | |
P478 | volume | 25 |
Q35083411 | Antagonistic interplay between necdin and Bmi1 controls proliferation of neural precursor cells in the embryonic mouse neocortex |
Q38317227 | Autonomic nervous system dysfunction in obesity and Prader-Willi syndrome: current evidence and implications for future obesity therapies. |
Q88815207 | Cognitive deficits in the Snord116 deletion mouse model for Prader-Willi syndrome |
Q51905205 | Dlxin-1, a MAGE family protein, induces accelerated neurite outgrowth and cell survival by enhanced and early activation of MEK and Akt signalling pathways in PC12 cells. |
Q98163300 | Firing activity of locus coeruleus noradrenergic neurons decreases in necdin-deficient mice, an animal model of Prader-Willi syndrome |
Q38296769 | From Prader-Willi syndrome to psychosis: translating parent-of-origin effects into schizophrenia research |
Q41791587 | Functional consequences of necdin nucleocytoplasmic localization. |
Q37961799 | Genetic and epigenetic dysregulation of imprinted genes in the brain |
Q34699894 | Genomic imprinting effects on brain development and function |
Q35416510 | Impaired hypothalamic regulation of endocrine function and delayed counterregulatory response to hypoglycemia in Magel2-null mice. |
Q37060646 | Imprinted genes and neuroendocrine function |
Q39732218 | Loss of Magel2 impairs the development of hypothalamic Anorexigenic circuits. |
Q48109362 | Loss of Necdin impairs myosin activation and delays cell polarization |
Q51953730 | Loss of the Prader-Willi syndrome protein necdin causes defective migration, axonal outgrowth, and survival of embryonic sympathetic neurons. |
Q34086646 | Lower brain-derived neurotrophic factor in patients with prader-willi syndrome compared to obese and lean control subjects |
Q38152232 | Mechanism of pain generation for endometriosis-associated pelvic pain |
Q38079927 | Mouse models of Prader-Willi Syndrome: a systematic review |
Q37456880 | Necdin and TrkA contribute to modulation by p75NTR of resistance to oxidant stress. |
Q34911741 | Necdin and neurotrophin receptors: interactors of relevance for neuronal resistance to oxidant stress |
Q39362556 | Necdin controls Foxo1 acetylation in hypothalamic arcuate neurons to modulate the thyroid axis. |
Q50892653 | Necdin controls proliferation and apoptosis of embryonic neural stem cells in an oxygen tension-dependent manner. |
Q34146581 | Necdin controls proliferation of white adipocyte progenitor cells |
Q48368860 | Necdin downregulates CDC2 expression to attenuate neuronal apoptosis. |
Q36119598 | Necdin mediates skeletal muscle regeneration by promoting myoblast survival and differentiation |
Q40278508 | Necdin promotes GABAergic neuron differentiation in cooperation with Dlx homeodomain proteins. |
Q42468550 | Necdin promotes tangential migration of neocortical interneurons from basal forebrain |
Q33727985 | Necdin promotes ubiquitin-dependent degradation of PIAS1 SUMO E3 ligase |
Q34017758 | Necdin protects embryonic motoneurons from programmed cell death |
Q46407035 | Necdin regulates p53 acetylation via Sirtuin1 to modulate DNA damage response in cortical neurons |
Q47230157 | Necdin shapes serotonergic development and SERT activity modulating breathing in a mouse model for Prader-Willi syndrome. |
Q37089973 | Necdin, a Prader-Willi syndrome candidate gene, regulates gonadotropin-releasing hormone neurons during development. |
Q34166515 | Necdin, a p53-target gene, is an inhibitor of p53-mediated growth arrest. |
Q51107047 | Novel NTRK1 Frameshift Mutation in Congenital Insensitivity to Pain With Anhidrosis. |
Q24304286 | Nucleoredoxin sustains Wnt/β-catenin signaling by retaining a pool of inactive dishevelled protein |
Q46262537 | On the origin of sensory impairment and altered pain perception in Prader-Willi syndrome: a neurophysiological study |
Q52605544 | Phenotypic heterogeneity of intellectual disability in patients with congenital insensitivity to pain with anhidrosis: A case report and literature review. |
Q64094464 | Preclinical Testing in Translational Animal Models of Prader-Willi Syndrome: Overview and Gap Analysis |
Q36690552 | Promotion of mitochondrial biogenesis by necdin protects neurons against mitochondrial insults |
Q28285118 | Psychosis and autism as diametrical disorders of the social brain |
Q44803556 | Recommendations for the investigation of animal models of Prader-Willi syndrome |
Q24674557 | Separate necdin domains bind ARNT2 and HIF1alpha and repress transcription |
Q31138426 | Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequences |
Q54292334 | Suppression of the p75 neurotrophin receptor in uninjured sensory neurons reduces neuropathic pain after nerve injury. |
Q24321957 | The Prader-Willi syndrome protein necdin interacts with the E1A-like inhibitor of differentiation EID-1 and promotes myoblast differentiation |
Q47706180 | The Prader-Willi syndrome proteins MAGEL2 and necdin regulate leptin receptor cell surface abundance through ubiquitination pathways |
Q35680716 | The neurobiology of mouse models syntenic to human chromosome 15q. |
Q37379467 | Thermosensory and mechanosensory perception in human genetic disease |
Q33732098 | p53 regulates hematopoietic stem cell quiescence. |
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