review article | Q7318358 |
scholarly article | Q13442814 |
P2093 | author name string | Koichi Iijima | |
Kanae Iijima-Ando | |||
P2860 | cites work | Abeta42-induced neurodegeneration via an age-dependent autophagic-lysosomal injury in Drosophila | Q21143886 |
Systematic in vivo analysis of the intrinsic determinants of amyloid Beta pathogenicity | Q21145889 | ||
Insulin-degrading enzyme regulates extracellular levels of amyloid beta-protein by degradation | Q22008492 | ||
The genetic epidemiology of neurodegenerative disease | Q22306301 | ||
Cell cycle regulation in the postmitotic neuron: oxymoron or new biology? | Q22337222 | ||
Mushroom body memoir: from maps to models | Q22337225 | ||
Identification of ubiquilin, a novel presenilin interactor that increases presenilin protein accumulation | Q24290515 | ||
Dissecting the pathological effects of human Abeta40 and Abeta42 in Drosophila: a potential model for Alzheimer's disease | Q24564815 | ||
Amyloid plaque core protein in Alzheimer disease and Down syndrome | Q24568384 | ||
A systematic analysis of human disease-associated gene sequences in Drosophila melanogaster | Q24619672 | ||
Retromer deficiency observed in Alzheimer's disease causes hippocampal dysfunction, neurodegeneration, and Abeta accumulation | Q24654821 | ||
Fenton chemistry and oxidative stress mediate the toxicity of the beta-amyloid peptide in a Drosophila model of Alzheimer's disease | Q24656549 | ||
Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease | Q24670696 | ||
A Drosophila model of ALS: human ALS-associated mutation in VAP33A suggests a dominant negative mechanism | Q27301268 | ||
The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics | Q27860914 | ||
Targeted gene expression as a means of altering cell fates and generating dominant phenotypes | Q27861039 | ||
Soluble protein oligomers in neurodegeneration: lessons from the Alzheimer's amyloid beta-peptide | Q28131750 | ||
Alzheimer's disease: genes, proteins, and therapy | Q28131806 | ||
Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition | Q28143799 | ||
A CBP binding transcriptional repressor produced by the PS1/epsilon-cleavage of N-cadherin is inhibited by PS1 FAD mutations | Q28185270 | ||
Can flies help humans treat neurodegenerative diseases? | Q28258885 | ||
Metabolism of amyloid-beta peptide and Alzheimer's disease | Q28268316 | ||
The Toll-->NFkappaB signaling pathway mediates the neuropathological effects of the human Alzheimer's Abeta42 polypeptide in Drosophila | Q28474328 | ||
Neprilysin degrades both amyloid beta peptides 1-40 and 1-42 most rapidly and efficiently among thiorphan- and phosphoramidon-sensitive endopeptidases | Q28505860 | ||
PS1 activates PI3K thus inhibiting GSK-3 activity and tau overphosphorylation: effects of FAD mutations | Q28587440 | ||
Transgenic Drosophila expressing human amyloid precursor protein show gamma-secretase activity and a blistered-wing phenotype | Q36796905 | ||
A drosophila model for amyotrophic lateral sclerosis reveals motor neuron damage by human SOD1. | Q36861919 | ||
Alzheimer disease and the role of free radicals in the pathogenesis of the disease | Q37090119 | ||
Dissociation of tau toxicity and phosphorylation: role of GSK-3beta, MARK and Cdk5 in a Drosophila model | Q37102130 | ||
Animal models of Alzheimer's disease and frontotemporal dementia | Q37196122 | ||
Distinctive roles of different beta-amyloid 42 aggregates in modulation of synaptic functions | Q37233197 | ||
Animal models of Alzheimer's disease: therapeutic implications. | Q37354734 | ||
Drosophila models of Alzheimer's amyloidosis: the challenge of dissecting the complex mechanisms of toxicity of amyloid-beta 42. | Q37354737 | ||
Modeling Tauopathy in the fruit fly Drosophila melanogaster | Q37354741 | ||
Conditioned behavior in Drosophila melanogaster | Q37433575 | ||
Recent advances in using Drosophila to model neurodegenerative diseases | Q37448324 | ||
Overexpression of neprilysin reduces alzheimer amyloid-beta42 (Abeta42)-induced neuron loss and intraneuronal Abeta42 deposits but causes a reduction in cAMP-responsive element-binding protein-mediated transcription, age-dependent axon pathology, an | Q39627415 | ||
Presynaptic localization of neprilysin contributes to efficient clearance of amyloid-beta peptide in mouse brain. | Q40594671 | ||
The role of presenilin cofactors in the gamma-secretase complex | Q40661006 | ||
Neprilysin gene transfer reduces human amyloid pathology in transgenic mice. | Q40661439 | ||
Hirano bodies and related neuronal inclusions | Q40765085 | ||
Degradation of the Alzheimer's amyloid beta peptide by endothelin-converting enzyme | Q40808050 | ||
Oxidative stress mediates tau-induced neurodegeneration in Drosophila | Q41044662 | ||
Amyloid precursor protein promotes post-developmental neurite arborization in the Drosophila brain | Q42263336 | ||
ubiquilin antagonizes presenilin and promotes neurodegeneration in Drosophila | Q43442095 | ||
Tauopathy in Drosophila: neurodegeneration without neurofibrillary tangles | Q43642001 | ||
Expression and targeting of Syrian hamster prion protein induced by heat shock in transgenic Drosophila melanogaster | Q43785458 | ||
Relationship between beta amyloid peptide generating molecules and neprilysin in Alzheimer disease and normal brain | Q43786476 | ||
Expression and characterization of the Drosophila X11-like/Mint protein during neural development | Q44030457 | ||
Post-transcriptional suppression of pathogenic prion protein expression in Drosophila neurons | Q44465712 | ||
Neurotoxicity and physicochemical properties of Abeta mutant peptides from cerebral amyloid angiopathy: implication for the pathogenesis of cerebral amyloid angiopathy and Alzheimer's disease | Q44565945 | ||
The Drosophila beta-amyloid precursor protein homolog promotes synapse differentiation at the neuromuscular junction. | Q52572881 | ||
Overexpression of human amyloid precursor protein in Drosophila. | Q52585343 | ||
Abnormal bundling and accumulation of F-actin mediates tau-induced neuronal degeneration in vivo. | Q52675363 | ||
Cytoskeleton proteins are modulators of mutant tau-induced neurodegeneration in Drosophila. | Q52677007 | ||
S/P and T/P phosphorylation is critical for tau neurotoxicity in Drosophila. | Q52677310 | ||
The H1c haplotype at the MAPT locus is associated with Alzheimer's disease. | Q53270733 | ||
Axonopathy in an APP/PS1 transgenic mouse model of Alzheimer's disease. | Q53276312 | ||
Physiochemical characterization of the Alzheimer's disease-related peptides A beta 1-42Arctic and A beta 1-42wt. | Q53278971 | ||
A68: a major subunit of paired helical filaments and derivatized forms of normal Tau. | Q53296439 | ||
Classical conditioning and retention in normal and mutantDrosophila melanogaster | Q55980197 | ||
Tau protein isoforms, phosphorylation and role in neurodegenerative disorders11These authors contributed equally to this work | Q57897029 | ||
In vivo aggregation of beta-amyloid peptide variants | Q64360819 | ||
Observation of metastable Abeta amyloid protofibrils by atomic force microscopy | Q73431442 | ||
Loss of presenilin function causes impairments of memory and synaptic plasticity followed by age-dependent neurodegeneration | Q28590719 | ||
A specific amyloid-beta protein assembly in the brain impairs memory | Q28854597 | ||
Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis | Q29547501 | ||
Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins | Q29547593 | ||
Alzheimer's disease is a synaptic failure | Q29547670 | ||
Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction | Q29547778 | ||
Twenty years of the Alzheimer's disease amyloid hypothesis: a genetic perspective | Q29614702 | ||
Tau suppression in a neurodegenerative mouse model improves memory function | Q29615831 | ||
A Drosophila model of Parkinson's disease | Q29617052 | ||
Abnormal phosphorylation of the microtubule-associated protein tau (tau) in Alzheimer cytoskeletal pathology | Q29617284 | ||
Microtubule-associated protein tau (tau) is a major antigenic component of paired helical filaments in Alzheimer disease | Q29617285 | ||
Tau-mediated neurodegeneration in Alzheimer's disease and related disorders | Q29618148 | ||
Opposing activities protect against age-onset proteotoxicity | Q29619758 | ||
Neurodegenerative tauopathies | Q29619895 | ||
Massive CA1/2 neuronal loss with intraneuronal and N-terminal truncated Abeta42 accumulation in a novel Alzheimer transgenic model | Q30478167 | ||
Alpha-helix targeting reduces amyloid-beta peptide toxicity | Q30488155 | ||
Reducing amyloid plaque burden via ex vivo gene delivery of an Abeta-degrading protease: a novel therapeutic approach to Alzheimer disease. | Q30833707 | ||
A survey of human disease gene counterparts in the Drosophila genome | Q30887434 | ||
Abeta42 mutants with different aggregation profiles induce distinct pathologies in Drosophila | Q33321266 | ||
Role of X11 and ubiquilin as in vivo regulators of the amyloid precursor protein in Drosophila | Q33346273 | ||
Long-term neprilysin gene transfer is associated with reduced levels of intracellular Abeta and behavioral improvement in APP transgenic mice | Q33384861 | ||
The role of cerebral amyloid beta accumulation in common forms of Alzheimer disease | Q33768345 | ||
Amyloid fibrillogenesis: themes and variations | Q33840755 | ||
Drosophila models of neurodegenerative disease | Q33844727 | ||
Nanoparticle-based detection in cerebral spinal fluid of a soluble pathogenic biomarker for Alzheimer's disease | Q33850134 | ||
Modeling human neurodegenerative diseases in Drosophila: on a wing and a prayer. | Q33870792 | ||
Correlation between elevated levels of amyloid beta-peptide in the brain and cognitive decline | Q33895797 | ||
Treatment with a copper-zinc chelator markedly and rapidly inhibits beta-amyloid accumulation in Alzheimer's disease transgenic mice. | Q34082498 | ||
The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Abeta protofibril formation | Q34088689 | ||
Cognitive and behavioral heterogeneity in Alzheimer's disease: seeking the neurobiological basis | Q34109698 | ||
Learning and memory deficits upon TAU accumulation in Drosophila mushroom body neurons | Q34340308 | ||
Strategies for disease modification in Alzheimer's disease | Q34342498 | ||
Abeta42 is essential for parenchymal and vascular amyloid deposition in mice | Q34374693 | ||
Intraneuronal Abeta, non-amyloid aggregates and neurodegeneration in a Drosophila model of Alzheimer's disease. | Q34404879 | ||
Neurotoxicity of a fragment of the amyloid precursor associated with Alzheimer's disease | Q34415666 | ||
What can we teach Drosophila? What can they teach us? | Q34446127 | ||
A genomic screen for modifiers of tauopathy identifies puromycin-sensitive aminopeptidase as an inhibitor of tau-induced neurodegeneration | Q34562996 | ||
Human wild-type tau interacts with wingless pathway components and produces neurofibrillary pathology in Drosophila | Q44662134 | ||
GSK-3beta inhibition reverses axonal transport defects and behavioural phenotypes in Drosophila | Q44782542 | ||
Age-dependent neurodegeneration and Alzheimer-amyloid plaque formation in transgenic Drosophila. | Q44854880 | ||
Analysis of the secondary structure of beta-amyloid (Abeta42) fibrils by systematic proline replacement | Q45086533 | ||
Attacking amyloid | Q45270079 | ||
Polyglutamine-expanded human huntingtin transgenes induce degeneration of Drosophila photoreceptor neurons. | Q45296540 | ||
Rapamycin alleviates toxicity of different aggregate-prone proteins | Q45299514 | ||
Human amyloid precursor protein ameliorates behavioral deficit of flies deleted for Appl gene | Q45729428 | ||
A reporter for amyloid precursor protein gamma-secretase activity in Drosophila | Q46183415 | ||
Glutaminyl cyclase inhibition attenuates pyroglutamate Abeta and Alzheimer's disease-like pathology | Q46330873 | ||
Over-expression of tau results in defective synaptic transmission in Drosophila neuromuscular junctions | Q46604113 | ||
Activation of PAR-1 kinase and stimulation of tau phosphorylation by diverse signals require the tumor suppressor protein LKB1. | Q46689310 | ||
Different conformations of amyloid beta induce neurotoxicity by distinct mechanisms in human cortical neurons. | Q46733868 | ||
Aggressive amyloidosis in mice expressing human amyloid peptides with the Arctic mutation | Q46814179 | ||
TOR-mediated cell-cycle activation causes neurodegeneration in a Drosophila tauopathy model. | Q46930440 | ||
Disruption of axonal transport and neuronal viability by amyloid precursor protein mutations in Drosophila. | Q46959740 | ||
Modeling clinically heterogeneous presenilin mutations with transgenic Drosophila | Q47069956 | ||
Isolation and characterization of the Drosophila ubiquilin ortholog dUbqln: in vivo interaction with early-onset Alzheimer disease genes | Q47070160 | ||
Fasciclin II signals new synapse formation through amyloid precursor protein and the scaffolding protein dX11/Mint. | Q47070740 | ||
A model for studying Alzheimer's Abeta42-induced toxicity in Drosophila melanogaster | Q47072796 | ||
Expanded polyglutamine protein forms nuclear inclusions and causes neural degeneration in Drosophila | Q47945602 | ||
PAR-1 kinase plays an initiator role in a temporally ordered phosphorylation process that confers tau toxicity in Drosophila | Q47987669 | ||
KUZ, a conserved metalloprotease-disintegrin protein with two roles in Drosophila neurogenesis | Q48060932 | ||
ApoE and clusterin cooperatively suppress Abeta levels and deposition: evidence that ApoE regulates extracellular Abeta metabolism in vivo | Q48087583 | ||
Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43). | Q48092766 | ||
Enhanced proteolysis of beta-amyloid in APP transgenic mice prevents plaque formation, secondary pathology, and premature death | Q48110123 | ||
Study of tauopathies by comparing Drosophila and human tau in Drosophila | Q48218465 | ||
Phosphorylation of Ser262 strongly reduces binding of tau to microtubules: distinction between PHF-like immunoreactivity and microtubule binding. | Q48253590 | ||
Identification and characterization of the Drosophila tau homolog. | Q48340828 | ||
Microtubule-associated protein tau. A component of Alzheimer paired helical filaments. | Q48344680 | ||
Enhanced amyloidogenic metabolism of the amyloid beta-protein precursor in the X11L-deficient mouse brain. | Q48399465 | ||
Intraneuronal beta-amyloid aggregates, neurodegeneration, and neuron loss in transgenic mice with five familial Alzheimer's disease mutations: potential factors in amyloid plaque formation. | Q48404445 | ||
Neuronal neprilysin overexpression is associated with attenuation of Abeta-related spatial memory deficit. | Q48410596 | ||
Neprilysin-sensitive synapse-associated amyloid-beta peptide oligomers impair neuronal plasticity and cognitive function. | Q48569616 | ||
Alzheimer's disease and Down's syndrome: Sharing of a unique cerebrovascular amyloid fibril protein | Q48634030 | ||
Somatodendritic localization of phosphorylated tau in neonatal and adult rat cerebral cortex. | Q48636064 | ||
The Arctic Alzheimer mutation facilitates early intraneuronal Abeta aggregation and senile plaque formation in transgenic mice. | Q48690756 | ||
Clearance of extracellular and cell-associated amyloid beta peptide through viral expression of neprilysin in primary neurons. | Q48701982 | ||
Small non-fibrillar assemblies of amyloid beta-protein bearing the Arctic mutation induce rapid neuritic degeneration. | Q48720650 | ||
Cathepsin D is involved in the clearance of Alzheimer's beta-amyloid protein. | Q48864600 | ||
Metabolic regulation of brain Abeta by neprilysin. | Q48869458 | ||
Degradation of amyloid beta-protein by a serine protease-alpha2-macroglobulin complex. | Q49012858 | ||
Water-soluble Abeta (N-40, N-42) oligomers in normal and Alzheimer disease brains. | Q49112901 | ||
Reduced neprilysin in high plaque areas of Alzheimer brain: a possible relationship to deficient degradation of beta-amyloid peptide. | Q49145268 | ||
Identifying genes that interact with Drosophila presenilin and amyloid precursor protein. | Q51800593 | ||
Gamma-secretase/presenilin inhibitors for Alzheimer's disease phenocopy Notch mutations in Drosophila. | Q52112599 | ||
Tau and tau reporters disrupt central projections of sensory neurons in Drosophila. | Q52162893 | ||
Systematic gain-of-function genetics in Drosophila. | Q52188274 | ||
Neuronal overexpression of APPL, the Drosophila homologue of the amyloid precursor protein (APP), disrupts axonal transport. | Q52535028 | ||
Presenilin-based genetic screens in Drosophila melanogaster identify novel notch pathway modifiers | Q34588474 | ||
Drosophila models of neurodegenerative diseases | Q34610331 | ||
Genetic modifiers of tauopathy in Drosophila. | Q34619083 | ||
An inhibitor of tau hyperphosphorylation prevents severe motor impairments in tau transgenic mice | Q34687145 | ||
Efficient inhibition of the Alzheimer's disease beta-secretase by membrane targeting | Q34774178 | ||
Amyloid-beta oligomers: their production, toxicity and therapeutic inhibition | Q34797468 | ||
Unfolding the role of protein misfolding in neurodegenerative diseases | Q35038274 | ||
Oxidative stress, cell cycle, and neurodegeneration | Q35086148 | ||
From fruit fly to bedside: translating lessons from Drosophila models of neurodegenerative disease | Q35180372 | ||
Neurotoxic effects induced by the Drosophila amyloid-beta peptide suggest a conserved toxic function | Q35565575 | ||
ADAMs family members as amyloid precursor protein alpha-secretases | Q35575838 | ||
Neurofibrillary tangles of Alzheimer disease share antigenic determinants with the axonal microtubule-associated protein tau (tau) | Q35609568 | ||
Intraneuronal Alzheimer abeta42 accumulates in multivesicular bodies and is associated with synaptic pathology | Q35747757 | ||
Intraneuronal Abeta42 accumulation in human brain. | Q35793325 | ||
Functional reconstitution of gamma-secretase through coordinated expression of presenilin, nicastrin, Aph-1, and Pen-2. | Q35833580 | ||
Clearance of Alzheimer's Abeta peptide: the many roads to perdition. | Q35875785 | ||
Loss of neprilysin function promotes amyloid plaque formation and causes cerebral amyloid angiopathy | Q35928266 | ||
Post-translational modifications of tau protein in Alzheimer's disease | Q35934376 | ||
Transgenic animal models of tauopathies | Q35992159 | ||
Genetic and molecular aspects of Alzheimer's disease shed light on new mechanisms of transcriptional regulation. | Q36089357 | ||
Presenilin function and gamma-secretase activity | Q36109130 | ||
Tau phosphorylation sites work in concert to promote neurotoxicity in vivo | Q36173962 | ||
Apoptotic activities of wild-type and Alzheimer's disease-related mutant presenilins in Drosophila melanogaster | Q36301441 | ||
Drosophila as a model for human neurodegenerative disease | Q36312196 | ||
Aberrant lysosomal carbohydrate storage accompanies endocytic defects and neurodegeneration in Drosophila benchwarmer | Q36320691 | ||
The development of amyloid beta protein deposits in the aged brain. | Q36416770 | ||
A decade of modeling Alzheimer's disease in transgenic mice. | Q36432964 | ||
Identification of novel genes that modify phenotypes induced by Alzheimer's beta-amyloid overexpression in Drosophila | Q36515716 | ||
Sorting through the cell biology of Alzheimer's disease: intracellular pathways to pathogenesis | Q36610341 | ||
Drosophila in the study of neurodegenerative disease | Q36610370 | ||
A century-old debate on protein aggregation and neurodegeneration enters the clinic | Q36628151 | ||
Aggregate-prone proteins are cleared from the cytosol by autophagy: therapeutic implications | Q36659535 | ||
Variation in MAPT is associated with cerebrospinal fluid tau levels in the presence of amyloid-beta deposition | Q36725676 | ||
P433 | issue | 2-3 | |
P921 | main subject | Alzheimer's disease | Q11081 |
Drosophila | Q312154 | ||
P304 | page(s) | 245-262 | |
P577 | publication date | 2009-12-05 | |
P1433 | published in | Brain Structure and Function | Q13444883 |
P1476 | title | Transgenic Drosophila models of Alzheimer's disease and tauopathies | |
P478 | volume | 214 |
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Q90732117 | A Human Embryonic Stem Cell Model of Aβ-Dependent Chronic Progressive Neurodegeneration |
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Q35233549 | Accumulation of amyloid-like Aβ1-42 in AEL (autophagy-endosomal-lysosomal) vesicles: potential implications for plaque biogenesis |
Q34031340 | Activation of JNK signaling mediates amyloid-ß-dependent cell death |
Q52644082 | Alzheimer's Disease and Sleep-Wake Disturbances: Amyloid, Astrocytes, and Animal Models. |
Q38532705 | Alzheimer's Disease, Drosophila melanogaster and Polyphenols. |
Q64880227 | Alzheimer’s Disease Associated Genes Ankyrin and Tau Cause Shortened Lifespan and Memory Loss in Drosophila. |
Q33815736 | Amyloid precursor proteins are protective in Drosophila models of progressive neurodegeneration |
Q38851223 | Amyloid-β induces sleep fragmentation that is rescued by fatty acid binding proteins in Drosophila |
Q26738294 | Analysis of Amyloid Precursor Protein Function in Drosophila melanogaster |
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Q89861787 | Effects of Gardenia jasminoides extracts on cognition and innate immune response in an adult Drosophila model of Alzheimer's disease |
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Q36760723 | Freeze-drying as sample preparation for micellar electrokinetic capillary chromatography-electrochemical separations of neurochemicals in Drosophila brains |
Q55409966 | Live Imaging Mitochondrial Transport in Neurons. |
Q27305748 | Loss of Polo ameliorates APP-induced Alzheimer's disease-like symptoms in Drosophila |
Q64235952 | Low-dose ionizing radiation alleviates Aβ42-induced cell death via regulating AKT and p38 pathways in Alzheimer's disease models |
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Q35048105 | Novel neuroprotective function of apical-basal polarity gene crumbs in amyloid beta 42 (aβ42) mediated neurodegeneration |
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Q37890901 | The power and richness of modelling tauopathies in Drosophila |
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