scholarly article | Q13442814 |
P50 | author | Michael Henry Malim | Q21166756 |
Susan J Little | Q90617607 | ||
Ramón Lorenzo-Redondo | Q42251907 | ||
John Archer | Q42833309 | ||
Steven M. Wolinsky | Q45517979 | ||
Will Fischer | Q54215194 | ||
Yoon-Seok Chung | Q55302534 | ||
Tanmoy Bhattacharya | Q55414889 | ||
Irina Maljkovic Berry | Q56364916 | ||
P2093 | author name string | Eun-Young Kim | |
Douglas D Richman | |||
Sudhir Penugonda | |||
Prabhjeet K Phalora | |||
P2860 | cites work | Dollo on Dollo's law: irreversibility and the status of evolutionary laws | Q15362938 |
HIV evolution in early infection: selection pressures, patterns of insertion and deletion, and the impact of APOBEC | Q21559422 | ||
Bayesian phylogenetics with BEAUti and the BEAST 1.7 | Q24286945 | ||
HIV-1 replication and APOBEC3 antiviral activity are not regulated by P bodies | Q24297395 | ||
Human and rhesus APOBEC3D, APOBEC3F, APOBEC3G, and APOBEC3H demonstrate a conserved capacity to restrict Vif-deficient HIV-1 | Q24321636 | ||
A second human antiretroviral factor, APOBEC3F, is suppressed by the HIV-1 and HIV-2 Vif proteins | Q24564187 | ||
MUSCLE: multiple sequence alignment with high accuracy and high throughput | Q24613456 | ||
HIV-1 Vif, APOBEC, and intrinsic immunity | Q24647833 | ||
Improved tools for biological sequence comparison | Q24652199 | ||
Polymorphisms and splice variants influence the antiretroviral activity of human APOBEC3H | Q24655418 | ||
Cytidine deamination induced HIV-1 drug resistance | Q24657298 | ||
Natural variation in Vif: differential impact on APOBEC3G/3F and a potential role in HIV-1 diversification | Q24817186 | ||
Multiple APOBEC3 restriction factors for HIV-1 and one Vif to rule them all | Q26823568 | ||
A Simple, Fast, and Accurate Algorithm to Estimate Large Phylogenies by Maximum Likelihood | Q27861000 | ||
Timing the ancestor of the HIV-1 pandemic strains | Q28137904 | ||
Retroelements versus APOBEC3 family members: No great escape from the magnificent seven | Q36162443 | ||
Suppression of HIV-1 infection by APOBEC3 proteins in primary human CD4(+) T cells is associated with inhibition of processive reverse transcription as well as excessive cytidine deamination | Q36559921 | ||
APOBEC-mediated viral restriction: not simply editing? | Q36736677 | ||
Conserved footprints of APOBEC3G on Hypermutated human immunodeficiency virus type 1 and human endogenous retrovirus HERV-K(HML2) sequences | Q36845949 | ||
Enhanced CD4+ T-cell recovery with earlier HIV-1 antiretroviral therapy | Q36854142 | ||
The APOBEC3 cytidine deaminases: an innate defensive network opposing exogenous retroviruses and endogenous retroelements | Q37096085 | ||
APOBEC proteins and intrinsic resistance to HIV-1 infection | Q37140468 | ||
A multi-site study using high-resolution HLA genotyping by next generation sequencing | Q38494977 | ||
Twin gradients in APOBEC3 edited HIV-1 DNA reflect the dynamics of lentiviral replication | Q39079333 | ||
APOBEC3G is a single-stranded DNA cytidine deaminase and functions independently of HIV reverse transcriptase | Q40899809 | ||
Polymorphism in human APOBEC3H affects a phenotype dominant for subcellular localization and antiviral activity | Q41900176 | ||
HIV-1 Vif adaptation to human APOBEC3H haplotypes | Q42261341 | ||
DNA deamination mediates innate immunity to retroviral infection | Q28179534 | ||
Induction of APOBEC3G Ubiquitination and Degradation by an HIV-1 Vif-Cul5-SCF Complex | Q28210666 | ||
APOBEC3F properties and hypermutation preferences indicate activity against HIV-1 in vivo | Q28275837 | ||
Not so different after all: a comparison of methods for detecting amino acid sites under selection | Q28306719 | ||
Viral population estimation using pyrosequencing | Q28472641 | ||
Likely role of APOBEC3G-mediated G-to-A mutations in HIV-1 evolution and drug resistance | Q28475150 | ||
Different mutagenic potential of HIV-1 restriction factors APOBEC3G and APOBEC3F is determined by distinct single-stranded DNA scanning mechanisms | Q28541264 | ||
Cytidine deamination of retroviral DNA by diverse APOBEC proteins | Q28578747 | ||
The evolutionary rate dynamically tracks changes in HIV-1 epidemics: application of a simple method for optimizing the evolutionary rate in phylogenetic trees with longitudinal data | Q28742086 | ||
Approximate likelihood-ratio test for branches: A fast, accurate, and powerful alternative | Q29547242 | ||
Datamonkey: rapid detection of selective pressure on individual sites of codon alignments | Q29615126 | ||
Single-strand specificity of APOBEC3G accounts for minus-strand deamination of the HIV genome | Q29618742 | ||
The antiretroviral enzyme APOBEC3G is degraded by the proteasome in response to HIV-1 Vif | Q29618938 | ||
Identification and characterization of transmitted and early founder virus envelopes in primary HIV-1 infection | Q29619510 | ||
Lower in vivo mutation rate of human immunodeficiency virus type 1 than that predicted from the fidelity of purified reverse transcriptase | Q29619994 | ||
Reduced APOBEC3H variant anti-viral activities are associated with altered RNA binding activities | Q31078477 | ||
Massive APOBEC3 editing of hepatitis B viral DNA in cirrhosis | Q33594457 | ||
The antiviral factor APOBEC3G improves CTL recognition of cultured HIV-infected T cells | Q33615181 | ||
Transmission of single HIV-1 genomes and dynamics of early immune escape revealed by ultra-deep sequencing | Q33680395 | ||
APOBEC3F and APOBEC3G inhibit HIV-1 DNA integration by different mechanisms. | Q33826968 | ||
Relationship between functional profile of HIV-1 specific CD8 T cells and epitope variability with the selection of escape mutants in acute HIV-1 infection | Q33828583 | ||
Human immunodeficiency virus type 1 DNA sequences genetically damaged by hypermutation are often abundant in patient peripheral blood mononuclear cells and may be generated during near-simultaneous infection and activation of CD4(+) T cells | Q33853620 | ||
Mutational and fitness landscapes of an RNA virus revealed through population sequencing | Q33953331 | ||
Genetic diversity of HIV-1: the moving target. | Q34087052 | ||
Selective escape from CD8+ T-cell responses represents a major driving force of human immunodeficiency virus type 1 (HIV-1) sequence diversity and reveals constraints on HIV-1 evolution | Q34092644 | ||
Human APOBEC3G-mediated editing can promote HIV-1 sequence diversification and accelerate adaptation to selective pressure | Q34120736 | ||
HIV-1 sequence variation: drift, shift, and attenuation | Q34175357 | ||
Analysis of high-depth sequence data for studying viral diversity: a comparison of next generation sequencing platforms using Segminator II. | Q34207464 | ||
APOBEC3G-induced hypermutation of human immunodeficiency virus type-1 is typically a discrete "all or nothing" phenomenon | Q34211562 | ||
Cellular immune responses to HIV. | Q34223069 | ||
Lack of evidence for proofreading mechanisms associated with an RNA virus polymerase | Q34239961 | ||
Vif hijacks CBF-β to degrade APOBEC3G and promote HIV-1 infection | Q34242229 | ||
High throughput HLA genotyping using 454 sequencing and the Fluidigm Access Array™ System for simplified amplicon library preparation. | Q34582241 | ||
Comparison of SIV and HIV-1 genomic RNA structures reveals impact of sequence evolution on conserved and non-conserved structural motifs | Q34671913 | ||
Analysis of human APOBEC3H haplotypes and anti-human immunodeficiency virus type 1 activity | Q34742607 | ||
The molecular clock of HIV-1 unveiled through analysis of a known transmission history | Q35631348 | ||
P275 | copyright license | Creative Commons CC0 License | Q6938433 |
P6216 | copyright status | copyrighted, dedicated to the public domain by copyright holder | Q88088423 |
P433 | issue | 7 | |
P304 | page(s) | e1004281 | |
P577 | publication date | 2014-07-31 | |
P1433 | published in | PLOS Pathogens | Q283209 |
P1476 | title | Human APOBEC3 induced mutation of human immunodeficiency virus type-1 contributes to adaptation and evolution in natural infection | |
P478 | volume | 10 |
Q37100587 | A Canonical Correlation Analysis of AIDS Restriction Genes and Metabolic Pathways Identifies Purine Metabolism as a Key Cooperator |
Q45324625 | APOBEC Enzymes as Targets for Virus and Cancer Therapy. |
Q47555165 | APOBEC mutagenesis in drug resistance and immune escape in HIV and cancer evolution |
Q36263411 | APOBEC3 Proteins in Viral Immunity. |
Q37264399 | APOBEC3 proteins can copackage and comutate HIV-1 genomes. |
Q39111231 | APOBEC3A/B-induced mutagenesis is responsible for 20% of heritable mutations in the TpCpW context |
Q61449156 | APOBEC3G Regulation of the Evolutionary Race Between Adaptive Immunity and Viral Immune Escape Is Deeply Imprinted in the HIV Genome |
Q37512973 | APOBEC3G-Mediated G-to-A Hypermutation of the HIV-1 Genome: The Missing Link in Antiviral Molecular Mechanisms |
Q38394187 | APOBECs and virus restriction |
Q41059063 | Antiretroviral activity and safety of once-daily etravirine in treatment-naive HIV-infected adults: 48-week results |
Q98159070 | Association between APOBEC3s and HPV16 E2 gene hypermutation in Uygur females with cervical cancer |
Q47380909 | Deep sequencing of HIV-1 reverse transcripts reveals the multifaceted antiviral functions of APOBEC3G. |
Q59351211 | Epstein-Barr virus BORF2 inhibits cellular APOBEC3B to preserve viral genome integrity |
Q57484166 | Estimating the mutational fitness effects distribution during early HIV infection |
Q57295397 | Genetic and mechanistic basis for APOBEC3H alternative splicing, retrovirus restriction, and counteraction by HIV-1 protease |
Q35834559 | HIV-1 and HIV-2 exhibit similar mutation frequencies and spectra in the absence of G-to-A hypermutation |
Q34473801 | HIV-1 and interferons: who's interfering with whom? |
Q46227536 | Increasing the CpG dinucleotide abundance in the HIV-1 genomic RNA inhibits viral replication. |
Q98665144 | Influenza virus repurposes the antiviral protein IFIT2 to promote translation of viral mRNAs |
Q59351278 | Inhibiting APOBEC3 Activity with Single-Stranded DNA Containing 2'-Deoxyzebularine Analogues |
Q35830651 | Interferon-α Subtypes in an Ex Vivo Model of Acute HIV-1 Infection: Expression, Potency and Effector Mechanisms |
Q30954229 | Intrinsic host restrictions to HIV-1 and mechanisms of viral escape. |
Q38688274 | Mechanisms of HIV-1 Control |
Q36019742 | Minimal Contribution of APOBEC3-Induced G-to-A Hypermutation to HIV-1 Recombination and Genetic Variation |
Q36811895 | Natural Single-Nucleotide Variations in the HIV-1 Genomic SA1prox Region Can Alter Viral Replication Ability by Regulating Vif Expression Levels |
Q36897321 | Persistent HIV-1 replication maintains the tissue reservoir during therapy |
Q93194984 | Polymorphisms of the cytidine deaminase APOBEC3F have different HIV-1 restriction efficiencies |
Q49205554 | Prediction of HIV-1 and HIV-2 proteins by using Chou's pseudo amino acid compositions and different classifiers. |
Q47554842 | Production of HIV-1 vif mRNA Is Modulated by Natural Nucleotide Variations and SLSA1 RNA Structure in SA1D2prox Genomic Region |
Q58583800 | RNA-Mediated Dimerization of the Human Deoxycytidine Deaminase APOBEC3H Influences Enzyme Activity and Interaction with Nucleic Acids |
Q38790689 | Reassessing APOBEC3G Inhibition by HIV-1 Vif-Derived Peptides |
Q35601655 | Reassessment of murine APOBEC1 as a retrovirus restriction factor in vivo |
Q33631993 | Recent advances in understanding HIV evolution |
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Q64109172 | Role of co-expressed APOBEC3F and APOBEC3G in inducing HIV-1 drug resistance |
Q37207674 | Role of the host restriction factor APOBEC3 on papillomavirus evolution |
Q35068754 | Sequence and structural determinants of human APOBEC3H deaminase and anti-HIV-1 activities |
Q39011114 | Sequence-Specific Sensing of Nucleic Acids |
Q37629738 | Structural basis for targeted DNA cytosine deamination and mutagenesis by APOBEC3A and APOBEC3B. |
Q39065035 | Viral reverse transcriptases. |
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