review article | Q7318358 |
scholarly article | Q13442814 |
P2093 | author name string | Jorge Monserrat | |
Melchor Alvarez-Mon | |||
Alfredo Prieto | |||
Raul de Pablo | |||
P2860 | cites work | Susceptibility to programmed cell death in T-lymphocytes from septic patients: a mechanism for lymphopenia and Th2 predominance. | Q44555497 |
Somatomedin-C as a fetal growth promoting factor and amino acid composition of cord blood in Japanese neonates | Q44691998 | ||
Tumour necrosis factor-alpha expression by activated monocytes and altered T-cell homeostasis in ascitic alcoholic cirrhosis: amelioration with norfloxacin | Q44805397 | ||
Mortality in patients with septic shock correlates with anti-inflammatory but not proinflammatory immunomodulatory molecules. | Q44829699 | ||
Interleukin-12 treatment restores normal resistance to bacterial challenge after burn injury | Q44973797 | ||
Caspase inhibitors improve survival in sepsis: a critical role of the lymphocyte | Q45018156 | ||
Trauma peptide induction of lymphocyte changes predictive of sepsis | Q46448754 | ||
Early activation of gammadelta T lymphocytes in patients with severe systemic inflammatory response syndrome | Q47331291 | ||
Direct bacterial protein PAMP recognition by human NK cells involves TLRs and triggers alpha-defensin production | Q47422836 | ||
Early changes in organ function predict eventual survival in severe sepsis | Q47713060 | ||
NKT cells: what's in a name? | Q47895830 | ||
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The role of regulatory T cells in the pathogenesis of sepsis and its clinical implication | Q27004368 | ||
Mechanisms underlying helper T-cell plasticity: implications for immune-mediated disease | Q27687830 | ||
Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells | Q27860460 | ||
Epidemiology of severe sepsis in the United States: analysis of incidence, outcome, and associated costs of care | Q27860820 | ||
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Transforming growth factor-beta induces development of the T(H)17 lineage | Q28237370 | ||
TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells | Q28296956 | ||
The epidemiology of sepsis in the United States from 1979 through 2000 | Q29547760 | ||
TH1 and TH2 cells: different patterns of lymphokine secretion lead to different functional properties | Q29547848 | ||
The pathophysiology and treatment of sepsis | Q29619103 | ||
The immunopathogenesis of sepsis | Q29622909 | ||
Both percentage of gammadelta T lymphocytes and CD3 expression are reduced during septic shock | Q33341913 | ||
Prevention of peripheral tolerance by a dendritic cell growth factor: flt3 ligand as an adjuvant | Q33368912 | ||
Differential role for CD80 and CD86 in the regulation of the innate immune response in murine polymicrobial sepsis | Q33492757 | ||
Immunotherapy - a potential new way forward in the treatment of sepsis | Q33749999 | ||
The efficacy of thymosin alpha 1 for severe sepsis (ETASS): a multicenter, single-blind, randomized and controlled trial | Q33750231 | ||
Early alterations of B cells in patients with septic shock | Q33752795 | ||
An anti-apoptotic peptide improves survival in lethal total body irradiation | Q33789426 | ||
The relationship between CD4+CD25+CD127- regulatory T cells and inflammatory response and outcome during shock states | Q33873468 | ||
Targeted adenovirus-induced expression of IL-10 decreases thymic apoptosis and improves survival in murine sepsis | Q33944663 | ||
Antimicrobial activity of mucosal-associated invariant T cells | Q34022899 | ||
T helper cell subset ratios in patients with severe sepsis. | Q51472529 | ||
Th memory for interleukin-17 expression is stable in vivo. | Q51961643 | ||
CD56brightCD16- killer Ig-like receptor- NK cells display longer telomeres and acquire features of CD56dim NK cells upon activation. | Q51990042 | ||
Peripheral human CD8(+)CD28(+)T lymphocytes give rise to CD28(-)progeny, but IL-4 prevents loss of CD28 expression. | Q52032875 | ||
Critical role of NK cells rather than V alpha 14(+)NKT cells in lipopolysaccharide-induced lethal shock in mice. | Q52956495 | ||
A divergent response of innate regulatory T-cells to sepsis in humans: circulating invariant natural killer T-cells are preserved. | Q53081823 | ||
Role of circulating soluble chemokines in septic shock. | Q53120623 | ||
Rapid increase in hospitalization and mortality rates for severe sepsis in the United States: a trend analysis from 1993 to 2003. | Q53238938 | ||
Identification of a detrimental role for NK cells in pneumococcal pneumonia and sepsis in immunocompromised hosts. | Q53676008 | ||
Early assessment of leukocyte alterations at diagnosis of septic shock. | Q54437566 | ||
Compensatory anti-inflammatory response syndrome | Q56270656 | ||
Decreased response to recall antigens is associated with depressed costimulatory receptor expression in septic critically ill patients | Q56906281 | ||
Apoptotic cell death in patients with sepsis, shock, and multiple organ dysfunction | Q57773627 | ||
Successful Cardiopulmonary Resuscitation After Cardiac Arrest as a “Sepsis-Like” Syndrome | Q58419479 | ||
A fatal cytokine-induced systemic inflammatory response reveals a critical role for NK cells | Q63968521 | ||
[Lymphocyte subpopulations in patients at risk of sepsis in a surgical intensive care unit] | Q68603361 | ||
Changes in major histocompatibility complex class II expression in monocytes and T cells of patients developing infection after surgery | Q70586254 | ||
Defective interferon-gamma production by T-lymphocytes from patients with acute brucellosis | Q71751061 | ||
Altered leukocyte immunophenotypes in septic shock. Studies of HLA-DR, CD11b, CD14, and IL-2R expression | Q72093787 | ||
Essential role for natural killer cells in the lethal lipopolysaccharide-induced Shwartzman-like reaction in mice | Q72416067 | ||
Lymphocyte subset responses to trauma and sepsis | Q72664456 | ||
Selective defects of T lymphocyte function in patients with lethal intraabdominal infection | Q73247605 | ||
Monocyte deactivation in septic patients: restoration by IFN-gamma treatment | Q73400029 | ||
Sepsis-induced apoptosis causes progressive profound depletion of B and CD4+ T lymphocytes in humans | Q73896312 | ||
Intrinsic differences in L-selectin expression levels affect T and B lymphocyte subset-specific recirculation pathways | Q74557624 | ||
Is Fas ligand or endotoxin responsible for mucosal lymphocyte apoptosis in sepsis? | Q77573339 | ||
Depletion of dendritic cells, but not macrophages, in patients with sepsis | Q77671545 | ||
CD69 is a stimulatory receptor for natural killer cell and its cytotoxic effect is blocked by CD94 inhibitory receptor | Q78127360 | ||
T lymphocytes | Q81599834 | ||
Harmful and protective roles of neutrophils in sepsis | Q81909493 | ||
Delayed administration of anti-PD-1 antibody reverses immune dysfunction and improves survival during sepsis | Q84255932 | ||
Newly discovered innate response activator B cells: crucial responders against microbial sepsis | Q84771180 | ||
The predictive role of early activation of natural killer cells in septic shock | Q36925691 | ||
gammadelta T cells: an important source of IL-17. | Q37012968 | ||
Associations of T helper 1, 2, 17 and regulatory T lymphocytes with mortality in severe sepsis | Q37015247 | ||
Induction and effector functions of T(H)17 cells. | Q37194494 | ||
Clinical relevance of the severe abnormalities of the T cell compartment in septic shock patients | Q37206791 | ||
Immunosuppression in sepsis: a novel understanding of the disorder and a new therapeutic approach | Q37233354 | ||
Regulatory NK-cell functions in inflammation and autoimmunity | Q37260883 | ||
Innovative therapeutic strategies for restoring lymphocyte functions in septic patients | Q37265016 | ||
Using surface molecule expression on lymphocytes to classify septic shock patients | Q37279882 | ||
Insights into the heterogeneity of human B cells: diverse functions, roles in autoimmunity, and use as therapeutic targets | Q37436035 | ||
The changing immune system in sepsis: is individualized immuno-modulatory therapy the answer? | Q37563023 | ||
The defensive alliance between neutrophils and NK cells as a novel arm of innate immunity. | Q37777529 | ||
Modulation of B cell responses by Toll-like receptors. | Q37786223 | ||
NKT cells: the culprits of sepsis? | Q37805051 | ||
B cells--masters of the immunoverse | Q37819339 | ||
The role of natural killer cells in sepsis | Q37883097 | ||
Anti-inflammatory mechanisms of sepsis | Q37887363 | ||
Natural killer (NK) cells in antibacterial innate immunity: angels or devils? | Q37959322 | ||
Immune regulatory function of B cells. | Q37973774 | ||
Integration of B cell responses through Toll-like receptors and antigen receptors | Q37994060 | ||
Bacterial sensing, cell signaling, and modulation of the immune response during sepsis | Q38024966 | ||
Thymosin α1 as a stimulatory agent of innate cell-mediated immune response. | Q38050836 | ||
Lymphocyte subset numbers depend on the bacterial origin of sepsis | Q39074723 | ||
Changes in lymphocyte subpopulations and CD3+/DR+ expression in sepsis | Q39074746 | ||
Late developmental plasticity in the T helper 17 lineage | Q39265291 | ||
Sepsis and the innate-like response | Q39298329 | ||
Delayed hypersensitivity: indicator of acquired failure of host defenses in sepsis and trauma | Q39823225 | ||
CD11b expression identifies CD8+CD28+ T lymphocytes with phenotype and function of both naive/memory and effector cells | Q40833951 | ||
Leukocyte phenotyping to stratify septic shock patients. | Q42237092 | ||
Specific MAIT cell behaviour among innate-like T lymphocytes in critically ill patients with severe infections. | Q42250131 | ||
Characterization of T-cell responses against immunodominant epitopes from hepatitis C virus E2 and NS4a proteins. | Q42997235 | ||
Improved survival in experimental sepsis with an orally administered inhibitor of apoptosis | Q43684928 | ||
Natural killer cells contribute to the lethality of a murine model of Escherichia coli infection | Q44130101 | ||
Increased lipopolysaccharide binding protein in cirrhotic patients with marked immune and hemodynamic derangement. | Q44261210 | ||
Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defense | Q34087551 | ||
IL-15 prevents apoptosis, reverses innate and adaptive immune dysfunction, and improves survival in sepsis. | Q34120958 | ||
Interleukin-7 (IL-7) treatment accelerates neutrophil recruitment through gamma delta T-cell IL-17 production in a murine model of sepsis. | Q34290886 | ||
Dendritic cells as a tool to induce anergic and regulatory T cells | Q34295082 | ||
Rapid activation of the T-cell tyrosine protein kinase pp56lck by the CD45 phosphotyrosine phosphatase | Q34297751 | ||
Immunodepression in sepsis and SIRS assessed by ex vivo cytokine production is not a generalized phenomenon: a review | Q34345653 | ||
The interaction of human natural killer cells with either unpolarized or polarized macrophages results in different functional outcomes | Q34411026 | ||
Phenotype and functions of natural killer cells in critically-ill septic patients | Q34510379 | ||
Marked elevation of human circulating CD4+CD25+ regulatory T cells in sepsis-induced immunoparalysis. | Q34535214 | ||
Monitoring immune dysfunctions in the septic patient: a new skin for the old ceremony | Q34585151 | ||
Increased circulating regulatory T cells (CD4(+)CD25 (+)CD127 (-)) contribute to lymphocyte anergy in septic shock patients | Q34599210 | ||
PD-1 expression by macrophages plays a pathologic role in altering microbial clearance and the innate inflammatory response to sepsis | Q34605998 | ||
CD45: a critical regulator of signaling thresholds in immune cells | Q34987899 | ||
Adult survivors of invasive pneumococcal disease exhibit defective B cell function | Q35019937 | ||
B cells enhance early innate immune responses during bacterial sepsis | Q35144572 | ||
Programmed death-1 levels correlate with increased mortality, nosocomial infection and immune dysfunctions in septic shock patients. | Q35559497 | ||
PD-L1 blockade improves survival in experimental sepsis by inhibiting lymphocyte apoptosis and reversing monocyte dysfunction | Q35561405 | ||
Early changes of CD4-positive lymphocytes and NK cells in patients with severe Gram-negative sepsis | Q35629894 | ||
Innate response activator B cells protect against microbial sepsis. | Q35757819 | ||
Clinical review: flow cytometry perspectives in the ICU - from diagnosis of infection to monitoring of injury-induced immune dysfunctions | Q35908002 | ||
Early natural killer cell counts in blood predict mortality in severe sepsis | Q35908269 | ||
Immunosuppression in patients who die of sepsis and multiple organ failure | Q35991015 | ||
Murine gammadelta T cells in infections: beneficial or deleterious? | Q36074898 | ||
NK but not CD1-restricted NKT cells facilitate systemic inflammation during polymicrobial intra-abdominal sepsis | Q36146173 | ||
Natural-killer cells and dendritic cells: "l'union fait la force". | Q36149272 | ||
Anti-CD23. | Q36284905 | ||
Prevention of lymphocyte apoptosis--a potential treatment of sepsis? | Q36291734 | ||
Differential tumor surveillance by natural killer (NK) and NKT cells. | Q36375962 | ||
CD40 and CD80/86 act synergistically to regulate inflammation and mortality in polymicrobial sepsis | Q36414862 | ||
The role of CD28 and cytotoxic T-lymphocyte antigen-4 (CTLA-4) in regulatory T-cell biology | Q36563520 | ||
Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3 | Q36742247 | ||
Prevention of lymphocyte cell death in sepsis improves survival in mice | Q36748242 | ||
Association of γδ T cells with disease severity and mortality in septic patients | Q36826310 | ||
P275 | copyright license | Creative Commons Attribution 3.0 Unported | Q14947546 |
P6216 | copyright status | copyrighted | Q50423863 |
P407 | language of work or name | English | Q1860 |
P921 | main subject | infection | Q166231 |
sepsis | Q183134 | ||
peptide | Q172847 | ||
cell | Q7868 | ||
cytokine | Q212354 | ||
adaptive immune system | Q1645075 | ||
biomedical investigative technique | Q66648976 | ||
hemic and immune systems | Q70202933 | ||
P304 | page(s) | 671087 | |
P577 | publication date | 2014-08-28 | |
P1433 | published in | BioMed Research International | Q17509958 |
P1476 | title | Role of circulating lymphocytes in patients with sepsis | |
P478 | volume | 2014 |
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