scholarly article | Q13442814 |
P2093 | author name string | J Y Lee | |
M I Polkey | |||
U Rosendahl | |||
P R Kemp | |||
M J D Griffiths | |||
T Syburra | |||
S A A Bloch | |||
P2860 | cites work | miR-181a promotes osteoblastic differentiation through repression of TGF-β signaling molecules. | Q43494942 |
Sustained elevation of circulating growth and differentiation factor-15 and a dynamic imbalance in mediators of muscle homeostasis are associated with the development of acute muscle wasting following cardiac surgery | Q44678534 | ||
Expression and regulation of CCN genes in murine osteoblasts | Q46823121 | ||
Muscle atrophy and preferential loss of myosin in prolonged critically ill patients | Q51007787 | ||
Muscle thickness, measured with ultrasound, may be an indicator of lean tissue wasting in multiple organ failure in the presence of edema | Q51595582 | ||
Novel events in the molecular regulation of muscle mass in critically ill patients. | Q54582240 | ||
Down-regulation of insulin-like growth factor I (IGF-I) in the mouse diaphragm during sepsis | Q82076052 | ||
The temporal responses of protein synthesis, gene expression and cell signalling in human quadriceps muscle and patellar tendon to disuse | Q24642532 | ||
The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation | Q24650204 | ||
Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources | Q27860739 | ||
Mechanisms of TGF-beta signaling from cell membrane to the nucleus | Q27860785 | ||
Serial evaluation of the SOFA score to predict outcome in critically ill patients | Q28191384 | ||
Downregulation of miR-133 and miR-590 contributes to nicotine-induced atrial remodelling in canines | Q28242982 | ||
The microRNA miR-181 targets the homeobox protein Hox-A11 during mammalian myoblast differentiation | Q28298317 | ||
Adaptive Induction of Growth Differentiation Factor 15 Attenuates Endothelial Cell Apoptosis in Response to High Glucose Stimulus | Q31120253 | ||
Uncoupling of expression of an intronic microRNA and its myosin host gene by exon skipping | Q33769212 | ||
Mammalian target of rapamycin regulates miRNA-1 and follistatin in skeletal myogenesis | Q33950260 | ||
Divergent molecular mechanisms underlying the pleiotropic functions of macrophage inhibitory cytokine-1 in cancer | Q34099900 | ||
Paresis acquired in the intensive care unit: a prospective multicenter study | Q34163357 | ||
miRWalk – Database: Prediction of possible miRNA binding sites by “walking” the genes of three genomes | Q34186822 | ||
Role of growth hormone, insulin-like growth factor-I, and insulin-like growth factor binding proteins in the catabolic response to injury and infection | Q34601336 | ||
Downregulation of the serum response factor/miR-1 axis in the quadriceps of patients with COPD. | Q35619124 | ||
Histone deacetylase inhibition suppresses myogenin-dependent atrogene activation in spinal muscular atrophy mice | Q36486738 | ||
Growth differentiation factor-15 and prognosis in acute respiratory distress syndrome: a retrospective cohort study | Q36997947 | ||
microRNAs and muscle disorders | Q37353526 | ||
Evidence of MyomiR network regulation of beta-myosin heavy chain gene expression during skeletal muscle atrophy | Q37462876 | ||
A framework for diagnosing and classifying intensive care unit-acquired weakness | Q37666319 | ||
The miRNA pathway in neurological and skeletal muscle disease: implications for pathogenesis and therapy. | Q37900519 | ||
Molecular mechanisms of intensive care unit-acquired weakness. | Q37941549 | ||
The central role of myostatin in skeletal muscle and whole body homeostasis | Q37984873 | ||
Intensive care unit-acquired weakness: clinical phenotypes and molecular mechanisms. | Q38064396 | ||
The multiple facets of the TGF-β family cytokine growth/differentiation factor-15/macrophage inhibitory cytokine-1 | Q38115886 | ||
Growth differentiation factor 15 acts anti-apoptotic and pro-hypertrophic in adult cardiomyocytes | Q38345677 | ||
Inhibition of myogenic microRNAs 1, 133, and 206 by inflammatory cytokines links inflammation and muscle degeneration in adult inflammatory myopathies. | Q39001797 | ||
Tumor-induced anorexia and weight loss are mediated by the TGF-beta superfamily cytokine MIC-1. | Q40056643 | ||
Acute skeletal muscle wasting in critical illness. | Q43466407 | ||
P433 | issue | 3 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | microRNA | Q310899 |
P304 | page(s) | 219-228 | |
P577 | publication date | 2014-12-16 | |
P1433 | published in | Thorax | Q7796158 |
P1476 | title | Increased expression of GDF-15 may mediate ICU-acquired weakness by down-regulating muscle microRNAs | |
P478 | volume | 70 |
Q90341015 | Circulatory factors associated with function and prognosis in patients with severe heart failure |
Q26799076 | Clinical review: intensive care unit acquired weakness |
Q50218185 | Critical illness myopathy and polyneuropathy |
Q36805120 | Effects of elastic band resistance training and nutritional supplementation on muscle quality and circulating muscle growth and degradation factors of institutionalized elderly women: the Vienna Active Ageing Study (VAAS) |
Q49339915 | Exercise increases circulating GDF15 in humans. |
Q90460790 | Growth Differentiation Factor-15 (GDF-15) is a Biomarker of Muscle Wasting and Renal Dysfunction in Preoperative Cardiovascular Surgery Patients |
Q47684686 | Growth differentiation factor 15 predicts advanced fibrosis in biopsy-proven non-alcoholic fatty liver disease. |
Q90167759 | Growth differentiation factor-15 as a biomarker of strength and recovery in survivors of acute respiratory failure |
Q36893044 | Growth differentiation factor-15 is associated with muscle mass in chronic obstructive pulmonary disease and promotes muscle wasting in vivo. |
Q63916376 | Growth/differentiation factor 15 causes TGFβ-activated kinase 1-dependent muscle atrophy in pulmonary arterial hypertension |
Q35492752 | MiR-181a: a potential biomarker of acute muscle wasting following elective high-risk cardiothoracic surgery |
Q47801556 | MicroRNA-542 Promotes Mitochondrial Dysfunction and SMAD Activity and Is Elevated in Intensive Care Unit-acquired Weakness. |
Q49399854 | Models of disuse muscle atrophy: therapeutic implications in critically ill patients |
Q37357978 | Muscle Regeneration after Critical Illness: Are Satellite Cells the Answer? |
Q42541271 | Muscle mitochondrial stress adaptation operates independently of endogenous FGF21 action |
Q47294663 | New device for nonvolitional evaluation of quadriceps force in ventilated patients |
Q55022891 | Sarcopenia: assessment of disease burden and strategies to improve outcomes. |
Q124149451 | Skeletal muscle alterations in patients with acute Covid‐19 and post‐acute sequelae of Covid‐19 |
Q39015167 | The increasing need for biomarkers in intensive care unit-acquired weakness--are microRNAs the solution? |
Q61798293 | Tocotrienol-Rich Fraction (TRF) Treatment Promotes Proliferation Capacity of Stress-Induced Premature Senescence Myoblasts and Modulates the Renewal of Satellite Cells: Microarray Analysis |
Q89911643 | Utility of Plasma GDF-15 for Diagnosis and Prognosis Assessment of ICU-Acquired Weakness in Mechanically Ventilated Patients: Prospective Observational Study |
Q64979492 | mTORC1 underlies age-related muscle fiber damage and loss by inducing oxidative stress and catabolism. |
Q89938818 | miR-1-5p targets TGF-βR1 and is suppressed in the hypertrophying hearts of rats with pulmonary arterial hypertension |
Q47688387 | miR-422a suppresses SMAD4 protein expression and promotes resistance to muscle loss |
Q47320378 | miR-424-5p reduces ribosomal RNA and protein synthesis in muscle wasting. |
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