Immune evasion strategies of Porphyromonas gingivalis.

scientific article published on January 2011

Immune evasion strategies of Porphyromonas gingivalis. is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1016/S1349-0079(11)80006-X
10.2330/JORALBIOSCI.53.233
P932PMC publication ID3231999
P698PubMed publication ID22162663

P50authorGeorge HajishengallisQ39672238
P2093author name stringGeorge Hajishengallis
P2860cites workEarly and preferential induction of IL-1 receptor-associated kinase-M in THP-1 cells by LPS derived from Porphyromonas gingivalisQ24304456
The C5a receptor impairs IL-12-dependent clearance of Porphyromonas gingivalis and is required for induction of periodontal bone lossQ24618908
Gingipain-dependent interactions with the host are important for survival of Porphyromonas gingivalisQ24630104
Pathogen induction of CXCR4/TLR2 cross-talk impairs host defense functionQ24652536
Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?Q24655246
Porphyromonas gingivalis fimbriae proactively modulate beta2 integrin adhesive activity and promote binding to and internalization by macrophagesQ24678639
Complement: a key system for immune surveillance and homeostasisQ29615484
Recognition of microorganisms and activation of the immune responseQ29617903
Microbial manipulation of receptor crosstalk in innate immunityQ30499616
Identification of a second lipopolysaccharide in Porphyromonas gingivalis W50Q33319075
Complement evasion by human pathogensQ33623972
Subgingival colonization by Porphyromonas gingivalisQ34129081
Periodontal diseases.Q34468885
Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection.Q34862986
Corruption of innate immunity by bacterial proteasesQ35003110
"Gum bug, leave my heart alone!"--epidemiologic and mechanistic evidence linking periodontal infections and atherosclerosis.Q35866675
Porphyromonas gingivalis interactions with complement receptor 3 (CR3): innate immunity or immune evasion?Q36814065
Pathogen subversion of cell-intrinsic innate immunityQ36977518
Binding of complement inhibitor C4b-binding protein contributes to serum resistance of Porphyromonas gingivalisQ37015215
Interpain A, a cysteine proteinase from Prevotella intermedia, inhibits complement by degrading complement factor C3.Q37097298
Induction of distinct TLR2-mediated proinflammatory and proadhesive signaling pathways in response to Porphyromonas gingivalis fimbriaeQ37200512
A new inflammatory cytokine on the block: re-thinking periodontal disease and the Th1/Th2 paradigm in the context of Th17 cells and IL-17.Q37249268
The chronicles of Porphyromonas gingivalis: the microbium, the human oral epithelium and their interplay.Q37285445
Surface components of Porphyromonas gingivalisQ37313453
LPS response and tolerance in the zebrafish (Danio rerio)Q37356543
Antimicrobial mechanisms of phagocytes and bacterial evasion strategiesQ37445493
The oral microbial consortium's interaction with the periodontal innate defense systemQ37478630
Complementary Tolls in the periodontium: how periodontal bacteria modify complement and Toll-like receptor responses to prevail in the hostQ37486358
Periodontitis: a polymicrobial disruption of host homeostasisQ37761827
Outer membrane vesicles function as offensive weapons in host-parasite interactionsQ37777812
Mechanisms of resistance of Porphyromonas gingivalis to killing by serum complementQ38481466
Microbial complexes in subgingival plaqueQ38553206
Porphyromonas gingivalis mediates the shedding and proteolysis of complement regulatory protein CD46 expressed by oral epithelial cellsQ39809096
Differential interactions of fimbriae and lipopolysaccharide from Porphyromonas gingivalis with the Toll-like receptor 2-centred pattern recognition apparatusQ40230062
TLR2 transmodulates monocyte adhesion and transmigration via Rac1- and PI3K-mediated inside-out signaling in response to Porphyromonas gingivalis fimbriaeQ40271727
MD-2 mediates the ability of tetra-acylated and penta-acylated lipopolysaccharides to antagonize Escherichia coli lipopolysaccharide at the TLR4 signaling complexQ40370317
Human Toll-like receptor 4 responses to P. gingivalis are regulated by lipid A 1- and 4'-phosphatase activitiesQ42714693
Microbial hijacking of complement-toll-like receptor crosstalkQ42911622
Periodontitis in RA-the citrullinated enolase connection.Q42916208
Complement receptor 3 blockade promotes IL-12-mediated clearance of Porphyromonas gingivalis and negates its virulence in vivoQ43985683
Integrin activation by bacterial fimbriae through a pathway involving CD14, Toll-like receptor 2, and phosphatidylinositol-3-kinaseQ45288377
"Complementing" toll signaling.Q53325906
Fimbrial proteins of porphyromonas gingivalis mediate in vivo virulence and exploit TLR2 and complement receptor 3 to persist in macrophages.Q54476982
Cutting Edge: TLR2 is required for the innate response to Porphyromonas gingivalis: activation leads to bacterial persistence and TLR2 deficiency attenuates induced alveolar bone resorptionQ79401044
Biphasic effect of gingipains from Porphyromonas gingivalis on the human complement systemQ80358557
P433issue3
P921main subjectevasion of host immune responseQ1660157
Porphyromonas gingivalisQ3214147
P304page(s)233-240
P577publication date2011-01-01
P1433published inJournal of oral biosciencesQ27720984
P1476titleImmune evasion strategies of Porphyromonas gingivalis
P478volume53

Reverse relations

cites work (P2860)
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