scholarly article | Q13442814 |
P2093 | author name string | Aaron M Stepanek | |
Corrella S Detweiler | |||
Diane E Brown | |||
Heidi J Nick | |||
Karyn E O'Connell | |||
Maria C Pilonieta | |||
Melissa W McCoy | |||
Ross Benik | |||
Sarah M Moreland | |||
Toni A Nagy | |||
P2860 | cites work | Hepcidin regulates cellular iron efflux by binding to ferroportin and inducing its internalization | Q24310115 |
Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method | Q25938999 | ||
Hemophagocytic macrophages harbor Salmonella enterica during persistent infection | Q27319444 | ||
Lack of a role for iron in the Lyme disease pathogen | Q28145506 | ||
Typhoid fever | Q28216285 | ||
The iron exporter ferroportin/Slc40a1 is essential for iron homeostasis | Q28264540 | ||
The Ity/Lsh/Bcg locus: natural resistance to infection with intracellular parasites is abrogated by disruption of the Nramp1 gene | Q28589797 | ||
Chronic murine typhoid fever is a natural model of secondary hemophagocytic lymphohistiocytosis | Q33388431 | ||
The battle for iron between bacterial pathogens and their vertebrate hosts | Q33658687 | ||
Pro-inflammatory cytokine-mediated anemia: regarding molecular mechanisms of erythropoiesis | Q33696464 | ||
Taming the elephant: Salmonella biology, pathogenesis, and prevention | Q33877264 | ||
Salmonella acquires ferrous iron from haemophagocytic macrophages | Q34161257 | ||
Sustained IL-4 exposure leads to a novel pathway for hemophagocytosis, inflammation, and tissue macrophage accumulation | Q34193528 | ||
Polarization dictates iron handling by inflammatory and alternatively activated macrophages | Q34255058 | ||
Anemia of chronic disease. | Q34401570 | ||
Animal models of Salmonella infections: enteritis versus typhoid fever. | Q34469859 | ||
Repeated TLR9 stimulation results in macrophage activation syndrome-like disease in mice. | Q35015661 | ||
Salmonella enterica infection stimulates macrophages to hemophagocytose | Q35072142 | ||
Innate immune activation during Salmonella infection initiates extramedullary erythropoiesis and splenomegaly | Q35109868 | ||
Hemophagocytosis causes a consumptive anemia of inflammation | Q35213574 | ||
Contribution of TonB- and Feo-mediated iron uptake to growth of Salmonella typhimurium in the mouse | Q35528349 | ||
Suppression of hepcidin during anemia requires erythropoietic activity | Q35848905 | ||
Persistent bacterial infections: the interface of the pathogen and the host immune system | Q35889600 | ||
The molecular regulation of iron metabolism | Q35901908 | ||
Slc11a1 (Nramp1) impairs growth of Salmonella enterica serovar typhimurium in macrophages via stimulation of lipocalin-2 expression | Q36091290 | ||
Hemophagocytic macrophages in murine typhoid fever have an anti-inflammatory phenotype | Q36277460 | ||
Slc11a1 limits intracellular growth of Salmonella enterica sv. Typhimurium by promoting macrophage immune effector functions and impairing bacterial iron acquisition. | Q36305784 | ||
Antimicrobial actions of the NADPH phagocyte oxidase and inducible nitric oxide synthase in experimental salmonellosis. I. Effects on microbial killing by activated peritoneal macrophages in vitro | Q36368785 | ||
Histopathology of the spleen | Q36635915 | ||
Iron depletion limits intracellular bacterial growth in macrophages | Q36787471 | ||
Nitric oxide-mediated regulation of ferroportin-1 controls macrophage iron homeostasis and immune function in Salmonella infection | Q36822684 | ||
The systemic immune state of super-shedder mice is characterized by a unique neutrophil-dependent blunting of TH1 responses | Q36908818 | ||
Immunolocalization of ferroportin in healthy and anemic mice | Q37020961 | ||
Salmonella enterica causes more severe inflammatory disease in C57/BL6 Nramp1G169 mice than Sv129S6 mice | Q37161148 | ||
Salmonella require the fatty acid regulator PPARδ for the establishment of a metabolic environment essential for long-term persistence | Q37202938 | ||
Iron metabolism in the anemia of chronic disease. | Q37266866 | ||
The expression of exogenous genes in macrophages: obstacles and opportunities | Q37433952 | ||
The Salmonella enterica serovar typhimurium divalent cation transport systems MntH and SitABCD are essential for virulence in an Nramp1G169 murine typhoid model | Q37521702 | ||
Hepcidin induction by pathogens and pathogen-derived molecules is strongly dependent on interleukin-6 | Q37546397 | ||
How to become a top model: impact of animal experimentation on human Salmonella disease research. | Q37845999 | ||
The utility of immunohistochemistry for the identification of hematopoietic and lymphoid cells in normal tissues and interpretation of proliferative and inflammatory lesions of mice and rats | Q37995585 | ||
Role of Ferroportin in Macrophage-Mediated Immunity | Q39656348 | ||
Interferon-gamma limits the availability of iron for intramacrophage Salmonella typhimurium. | Q39968051 | ||
The iron efflux protein ferroportin regulates the intracellular growth of Salmonella enterica | Q40290550 | ||
Cytokine patterns at the site of mycobacterial infection | Q40505087 | ||
Mycobacterium avium complex-associated hemophagocytic syndrome in systemic lupus erythematosus patient: report of one case | Q40559772 | ||
Evaluation of the erythroid regenerative response in two different models of experimentally induced iron deficiency anemia | Q44877853 | ||
The co-ordinated regulation of iron homeostasis in murine macrophages limits the availability of iron for intracellular Salmonella typhimurium | Q50072262 | ||
Elevated neutrophil, macrophage and dendritic cell numbers characterize immune cell populations in mice chronically infected with Salmonella | Q50079634 | ||
Cachectin/tumor necrosis factor-alpha alters red blood cell kinetics and induces anemia in vivo | Q50195798 | ||
Aromatic-dependent Salmonella typhimurium as modified live vaccines for calves | Q50209788 | ||
Regulation of iron homeostasis in anemia of chronic disease and iron deficiency anemia: diagnostic and therapeutic implications. | Q51764368 | ||
CD40 and tumour necrosis factor-α co-operate to up-regulate inducuble nitric oxide synthase expression in macrophages. | Q53208850 | ||
Differential regulation of iron homeostasis during human macrophage polarized activation. | Q53735318 | ||
Release of reactive nitrogen intermediates and reactive oxygen intermediates from mouse peritoneal macrophages. Comparison of activating cytokines and evidence for independent production | Q70405593 | ||
P433 | issue | 6 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Salmonella enterica | Q2264864 |
P304 | page(s) | 2290-2299 | |
P577 | publication date | 2015-03-30 | |
P1433 | published in | Infection and Immunity | Q6029193 |
P1476 | title | Increased ferroportin-1 expression and rapid splenic iron loss occur with anemia caused by Salmonella enterica Serovar Typhimurium infection in mice | |
P478 | volume | 83 |
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