Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression

scientific article published on May 2007

Direct glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.1101/GAD.426007
P932PMC publication ID1865487
P698PubMed publication ID17504935
P5875ResearchGate publication ID6327333

P50authorRichard MorigglQ38327183
Jan-Wilhelm KornfeldQ39271588
Lothar HennighausenQ51971727
P2093author name stringAndreas Reimann
Hartmut Beug
Günther Schütz
David Engblom
Lukas Schwake
Francois Tronche
P2860cites workMAPPFinder: using Gene Ontology and GenMAPP to create a global gene-expression profile from microarray dataQ24804397
Cellular localization and regulation of gene expression for components of the insulin-like growth factor ternary binding protein complexQ28286146
Functional interactions between Stat5 and the glucocorticoid receptorQ28294034
Stat5a and Stat5b proteins have essential and nonessential, or redundant, roles in cytokine responsesQ28592023
In vivo analysis of growth hormone receptor signaling domains and their associated transcriptsQ33551712
Clarifying the role of Stat5 in lymphoid development and Abelson-induced transformationQ33874580
Circulating levels of IGF-1 directly regulate bone growth and densityQ34149771
Stat5a/b are essential for normal lymphoid development and differentiationQ34253065
DNA binding of the glucocorticoid receptor is not essential for survivalQ34469789
Liver-derived insulin-like growth factor I (IGF-I) is the principal source of IGF-I in blood but is not required for postnatal body growth in miceQ36392687
Normal growth and development in the absence of hepatic insulin-like growth factor IQ36394699
Deletion of the carboxyl-terminal transactivation domain of MGF-Stat5 results in sustained DNA binding and a dominant negative phenotype.Q36562956
Requirement of STAT5b for sexual dimorphism of body growth rates and liver gene expressionQ36653664
Glucocorticoid receptor function in hepatocytes is essential to promote postnatal body growthQ37092115
Inactivation of Stat5 in mouse mammary epithelium during pregnancy reveals distinct functions in cell proliferation, survival, and differentiationQ37493064
Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptorQ38293939
Specific DNA binding of Stat5, but not of glucocorticoid receptor, is required for their functional cooperation in the regulation of gene transcriptionQ40023915
Interleukin-2 inhibits glucocorticoid receptor transcriptional activity through a mechanism involving STAT5 (signal transducer and activator of transcription 5) but not AP-1.Q40795105
Hepatocyte-specific expression of Cre recombinaseQ43604415
Sex-dependent liver gene expression is extensive and largely dependent upon signal transducer and activator of transcription 5b (STAT5b): STAT5b-dependent activation of male genes and repression of female genes revealed by microarray analysis.Q51819758
Stat5 tetramer formation is associated with leukemogenesisQ58326863
Growth hormone stimulates longitudinal bone growth directlyQ70357932
Postnatal body growth is dependent on the transcription factors signal transducers and activators of transcription 5a/b in muscle: a role for autocrine/paracrine insulin-like growth factor IQ79421896
P433issue10
P304page(s)1157-1162
P577publication date2007-05-01
P1433published inGenes & DevelopmentQ1524533
P1476titleDirect glucocorticoid receptor-Stat5 interaction in hepatocytes controls body size and maturation-related gene expression
P478volume21

Reverse relations

cites work (P2860)
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