scholarly article | Q13442814 |
P50 | author | Elliott D. Kieff | Q43399614 |
P2093 | author name string | C Miller | |
J Lin | |||
E S Robertson | |||
B Tomkinson | |||
E Johannsen | |||
S Grossman | |||
P2860 | cites work | TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms | Q24320364 |
A Ca2+/calmodulin-dependent protein kinase, CaM kinase-Gr, expressed after transformation of primary human B lymphocytes by Epstein-Barr virus (EBV) is induced by the EBV oncogene LMP1 | Q24323156 | ||
Transcriptional regulation in mammalian cells by sequence-specific DNA binding proteins | Q27861094 | ||
The macrophage and B cell-specific transcription factor PU.1 is related to the ets oncogene | Q28116777 | ||
Scissors-grip model for DNA recognition by a family of leucine zipper proteins | Q28272775 | ||
CAT constructions with multiple unique restriction sites for the functional analysis of eukaryotic promoters and regulatory elements | Q29618195 | ||
Epstein-Barr virus-encoded nuclear antigen 2 activates the viral latent membrane protein promoter by modulating the activity of a negative regulatory element | Q33810690 | ||
DNA sequence and expression of the B95-8 Epstein-Barr virus genome. | Q34245077 | ||
Epstein-Barr virus nuclear protein 2 is a key determinant of lymphocyte transformation | Q34324039 | ||
The Epstein-Barr virus nuclear antigen 2 interacts with an EBNA2 responsive cis-element of the terminal protein 1 gene promoter | Q34355226 | ||
Epstein-Barr virus (EBV) induces expression of B-cell activation markers on in vitro infection of EBV-negative B-lymphoma cells | Q34366291 | ||
Epstein-Barr virus nuclear antigen 2 specifically induces expression of the B-cell activation antigen CD23. | Q34625121 | ||
The Epstein-Barr virus nuclear antigen 2 transactivator is directed to response elements by the J kappa recombination signal binding protein | Q35648541 | ||
Epstein-Barr virus nuclear protein 2 transactivation of the latent membrane protein 1 promoter is mediated by J kappa and PU.1. | Q35829475 | ||
The Epstein-Barr virus nuclear protein 2 acidic domain can interact with TFIIB, TAF40, and RPA70 but not with TATA-binding protein. | Q35830605 | ||
Reducing the complexity of the transforming Epstein-Barr virus genome to 64 kilobase pairs. | Q35832104 | ||
A cis-acting element from the Epstein-Barr viral genome that permits stable replication of recombinant plasmids in latently infected cells | Q36263086 | ||
Epstein-Barr virus latent membrane protein 1 is essential for B-lymphocyte growth transformation | Q36575274 | ||
The Epstein-Barr virus immortalizing protein EBNA-2 is targeted to DNA by a cellular enhancer-binding protein | Q36581891 | ||
Epstein-Barr virus recombinant molecular genetic analysis of the LMP1 amino-terminal cytoplasmic domain reveals a probable structural role, with no component essential for primary B-lymphocyte growth transformation | Q36622140 | ||
An Epstein-Barr virus with a 58-kilobase-pair deletion that includes BARF0 transforms B lymphocytes in vitro | Q36628631 | ||
The EBNA-2 arginine-glycine domain is critical but not essential for B-lymphocyte growth transformation; the rest of region 3 lacks essential interactive domains. | Q36635773 | ||
Deletion of DNA encoding the first five transmembrane domains of Epstein-Barr virus latent membrane proteins 2A and 2B. | Q36651589 | ||
The recombination signal sequence-binding protein RBP-2N functions as a transcriptional repressor | Q36652805 | ||
Mouse beta-globin DNA-binding protein B1 is identical to a proto-oncogene, the transcription factor Spi-1/PU.1, and is restricted in expression to hematopoietic cells and the testis | Q36682511 | ||
The only domain which distinguishes Epstein-Barr virus latent membrane protein 2A (LMP2A) from LMP2B is dispensable for lymphocyte infection and growth transformation in vitro; LMP2A is therefore nonessential | Q36685081 | ||
The last seven transmembrane and carboxy-terminal cytoplasmic domains of Epstein-Barr virus latent membrane protein 2 (LMP2) are dispensable for lymphocyte infection and growth transformation in vitro. | Q36686042 | ||
Epstein-Barr virus nuclear proteins EBNA-3A and EBNA-3C are essential for B-lymphocyte growth transformation. | Q36686055 | ||
Epstein-Barr virus nuclear protein 2 mutations define essential domains for transformation and transactivation | Q36689004 | ||
Identification of critical cis elements involved in mediating Epstein-Barr virus nuclear antigen 2-dependent activity of an enhancer located upstream of the viral BamHI C promoter | Q36697355 | ||
Use of second-site homologous recombination to demonstrate that Epstein-Barr virus nuclear protein 3B is not important for lymphocyte infection or growth transformation in vitro | Q36697392 | ||
Epstein-Barr virus types 1 and 2 differ in their EBNA-3A, EBNA-3B, and EBNA-3C genes | Q36782491 | ||
Epstein-Barr virus latent membrane protein (LMP1) and nuclear proteins 2 and 3C are effectors of phenotypic changes in B lymphocytes: EBNA-2 and LMP1 cooperatively induce CD23 | Q36806063 | ||
The level of c-fgr RNA is increased by EBNA-2, an Epstein-Barr virus gene required for B-cell immortalization | Q36806641 | ||
Epstein-Barr virus nuclear antigen 2 transactivates latent membrane protein LMP1. | Q36810617 | ||
An Epstein-Barr virus nuclear protein 2 domain essential for transformation is a direct transcriptional activator | Q36820857 | ||
A promoter of Epstein-Barr virus that can function during latent infection can be transactivated by EBNA-1, a viral protein required for viral DNA replication during latent infection. | Q36828303 | ||
Delineation of the cis-acting element mediating EBNA-2 transactivation of latent infection membrane protein expression | Q36828413 | ||
The Epstein-Barr virus nuclear protein encoded by the leader of the EBNA RNAs is important in B-lymphocyte transformation | Q36828608 | ||
A fifth Epstein-Barr virus nuclear protein (EBNA3C) is expressed in latently infected growth-transformed lymphocytes | Q36866232 | ||
Epstein-Barr virus latent infection membrane protein alters the human B-lymphocyte phenotype: deletion of the amino terminus abolishes activity | Q36881880 | ||
A vector that replicates as a plasmid and can be efficiently selected in B-lymphoblasts transformed by Epstein-Barr virus | Q36885800 | ||
Recombinant Epstein-Barr virus with small RNA (EBER) genes deleted transforms lymphocytes and replicates in vitro | Q37409177 | ||
Recognition sequence of a highly conserved DNA binding protein RBP-J kappa | Q38309890 | ||
Epstein-Barr virus nuclear antigen EBNA3C/6 expression maintains the level of latent membrane protein 1 in G1-arrested cells | Q40040836 | ||
Crucial sequences within the Epstein-Barr virus TP1 promoter for EBNA2-mediated transactivation and interaction of EBNA2 with its responsive element | Q40042560 | ||
Host cell and EBNA-2 regulation of Epstein-Barr virus latent-cycle promoter activity in B lymphocytes. | Q40049217 | ||
Epstein-Barr virus nuclear antigen 2 induces expression of the virus-encoded latent membrane protein. | Q40108807 | ||
Epstein-Barr virus nuclear antigen 2 exerts its transactivating function through interaction with recombination signal binding protein RBP-J kappa, the homologue of Drosophila Suppressor of Hairless. | Q40793575 | ||
Biochemical and immunological characterization of the DNA binding protein (RBP-J kappa) to mouse J kappa recombination signal sequence | Q41099910 | ||
Precipitation of the Epstein-Barr virus protein EBNA 2 by an EBNA 3c-specific monoclonal antibody | Q41479044 | ||
Cell phenotype-dependent control of Epstein-Barr virus latent membrane protein 1 gene regulatory sequences | Q41543823 | ||
Suppressor of Hairless, the Drosophila homolog of the mouse recombination signal-binding protein gene, controls sensory organ cell fates | Q43575150 | ||
Genetic analysis of immortalizing functions of Epstein-Barr virus in human B lymphocytes | Q43679392 | ||
Expression of Epstein-Barr virus transformation-associated genes in tissues of patients with EBV lymphoproliferative disease | Q43936463 | ||
Mediation of Epstein-Barr virus EBNA2 transactivation by recombination signal-binding protein J kappa | Q45781476 | ||
Enhancement of Epstein-Barr virus membrane protein (LMP) expression by serum, TPA, or n-butyrate in latently infected Raji cells | Q45834584 | ||
Epstein-Barr virus latent gene expression during the initiation of B cell immortalization | Q45845786 | ||
Subnuclear localization and phosphorylation of Epstein-Barr virus latent infection nuclear proteins | Q45851423 | ||
Early events in Epstein-Barr virus infection of human B lymphocytes | Q45855126 | ||
The suppressor of hairless protein participates in notch receptor signaling | Q47072502 | ||
The Drosophila homolog of the immunoglobulin recombination signal-binding protein regulates peripheral nervous system development. | Q52444074 | ||
An EBV membrane protein expressed in immortalized lymphocytes transforms established rodent cells. | Q53541156 | ||
Genetic and biochemical evidence that EBNA 2 interaction with a 63-kDa cellular GTG-binding protein is essential for B lymphocyte growth transformation by EBV | Q72756611 | ||
P433 | issue | 5 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | Epstein–Barr virus | Q6900 |
P1104 | number of pages | 9 | |
P304 | page(s) | 3108-3116 | |
P577 | publication date | 1995-05-01 | |
P1433 | published in | Journal of Virology | Q1251128 |
P1476 | title | Epstein-Barr virus nuclear protein 3C modulates transcription through interaction with the sequence-specific DNA-binding protein J kappa | |
P478 | volume | 69 |
Q24682619 | A conserved domain of the Epstein-Barr virus nuclear antigens 3A and 3C binds to a discrete domain of Jkappa |
Q40423684 | Activation of the Notch-regulated transcription factor CBF1/RBP-Jkappa through the 13SE1A oncoprotein |
Q39601084 | Amino acids of Epstein-Barr virus nuclear antigen 3A essential for repression of Jkappa-mediated transcription and their evolutionary conservation |
Q38664787 | An essential EBV latent antigen 3C binds Bcl6 for targeted degradation and cell proliferation. |
Q24675704 | BRCA1 is a component of the RNA polymerase II holoenzyme |
Q39756123 | Biophysical and mutational analysis of the putative bZIP domain of Epstein-Barr virus EBNA 3C. |
Q22008610 | CIR, a corepressor linking the DNA binding factor CBF1 to the histone deacetylase complex |
Q35192797 | Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latency |
Q22010174 | Characterization of DP103, a novel DEAD box protein that binds to the Epstein-Barr virus nuclear proteins EBNA2 and EBNA3C |
Q38341023 | Characterization of the CBF2 binding site within the Epstein-Barr virus latency C promoter and its role in modulating EBNA2-mediated transactivation. |
Q38728190 | Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression |
Q34234140 | Cytokine signaling and Epstein-Barr virus-mediated cell transformation |
Q38337443 | Deregulation of the cell cycle machinery by Epstein-Barr virus nuclear antigen 3C. |
Q39392125 | Downregulation of integrin receptor-signaling genes by Epstein-Barr virus EBNA 3C via promoter-proximal and -distal binding elements |
Q38174306 | Dynamic Epstein-Barr virus gene expression on the path to B-cell transformation |
Q35862488 | EBNA-2 and EBNA-3C extensively and mutually exclusively associate with RBPJkappa in Epstein-Barr virus-transformed B lymphocytes |
Q35889902 | EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells |
Q34552733 | EBNA3C coactivation with EBNA2 requires a SUMO homology domain |
Q61450799 | EBNA3C facilitates RASSF1A downregulation through ubiquitin-mediated degradation and promoter hypermethylation to drive B-cell proliferation |
Q36109083 | EBV Nuclear Antigen 3C Mediates Regulation of E2F6 to Inhibit E2F1 Transcription and Promote Cell Proliferation. |
Q36535405 | EBV-encoded EBNA-6 binds and targets MRS18-2 to the nucleus, resulting in the disruption of pRb-E2F1 complexes |
Q60920702 | Epstein-Barr Virus (EBV)-Related Lymphoproliferative Disorders in Ataxia Telangiectasia: Does ATM Regulate EBV Life Cycle? |
Q59357136 | Epstein-Barr Virus Nuclear Antigen 3C Facilitates Cell Proliferation by Regulating Cyclin D2 |
Q35685797 | Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. |
Q36525145 | Epstein-Barr Virus nuclear protein EBNA3A is critical for maintaining lymphoblastoid cell line growth |
Q28074586 | Epstein-Barr Virus: Diseases Linked to Infection and Transformation |
Q35539925 | Epstein-Barr nuclear antigen leader protein coactivates transcription through interaction with histone deacetylase 4. |
Q39306073 | Epstein-Barr virus EBNA-3C is targeted to and regulates expression from the bidirectional LMP-1/2B promoter. |
Q38353695 | Epstein-Barr virus EBNA3A and EBNA3C proteins both repress RBP-J kappa-EBNA2-activated transcription by inhibiting the binding of RBP-J kappa to DNA. |
Q39882226 | Epstein-Barr virus EBNA3C represses Cp, the major promoter for EBNA expression, but has no effect on the promoter of the cell gene CD21. |
Q24675055 | Epstein-Barr virus and virus human protein interaction maps |
Q39686312 | Epstein-Barr virus encoded EBNA-3 binds to vitamin D receptor and blocks activation of its target genes. |
Q24678677 | Epstein-Barr virus immortalization: Notch2 interacts with CBF1 and blocks differentiation |
Q35008870 | Epstein-Barr virus in the pathogenesis of NPC. |
Q37254393 | Epstein-Barr virus induces global changes in cellular mRNA isoform usage that are important for the maintenance of latency |
Q34328170 | Epstein-Barr virus infection and human malignancies |
Q28082701 | Epstein-Barr virus latent genes |
Q35891377 | Epstein-Barr virus leader protein enhances EBNA-2-mediated transactivation of latent membrane protein 1 expression: a role for the W1W2 repeat domain |
Q28594944 | Epstein-Barr virus nuclear antigen 1 does not cause lymphoma in C57BL/6J mice |
Q41063703 | Epstein-Barr virus nuclear antigen 2 is a transcriptional suppressor of the immunoglobulin mu gene: implications for the expression of the translocated c-myc gene in Burkitt's lymphoma cells. |
Q34985686 | Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes |
Q24537547 | Epstein-Barr virus nuclear antigen 3C and prothymosin alpha interact with the p300 transcriptional coactivator at the CH1 and CH3/HAT domains and cooperate in regulation of transcription and histone acetylation. |
Q39879529 | Epstein-Barr virus nuclear antigen 3C augments Mdm2-mediated p53 ubiquitination and degradation by deubiquitinating Mdm2. |
Q37474974 | Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. |
Q39594681 | Epstein-Barr virus nuclear antigen 3C interacts with histone deacetylase to repress transcription |
Q39874105 | Epstein-Barr virus nuclear antigen 3C is a powerful repressor of transcription when tethered to DNA. |
Q24540313 | Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2. |
Q34782258 | Epstein-Barr virus nuclear antigen 3C recruits histone deacetylase activity and associates with the corepressors mSin3A and NCoR in human B-cell lines |
Q34472101 | Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines |
Q40594951 | Epstein-Barr virus nuclear antigen 3C regulates cyclin A/p27 complexes and enhances cyclin A-dependent kinase activity |
Q34102722 | Epstein-Barr virus nuclear antigen 3C stabilizes Gemin3 to block p53-mediated apoptosis |
Q34872837 | Epstein-Barr virus nuclear antigen 3C targets p53 and modulates its transcriptional and apoptotic activities |
Q34549944 | Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression |
Q24681418 | Epstein-Barr virus nuclear protein 2 (EBNA2) binds to a component of the human SNF-SWI complex, hSNF5/Ini1 |
Q33911878 | Epstein-Barr virus nuclear protein 3A domains essential for growth of lymphoblasts: transcriptional regulation through RBP-Jkappa/CBF1 is critical |
Q35014692 | Epstein-Barr virus nuclear protein 3C binds to the N-terminal (NTD) and beta trefoil domains (BTD) of RBP/CSL; only the NTD interaction is essential for lymphoblastoid cell growth |
Q37452130 | Epstein-Barr virus nuclear protein 3C domains necessary for lymphoblastoid cell growth: interaction with RBP-Jkappa regulates TCL1. |
Q38303570 | Epstein-Barr virus nuclear protein EBNA-3C interacts with the human metastatic suppressor Nm23-H1: a molecular link to cancer metastasis |
Q35540042 | Epstein-Barr virus nuclear protein EBNA3C is required for cell cycle progression and growth maintenance of lymphoblastoid cells |
Q37132783 | Epstein-Barr virus nuclear protein EBNA3C residues critical for maintaining lymphoblastoid cell growth |
Q35891360 | Epstein-Barr virus nuclear protein LP stimulates EBNA-2 acidic domain-mediated transcriptional activation |
Q90095448 | Epstein-Barr virus subverts mevalonate and fatty acid pathways to promote infected B-cell proliferation and survival |
Q37248930 | Epstein-Barr virus vectors for gene delivery to B lymphocytes |
Q33787090 | Epstein-barr virus nuclear antigen 3C activates the latent membrane protein 1 promoter in the presence of Epstein-Barr virus nuclear antigen 2 through sequences encompassing an spi-1/Spi-B binding site |
Q34347958 | Epstein-barr virus-induced changes in B-lymphocyte gene expression |
Q40288802 | Expression of alpha V integrin is modulated by Epstein-Barr virus nuclear antigen 3C and the metastasis suppressor Nm23-H1 through interaction with the GATA-1 and Sp1 transcription factors |
Q33749934 | Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification |
Q45729115 | High level of transgene expression in primary chronic lymphocytic leukemia cells using helper-virus-free recombinant Epstein-Barr virus vectors |
Q34273467 | Host cell-dependent expression of latent Epstein-Barr virus genomes: regulation by DNA methylation |
Q34998257 | Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 |
Q33843211 | Induction of Kaposi's sarcoma-associated herpesvirus latency-associated nuclear antigen by the lytic transactivator RTA: a novel mechanism for establishment of latency |
Q39588351 | Intracellular forms of human NOTCH1 functionally activate essential Epstein-Barr virus major latent promoters in the Burkitt's lymphoma BJAB cell line but repress these promoters in Jurkat cells |
Q33640843 | Intron retention may regulate expression of Epstein-Barr virus nuclear antigen 3 family genes |
Q35865472 | Isolation of intertypic recombinants of Epstein-Barr virus from T-cell-immunocompromised individuals |
Q33737793 | Kaposi's sarcoma-associated herpesvirus reactivation is regulated by interaction of latency-associated nuclear antigen with recombination signal sequence-binding protein Jkappa, the major downstream effector of the Notch signaling pathway |
Q37217649 | MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. |
Q64990535 | Manipulation of Epithelial Differentiation by HPV Oncoproteins. |
Q39880772 | Mapping promoter regions that are hypersensitive to methylation-mediated inhibition of transcription: application of the methylation cassette assay to the Epstein-Barr virus major latency promoter. |
Q34998243 | Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming |
Q28117643 | Modulation of histone acetyltransferase activity through interaction of epstein-barr nuclear antigen 3C with prothymosin alpha |
Q34225554 | Molecular virology of Epstein-Barr virus |
Q39577674 | Multiple functions within the Epstein-Barr virus EBNA-3A protein. |
Q22254088 | NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells |
Q35008880 | Nasopharyngeal carcinoma and the EBV-specific T cell response: prospects for immunotherapy |
Q36402940 | Notch1 modulates timing of G1-S progression by inducing SKP2 transcription and p27 Kip1 degradation |
Q33604108 | Promoter-specific targeting of human SWI-SNF complex by Epstein-Barr virus nuclear protein 2. |
Q40397055 | RBP-J kappa repression activity is mediated by a co-repressor and antagonized by the Epstein-Barr virus transcription factor EBNA2. |
Q39877574 | Regulation of EBNA gene transcription in lymphoblastoid cell lines: characterization of sequences downstream of BCR2 (Cp). |
Q40398026 | Regulation of matrix metalloproteinase 9 expression by Epstein-Barr virus nuclear antigen 3C and the suppressor of metastasis Nm23-H1. |
Q33786088 | Residues 231 to 280 of the Epstein-Barr virus nuclear protein 2 are not essential for primary B-lymphocyte growth transformation. |
Q39591670 | Structural, functional, and genetic comparisons of Epstein-Barr virus nuclear antigen 3A, 3B, and 3C homologues encoded by the rhesus lymphocryptovirus |
Q54251658 | The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency. |
Q28546003 | The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth |
Q34711793 | The EBV Latent Antigen 3C Inhibits Apoptosis through Targeted Regulation of Interferon Regulatory Factors 4 and 8. |
Q42702063 | The Epstein-Barr virus EBNA-LP protein preferentially coactivates EBNA2-mediated stimulation of latent membrane proteins expressed from the viral divergent promoter |
Q34275895 | The Epstein-Barr virus and post-transplant lymphoproliferative disease: interplay of immunosuppression, EBV, and the immune system in disease pathogenesis |
Q40588233 | The Epstein-Barr virus nuclear antigen-6 protein co-localizes with EBNA-3 and survival of motor neurons protein |
Q24310605 | The amino-terminal domains of Epstein-Barr virus nuclear proteins 3A, 3B, and 3C interact with RBPJ(kappa) |
Q34362138 | The metastatic suppressor Nm23-H1 interacts with EBNA3C at sequences located between the glutamine- and proline-rich domains and can cooperate in activation of transcription |
Q39748937 | Transcriptional Regulatory Properties of Epstein-Barr Virus Nuclear Antigen 3C Are Conserved in Simian Lymphocryptoviruses |
Q34039189 | Viral latency and its regulation: lessons from the gamma-herpesviruses |
Q24653900 | Viruses associated with human cancer |
Q35867579 | oriP is essential for EBNA gene promoter activity in Epstein-Barr virus-immortalized lymphoblastoid cell lines. |