scholarly article | Q13442814 |
P50 | author | Kostas Paschos | Q59384393 |
Quentin Bazot | Q59691365 | ||
Adam C T Gillman | Q87048798 | ||
Martin J Allday | Q96115879 | ||
P2093 | author name string | Bettina Kempkes | |
Jens S Kalchschmidt | |||
P2860 | cites work | Mastermind is a putative activator for Notch. | Q52580782 |
Histone H2A monoubiquitination promotes histone H3 methylation in Polycomb repression. | Q52771957 | ||
Control of cell fate in C. elegans by a GLP-1 peptide consisting primarily of ankyrin repeats | Q60065785 | ||
Genomewide analysis of PRC1 and PRC2 occupancy identifies two classes of bivalent domains | Q21092474 | ||
CIR, a corepressor linking the DNA binding factor CBF1 to the histone deacetylase complex | Q22008610 | ||
MAML1, a human homologue of Drosophila mastermind, is a transcriptional co-activator for NOTCH receptors | Q24290574 | ||
RBP-Jkappa/SHARP recruits CtIP/CtBP corepressors to silence Notch target genes | Q24294890 | ||
The X-linked mental retardation gene SMCX/JARID1C defines a family of histone H3 lysine 4 demethylases | Q24298299 | ||
The amino-terminal domains of Epstein-Barr virus nuclear proteins 3A, 3B, and 3C interact with RBPJ(kappa) | Q24310605 | ||
SHARP is a novel component of the Notch/RBP-Jkappa signalling pathway | Q24534941 | ||
A histone deacetylase corepressor complex regulates the Notch signal transduction pathway | Q24598533 | ||
RYBP-PRC1 complexes mediate H2A ubiquitylation at polycomb target sites independently of PRC2 and H3K27me3 | Q24631517 | ||
A conserved domain of the Epstein-Barr virus nuclear antigens 3A and 3C binds to a discrete domain of Jkappa | Q24682619 | ||
Histone acetyltransferases and histone deacetylases in B- and T-cell development, physiology and malignancy | Q28082536 | ||
Notch signaling: from the outside in | Q28139768 | ||
Control of developmental regulators by Polycomb in human embryonic stem cells | Q28235841 | ||
Physical interaction between a novel domain of the receptor Notch and the transcription factor RBP-J kappa/Su(H) | Q28286560 | ||
RBP2 belongs to a family of demethylases, specific for tri-and dimethylated lysine 4 on histone 3 | Q28289886 | ||
Two nonconsensus sites in the Epstein-Barr virus oncoprotein EBNA3A cooperate to bind the co-repressor carboxyl-terminal-binding protein (CtBP) | Q28579873 | ||
Notch signalling: a simple pathway becomes complex | Q29555851 | ||
Epstein-Barr virus: 40 years on | Q29617969 | ||
NOTCH1 nuclear interactome reveals key regulators of its transcriptional activity and oncogenic function. | Q30668571 | ||
Induction of p16(INK4a) is the major barrier to proliferation when Epstein-Barr virus (EBV) transforms primary B cells into lymphoblastoid cell lines | Q31111648 | ||
Physical and functional interactions between the corepressor CtBP and the Epstein-Barr virus nuclear antigen EBNA3C | Q33292390 | ||
PcG proteins, DNA methylation, and gene repression by chromatin looping. | Q33389249 | ||
Epigenetic repression of p16(INK4A) by latent Epstein-Barr virus requires the interaction of EBNA3A and EBNA3C with CtBP. | Q33604848 | ||
Extensive co-operation between the Epstein-Barr virus EBNA3 proteins in the manipulation of host gene expression and epigenetic chromatin modification | Q33749934 | ||
GC-rich sequence elements recruit PRC2 in mammalian ES cells | Q33775517 | ||
Targeting polycomb to pericentric heterochromatin in embryonic stem cells reveals a role for H2AK119u1 in PRC2 recruitment. | Q33775664 | ||
A somatic knockout of CBF1 in a human B-cell line reveals that induction of CD21 and CCR7 by EBNA-2 is strictly CBF1 dependent and that downregulation of immunoglobulin M is partially CBF1 independent | Q33884014 | ||
LAG-3 is a putative transcriptional activator in the C. elegans Notch pathway | Q33903634 | ||
Direct recruitment of polycomb repressive complex 1 to chromatin by core binding transcription factors. | Q34030648 | ||
PROMO: detection of known transcription regulatory elements using species-tailored searches | Q34114282 | ||
Notch and Presenilin: regulated intramembrane proteolysis links development and degeneration. | Q34194714 | ||
The human J kappa recombination signal sequence binding protein (RBP-J kappa) targets the Epstein-Barr virus EBNA2 protein to its DNA responsive elements. | Q34327455 | ||
EBNA2 and Notch signalling in Epstein-Barr virus mediated immortalization of B lymphocytes | Q34415971 | ||
Epstein-Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines | Q34472101 | ||
The histone H3K4 demethylase SMCX links REST target genes to X-linked mental retardation. | Q34624060 | ||
RBP2 is an MRG15 complex component and down-regulates intragenic histone H3 lysine 4 methylation. | Q34638245 | ||
Recruitment of PRC1 function at the initiation of X inactivation independent of PRC2 and silencing | Q34767759 | ||
Epstein-Barr virus nuclear antigen 3C recruits histone deacetylase activity and associates with the corepressors mSin3A and NCoR in human B-cell lines | Q34782258 | ||
Gamma-secretase-mediated proteolysis in cell-surface-receptor signalling | Q34810184 | ||
EBV nuclear antigen EBNALP dismisses transcription repressors NCoR and RBPJ from enhancers and EBNA2 increases NCoR-deficient RBPJ DNA binding | Q34977875 | ||
Epstein-Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes | Q34985686 | ||
Modulation of enhancer looping and differential gene targeting by Epstein-Barr virus transcription factors directs cellular reprogramming | Q34998243 | ||
Inactivation of intergenic enhancers by EBNA3A initiates and maintains polycomb signatures across a chromatin domain encoding CXCL10 and CXCL9 | Q34998257 | ||
Epstein-Barr virus nuclear protein 3C binds to the N-terminal (NTD) and beta trefoil domains (BTD) of RBP/CSL; only the NTD interaction is essential for lymphoblastoid cell growth | Q35014692 | ||
RBPJ, the major transcriptional effector of Notch signaling, remains associated with chromatin throughout mitosis, suggesting a role in mitotic bookmarking | Q35112753 | ||
The mammalian transcriptional repressor RBP (CBF1) targets TFIID and TFIIA to prevent activated transcription | Q35201509 | ||
Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth | Q35202714 | ||
The Epstein-Barr virus nuclear antigen 2 transactivator is directed to response elements by the J kappa recombination signal binding protein | Q35648541 | ||
Epstein-Barr Virus Proteins EBNA3A and EBNA3C Together Induce Expression of the Oncogenic MicroRNA Cluster miR-221/miR-222 and Ablate Expression of Its Target p57KIP2. | Q35685797 | ||
Hyperconserved CpG domains underlie Polycomb-binding sites | Q35720547 | ||
Epstein-Barr virus nuclear protein 3C modulates transcription through interaction with the sequence-specific DNA-binding protein J kappa | Q35839108 | ||
Viral interactions with the Notch pathway | Q35853119 | ||
The polycomb group protein Suz12 is required for embryonic stem cell differentiation | Q35856936 | ||
EBNA-2 and EBNA-3C extensively and mutually exclusively associate with RBPJkappa in Epstein-Barr virus-transformed B lymphocytes | Q35862488 | ||
Carboxyl-terminal amino acids 1052 to 1082 of the latency-associated nuclear antigen (LANA) interact with RBP-Jκ and are responsible for LANA-mediated RTA repression. | Q35943718 | ||
Propagation and recovery of intact, infectious Epstein-Barr virus from prokaryotic to human cells | Q36173276 | ||
BIM promoter directly targeted by EBNA3C in polycomb-mediated repression by EBV | Q36180229 | ||
Notch and Wnt signaling: mimicry and manipulation by gamma herpesviruses | Q36480684 | ||
A novel 6C assay uncovers Polycomb-mediated higher order chromatin conformations | Q36804768 | ||
Notch signaling at a glance | Q36903753 | ||
Spectrum of Epstein-Barr virus-associated diseases | Q37014791 | ||
Epstein-Barr virus nuclear protein EBNA3C residues critical for maintaining lymphoblastoid cell growth | Q37132783 | ||
Differential gene expression patterns of EBV infected EBNA-3A positive and negative human B lymphocytes | Q37237334 | ||
Epstein-Barr virus nuclear protein 3C domains necessary for lymphoblastoid cell growth: interaction with RBP-Jkappa regulates TCL1. | Q37452130 | ||
Epstein-Barr virus nuclear antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A. | Q37474974 | ||
Physiological roles of class I HDAC complex and histone demethylase. | Q37806964 | ||
Molecular mechanisms and potential functions of histone demethylases | Q37999690 | ||
Polycomb silencing: from linear chromatin domains to 3D chromosome folding | Q38179206 | ||
The retinoblastoma binding protein RBP2 is an H3K4 demethylase | Q38304373 | ||
Epstein-Barr virus EBNA3A and EBNA3C proteins both repress RBP-J kappa-EBNA2-activated transcription by inhibiting the binding of RBP-J kappa to DNA. | Q38353695 | ||
Epstein-Barr virus (EBV) nuclear antigen 6 induces expression of the EBV latent membrane protein and an activated phenotype in Raji cells | Q38869720 | ||
Dynamic binding of RBPJ is determined by Notch signaling status. | Q39155079 | ||
Downregulation of integrin receptor-signaling genes by Epstein-Barr virus EBNA 3C via promoter-proximal and -distal binding elements | Q39392125 | ||
Epstein-Barr virus nuclear antigen 3C interacts with histone deacetylase to repress transcription | Q39594681 | ||
LIM protein KyoT2 negatively regulates transcription by association with the RBP-J DNA-binding protein | Q39631445 | ||
Roles of the ankyrin repeats and C-terminal region of the mouse notch1 intracellular region | Q39725752 | ||
Long-term stability of large insert genomic DNA episomal shuttle vectors in human cells | Q39727680 | ||
Cellular factors associated with latency and spontaneous Epstein-Barr virus reactivation in B-lymphoblastoid cell lines. | Q39740136 | ||
Identification of patterns in biological sequences at the ALGGEN server: PROMO and MALGEN. | Q39804548 | ||
Epstein-Barr virus nuclear antigen 3C is a transcriptional regulator | Q39870334 | ||
Epstein-Barr virus nuclear antigen 3C is a powerful repressor of transcription when tethered to DNA. | Q39874105 | ||
Epstein-Barr virus EBNA3C represses Cp, the major promoter for EBNA expression, but has no effect on the promoter of the cell gene CD21. | Q39882226 | ||
Establishment in continuous culture of a new type of lymphocyte from a “burkitt-like” malignant lymphoma (line d.g.-75) | Q40039870 | ||
Polycomb response elements mediate the formation of chromosome higher-order structures in the bithorax complex. | Q40082937 | ||
Physical and functional association of a trimethyl H3K4 demethylase and Ring6a/MBLR, a polycomb-like protein | Q40167322 | ||
Variant PRC1 complex-dependent H2A ubiquitylation drives PRC2 recruitment and polycomb domain formation. | Q40406465 | ||
Generation and precise modification of a herpesvirus saimiri bacterial artificial chromosome demonstrates that the terminal repeats are required for both virus production and episomal persistence | Q40611661 | ||
Epstein-Barr virus nuclear antigen 2 exerts its transactivating function through interaction with recombination signal binding protein RBP-J kappa, the homologue of Drosophila Suppressor of Hairless. | Q40793575 | ||
Involvement of RBP-J in biological functions of mouse Notch1 and its derivatives. | Q41078359 | ||
Epstein-Barr nuclear antigen-3 and -4 interact with RBP-2N, a major isoform of RBP-J kappa in B lymphocytes | Q41145087 | ||
The Epstein-Barr virus determined nuclear antigens EBNA-3A, -3B, and -3C repress EBNA-2-mediated transactivation of the viral terminal protein 1 gene promoter | Q41415414 | ||
Notch activation stimulates transient and selective binding of Su(H)/CSL to target enhancers | Q41882770 | ||
Two Epstein-Barr virus (EBV) oncoproteins cooperate to repress expression of the proapoptotic tumour-suppressor Bim: clues to the pathogenesis of Burkitt's lymphoma. | Q45401541 | ||
Epstein-Barr virus EBNA3 proteins bind to the C8/alpha7 subunit of the 20S proteasome and are degraded by 20S proteasomes in vitro, but are very stable in latently infected B cells | Q45482113 | ||
Transcriptional repression by the Epstein-Barr virus EBNA3A protein tethered to DNA does not require RBP-Jkappa. | Q45757839 | ||
Mediation of Epstein-Barr virus EBNA2 transactivation by recombination signal-binding protein J kappa | Q45781476 | ||
P275 | copyright license | Creative Commons Attribution 4.0 International | Q20007257 |
P6216 | copyright status | copyrighted | Q50423863 |
P433 | issue | 1 | |
P304 | page(s) | e1005383 | |
P577 | publication date | 2016-01-11 | |
P1433 | published in | PLOS Pathogens | Q283209 |
P1476 | title | EBNA3C Directs Recruitment of RBPJ (CBF1) to Chromatin during the Process of Gene Repression in EBV Infected B Cells | |
P478 | volume | 12 |
Q57025486 | An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model |
Q38728190 | Core binding factor (CBF) is required for Epstein-Barr virus EBNA3 proteins to regulate target gene expression |
Q41926654 | EBF1 binds to EBNA2 and promotes the assembly of EBNA2 chromatin complexes in B cells. |
Q61450799 | EBNA3C facilitates RASSF1A downregulation through ubiquitin-mediated degradation and promoter hypermethylation to drive B-cell proliferation |
Q38644507 | EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency |
Q47549184 | Epstein-Barr Virus (EBV) Latent Protein EBNA3A Directly Targets and Silences the STK39 Gene in B Cells Infected by EBV. |
Q58740108 | Epstein-Barr Virus Nuclear Antigen 3C Inhibits Expression of and the Locus via Interaction with the Histone Lysine Demethylase KDM2B |
Q38768957 | Epstein-Barr virus nuclear protein EBNA3C directly induces expression of AID and somatic mutations in B cells |
Q37217649 | MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. |
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Q54251658 | The Cooperative Functions of the EBNA3 Proteins Are Central to EBV Persistence and Latency. |
Q41917994 | The Epstein-Barr Virus Regulome in Lymphoblastoid Cells. |
Q54978478 | Transcriptional and epigenetic modulation of autophagy promotes EBV oncoprotein EBNA3C induced B-cell survival. |
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