Cell-autonomous axon growth of young motoneurons is triggered by a voltage-gated sodium channel

scientific article published on December 13, 2012

Cell-autonomous axon growth of young motoneurons is triggered by a voltage-gated sodium channel is …
instance of (P31):
scholarly articleQ13442814

External links are
P356DOI10.4161/CHAN.23153
P953full work available online athttp://www.tandfonline.com/doi/pdf/10.4161/chan.23153
https://europepmc.org/articles/PMC3589283
https://europepmc.org/articles/PMC3589283?pdf=render
https://www.tandfonline.com/doi/pdf/10.4161/chan.23153?needAccess=true
P932PMC publication ID3589283
P698PubMed publication ID23238424
P5875ResearchGate publication ID233914073

P50authorRobert BlumQ42172829
Sibylle JablonkaQ90711517
P2093author name stringAndrea Wetzel
P2860cites workIdentification of a novel human voltage-gated sodium channel alpha subunit gene, SCN12AQ22011020
Neurotrophin-evoked depolarization requires the sodium channel Na(V)1.9Q28208066
Specific interaction of Smn, the spinal muscular atrophy determining gene product, with hnRNP-R and gry-rbp/hnRNP-Q: a role for Smn in RNA processing in motor axons?Q28214361
International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channelsQ28289125
International Union of Pharmacology. XLVIII. Nomenclature and structure-function relationships of voltage-gated calcium channelsQ28289138
A novel persistent tetrodotoxin-resistant sodium current in SNS-null and wild-type small primary sensory neuronsQ28377530
Smn, the spinal muscular atrophy-determining gene product, modulates axon growth and localization of beta-actin mRNA in growth cones of motoneuronsQ28576647
Contribution of the tetrodotoxin-resistant voltage-gated sodium channel NaV1.9 to sensory transmission and nociceptive behaviorQ33878908
Activation of a TRPC3-dependent cation current through the neurotrophin BDNF.Q33890263
Activation of Trk neurotrophin receptors in the absence of neurotrophinsQ33937877
Role of Na(v)1.9 in activity-dependent axon growth in motoneuronsQ34284951
Neurotrophin-mediated rapid signaling in the central nervous system: mechanisms and functionsQ36011413
Adenosine receptor A2A-R contributes to motoneuron survival by transactivating the tyrosine kinase receptor TrkBQ36089203
NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomyQ36229128
Spinal muscular atrophy: a deficiency in a ubiquitous protein; a motor neuron-specific diseaseQ36347017
Electrical activity in early neuronal developmentQ36676053
Embryonic motor axon development in the severe SMA mouseQ37294791
Defective Ca2+ channel clustering in axon terminals disturbs excitability in motoneurons in spinal muscular atrophyQ39751507
Endogenous brain-derived neurotrophic factor triggers fast calcium transients at synapses in developing dendrites.Q40176546
Efficient expression of rat brain type IIA Na+ channel α subunits in a somatic cell lineQ41651477
Mechanosensitivity of Nav1.5, a voltage-sensitive sodium channelQ42680525
Postsynaptic Induction of BDNF-Mediated Long-Term PotentiationQ43901725
Pre-symptomatic development of lower motor neuron connectivity in a mouse model of severe spinal muscular atrophyQ43932877
Characterization of the circuits that generate spontaneous episodes of activity in the early embryonic mouse spinal cord.Q44281907
GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neuronesQ44372230
Brain-derived neurotrophic factor prevents the death of motoneurons in newborn rats after nerve sectionQ44602903
Distinct aspects of neuronal differentiation encoded by frequency of spontaneous Ca2+ transientsQ46567447
GTP up-regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9.Q46838525
Altered expression of ion channel isoforms at the node of Ranvier in P0-deficient myelin mutantsQ51037640
Selective expression of a persistent tetrodotoxin-resistant Na+ current and NaV1.9 subunit in myenteric sensory neurons.Q51300821
Persistent TTX-resistant Na+ current affects resting potential and response to depolarization in simulated spinal sensory neuronsQ51348354
The development of hindlimb motor activity studied in the isolated spinal cord of the chick embryoQ52255000
Laminin induced local axonal translation of β-actin mRNA is impaired in SMN-deficient motoneuronsQ53151357
P433issue1
P407language of work or nameEnglishQ1860
P921main subjectvoltage-gated sodium channelQ424960
P304page(s)51-56
P577publication date2012-12-13
P1433published inChannelsQ15764469
P1476titleCell-autonomous axon growth of young motoneurons is triggered by a voltage-gated sodium channel
P478volume7

Reverse relations

cites work (P2860)
Q28298515A de novo gain-of-function mutation in SCN11A causes loss of pain perception
Q47797338Age-dependent expression of Nav1.9 channels in medial prefrontal cortex pyramidal neurons in rats.
Q55316384An open source tool for automatic spatiotemporal assessment of calcium transients and local 'signal-close-to-noise' activity in calcium imaging data.
Q47150601BDNF/trkB Induction of Calcium Transients through Cav2.2 Calcium Channels in Motoneurons Corresponds to F-actin Assembly and Growth Cone Formation on β2-Chain Laminin (221).
Q37590635Back to the tubule: microtubule dynamics in Parkinson's disease
Q100454969Caveolin 1 is required for axonal outgrowth of motor neurons and affects Xenopus neuromuscular development
Q42013925Differentiation defects in primary motoneurons from a SMARD1 mouse model that are insensitive to treatment with low dose PEGylated IGF1
Q38881907Heterologous expression of NaV1.9 chimeras in various cell systems
Q36583137Impaired Axonal Na(+) Current by Hindlimb Unloading: Implication for Disuse Neuromuscular Atrophy
Q49577397NaV1.9 Potentiates Oxidized Phospholipid-Induced TRP Responses Only under Inflammatory Conditions.

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