| scholarly article | Q13442814 |
| P2093 | author name string | Scot R Kimball | |
| Charles H Lang | |||
| Jennifer L Steiner | |||
| Kristen T Crowell | |||
| P2860 | cites work | Mechanism of activation of protein kinase B by insulin and IGF-1 | Q24309528 |
| REDD1, a developmentally regulated transcriptional target of p63 and p53, links p63 to regulation of reactive oxygen species | Q24321795 | ||
| Regulation of mTOR function in response to hypoxia by REDD1 and the TSC1/TSC2 tumor suppressor complex | Q24559347 | ||
| Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method | Q25938999 | ||
| Misregulation of autophagy and protein degradation systems in myopathies and muscular dystrophies | Q27027197 | ||
| TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling | Q28131740 | ||
| Amino acid-induced stimulation of translation initiation in rat skeletal muscle | Q28140523 | ||
| Akt/mTOR pathway is a crucial regulator of skeletal muscle hypertrophy and can prevent muscle atrophy in vivo | Q28206290 | ||
| AMP-activated protein kinase suppresses protein synthesis in rat skeletal muscle through down-regulated mammalian target of rapamycin (mTOR) signaling | Q28216932 | ||
| Stimulation of the AMP-activated protein kinase leads to activation of eukaryotic elongation factor 2 kinase and to its phosphorylation at a novel site, serine 398 | Q28237559 | ||
| REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy. | Q45884215 | ||
| IL-6-induced skeletal muscle atrophy | Q46172460 | ||
| Endotoxin stimulates in vivo expression of inflammatory cytokines tumor necrosis factor alpha, interleukin-1beta, -6, and high-mobility-group protein-1 in skeletal muscle | Q47863220 | ||
| Autophagy is required to maintain muscle mass | Q51411361 | ||
| Myostatin induces cachexia by activating the ubiquitin proteolytic system through an NF-kappaB-independent, FoxO1-dependent mechanism | Q51805331 | ||
| Glycogen synthase kinase-3 is the predominant insulin-regulated eukaryotic initiation factor 2B kinase in skeletal muscle | Q52569751 | ||
| Critical illness polyneuropathy: risk factors and clinical consequences. A cohort study in septic patients | Q59235137 | ||
| Sepsis upregulates the gene expression of multiple ubiquitin ligases in skeletal muscle | Q73208228 | ||
| TNF-binding protein ameliorates inhibition of skeletal muscle protein synthesis during sepsis | Q77306855 | ||
| Implication of eIF2B rather than eIF4E in the regulation of global protein synthesis by amino acids in L6 myoblasts | Q77545079 | ||
| beta-Adrenergic blockade during systemic inflammation: impact on cellular immune functions and survival in a murine model of sepsis | Q79374040 | ||
| Animal research. Harmonization of animal care and use guidance | Q83289343 | ||
| Protein degradation by the ubiquitin-proteasome pathway in normal and disease states | Q28243121 | ||
| Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1 | Q28276788 | ||
| Impairment of starvation-induced and constitutive autophagy in Atg7-deficient mice | Q28588080 | ||
| The autophagy initiating kinase ULK1 is regulated via opposing phosphorylation by AMPK and mTOR | Q28610067 | ||
| FoxO3 coordinately activates protein degradation by the autophagic/lysosomal and proteasomal pathways in atrophying muscle cells | Q29614482 | ||
| Targeting Cullin-RING ligases by MLN4924 induces autophagy via modulating the HIF1-REDD1-TSC1-mTORC1-DEPTOR axis | Q30525356 | ||
| Ribosomal S6 kinase signaling and the control of translation | Q33783527 | ||
| Altered nutrient response of mTORC1 as a result of changes in REDD1 expression: effect of obesity vs. REDD1 deficiency | Q34006278 | ||
| REDD1 enhances protein phosphatase 2A-mediated dephosphorylation of Akt to repress mTORC1 signaling. | Q34092837 | ||
| Autophagy and Skeletal Muscles in Sepsis | Q34442945 | ||
| Sepsis-induced suppression of skeletal muscle translation initiation mediated by tumor necrosis factor alpha | Q34589839 | ||
| Novel insights into the regulation of skeletal muscle protein synthesis as revealed by a new nonradioactive in vivo technique | Q34592377 | ||
| Regulation of muscle protein synthesis during sepsis and inflammation | Q34628735 | ||
| Elevated corticosterone associated with food deprivation upregulates expression in rat skeletal muscle of the mTORC1 repressor, REDD1. | Q34964972 | ||
| Sepsis attenuates the anabolic response to skeletal muscle contraction | Q35177499 | ||
| Deep proteomics of mouse skeletal muscle enables quantitation of protein isoforms, metabolic pathways, and transcription factors | Q35325626 | ||
| Mnk2 and Mnk1 are essential for constitutive and inducible phosphorylation of eukaryotic initiation factor 4E but not for cell growth or development | Q35663786 | ||
| Sepsis stimulates nonlysosomal, energy-dependent proteolysis and increases ubiquitin mRNA levels in rat skeletal muscle | Q35824460 | ||
| Alteration of somatotropic function by proinflammatory cytokines | Q35912023 | ||
| Roles of mitogen-activated protein kinase signal-integrating kinases 1 and 2 in oxidant-mediated eIF4E phosphorylation | Q36026674 | ||
| Loss of muscle strength during sepsis is in part regulated by glucocorticoids and is associated with reduced muscle fiber stiffness | Q36454351 | ||
| mTor Signaling in Skeletal Muscle During Sepsis and Inflammation: Where Does It All Go Wrong? | Q36711877 | ||
| Activation of AMP-activated protein kinase by 5-aminoimidazole-4-carboxamide-1-beta-D-ribonucleoside prevents leucine-stimulated protein synthesis in rat skeletal muscle | Q37000863 | ||
| REDD2 is enriched in skeletal muscle and inhibits mTOR signaling in response to leucine and stretch | Q37139068 | ||
| Local insulin-like growth factor I prevents sepsis-induced muscle atrophy | Q37381871 | ||
| Myostatin and the skeletal muscle atrophy and hypertrophy signaling pathways | Q38235659 | ||
| Regulated in DNA damage and development 1 (REDD1) promotes cell survival during serum deprivation by sustaining repression of signaling through the mechanistic target of rapamycin in complex 1 (mTORC1). | Q39098298 | ||
| Acute alcohol intoxication increases REDD1 in skeletal muscle | Q40003242 | ||
| A REDD1/TXNIP pro-oxidant complex regulates ATG4B activity to control stress-induced autophagy and sustain exercise capacity. | Q40343998 | ||
| Regulation of eIF-4E BP1 phosphorylation by mTOR. | Q41087062 | ||
| Lack of REDD1 reduces whole body glucose and insulin tolerance, and impairs skeletal muscle insulin signaling | Q41760472 | ||
| Nutrient-induced stimulation of protein synthesis in mouse skeletal muscle is limited by the mTORC1 repressor REDD1. | Q41862322 | ||
| Reduced REDD1 expression contributes to activation of mTORC1 following electrically induced muscle contraction | Q42948178 | ||
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P433 | issue | 12 | |
| P921 | main subject | proteolysis | Q33123 |
| sepsis | Q183134 | ||
| P304 | page(s) | E981-94 | |
| P577 | publication date | 2015-10-20 | |
| P1433 | published in | American Journal of Physiology - Endocrinology and Metabolism | Q15765671 |
| P1476 | title | Disruption of REDD1 gene ameliorates sepsis-induced decrease in mTORC1 signaling but has divergent effects on proteolytic signaling in skeletal muscle | |
| P478 | volume | 309 |
| Q90295543 | Altered protein turnover signaling and myogenesis during impaired recovery of inflammation-induced muscle atrophy in emphysematous mice |
| Q41140239 | Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylation |
| Q42289835 | Differential regulation of muscle protein turnover in response to emphysema and acute pulmonary inflammation |
| Q37139408 | Emerging role for regulated in development and DNA damage 1 (REDD1) in the regulation of skeletal muscle metabolism |
| Q55331913 | Glucocorticoid-dependent REDD1 expression reduces muscle metabolism to enable adaptation under energetic stress. |
| Q58776998 | Impact of Very Early Physical Therapy During Septic Shock on Skeletal Muscle: A Randomized Controlled Trial |
| Q39013918 | Inability to replete white adipose tissue during recovery phase of sepsis is associated with increased autophagy, apoptosis, and proteasome activity |
| Q28075486 | Is REDD1 a Metabolic Éminence Grise? |
| Q37419009 | Marginal dietary zinc deprivation augments sepsis-induced alterations in skeletal muscle TNF-α but not protein synthesis |
| Q55033522 | Pulmonary inflammation-induced loss and subsequent recovery of skeletal muscle mass require functional poly-ubiquitin conjugation. |
| Q51384750 | Restorative Mechanisms Regulating Protein Balance in Skeletal Muscle During Recovery From Sepsis. |
| Q90732384 | Skeletal Muscle Atrophy Was Alleviated by Salidroside Through Suppressing Oxidative Stress and Inflammation During Denervation |
| Q55036220 | Systemic bioinformatics analysis of skeletal muscle gene expression profiles of sepsis. |
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