| scholarly article | Q13442814 |
| P50 | author | Stanley J. Korsmeyer | Q138795 |
| P2093 | author name string | Hyungjin Kim | |
| Emily H-Y Cheng | |||
| Ho-Chou Tu | |||
| James J-D Hsieh | |||
| Todd D Westergard | |||
| Decheng Ren | |||
| Jill K Fisher | |||
| Jeff A Rubens | |||
| P2860 | cites work | A critical role for Dnmt1 and DNA methylation in T cell development, function, and survival. | Q52126962 |
| Deficiency in Bak and Bax perturbs thymic selection and lymphoid homeostasis. | Q52547440 | ||
| In vivo calcium elevations in thymocytes with T cell receptors that are specific for self ligands | Q68188967 | ||
| Hierarchical regulation of mitochondrion-dependent apoptosis by BCL-2 subfamilies | Q79370601 | ||
| tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c | Q24290118 | ||
| Proapoptotic Bak is sequestered by Mcl-1 and Bcl-xL, but not Bcl-2, until displaced by BH3-only proteins | Q24302494 | ||
| Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members | Q24337108 | ||
| Structure of Bax: coregulation of dimer formation and intracellular localization | Q27628839 | ||
| BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis | Q28189945 | ||
| BH3-only Bcl-2 family member Bim is required for apoptosis of autoreactive thymocytes | Q28203083 | ||
| Apoptosis in the pathogenesis and treatment of disease | Q28235731 | ||
| Cell death: critical control points | Q28240722 | ||
| Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death | Q28297152 | ||
| Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death | Q28363890 | ||
| The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues | Q28512628 | ||
| Essential role of BAX,BAK in B cell homeostasis and prevention of autoimmune disease | Q28513752 | ||
| Bax-deficient mice with lymphoid hyperplasia and male germ cell death | Q28589360 | ||
| VDAC2 inhibits BAK activation and mitochondrial apoptosis | Q28594000 | ||
| Mitochondrial membrane permeabilization in cell death | Q29547425 | ||
| BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly | Q29617135 | ||
| Distinct BH3 domains either sensitize or activate mitochondrial apoptosis, serving as prototype cancer therapeutics | Q29620467 | ||
| Immotile sperm and infertility in mice lacking mitochondrial voltage-dependent anion channel type 3. | Q34086981 | ||
| Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c. | Q34139321 | ||
| DNA damage response and MCL-1 destruction initiate apoptosis in adenovirus-infected cells | Q34278532 | ||
| Proteolysis of MLL family proteins is essential for taspase1-orchestrated cell cycle progression. | Q34563234 | ||
| RAS-RAF-MEK-dependent oxidative cell death involving voltage-dependent anion channels | Q34618715 | ||
| Apoptosis in the development and maintenance of the immune system | Q35116684 | ||
| Proapoptotic multidomain Bcl-2/Bax-family proteins: mechanisms, physiological roles, and therapeutic opportunities | Q36490192 | ||
| BCL-2 in the crosshairs: tipping the balance of life and death | Q36501184 | ||
| The mitochondrial permeability transition pore and its involvement in cell death and in disease pathogenesis | Q36733673 | ||
| How do BCL-2 proteins induce mitochondrial outer membrane permeabilization? | Q37100016 | ||
| Recent progress in elucidating the molecular mechanism of the mitochondrial permeability transition pore. | Q37137108 | ||
| Altered mitochondrial sensitivity for ADP and maintenance of creatine-stimulated respiration in oxidative striated muscles from VDAC1-deficient mice | Q43508632 | ||
| Thymocyte negative selection is mediated by protein kinase C- and Ca2+-dependent transcriptional induction of bim [corrected]. | Q46925852 | ||
| Bcl-2-family proteins: the role of the BH3 domain in apoptosis | Q47686942 | ||
| P433 | issue | 85 | |
| P921 | main subject | thymocyte | Q7799635 |
| P304 | page(s) | ra48 | |
| P577 | publication date | 2009-08-25 | |
| P1433 | published in | Science Signaling | Q7433604 |
| P1476 | title | The VDAC2-BAK rheostat controls thymocyte survival | |
| P478 | volume | 2 |
| Q39080264 | A chemical glycoproteomics platform reveals O-GlcNAcylation of mitochondrial voltage-dependent anion channel 2. |
| Q35816653 | Analysis of proteome changes in doxorubicin-treated adult rat cardiomyocyte |
| Q34179568 | Artesunate induces apoptosis via a Bak-mediated caspase-independent intrinsic pathway in human lung adenocarcinoma cells |
| Q34486441 | Bax targets mitochondria by distinct mechanisms before or during apoptotic cell death: a requirement for VDAC2 or Bak for efficient Bax apoptotic function |
| Q37807748 | Bcl-2 proteins and mitochondria—Specificity in membrane targeting for death |
| Q35293180 | Bim and VDAC1 are hierarchically essential for mitochondrial ATF2 mediated cell death |
| Q28300914 | Building blocks of the apoptotic pore: how Bax and Bak are activated and oligomerize during apoptosis |
| Q35884065 | Characterization of Oyster Voltage-Dependent Anion Channel 2 (VDAC2) Suggests Its Involvement in Apoptosis and Host Defense |
| Q64052715 | Clinical and molecular cytogenetic characterization of a novel 10q interstitial deletion: a case report and review of the literature |
| Q38711737 | Complex formation and turnover of mitochondrial transporters and ion channels |
| Q30153423 | Cysteine residues impact the stability and micelle interaction dynamics of the human mitochondrial β-barrel anion channel hVDAC-2 |
| Q35552682 | Identification of Bax-voltage-dependent anion channel 1 complexes in digitonin-solubilized cerebellar granule neurons |
| Q34299265 | Inhibition of Bak activation by VDAC2 is dependent on the Bak transmembrane anchor |
| Q34630758 | Is mPTP the gatekeeper for necrosis, apoptosis, or both? |
| Q36111722 | MYBL2 guides autophagy suppressor VDAC2 in the developing ovary to inhibit autophagy through a complex of VDAC2-BECN1-BCL2L1 in mammals. |
| Q33838448 | Mcl-1 promotes survival of thymocytes by inhibition of Bak in a pathway separate from Bcl-2. |
| Q33645577 | Mitochondrial and postmitochondrial survival signaling in cancer. |
| Q38201407 | Mitochondrial channels: ion fluxes and more |
| Q36179374 | Motifs of VDAC2 required for mitochondrial Bak import and tBid-induced apoptosis |
| Q91973521 | Novel insights into molecular mechanisms of Pseudourostyla cristata encystment using comparative transcriptomics |
| Q26781203 | Regulation of Bim in Health and Disease |
| Q38268983 | Stepwise activation of BAX and BAK by tBID, BIM, and PUMA initiates mitochondrial apoptosis. |
| Q35687111 | VDAC1 selectively transfers apoptotic Ca2+ signals to mitochondria. |
| Q59333353 | VDAC2 enables BAX to mediate apoptosis and limit tumor development |
| Q41633850 | VDAC2-specific cellular functions and the underlying structure |
| Q37952424 | Voltage-dependant anion channels: novel insights into isoform function through genetic models. |
Search more.