Secretion of IL-1β from imatinib-resistant chronic myeloid leukemia cells contributes to BCR-ABL mutation-independent imatinib resistance.

scientific article published on 6 January 2016

Secretion of IL-1β from imatinib-resistant chronic myeloid leukemia cells contributes to BCR-ABL mutation-independent imatinib resistance. is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1002/1873-3468.12057
P698PubMed publication ID26831735

P50authorHye-Eun KimQ42920357
Sung-Gyoo ParkQ83144987
P2093author name stringCho-Rong Lee
Jung-Ah Kang
Taewoo Yang
Yegyun Choi
P2860cites workCloning, sequence and expression of two distinct human interleukin-1 complementary DNAsQ24294766
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Potentiation of the T-lymphocyte response to mitogens. I. The responding cellQ24679275
Two different point mutations in ABL gene ATP-binding domain conferring primary imatinib resistance in a chronic myeloid leukemia (CML) patient: A case reportQ24800045
Role of cyclooxygenase-2 in the angiogenesis of colorectal cancerQ28196352
The balance between IL-1 and IL-1Ra in diseaseQ28200818
Philadelphia chromosome-positive leukemias: from basic mechanisms to molecular therapeuticsQ28205012
Properties of the K562 cell line, derived from a patient with chronic myeloid leukemiaQ28236440
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IL-1 is required for tumor invasiveness and angiogenesisQ28594748
PTEN is a tumor suppressor in CML stem cells and BCR-ABL-induced leukemias in miceQ33608760
The biology of chronic myeloid leukemiaQ33686190
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Isolation and killing of candidate chronic myeloid leukemia stem cells by antibody targeting of IL-1 receptor accessory proteinQ34136730
A therapeutically targetable mechanism of BCR-ABL-independent imatinib resistance in chronic myeloid leukemia.Q34170309
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Many actions of cyclooxygenase-2 in cellular dynamics and in cancerQ34539982
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Second generation inhibitors of BCR-ABL for the treatment of imatinib-resistant chronic myeloid leukaemiaQ34622576
Kinase domain mutants of Bcr-Abl exhibit altered transformation potency, kinase activity, and substrate utilization, irrespective of sensitivity to imatinibQ35071103
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Rottlerin enhances IL-1β-induced COX-2 expression through sustained p38 MAPK activation in MDA-MB-231 human breast cancer cellsQ35667259
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Cytokines as a key component of cancer-related inflammationQ37243051
Ponatinib overcomes FGF2-mediated resistance in CML patients without kinase domain mutationsQ37623532
BCR/ABL kinase induces self-mutagenesis via reactive oxygen species to encode imatinib resistanceQ40306743
The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET proteinQ40351786
Regulation of hematopoietic growth factor production by genetically modified human bone marrow stromal cells expressing interleukin-1beta antisense RNA.Q40768282
Bcr-Abl protein tyrosine kinase activity induces a loss of p53 protein that mediates a delay in myeloid differentiationQ40837798
Mutator phenotype of BCR--ABL transfected Ba/F3 cell lines and its association with enhanced expression of DNA polymerase betaQ40951632
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Result of high-dose imatinib mesylate in patients with Philadelphia chromosome-positive chronic myeloid leukemia after failure of interferon-alphaQ44361989
Frequency of major molecular responses to imatinib or interferon alfa plus cytarabine in newly diagnosed chronic myeloid leukemiaQ44611047
P433issue3
P407language of work or nameEnglishQ1860
P921main subjectimatinibQ177094
chronic myeloid leukemiaQ729735
P304page(s)358-368
P577publication date2016-01-25
P1433published inFEBS LettersQ1388051
P1476titleSecretion of IL-1β from imatinib-resistant chronic myeloid leukemia cells contributes to BCR-ABL mutation-independent imatinib resistance
P478volume590