scholarly article | Q13442814 |
P356 | DOI | 10.1007/S00418-014-1204-3 |
P698 | PubMed publication ID | 24664425 |
P50 | author | Jin Won Cho | Q12617092 |
Yukishige Ito | Q37376248 | ||
P2093 | author name string | Christian Zuber | |
Jürgen Roth | |||
Ichiro Matsuo | |||
Yangsin Lee | |||
Insook Jang | |||
Sujin Park | |||
P2860 | cites work | p62/SQSTM1 and ALFY interact to facilitate the formation of p62 bodies/ALIS and their degradation by autophagy | Q24300134 |
Alfy, a novel FYVE-domain-containing protein associated with protein granules and autophagic membranes | Q24300526 | ||
The deacetylase HDAC6 regulates aggresome formation and cell viability in response to misfolded protein stress | Q24301895 | ||
The selective macroautophagic degradation of aggregated proteins requires the PI3P-binding protein Alfy | Q24307759 | ||
p62, a phosphotyrosine-independent ligand of the SH2 domain of p56lck, belongs to a new class of ubiquitin-binding proteins | Q24311839 | ||
p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy | Q24312147 | ||
Human OS-9, a lectin required for glycoprotein endoplasmic reticulum-associated degradation, recognizes mannose-trimmed N-glycans | Q24313146 | ||
A role for NBR1 in autophagosomal degradation of ubiquitinated substrates | Q24316135 | ||
HDAC6 and microtubules are required for autophagic degradation of aggregated huntingtin | Q24337155 | ||
EDEM1 recognition and delivery of misfolded proteins to the SEL1L-containing ERAD complex | Q24338144 | ||
A novel ER alpha-mannosidase-like protein accelerates ER-associated degradation | Q24522524 | ||
A glycosylated type I membrane protein becomes cytosolic when peptide: N-glycanase is compromised | Q24535941 | ||
Identification of proteins that interact with mammalian peptide:N-glycanase and implicate this hydrolase in the proteasome-dependent pathway for protein degradation | Q24555191 | ||
LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing | Q24597817 | ||
Misfolded proteins partition between two distinct quality control compartments | Q24651093 | ||
EDEM1 reveals a quality control vesicular transport pathway out of the endoplasmic reticulum not involving the COPII exit sites | Q24683239 | ||
Structural basis for sorting mechanism of p62 in selective autophagy | Q27650745 | ||
Autophagy fights disease through cellular self-digestion | Q27860902 | ||
A luminal surveillance complex that selects misfolded glycoproteins for ER-associated degradation | Q27930372 | ||
Exploration of the topological requirements of ERAD identifies Yos9p as a lectin sensor of misfolded glycoproteins in the ER lumen | Q27930448 | ||
Defining the glycan destruction signal for endoplasmic reticulum-associated degradation | Q27932810 | ||
A complex of Pdi1p and the mannosidase Htm1p initiates clearance of unfolded glycoproteins from the endoplasmic reticulum | Q27933028 | ||
Yos9p detects and targets misfolded glycoproteins for ER-associated degradation | Q27933494 | ||
Yos9 protein is essential for degradation of misfolded glycoproteins and may function as lectin in ERAD. | Q27934679 | ||
Mnl1p, an alpha -mannosidase-like protein in yeast Saccharomyces cerevisiae, is required for endoplasmic reticulum-associated degradation of glycoproteins | Q27937049 | ||
Degradation of misfolded endoplasmic reticulum glycoproteins in Saccharomyces cerevisiae is determined by a specific oligosaccharide structure | Q27938571 | ||
Retrotranslocation of a misfolded luminal ER protein by the ubiquitin-ligase Hrd1p | Q27938951 | ||
Htm1 protein generates the N-glycan signal for glycoprotein degradation in the endoplasmic reticulum | Q27939524 | ||
Htm1p, a mannosidase-like protein, is involved in glycoprotein degradation in yeast | Q27939809 | ||
Aggresomes: a cellular response to misfolded proteins | Q28292275 | ||
A novel stress-induced EDEM variant regulating endoplasmic reticulum-associated glycoprotein degradation | Q28296585 | ||
Selective autophagy mediated by autophagic adapter proteins | Q28301940 | ||
EDEM1 accelerates the trimming of alpha1,2-linked mannose on the C branch of N-glycans | Q28510507 | ||
Immunolocalization of UDP-glucose:glycoprotein glucosyltransferase indicates involvement of pre-Golgi intermediates in protein quality control | Q28582340 | ||
Autophagy: renovation of cells and tissues | Q29547206 | ||
Methods in mammalian autophagy research | Q29547276 | ||
p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death | Q29547297 | ||
The role of Atg proteins in autophagosome formation | Q29547418 | ||
Distinct classes of phosphatidylinositol 3'-kinases are involved in signaling pathways that control macroautophagy in HT-29 cells | Q29614181 | ||
Intracellular Aspects of the Process of Protein Synthesis | Q29615237 | ||
Roles of N-linked glycans in the endoplasmic reticulum | Q29616458 | ||
HDAC6 controls autophagosome maturation essential for ubiquitin-selective quality-control autophagy | Q30014829 | ||
Role of the SEL1L:LC3-I complex as an ERAD tuning receptor in the mammalian ER. | Q44455557 | ||
Misfolding of glycoproteins is a prerequisite for peptide: N-glycanase mediated deglycosylation. | Q45238415 | ||
Systematic synthesis and inhibitory activity of haloacetamidyl oligosaccharide derivatives toward cytoplasmic peptide:N-glycanase | Q46434284 | ||
Use of poly(vinylpyrrolidone) and poly(vinyl alcohol) for cryoultramicrotomy | Q46472458 | ||
Chemistry and biology of wortmannin | Q46511165 | ||
z-VAD-fmk inhibits peptide:N-glycanase and may result in ER stress | Q46589271 | ||
A cell culture system for the induction of Mallory bodies: Mallory bodies and aggresomes represent different types of inclusion bodies | Q47973741 | ||
A complex of Yos9p and the HRD ligase integrates endoplasmic reticulum quality control into the degradation machinery. | Q52931787 | ||
Large protein complexes retained in the ER are dislocated by non-COPII vesicles and degraded by selective autophagy. | Q54455138 | ||
Prevention of non-specific interactions of gold-labeled reagents on tissue sections | Q69094922 | ||
Postembedding labeling on Lowicryl K4M tissue sections: detection and modification of cellular components | Q69536946 | ||
Ubiquitylation in the ERAD Pathway | Q83165590 | ||
The ubiquitin clan: A protein family essential for life | Q84828995 | ||
A genome-wide screen identifies Yos9p as essential for ER-associated degradation of glycoproteins | Q31130967 | ||
Dual role of 3-methyladenine in modulation of autophagy via different temporal patterns of inhibition on class I and III phosphoinositide 3-kinase | Q33796255 | ||
Delivery of ubiquitinated substrates to protein-unfolding machines | Q33989072 | ||
ALIS are stress-induced protein storage compartments for substrates of the proteasome and autophagy | Q33998133 | ||
A role for N-glycanase in the cytosolic turnover of glycoproteins | Q34179944 | ||
Ultrastructural localization of intracellular antigens by the use of protein A-gold complex | Q34186780 | ||
The structure and biological features of fibrinogen and fibrin. | Q34311771 | ||
Identification and distribution of peptide:N-glycanase (PNGase) in mouse organs | Q34313410 | ||
Identification of peptide:N-glycanase activity in mammalian-derived cultured cells | Q34352953 | ||
Potent histone deacetylase inhibitors built from trichostatin A and cyclic tetrapeptide antibiotics including trapoxin | Q34584029 | ||
Golgi apparatus immunolocalization of endomannosidase suggests post-endoplasmic reticulum glucose trimming: implications for quality control | Q34783037 | ||
Characterization of early EDEM1 protein maturation events and their functional implications | Q35107478 | ||
Rad23 and Rpn10: perennial wallflowers join the melee | Q35950681 | ||
The Png1-Rad23 complex regulates glycoprotein turnover | Q36116808 | ||
Immunolocalization of the oligosaccharide trimming enzyme glucosidase II | Q36215161 | ||
The organization of endoplasmic reticulum export complexes | Q36237498 | ||
Cytoplasmic peptide:N-glycanase and catabolic pathway for free N-glycans in the cytosol | Q36976680 | ||
Misfolded membrane proteins are specifically recognized by the transmembrane domain of the Hrd1p ubiquitin ligase | Q37260547 | ||
The proteostasis boundary in misfolding diseases of membrane traffic | Q37587095 | ||
Autophagy and misfolded proteins in neurodegeneration | Q37811463 | ||
Mitochondria removal by autophagy | Q37831014 | ||
Protein N-glycosylation, protein folding, and protein quality control. | Q37845738 | ||
Protein folding and modification in the mammalian endoplasmic reticulum. | Q37865523 | ||
Fluorescently labeled inhibitor for profiling cytoplasmic peptide:N-glycanase | Q38299876 | ||
Rad23 provides a link between the Png1 deglycosylating enzyme and the 26 S proteasome in yeast. | Q38303135 | ||
p62/SQSTM1 in autophagic clearance of a non-ubiquitylated substrate | Q39504135 | ||
A reporter cell system to monitor autophagy based on p62/SQSTM1. | Q39688848 | ||
Basal autophagy is involved in the degradation of the ERAD component EDEM1. | Q39876239 | ||
Segregation and rapid turnover of EDEM1 by an autophagy-like mechanism modulates standard ERAD and folding activities | Q39985945 | ||
EDEM1 regulates ER-associated degradation by accelerating de-mannosylation of folding-defective polypeptides and by inhibiting their covalent aggregation. | Q40229528 | ||
Using a small molecule inhibitor of peptide: N-glycanase to probe its role in glycoprotein turnover. | Q40478768 | ||
p62 Is a common component of cytoplasmic inclusions in protein aggregation diseases | Q42184099 | ||
Inhibition of human caspases by peptide-based and macromolecular inhibitors | Q42538024 | ||
Wortmannin, a potent and selective inhibitor of phosphatidylinositol-3-kinase | Q42799184 | ||
Nucleocytoplasmic shuttling of p62/SQSTM1 and its role in recruitment of nuclear polyubiquitinated proteins to promyelocytic leukemia bodies. | Q42917978 | ||
P433 | issue | 2 | |
P921 | main subject | autophagy | Q288322 |
P304 | page(s) | 153-169 | |
P577 | publication date | 2014-03-25 | |
P1433 | published in | Histochemistry and Cell Biology | Q1524012 |
P1476 | title | ERADication of EDEM1 occurs by selective autophagy and requires deglycosylation by cytoplasmic peptide N-glycanase | |
P478 | volume | 142 |
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Q37319145 | Identification of the nuclear localisation signal of O-GlcNAc transferase and its nuclear import regulation |
Q38996383 | Quality control of glycoprotein folding and ERAD: the role of N-glycan handling, EDEM1 and OS-9. |
Q38368268 | The Histochemistry and Cell Biology pandect: the year 2014 in review |
Q38507330 | The Role of Lectin-Carbohydrate Interactions in the Regulation of ER-Associated Protein Degradation. |
Q30274598 | The cytoplasmic peptide:N-glycanase (NGLY1) - Structure, expression and cellular functions |
Q34448040 | The cytoplasmic peptide:N-glycanase (Ngly1)-basic science encounters a human genetic disorder |
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