scholarly article | Q13442814 |
P50 | author | Nadja Zaborsky | Q64213596 |
Roland Geisberger | Q42399953 | ||
Thomas K Felder | Q57631136 | ||
Franz Josef Gassner | Q64213594 | ||
P2093 | author name string | Richard Greil | |
Michael Huemer | |||
Kemal Catakovic | |||
Stefan Rebhandl | |||
P2860 | cites work | Solubility-based genetic screen identifies RING finger protein 126 as an E3 ligase for activation-induced cytidine deaminase | Q24307502 |
Activation-induced cytidine deaminase shuttles between nucleus and cytoplasm like apolipoprotein B mRNA editing catalytic polypeptide 1 | Q24307678 | ||
Molecules and their functions in autophagy | Q24622004 | ||
MicroRNA-155 is a negative regulator of activation-induced cytidine deaminase | Q24653222 | ||
Correlation between protein and mRNA abundance in yeast | Q28137554 | ||
Chronic lymphocytic leukemia B cells expressing AID display dissociation between class switch recombination and somatic hypermutation | Q28202500 | ||
Activation-induced cytidine deaminase splice variants are defective because of the lack of structural support for the catalytic site. | Q30385115 | ||
Integrity of the AID serine-38 phosphorylation site is critical for class switch recombination and somatic hypermutation in mice | Q30857687 | ||
The AID antibody diversification enzyme is regulated by protein kinase A phosphorylation | Q33226024 | ||
Haploinsufficiency of activation-induced deaminase for antibody diversification and chromosome translocations both in vitro and in vivo | Q33392701 | ||
Human chronic lymphocytic leukemia modeled in mouse by targeted TCL1 expression | Q34068006 | ||
Lysine residue at position 22 of the AID protein regulates its class switch activity. | Q34170147 | ||
Regulation of activation-induced deaminase stability and antibody gene diversification by Hsp90. | Q34340626 | ||
Regulation of hypermutation by activation-induced cytidine deaminase phosphorylation | Q34695460 | ||
The splicing factor SF2/ASF regulates translation initiation by enhancing phosphorylation of 4E-BP1. | Q34774397 | ||
Mutations associated with Charcot-Marie-Tooth disease cause SIMPLE protein mislocalization and degradation by the proteasome and aggresome-autophagy pathways | Q35227491 | ||
Translocation-capture sequencing reveals the extent and nature of chromosomal rearrangements in B lymphocytes | Q35304991 | ||
Retrovirus-mediated gene transfer and expression cloning: powerful tools in functional genomics | Q35571380 | ||
REG-γ associates with and modulates the abundance of nuclear activation-induced deaminase | Q35669089 | ||
The regulation of somatic hypermutation | Q35692214 | ||
Regulation of AID expression in the immune response | Q36229307 | ||
IGHV-unmutated and IGHV-mutated chronic lymphocytic leukemia cells produce activation-induced deaminase protein with a full range of biologic functions | Q36462397 | ||
Immunoglobulin class-switch DNA recombination: induction, targeting and beyond | Q36534234 | ||
Proteasomal degradation restricts the nuclear lifespan of AID | Q36699771 | ||
Molecular mechanisms of antibody somatic hypermutation. | Q36747477 | ||
Regulation of class switch recombination and somatic mutation by AID phosphorylation | Q36946561 | ||
Rate-limiting steps in yeast protein translation | Q36959359 | ||
Alternative splicing regulates activation-induced cytidine deaminase (AID): implications for suppression of AID mutagenic activity in normal and malignant B cells | Q37001985 | ||
The stability of AID and its function in class-switching are critically sensitive to the identity of its nuclear-export sequence | Q37167699 | ||
AID is required for the chromosomal breaks in c-myc that lead to c-myc/IgH translocations | Q37269390 | ||
Genome-wide translocation sequencing reveals mechanisms of chromosome breaks and rearrangements in B cells | Q38626707 | ||
Activation of human B cells by phosphorothioate oligodeoxynucleotides | Q39770135 | ||
Expression of activation-induced cytidine deaminase in human hepatocytes via NF-kappaB signaling | Q40150411 | ||
Amino-terminal phosphorylation of activation-induced cytidine deaminase suppresses c-myc/IgH translocation | Q42582794 | ||
MEC1 and MEC2: two new cell lines derived from B-chronic lymphocytic leukaemia in prolymphocytoid transformation | Q42820324 | ||
The activation-induced deaminase functions in a postcleavage step of the somatic hypermutation process | Q42944711 | ||
High expression of AID and active class switch recombination might account for a more aggressive disease in unmutated CLL patients: link with an activated microenvironment in CLL disease | Q43127229 | ||
Activation-induced cytidine deaminase splicing patterns in chronic lymphocytic leukemia | Q43179782 | ||
High expression of activation-induced cytidine deaminase (AID) and splice variants is a distinctive feature of poor-prognosis chronic lymphocytic leukemia | Q44315242 | ||
Activation-induced cytidine deaminase in chronic lymphocytic leukemia B cells: expression as multiple forms in a dynamic, variably sized fraction of the clone. | Q44509645 | ||
AID splice variants lack deaminase activity. | Q46116559 | ||
Two levels of protection for the B cell genome during somatic hypermutation. | Q46755254 | ||
AID is required for germinal center-derived lymphomagenesis | Q46861217 | ||
Regulatory T cells predict the time to initial treatment in early stage chronic lymphocytic leukemia. | Q53251673 | ||
P433 | issue | 7 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | leukemia | Q29496 |
stoichiometry | Q213185 | ||
lymphocyte | Q715347 | ||
chronic lymphocytic leukemia | Q1088156 | ||
P304 | page(s) | 2175-2187 | |
P577 | publication date | 2014-04-15 | |
P1433 | published in | European Journal of Immunology | Q5412727 |
P1476 | title | Alternative splice variants of AID are not stoichiometrically present at the protein level in chronic lymphocytic leukemia | |
P478 | volume | 44 |
Q34770449 | AID induces intraclonal diversity and genomic damage in CD86(+) chronic lymphocytic leukemia cells |
Q28085553 | AID/APOBEC deaminases and cancer |
Q37593573 | Reactivation of dormant anti-tumor immunity - a clinical perspective of therapeutic immune checkpoint modulation |
Q35585086 | Splice variants of activation induced deaminase (AID) do not affect the efficiency of class switch recombination in murine CH12F3 cells |
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