scholarly article | Q13442814 |
P50 | author | Stephanie Bleicken | Q57054678 |
Ana J García-Sáez | Q78568898 | ||
P2093 | author name string | Gorka Basañez | |
Olatz Landeta | |||
Ane Landajuela | |||
P2860 | cites work | Bcl-x(L) retrotranslocates Bax from the mitochondria into the cytosol | Q24298758 |
Membrane remodeling induced by the dynamin-related protein Drp1 stimulates Bax oligomerization | Q24300125 | ||
Bax crystal structures reveal how BH3 domains activate Bax and nucleate its oligomerization to induce apoptosis | Q24313049 | ||
Membrane binding by tBid initiates an ordered series of events culminating in membrane permeabilization by Bax | Q24323186 | ||
Endophilin B1/Bif-1 stimulates BAX activation independently from its capacity to produce large scale membrane morphological rearrangements | Q24324817 | ||
Movement of Bax from the cytosol to mitochondria during apoptosis | Q24677881 | ||
A new view of the lethal apoptotic pore | Q27000479 | ||
High resolution X-ray crystallographic structure of bovine heart cytochrome c and its application to the design of an electron transfer biosensor | Q27646756 | ||
BID-induced structural changes in BAK promote apoptosis | Q27677468 | ||
Isolation, crystallization, crystal structure analysis and refinement of allophycocyanin from the cyanobacterium Spirulina platensis at 2.3 A resolution | Q27730274 | ||
Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death | Q28363890 | ||
The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues | Q28512628 | ||
Bid induces the oligomerization and insertion of Bax into the outer mitochondrial membrane | Q28647586 | ||
Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane | Q29616354 | ||
Structure of transmembrane pore induced by Bax-derived peptide: evidence for lipidic pores. | Q30157467 | ||
Different mitochondrial intermembrane space proteins are released during apoptosis in a manner that is coordinately initiated but can vary in duration | Q30477672 | ||
Antimicrobial peptides bind more strongly to membrane pores | Q33912494 | ||
BAX-dependent transport of cytochrome c reconstituted in pure liposomes | Q33912996 | ||
Dynamic interaction of cBid with detergents, liposomes and mitochondria. | Q34249427 | ||
XIAP impairs Smac release from the mitochondria during apoptosis. | Q34545370 | ||
A lipocentric view of peptide-induced pores. | Q34757093 | ||
Membrane protein assembly into Nanodiscs | Q35008328 | ||
BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak | Q35079498 | ||
Mitochondria in apoptosis: Bcl-2 family members and mitochondrial dynamics. | Q35165455 | ||
Translocation of a Bak C-terminus mutant from cytosol to mitochondria to mediate cytochrome C release: implications for Bak and Bax apoptotic function | Q35839831 | ||
Mitochondrial release of apoptosis-inducing factor occurs downstream of cytochrome c release in response to several proapoptotic stimuli | Q36325547 | ||
Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations | Q36353924 | ||
Where killers meet--permeabilization of the outer mitochondrial membrane during apoptosis | Q36629743 | ||
Direct activation of full-length proapoptotic BAK | Q36692849 | ||
Three-dimensional structure of Bax-mediated pores in membrane bilayers | Q36985116 | ||
Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation | Q37158414 | ||
Assembly of the mitochondrial apoptosis-induced channel, MAC. | Q37169502 | ||
Spatial and temporal dynamics of mitochondrial membrane permeability waves during apoptosis | Q37392644 | ||
Conformational changes and protein stability of the pro-apoptotic protein Bax. | Q37424410 | ||
The physicochemical properties of cardiolipin bilayers and cardiolipin-containing lipid membranes | Q37425910 | ||
Permeabilization of the Outer Mitochondrial Membrane by Bcl-2 Proteins | Q37778022 | ||
Homeostatic functions of BCL-2 proteins beyond apoptosis | Q37797382 | ||
Apoptosis and oncogenesis: give and take in the BCL-2 family | Q37828794 | ||
The secrets of the Bcl-2 family | Q38039293 | ||
Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis | Q38326924 | ||
Sphingosine-induced apoptosis in rhabdomyosarcoma cell lines is dependent on pre-mitochondrial Bax activation and post-mitochondrial caspases | Q40181636 | ||
Peptides derived from apoptotic Bax and Bid reproduce the poration activity of the parent full-length proteins | Q40334590 | ||
Real-time single cell analysis of Smac/DIABLO release during apoptosis | Q40634471 | ||
Bax exists in a dynamic equilibrium between the cytosol and mitochondria to control apoptotic priming | Q41263641 | ||
Reconstitution of proapoptotic BAK function in liposomes reveals a dual role for mitochondrial lipids in the BAK-driven membrane permeabilization process | Q41975085 | ||
Pore formation and translocation of melittin | Q42029147 | ||
Pores formed by Baxα5 relax to a smaller size and keep at equilibrium | Q42183300 | ||
Pore formation by a Bax-derived peptide: effect on the line tension of the membrane probed by AFM. | Q42183606 | ||
Transient binding of an activator BH3 domain to the Bak BH3-binding groove initiates Bak oligomerization | Q42800693 | ||
Molecular details of Bax activation, oligomerization, and membrane insertion. | Q42910802 | ||
Mechanistic differences in the membrane activity of Bax and Bcl-xL correlate with their opposing roles in apoptosis. | Q42928576 | ||
Bid induces cytochrome c-impermeable Bax channels in liposomes | Q43001510 | ||
Transbilayer lipid diffusion promoted by Bax: implications for apoptosis. | Q44684385 | ||
Membrane promotes tBID interaction with BCL(XL). | Q45815145 | ||
Magainin 2-induced pore formation in the lipid membranes depends on its concentration in the membrane interface | Q46225719 | ||
BIM and tBID are not mechanistically equivalent when assisting BAX to permeabilize bilayer membranes | Q46808299 | ||
Lipidic pore formation by the concerted action of proapoptotic BAX and tBID. | Q47618625 | ||
Molecular mechanism of Peptide-induced pores in membranes | Q49260601 | ||
Peptides corresponding to helices 5 and 6 of Bax can independently form large lipid pores | Q57824114 | ||
Mitochondrial contact sites. Lipid composition and dynamics | Q68483792 | ||
Regulation of antiapoptotic MCL-1 function by gossypol: mechanistic insights from in vitro reconstituted systems | Q81908401 | ||
Structure of fully hydrated fluid phase lipid bilayers with monounsaturated chains | Q83129856 | ||
P4510 | describes a project that uses | ImageJ | Q1659584 |
P433 | issue | 46 | |
P407 | language of work or name | English | Q1860 |
P1104 | number of pages | 12 | |
P304 | page(s) | 33241-33252 | |
P577 | publication date | 2013-10-07 | |
P1433 | published in | Journal of Biological Chemistry | Q867727 |
P1476 | title | Proapoptotic Bax and Bak proteins form stable protein-permeable pores of tunable size | |
P478 | volume | 288 |
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Q36996518 | In Situ Characterization of Bak Clusters Responsible for Cell Death Using Single Molecule Localization Microscopy. |
Q41570585 | Live-cell imaging to measure BAX recruitment kinetics to mitochondria during apoptosis |
Q96023628 | Location of Peptide-Induced Submicron Discontinuities in the Membranes of Vesicles Using ImageJ |
Q48161032 | Melilotus indicus extract induces apoptosis in hepatocellular carcinoma cells via a mechanism involving mitochondria-mediated pathways. |
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Q38952396 | Milestones and recent discoveries on cell death mediated by mitochondria and their interactions with biologically active amines |
Q28383717 | Minimalist Model Systems Reveal Similarities and Differences between Membrane Interaction Modes of MCL1 and BAK |
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Q90518992 | Mitochondrial inner membrane permeabilisation enables mtDNA release during apoptosis |
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Q34853156 | Pycnogenol Induces Nuclear Translocation of Apoptosis-inducing Factor and Caspase-independent Apoptosis in MC-3 Human Mucoepidermoid Carcinoma Cell Line |
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Q52721187 | Regulatory role of calpain in neuronal death. |
Q33752784 | Release of Cytochrome C from Bax Pores at the Mitochondrial Membrane |
Q42166522 | Structural model of active Bax at the membrane. |
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Q48374835 | The membrane activity of BOK involves formation of large, stable toroidal pores and is promoted by cBID. |
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Q34960771 | Visual and functional demonstration of growing Bax-induced pores in mitochondrial outer membranes |
Q40757128 | cBid, Bax and Bcl-xL exhibit opposite membrane remodeling activities. |
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