| scholarly article | Q13442814 |
| P50 | author | Jerry E. Chipuk | Q38325376 |
| Roger Schneiter | Q57209447 | ||
| Vineet Choudhary | Q83396290 | ||
| P2093 | author name string | Tomomi Kuwana | |
| Han Du | |||
| Joel Quispe | |||
| Blanca Schafer | |||
| Teddy G Ajero | |||
| P2860 | cites work | Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC | Q22009974 |
| A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death | Q24291746 | ||
| Interaction with a membrane surface triggers a reversible conformational change in Bax normally associated with induction of apoptosis | Q24296781 | ||
| Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c | Q24309066 | ||
| Oligomeric Bax is a component of the putative cytochrome c release channel MAC, mitochondrial apoptosis-induced channel | Q24523479 | ||
| Mitochondrial carrier homolog 2 is a target of tBID in cells signaled to die by tumor necrosis factor alpha | Q24529078 | ||
| Conformation of the Bax C-terminus regulates subcellular location and cell death | Q24534099 | ||
| Pharmacological manipulation of cell death: clinical applications in sight? | Q24536101 | ||
| A novel, high conductance channel of mitochondria linked to apoptosis in mammalian cells and Bax expression in yeast | Q24685400 | ||
| The pro-apoptotic Bcl-2 family member tBid localizes to mitochondrial contact sites | Q24794830 | ||
| Structure of the outer membrane protein A transmembrane domain | Q27765990 | ||
| Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition | Q28115131 | ||
| Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins | Q28117928 | ||
| Structural basis of pore formation by the bacterial toxin pneumolysin | Q28246818 | ||
| Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death | Q28297152 | ||
| Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death | Q28363890 | ||
| The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues | Q28512628 | ||
| The BCL-2 protein family: opposing activities that mediate cell death | Q29547380 | ||
| Automated molecular microscopy: the new Leginon system | Q29614290 | ||
| The Bcl2 family: regulators of the cellular life-or-death switch | Q29614982 | ||
| Bid, Bax, and lipids cooperate to form supramolecular openings in the outer mitochondrial membrane | Q29616354 | ||
| BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly | Q29617135 | ||
| Structure of transmembrane pore induced by Bax-derived peptide: evidence for lipidic pores. | Q30157467 | ||
| Correct folding of the beta-barrel of the human membrane protein VDAC requires a lipid bilayer | Q30158037 | ||
| Gene duplication of the eight-stranded beta-barrel OmpX produces a functional pore: a scenario for the evolution of transmembrane beta-barrels | Q30159488 | ||
| The mechanism of pore formation by bacterial toxins | Q30159862 | ||
| Folding and assembly of beta-barrel membrane proteins | Q30160417 | ||
| Bcl-XL inhibits membrane permeabilization by competing with Bax. | Q33343021 | ||
| Mechanism of the binding, insertion and destabilization of phospholipid bilayer membranes by alpha-helical antimicrobial and cell non-selective membrane-lytic peptides | Q33789716 | ||
| BAX-dependent transport of cytochrome c reconstituted in pure liposomes | Q33912996 | ||
| Mixed micelles and other structures in the solubilization of bilayer lipid membranes by surfactants | Q34088694 | ||
| Mitochondrial carrier homolog 2: a clue to cracking the BCL-2 family riddle? | Q34451522 | ||
| Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria | Q35009655 | ||
| Bcl-2-family proteins and the role of mitochondria in apoptosis | Q35595911 | ||
| Intrinsic tumour suppression | Q35953133 | ||
| Promoting apoptosis as a strategy for cancer drug discovery | Q36292878 | ||
| Bax, but not Bcl-xL, decreases the lifetime of planar phospholipid bilayer membranes at subnanomolar concentrations | Q36353924 | ||
| Bax and Bak coalesce into novel mitochondria-associated clusters during apoptosis | Q36364894 | ||
| Role of cardiolipin in cytochrome c release from mitochondria | Q36790564 | ||
| Embedded together: the life and death consequences of interaction of the Bcl-2 family with membranes | Q36799170 | ||
| BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases | Q37044625 | ||
| Mitochondria potential, bax "activation," and programmed cell death | Q37050814 | ||
| Refolding and oriented insertion of a membrane protein into a lipid bilayer | Q37150531 | ||
| Cardiolipin Is Not Required for Bax-mediated Cytochrome c Release from Yeast Mitochondria | Q38349383 | ||
| Caspase inhibition blocks cell death and results in cell cycle arrest in cytokine-deprived hematopoietic cells | Q40208906 | ||
| Liposomes, disks, and spherical micelles: aggregate structure in mixtures of gel phase phosphatidylcholines and poly(ethylene glycol)-phospholipids | Q40263969 | ||
| Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome c. | Q40886623 | ||
| Inhibition of Ced-3/ICE-related proteases does not prevent cell death induced by oncogenes, DNA damage, or the Bcl-2 homologue Bak | Q41134694 | ||
| Cryoelectron microscopy of liposomes | Q41614670 | ||
| The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant | Q41723645 | ||
| Pore formation by a Bax-derived peptide: effect on the line tension of the membrane probed by AFM. | Q42183606 | ||
| Cardiolipin and phosphatidylglycerol are not required for the in vivo action of Bcl-2 family proteins | Q42214140 | ||
| Bid induces cytochrome c-impermeable Bax channels in liposomes | Q43001510 | ||
| Bax oligomerization in mitochondrial membranes requires tBid (caspase-8-cleaved Bid) and a mitochondrial protein. | Q43002476 | ||
| Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells | Q43514949 | ||
| Cardiolipin and its metabolites move from mitochondria to other cellular membranes during death receptor-mediated apoptosis. | Q44924644 | ||
| Do inducers of apoptosis trigger caspase-independent cell death? | Q45267818 | ||
| The role of apoptosis in cancer development and treatment response | Q46136338 | ||
| Contributions to Bax insertion and oligomerization of lipids of the mitochondrial outer membrane | Q46765170 | ||
| Bax activation and stress-induced apoptosis delayed by the accumulation of cholesterol in mitochondrial membranes | Q46847162 | ||
| Lipidic pore formation by the concerted action of proapoptotic BAX and tBID. | Q47618625 | ||
| GAPDH and autophagy preserve survival after apoptotic cytochrome c release in the absence of caspase activation | Q50681145 | ||
| Bax-type apoptotic proteins porate pure lipid bilayers through a mechanism sensitive to intrinsic monolayer curvature | Q54536632 | ||
| Mitochondrial contact sites. Lipid composition and dynamics | Q68483792 | ||
| Cardiolipin provides specificity for targeting of tBid to mitochondria | Q73062575 | ||
| Strategy for membrane protein crystallization exemplified with OmpA and OmpX | Q74449398 | ||
| The mitochondrial apoptosis-induced channel (MAC) corresponds to a late apoptotic event | Q80490091 | ||
| Effect of lipids with different spontaneous curvature on the channel activity of colicin E1: evidence in favor of a toroidal pore | Q80841194 | ||
| P433 | issue | 8 | |
| P921 | main subject | mitochondrion | Q39572 |
| membrane protein | Q423042 | ||
| P304 | page(s) | 2276-2285 | |
| P577 | publication date | 2009-02-25 | |
| P1433 | published in | Molecular Biology of the Cell | Q2338259 |
| P1476 | title | Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation | |
| P478 | volume | 20 |
| Q84566130 | A BID on mitochondria with MTCH2 |
| Q46920040 | Activation of Bax by joint action of tBid and mitochondrial outer membrane: Monte Carlo simulations |
| Q34384160 | Activation of mitochondrial protease OMA1 by Bax and Bak promotes cytochrome c release during apoptosis |
| Q34963114 | Anti-apoptotic activity and proteasome-mediated degradation of Xenopus Mcl-1 protein in egg extracts |
| Q37828794 | Apoptosis and oncogenesis: give and take in the BCL-2 family |
| Q38181031 | Apoptosis regulation at the mitochondrial outer membrane |
| Q37796181 | Apoptosis-induced changes in mitochondrial lipids |
| Q28831381 | Assembly of Bak homodimers into higher order homooligomers in the mitochondrial apoptotic pore |
| Q37151053 | Assembly of the Bak apoptotic pore: a critical role for the Bak protein α6 helix in the multimerization of homodimers during apoptosis |
| Q36478810 | BH3-in-groove dimerization initiates and helix 9 dimerization expands Bax pore assembly in membranes |
| Q37629756 | BH3-only proteins: the death-puppeteer's wires |
| Q28483986 | Bax activation initiates the assembly of a multimeric catalyst that facilitates Bax pore formation in mitochondrial outer membranes |
| Q39033533 | Bax and Bak Pores: Are We Closing the Circle? |
| Q38494665 | Bax assembles into large ring-like structures remodeling the mitochondrial outer membrane in apoptosis |
| Q47305952 | Bax, Bak and beyond - mitochondrial performance in apoptosis |
| Q34310255 | Beta-amyloid oligomers activate apoptotic BAK pore for cytochrome c release. |
| Q83728527 | Bid and Bax Are Involved in Granulosa Cell Apoptosis During Follicular Atresia in Porcine Ovaries |
| Q28240923 | Bioactive lipids and the control of Bax pro-apoptotic activity |
| Q37098808 | Cardiolipin or MTCH2 can serve as tBID receptors during apoptosis |
| Q35869797 | Ceramide channels: Influence of molecular structure on channel formation in membranes |
| Q92494358 | Contribution of Mitochondrial Ion Channels to Chemo-Resistance in Cancer Cells |
| Q28397122 | Cytochrome c causes pore formation in cardiolipin-containing membranes |
| Q34671696 | Data-driven modeling of SRC control on the mitochondrial pathway of apoptosis: implication for anticancer therapy optimization |
| Q39448418 | Effect of RNA Interference of BID and BAX mRNAs on Apoptosis in Granulosa Cell-derived KGN Cells |
| Q28477131 | Efficient Elimination of Cancer Cells by Deoxyglucose-ABT-263/737 Combination Therapy |
| Q38092535 | Emerging roles of lipids in BCL-2 family-regulated apoptosis |
| Q39352250 | Emodin Regulates Apoptotic Pathway in Human Liver Cancer Cells |
| Q47160842 | Get1p and Get2p are required for maintenance of mitochondrial morphology and normal cardiolipin levels |
| Q43268599 | Interaction of the alpha-helical H6 peptide from the pro-apoptotic protein tBid with cardiolipin |
| Q47148226 | MOMP, cell suicide as a BCL-2 family business |
| Q28594262 | MTCH2/MIMP is a major facilitator of tBID recruitment to mitochondria |
| Q36932838 | Mechanisms of action of Bcl-2 family proteins |
| Q29615459 | Mitochondria and cell death: outer membrane permeabilization and beyond |
| Q35165455 | Mitochondria in apoptosis: Bcl-2 family members and mitochondrial dynamics. |
| Q45969652 | Mitochondria, Bioenergetics and Apoptosis in Cancer. |
| Q38043576 | Mitochondrial Outer Membrane Channels |
| Q35087797 | Mitochondrial cardiolipin involved in outer-membrane protein biogenesis: implications for Barth syndrome |
| Q41579498 | Mitochondrial outer membrane permeabilization: a focus on the role of mitochondrial membrane structural organization |
| Q38134062 | Mitochondrial regulation of cell death. |
| Q39447472 | Mitochondrial swelling and incipient outer membrane rupture in preapoptotic and apoptotic cells |
| Q37517187 | Mitochondrial targeting of tBid/Bax: a role for the TOM complex? |
| Q61799950 | Molecular and topological reorganizations in mitochondrial architecture interplay during Bax-mediated steps of apoptosis |
| Q42910802 | Molecular details of Bax activation, oligomerization, and membrane insertion. |
| Q37536444 | Organization of the mitochondrial apoptotic BAK pore: oligomerization of the BAK homodimers |
| Q37778022 | Permeabilization of the Outer Mitochondrial Membrane by Bcl-2 Proteins |
| Q92632027 | Phenotypic selection with an intrabody library reveals an anti-apoptotic function of PKM2 requiring Mitofusin-1 |
| Q36213869 | Phosphatidic acid mediates the targeting of tBid to induce lysosomal membrane permeabilization and apoptosis |
| Q38921056 | Physiological and Pharmacological Control of BAK, BAX, and Beyond |
| Q39384356 | Pore formation by dimeric Bak and Bax: an unusual pore? |
| Q42183300 | Pores formed by Baxα5 relax to a smaller size and keep at equilibrium |
| Q30388868 | Pro-apoptotic Bax molecules densely populate the edges of membrane pores |
| Q41955014 | Proapoptotic Bax and Bak proteins form stable protein-permeable pores of tunable size |
| Q41975085 | Reconstitution of proapoptotic BAK function in liposomes reveals a dual role for mitochondrial lipids in the BAK-driven membrane permeabilization process |
| Q38268983 | Stepwise activation of BAX and BAK by tBID, BIM, and PUMA initiates mitochondrial apoptosis. |
| Q37773789 | Still embedded together binding to membranes regulates Bcl-2 protein interactions. |
| Q36150746 | The Dynamics of Bax Channel Formation: Influence of Ionic Strength |
| Q38668186 | The deadly landscape of pro-apoptotic BCL-2 proteins in the outer mitochondrial membrane |
| Q38331196 | The mystery of BCL2 family: Bcl-2 proteins and apoptosis: an update. |
| Q34038716 | The rheostat in the membrane: BCL-2 family proteins and apoptosis |
| Q38039293 | The secrets of the Bcl-2 family |
| Q37774394 | There is more to life than revascularization: therapeutic targeting of myocardial ischemia/reperfusion injury |
| Q36985116 | Three-dimensional structure of Bax-mediated pores in membrane bilayers |
| Q91304407 | Topology of active, membrane-embedded Bax in the context of a toroidal pore |
| Q34960771 | Visual and functional demonstration of growing Bax-induced pores in mitochondrial outer membranes |
| Q36629743 | Where Killers Meet--Permeabilization of the Outer Mitochondrial Membrane during Apoptosis |
| Q40757128 | cBid, Bax and Bcl-xL exhibit opposite membrane remodeling activities. |
Search more.