| scholarly article | Q13442814 |
| P356 | DOI | 10.1016/J.EXPNEUROL.2009.02.007 |
| P8608 | Fatcat ID | release_2xh4tmlwxngnna6yaagax34dii |
| P932 | PMC publication ID | 2710407 |
| P698 | PubMed publication ID | 19236863 |
| P5875 | ResearchGate publication ID | 24034579 |
| P2093 | author name string | Nickolay Brustovetsky | |
| Tatiana Brustovetsky | |||
| Viacheslav Li | |||
| P2860 | cites work | A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding | Q25938984 |
| Increased susceptibility of striatal mitochondria to calcium-induced permeability transition | Q28183063 | ||
| Calcium-induced cytochrome c release from CNS mitochondria is associated with the permeability transition and rupture of the outer membrane | Q28208002 | ||
| Mitochondrial permeability transition in acute neurodegeneration | Q28218571 | ||
| Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death | Q28242641 | ||
| Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death | Q28242653 | ||
| Intracellular calcium levels during the period of delayed excitotoxicity | Q28248689 | ||
| Delayed increase of Ca2+ influx elicited by glutamate: role in neuronal death | Q28257073 | ||
| Calcineurin is a common target of cyclophilin-cyclosporin A and FKBP-FK506 complexes | Q28277749 | ||
| Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure | Q28289036 | ||
| Mitochondrial dysfunction is a primary event in glutamate neurotoxicity | Q28290602 | ||
| Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia | Q28585666 | ||
| Regulation of cell death: the calcium-apoptosis link | Q29615554 | ||
| Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart. | Q31552541 | ||
| Critical role of calpain-mediated cleavage of calcineurin in excitotoxic neurodegeneration. | Q33195171 | ||
| Mitochondrial transport of cations: channels, exchangers, and permeability transition. | Q33744633 | ||
| Mitochondria and neuronal survival | Q33807218 | ||
| Mitochondrial membrane potential and the permeability transition in excitotoxicity | Q33836193 | ||
| Continuous infusion of cyclosporin A postinjury significantly ameliorates cortical damage following traumatic brain injury | Q33891081 | ||
| Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function | Q34296896 | ||
| Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D. | Q34406704 | ||
| Glutamate neurotoxicity and diseases of the nervous system | Q34560997 | ||
| Cyclophilin D inactivation protects axons in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis | Q35762075 | ||
| Glutamate-induced deregulation of calcium homeostasis and mitochondrial dysfunction in mammalian central neurones | Q35853368 | ||
| Mitochondrial permeability transition in CNS trauma: cause or effect of neuronal cell death? | Q35968596 | ||
| The mitochondrial permeability transition pore and its involvement in cell death and in disease pathogenesis | Q36733673 | ||
| Cyclosporin A delays mitochondrial depolarization induced by N-methyl-D-aspartate in cortical neurons: evidence of the mitochondrial permeability transition | Q36834803 | ||
| Calcium-induced precipitate formation in brain mitochondria: composition, calcium capacity, and retention | Q36935277 | ||
| Lithium desensitizes brain mitochondria to calcium, antagonizes permeability transition, and diminishes cytochrome C release | Q39272817 | ||
| Glutamate-induced mitochondrial depolarisation and perturbation of calcium homeostasis in cultured rat hippocampal neurones | Q39515826 | ||
| Mitochondria are morphologically and functionally heterogeneous within cells. | Q39646958 | ||
| Molecular Mechanisms of Immunosuppression by Cyclosporins | Q40489674 | ||
| The permeability transition pore as a mitochondrial calcium release channel: a critical appraisal | Q41456661 | ||
| Calcineurin regulation of synaptic function: from ion channels to transmitter release and gene transcription | Q41468953 | ||
| Glutamate-induced calcium transient triggers delayed calcium overload and neurotoxicity in rat hippocampal neurons | Q42453073 | ||
| Inhibition of glutamate-induced delayed calcium deregulation by 2-APB and La3+ in cultured cortical neurones | Q42468259 | ||
| Regulation of the intracellular free calcium concentration in single rat dorsal root ganglion neurones in vitro | Q42481777 | ||
| Inability to restore resting intracellular calcium levels as an early indicator of delayed neuronal cell death | Q42513068 | ||
| Dominant role of mitochondria in clearance of large Ca2+ loads from rat adrenal chromaffin cells. | Q42514013 | ||
| Sequestration of glutamate-induced Ca2+ loads by mitochondria in cultured rat hippocampal neurons | Q42526106 | ||
| Regulation of the permeability transition pore in skeletal muscle mitochondria. Modulation By electron flow through the respiratory chain complex i. | Q42677114 | ||
| Inhibition by cyclosporin A of a Ca2+-dependent pore in heart mitochondria activated by inorganic phosphate and oxidative stress | Q42818406 | ||
| Intracellular calcium concentrations during "chemical hypoxia" and excitotoxic neuronal injury | Q43414054 | ||
| Calcium and pyridine nucleotide interaction in mitochondrial membranes | Q43507689 | ||
| A transient inhibition of mitochondrial ATP synthesis by nitric oxide synthase activation triggered apoptosis in primary cortical neurons. | Q43572927 | ||
| Effects of dantrolene on extracellular glutamate concentration and neuronal death in the rat hippocampal CA1 region subjected to transient ischemia | Q43901888 | ||
| Mitochondrial permeability transition and calcium dynamics in striatal neurons upon intense NMDA receptor activation | Q43924738 | ||
| The relationship between free and total calcium concentrations in the matrix of liver and brain mitochondria. | Q44378787 | ||
| Glucose deprivation produces a prolonged increase in sensitivity to glutamate in cultured rat cortical neurons | Q44614371 | ||
| Cyclosporin A limits calcium-induced axonal damage following traumatic brain injury | Q44859730 | ||
| Excitotoxic calcium overload in a subpopulation of mitochondria triggers delayed death in hippocampal neurons. | Q44939763 | ||
| Dearth of glutamate transporters contributes to striatal excitotoxicity | Q45068369 | ||
| Cleavage of the plasma membrane Na+/Ca2+ exchanger in excitotoxicity | Q45244063 | ||
| Determination of amino acids associated with cerebral ischaemia in rat brain microdialysates using narrowbore liquid chromatography and fluorescence detection | Q46452335 | ||
| Excitotoxic injury to mitochondria isolated from cultured neurons. | Q46525122 | ||
| Calpain-mediated mGluR1alpha truncation: a key step in excitotoxicity | Q46769581 | ||
| Measurements of mitochondrial pH in cultured cortical neurons clarify contribution of mitochondrial pore to the mechanism of glutamate-induced delayed Ca2+ deregulation | Q46882934 | ||
| Attenuation of acute mitochondrial dysfunction after traumatic brain injury in mice by NIM811, a non-immunosuppressive cyclosporin A analog | Q46894153 | ||
| Neuroexcitatory amino acids and their relation to infarct size and neurological deficit in ischemic stroke | Q47331087 | ||
| Cyclosporin A significantly ameliorates cortical damage following experimental traumatic brain injury in rodents | Q48091139 | ||
| Cyclosporin A and its nonimmunosuppressive analogue N-Me-Val-4-cyclosporin A mitigate glucose/oxygen deprivation-induced damage to rat cultured hippocampal neurons | Q48098658 | ||
| High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition. | Q48107838 | ||
| Protective effects of NIM811 in transient focal cerebral ischemia suggest involvement of the mitochondrial permeability transition. | Q48159910 | ||
| Cell-permeant Ca2+ chelators reduce early excitotoxic and ischemic neuronal injury in vitro and in vivo. | Q48244590 | ||
| Glutamate receptor-induced 45Ca2+ accumulation in cortical cell culture correlates with subsequent neuronal degeneration | Q48300242 | ||
| Secondary Ca2+ overload indicates early neuronal injury which precedes staining with viability indicators. | Q48305312 | ||
| Mitochondrial control of acute glutamate excitotoxicity in cultured cerebellar granule cells. | Q48329711 | ||
| Calcium-dependent spontaneously reversible remodeling of brain mitochondria | Q48390266 | ||
| Mitochondria and neuronal glutamate excitotoxicity | Q48406562 | ||
| Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis | Q48609930 | ||
| Mitochondria accumulate Ca2+ following intense glutamate stimulation of cultured rat forebrain neurones | Q48811571 | ||
| Mitochondria, calcium regulation, and acute glutamate excitotoxicity in cultured cerebellar granule cells | Q48856939 | ||
| Neurotoxic glutamate treatment of cultured cerebellar granule cells induces Ca2+ -dependent collapse of mitochondrial membrane potential and ultrastructural alterations of mitochondria | Q48939941 | ||
| Modulation of the mitochondrial cyclosporin A-sensitive permeability transition pore. I. Evidence for two separate Me2+ binding sites with opposing effects on the pore open probability. | Q51673314 | ||
| Inhibition of the mitochondrial permeability transition by cyclosporin A during long time frame experiments: relationship between pore opening and the activity of mitochondrial phospholipases. | Q53998526 | ||
| Cyclosporin A attenuates acute mitochondrial dysfunction following traumatic brain injury. | Q54066088 | ||
| Properties of neuroprotective cell-permeant Ca2+ chelators: effects on [Ca2+]i and glutamate neurotoxicity in vitro. | Q54194410 | ||
| Modulation of the mitochondrial permeability transition pore. Effect of protons and divalent cations | Q67749578 | ||
| Cyclosporin A-sensitive and insensitive mechanisms produce the permeability transition in mitochondria | Q69717470 | ||
| An obligate role for oxygen in the early stages of glutamate-induced, delayed neuronal death | Q71511512 | ||
| Progression of ischaemic stroke and excitotoxic aminoacids | Q71975978 | ||
| Source specificity of early calcium neurotoxicity in cultured embryonic spinal neurons | Q72216851 | ||
| Glutamate-induced destabilization of intracellular calcium concentration homeostasis in cultured cerebellar granule cells: role of mitochondria in calcium buffering | Q72478477 | ||
| Limitations of cyclosporin A inhibition of the permeability transition in CNS mitochondria | Q73168250 | ||
| Pretreatment with the cyclosporin derivative, NIM811, improves the function of synaptic mitochondria following spinal cord contusion in rats | Q80170100 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | excitotoxicity | Q901117 |
| P304 | page(s) | 171-182 | |
| P577 | publication date | 2009-02-21 | |
| P1433 | published in | Experimental Neurology | Q5421127 |
| P1476 | title | Role of cyclophilin D-dependent mitochondrial permeability transition in glutamate-induced calcium deregulation and excitotoxic neuronal death | |
| P478 | volume | 218 |
| Q28830612 | A mitochondrial division inhibitor, Mdivi-1, inhibits mitochondrial fragmentation and attenuates kainic acid-induced hippocampal cell death |
| Q35245867 | Activation of the mitochondrial permeability transition pore modulates Ca2+ responses to physiological stimuli in adult neurons |
| Q36455346 | An approach to experimental synaptic pathology using green fluorescent protein-transgenic mice and gene knockout mice to show mitochondrial permeability transition pore-driven excitotoxicity in interneurons and motoneurons |
| Q38915418 | Bax regulates neuronal Ca2+ homeostasis. |
| Q36372727 | Calcium-dependent mitochondrial function and dysfunction in neurons |
| Q37591990 | Caloric restriction increases brain mitochondrial calcium retention capacity and protects against excitotoxicity |
| Q39339387 | Calpain activation and Na+/Ca2+ exchanger degradation occur downstream of calcium deregulation in hippocampal neurons exposed to excitotoxic glutamate |
| Q37635778 | Collapsin response mediator protein 2 (CRMP2) interacts with N-methyl-D-aspartate (NMDA) receptor and Na+/Ca2+ exchanger and regulates their functional activity |
| Q36018983 | Comparative impact of voltage-gated calcium channels and NMDA receptors on mitochondria-mediated neuronal injury |
| Q42105310 | Delayed calcium dysregulation in neurons requires both the NMDA receptor and the reverse Na+/Ca2+ exchanger |
| Q57021971 | Deletion of mitochondrial calcium uniporter incompletely inhibits calcium uptake and induction of the permeability transition pore in brain mitochondria |
| Q30493042 | Differential NMDA receptor-dependent calcium loading and mitochondrial dysfunction in CA1 vs. CA3 hippocampal neurons |
| Q33906624 | ER-mitochondrial calcium flow underlies vulnerability of mechanosensory hair cells to damage |
| Q58803405 | Excitatory Dendritic Mitochondrial Calcium Toxicity: Implications for Parkinson's and Other Neurodegenerative Diseases |
| Q36188246 | Ifenprodil, a NR2B-selective antagonist of NMDA receptor, inhibits reverse Na+/Ca2+ exchanger in neurons |
| Q35786606 | Investigating the biology of alpha herpesviruses with MS-based proteomics |
| Q41539954 | KB-R7943, an inhibitor of the reverse Na+ /Ca2+ exchanger, blocks N-methyl-D-aspartate receptor and inhibits mitochondrial complex I. |
| Q48221540 | Mitochondrial ATP-Mg/Pi carrier SCaMC-3/Slc25a23 counteracts PARP-1-dependent fall in mitochondrial ATP caused by excitotoxic insults in neurons |
| Q38244718 | Mitochondrial bioenergetics and neuronal survival modelled in primary neuronal culture and isolated nerve terminals |
| Q35566016 | Mitochondrial calcium and its regulation in neurodegeneration induced by oxidative stress |
| Q57170869 | Mitochondrial permeability transition pore: a potential drug target for neurodegeneration |
| Q87906376 | Neuronal Cell Death |
| Q37530288 | Oxidative metabolism and Ca2+ handling in isolated brain mitochondria and striatal neurons from R6/2 mice, a model of Huntington's disease |
| Q38094017 | Physiologic functions of cyclophilin D and the mitochondrial permeability transition pore. |
| Q34003451 | The beneficial effect of melatonin in brain endothelial cells against oxygen-glucose deprivation followed by reperfusion-induced injury |
| Q36807516 | The proteome of Toll-like receptor 3-stimulated human immortalized fibroblasts: implications for susceptibility to herpes simplex virus encephalitis |
| Q33664175 | Unlocking the Door to Neuronal Woes in Alzheimer's Disease: Aβ and Mitochondrial Permeability Transition Pore |
| Q36242512 | Wnt Signaling Prevents the Aβ Oligomer-Induced Mitochondrial Permeability Transition Pore Opening Preserving Mitochondrial Structure in Hippocampal Neurons |
| Q64076328 | injection protects cerebral microvascular endothelial cells against oxygen-glucose deprivation reperfusion |
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