scholarly article | Q13442814 |
P2093 | author name string | Ekihiro Seki | |
Hisataka Moriwaki | |||
Atsushi Suetsugu | |||
Yosuke Osawa | |||
Masahito Nagaki | |||
Hiroyasu Ito | |||
Mitsuru Seishima | |||
Masayuki Adachi | |||
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Roles of AKT and sphingosine kinase in the antiapoptotic effects of bile duct ligation in mouse liver | Q46827700 | ||
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Severe impairment in early host defense against Listeria monocytogenes in mice deficient in acid sphingomyelinase | Q50105214 | ||
SMAD and p38 MAPK signaling pathways independently regulate alpha1(I) collagen gene expression in unstimulated and transforming growth factor-beta-stimulated hepatic stellate cells. | Q50781080 | ||
Acid sphingomyelinase inhibition suppresses lipopolysaccharide-mediated release of inflammatory cytokines from macrophages and protects against disease pathology in dextran sulphate sodium-induced colitis in mice. | Q51020334 | ||
Altered Kupffer cell function in biliary obstruction. | Q51841407 | ||
Kupffer Cell-Expressed Membrane-Bound TNF Mediates Melphalan Hepatotoxicity via Activation of Both TNF Receptors | Q58858794 | ||
Expression of the NF-kappa B target gene X-ray-inducible immediate early response factor-1 short enhances TNF-alpha-induced hepatocyte apoptosis by inhibiting Akt activation | Q73229802 | ||
Inflammation in response to hepatocellular apoptosis | Q77823653 | ||
Kupffer cell engulfment of apoptotic bodies stimulates death ligand and cytokine expression | Q79209033 | ||
Systemic mediators induce fibrogenic effects in normal liver after partial bile duct ligation | Q79219306 | ||
Mannose-conjugated alendronate selectively depletes Kupffer cells and inhibits endotoxemic shock in the mice | Q79860542 | ||
Akt-mediated liver growth promotes induction of cyclin E through a novel translational mechanism and a p21-mediated cell cycle arrest | Q80367693 | ||
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TLR4 enhances TGF-beta signaling and hepatic fibrosis | Q29620016 | ||
Defective TNF-alpha-mediated hepatocellular apoptosis and liver damage in acidic sphingomyelinase knockout mice | Q30881683 | ||
Bile acid-induced rat hepatocyte apoptosis is inhibited by antioxidants and blockers of the mitochondrial permeability transition | Q32075959 | ||
Kupffer cells abrogate cholestatic liver injury in mice | Q33236130 | ||
Roles for C16-ceramide and sphingosine 1-phosphate in regulating hepatocyte apoptosis in response to tumor necrosis factor-alpha | Q34425224 | ||
Bile acid regulation of hepatic physiology: IV. Bile acids and death receptors | Q35102867 | ||
Ceramide-enriched membrane domains | Q36286893 | ||
"Let there be bile"--understanding hepatic injury in cholestasis | Q36526816 | ||
Role of the Kupffer cell in mediating hepatic toxicity and carcinogenesis | Q36661322 | ||
Common pathogenic mechanism in development progression of liver injury caused by non-alcoholic or alcoholic steatohepatitis | Q37058328 | ||
Inflammatory pathways in liver homeostasis and liver injury | Q37207091 | ||
Fas enhances fibrogenesis in the bile duct ligated mouse: a link between apoptosis and fibrosis. | Q39632325 | ||
Functions of ceramide in coordinating cellular responses to stress | Q41243489 | ||
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Protective role of Kupffer cells in acetaminophen-induced hepatic injury in mice | Q44249123 | ||
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P433 | issue | 1 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 237-245 | |
P577 | publication date | 2010-01-01 | |
P1433 | published in | Hepatology | Q15724398 |
P1476 | title | Role of acid sphingomyelinase of Kupffer cells in cholestatic liver injury in mice | |
P478 | volume | 51 |
Q34086900 | Acidic sphingomyelinase controls hepatic stellate cell activation and in vivo liver fibrogenesis |
Q55110755 | Cellular Mechanisms of Hepatoprotection Mediated by M2-Like Macrophages. |
Q36517945 | Contrasting responses of Kupffer cells and inflammatory mononuclear phagocytes to biliary obstruction in a mouse model of cholestatic liver injury |
Q89497645 | Crosstalk Between Acid Sphingomyelinase and Inflammasome Signaling and Their Emerging Roles in Tissue Injury and Fibrosis |
Q100517651 | Hepatic macrophages in liver homeostasis and diseases-diversity, plasticity and therapeutic opportunities |
Q36541005 | Inhibition of Cyclic Adenosine Monophosphate (cAMP)-response Element-binding Protein (CREB)-binding Protein (CBP)/β-Catenin Reduces Liver Fibrosis in Mice. |
Q46331739 | Isofraxidin, a coumarin component improves high-fat diet induced hepatic lipid homeostasis disorder and macrophage inflammation in mice |
Q35235774 | Kupffer cells influence parenchymal invasion and phenotypic orientation, but not the proliferation, of liver progenitor cells in a murine model of liver injury |
Q35266698 | L-tryptophan-mediated enhancement of susceptibility to nonalcoholic fatty liver disease is dependent on the mammalian target of rapamycin |
Q34796252 | Lithocholic acid disrupts phospholipid and sphingolipid homeostasis leading to cholestasis in mice. |
Q64946007 | Liver Macrophages: Old Dogmas and New Insights. |
Q36580354 | Liver acid sphingomyelinase inhibits growth of metastatic colon cancer |
Q33740461 | M2-like Kupffer cells in fibrotic liver may protect against acute insult |
Q41620915 | M2-like macrophages in the fibrotic liver protect mice against lethal insults through conferring apoptosis resistance to hepatocytes |
Q39367594 | Mechanisms of bile acid mediated inflammation in the liver |
Q38906942 | Pathogenesis of Kupffer Cells in Cholestatic Liver Injury |
Q91897717 | Phenotypic switch of human and mouse macrophages and resultant effects on apoptosis resistance in hepatocytes |
Q64111310 | Role of macrophages in experimental liver injury and repair in mice |
Q34766246 | Tumor necrosis factor-α promotes cholestasis-induced liver fibrosis in the mouse through tissue inhibitor of metalloproteinase-1 production in hepatic stellate cells |
Q52605838 | Tumor necrosis factor-α-mediated hepatocyte apoptosis stimulates fibrosis in the steatotic liver in mice. |
Q57478274 | Wnt/β-Catenin Signaling as a Potential Target for the Treatment of Liver Cirrhosis Using Antifibrotic Drugs |
Q36442343 | β-Arrestin 2 Promotes Hepatocyte Apoptosis by Inhibiting Akt Protein |
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