| P2093 | author name string | Jeesun Kim | |
| | Alexander M Kleschevnikov | |
| | William C Mobley | |
| | Y Eugene Yu | |
| | Jessica Yu | |
| | Zheng Zeng | |
| | Larisa V Lysenko | |
| P2860 | cites work | Upregulation of GABA neurotransmission suppresses hippocampal excitability and prevents long-term potentiation in transgenic superoxide dismutase-overexpressing mice | Q73263708 |
| | Implicit and explicit memory: a functional dissociation in persons with Down syndrome | Q73457679 |
| | Involvement of GIRK2 in postnatal development of the weaver cerebellum | Q73599762 |
| | Chromosomic diagnosis of mongolism | Q78820342 |
| | Working memory and Down syndrome | Q81571734 |
| | Clinical manifestations of the deletion of Down syndrome critical region including DYRK1A and KCNJ6 | Q83120929 |
| | Symptoms of attention-deficit/hyperactivity disorder in Down syndrome: effects of the dopamine receptor D4 gene | Q86433319 |
| | Down Syndrome and Attention-Deficit/Hyperactivity Disorder (ADHD) | Q22241820 |
| | On the promise of pharmacotherapies targeted at cognitive and neurodegenerative components of Down syndrome | Q22241936 |
| | Molecular and cellular characterization of the Down syndrome critical region protein 2 | Q24292695 |
| | Normal cerebellar development but susceptibility to seizures in mice lacking G protein-coupled, inwardly rectifying K+ channel GIRK2 | Q24672204 |
| | Mouse models of Down syndrome as a tool to unravel the causes of mental disabilities | Q26863005 |
| | Neurological phenotypes for Down syndrome across the life span | Q27025698 |
| | Down Syndrome Cognitive Phenotypes Modeled in Mice Trisomic for All HSA 21 Homologues | Q27300997 |
| | A potassium channel mutation in weaver mice implicates membrane excitability in granule cell differentiation | Q28115658 |
| | Trisomy for synaptojanin1 in Down syndrome is functionally linked to the enlargement of early endosomes | Q28118988 |
| | Subunit interactions in the assembly of neuronal Kir3.0 inwardly rectifying K+ channels | Q28245514 |
| | G protein regulation of potassium ion channels | Q28292042 |
| | Heteromultimerization of G-protein-gated inwardly rectifying K+ channel proteins GIRK1 and GIRK2 and their altered expression in weaver brain | Q28297400 |
| | Working memory in the Ts65Dn mouse, a model for Down syndrome. | Q51985856 |
| | Long-term memory in mental retardation: evidence for a specific impairment in subjects with Down's syndrome. | Q52005572 |
| | A mouse model for Down syndrome exhibits learning and behaviour deficits. | Q52013436 |
| | Reversible impairment of long-term potentiation in transgenic Cu/Zn-SOD mice. | Q52037580 |
| | DYRK1A (dual-specificity tyrosine-phosphorylated and -regulated kinase 1A): a gene with dosage effect during development and neurogenesis. | Q53581383 |
| | Keppen-Lubinsky syndrome: Expanding the phenotype. | Q55052070 |
| | Monoacylglycerol lipase inhibitor JZL184 improves behavior and neural properties in Ts65Dn mice, a model of down syndrome | Q28542515 |
| | Comprehensive behavioral phenotyping of Ts65Dn mouse model of Down syndrome: activation of β1-adrenergic receptor by xamoterol as a potential cognitive enhancer | Q30459302 |
| | The use of mouse models to understand and improve cognitive deficits in Down syndrome | Q30474023 |
| | Effects of individual segmental trisomies of human chromosome 21 syntenic regions on hippocampal long-term potentiation and cognitive behaviors in mice. | Q30497997 |
| | Dyrk1A haploinsufficiency affects viability and causes developmental delay and abnormal brain morphology in mice | Q30531707 |
| | Genetic dissection of the Down syndrome critical region | Q30670043 |
| | Understanding the basis for Down syndrome phenotypes | Q33238943 |
| | Gene expression signature of cerebellar hypoplasia in a mouse model of Down syndrome during postnatal development | Q33424071 |
| | A chromosome 21 critical region does not cause specific Down syndrome phenotypes. | Q33605833 |
| | Behavioral characterization of a mouse model overexpressing DSCR1/ RCAN1. | Q33833885 |
| | G-protein mediated gating of inward-rectifier K+ channels | Q33918594 |
| | An aneuploid mouse strain carrying human chromosome 21 with Down syndrome phenotypes | Q33990273 |
| | Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome | Q34010790 |
| | Segmental trisomy of murine chromosome 16: a new model system for studying Down syndrome | Q34176485 |
| | Down's syndrome | Q34191091 |
| | Critical role of the D21S55 region on chromosome 21 in the pathogenesis of Down syndrome | Q34294835 |
| | Keppen-Lubinsky syndrome is caused by mutations in the inwardly rectifying K+ channel encoded by KCNJ6. | Q34459609 |
| | Contribution of GIRK2-mediated postsynaptic signaling to opiate and alpha 2-adrenergic analgesia and analgesic sex differences | Q34467801 |
| | Inhibition of G protein-activated inwardly rectifying K+ channels by fluoxetine (Prozac). | Q35045125 |
| | Specific targeting of the GABA-A receptor α5 subtype by a selective inverse agonist restores cognitive deficits in Down syndrome mice | Q35177041 |
| | Down's syndrome: a genetic disorder in biobehavioral perspective | Q35204303 |
| | Increased efficiency of the GABAA and GABAB receptor-mediated neurotransmission in the Ts65Dn mouse model of Down syndrome | Q35675287 |
| | Trisomy of the G protein-coupled K+ channel gene, Kcnj6, affects reward mechanisms, cognitive functions, and synaptic plasticity in mice | Q35787216 |
| | My close encounter with GABA(B) receptors | Q35903187 |
| | Working memory in the aged Ts65Dn mouse, a model for Down syndrome | Q36004329 |
| | Deficits in cognition and synaptic plasticity in a mouse model of Down syndrome ameliorated by GABAB receptor antagonists. | Q36138353 |
| | Localization of a retroviral element within the rd gene coding for the beta subunit of cGMP phosphodiesterase | Q36209453 |
| | Practitioner review: short-term and working memory impairments in neurodevelopmental disorders: diagnosis and remedial support | Q36365489 |
| | Beta-amyloid, oxidative stress and down syndrome | Q36683240 |
| | A transcript map of the Down syndrome critical region on chromosome 21. | Q36715199 |
| | An Anti-β-Amyloid Vaccine for Treating Cognitive Deficits in a Mouse Model of Down Syndrome | Q36739111 |
| | Psychiatric and behavioral disorders in persons with Down syndrome | Q36957680 |
| | Aging in Down Syndrome and the Development of Alzheimer's Disease Neuropathology | Q37102001 |
| | The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome | Q37361341 |
| | Size does not always matter: Ts65Dn Down syndrome mice show cerebellum-dependent motor learning deficits that cannot be rescued by postnatal SAG treatment | Q37381873 |
| | Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalities | Q37394402 |
| | A new mouse model for the trisomy of the Abcg1-U2af1 region reveals the complexity of the combinatorial genetic code of down syndrome | Q37423613 |
| | Developmental regulation of G protein-gated inwardly-rectifying K+ (GIRK/Kir3) channel subunits in the brain | Q37606833 |
| | GABAB-GIRK2-mediated signaling in Down syndrome. | Q37775217 |
| | Molecular mapping of the Down syndrome phenotype | Q37862656 |
| | Human and mouse model cognitive phenotypes in Down syndrome: implications for assessment. | Q38006085 |
| | Discoveries in Down syndrome: moving basic science to clinical care. | Q38006088 |
| | Treating enhanced GABAergic inhibition in Down syndrome: use of GABA α5-selective inverse agonists. | Q38177515 |
| | Clinical Assessment of Cognitive Decline in Adults with Down Syndrome | Q38590507 |
| | Dentate gyrus mediates cognitive function in the Ts65Dn/DnJ mouse model of Down syndrome | Q39294662 |
| | GABAergic hyperinnervation of dentate granule cells in the Ts65Dn mouse model of down syndrome: Exploring the role of App. | Q39324113 |
| | Inhibition of serotonin reuptake | Q39624473 |
| | Growth of children with Down's syndrome: birth to age 3 years | Q39856812 |
| | Functional Implications of Gene Dosage Effects in Trisomy 21 | Q40105588 |
| | Short-term memory in Down syndrome: applying the working memory model | Q40676242 |
| | Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: a model of Down syndrome | Q41978368 |
| | Hippocampal long-term potentiation suppressed by increased inhibition in the Ts65Dn mouse, a genetic model of Down syndrome. | Q42467654 |
| | Increased App expression in a mouse model of Down's syndrome disrupts NGF transport and causes cholinergic neuron degeneration. | Q42497675 |
| | GIRK2 deficient mice. Evidence for hyperactivity and reduced anxiety | Q42659945 |
| | Early pharmacotherapy restores neurogenesis and cognitive performance in the Ts65Dn mouse model for Down syndrome | Q42997861 |
| | Restoration of norepinephrine-modulated contextual memory in a mouse model of Down syndrome. | Q43109273 |
| | Evaluation of the impact of abdominal obesity on glucose and lipid metabolism disorders in adults with Down syndrome. | Q44918752 |
| | Pharmacotherapy for cognitive impairment in a mouse model of Down syndrome. | Q46020053 |
| | G protein-coupled inwardly rectifying K+ channels (GIRKs) mediate postsynaptic but not presynaptic transmitter actions in hippocampal neurons | Q46524326 |
| | Down syndrome: genes, model systems, and progress towards pharmacotherapies and clinical trials for cognitive deficits. | Q46531220 |
| | Normalization of Dyrk1A expression by AAV2/1-shDyrk1A attenuates hippocampal-dependent defects in the Ts65Dn mouse model of Down syndrome | Q46683684 |
| | Down syndrome mouse models Ts65Dn, Ts1Cje, and Ms1Cje/Ts65Dn exhibit variable severity of cerebellar phenotypes. | Q47367835 |
| | Mouse models of Down syndrome: how useful can they be? Comparison of the gene content of human chromosome 21 with orthologous mouse genomic regions | Q47415659 |
| | Chromosome 21 KIR channels in brain development. | Q47807172 |
| | Engineering DYRK1A overdosage yields Down syndrome-characteristic cortical splicing aberrations. | Q48141056 |
| | Thigmotaxis as an index of anxiety in mice. Influence of dopaminergic transmissions | Q48153919 |
| | Inhibitory modulation of long-term potentiation via the 5-HT1A receptor in slices of the rat hippocampal dentate gyrus | Q48267615 |
| | Reversing excitatory GABAAR signaling restores synaptic plasticity and memory in a mouse model of Down syndrome. | Q48278664 |
| | Down syndrome: the brain in trisomic mode | Q48287894 |
| | Frontal cortex BDNF levels correlate with working memory in an animal model of Down syndrome | Q48362366 |
| | Ts65Dn, a mouse model of Down syndrome, exhibits increased GABAB-induced potassium current | Q48375391 |
| | Object recognition in rats and mice: a one-trial non-matching-to-sample learning task to study 'recognition memory'. | Q48406875 |
| | Impaired spatial working and reference memory in segmental trisomy (Ts65Dn) mice | Q48498793 |
| | Molecular mapping of Alzheimer-type dementia in Down's syndrome | Q48512105 |
| | An immunocytochemical study on the distribution of two G-protein-gated inward rectifier potassium channels (GIRK2 and GIRK4) in the adult rat brain | Q48631496 |
| | Impaired short- and long-term memory in Ts65Dn mice, a model for Down syndrome | Q48718482 |
| | Excitation/inhibition balance and learning are modified by Dyrk1a gene dosage. | Q50472132 |
| | Working memory and cognitive skills in individuals with Down syndrome. | Q51939030 |
| P4510 | describes a project that uses | ImageJ | Q1659584 |
| P921 | main subject | synaptic plasticity | Q1551556 |
| P304 | page(s) | 1-10 | |
| P577 | publication date | 2017-03-22 | |
| P1433 | published in | Neurobiology of Disease | Q15716606 |
| P1476 | title | Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome | |
| P478 | volume | 103 | |