| scholarly article | Q13442814 |
| P50 | author | Andrea M Harrington | Q43057655 |
| Stuart M. Brierley | Q54251000 | ||
| Joel Castro | Q87190869 | ||
| Luke Grundy | Q87312979 | ||
| P2093 | author name string | Sonia Garcia-Caraballo | |
| Ashlee Caldwell | |||
| Andelain Erickson | |||
| Annemie Deiteren | |||
| P2860 | cites work | Molecular cloning and functional expression of the human sodium channel β1B subunit, a novel splicing variant of the β1 subunit | Q24299811 |
| Structure and functional expression of a new member of the tetrodotoxin-sensitive voltage-activated sodium channel family from human neuroendocrine cells | Q24314768 | ||
| Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain | Q24562888 | ||
| The neural control of micturition | Q24619130 | ||
| A multicentre open-label safety and efficacy study of tetrodotoxin for cancer pain | Q24623970 | ||
| Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons | Q24632552 | ||
| Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia | Q24675844 | ||
| Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8. | Q24814946 | ||
| Tetrodotoxin, an Extremely Potent Marine Neurotoxin: Distribution, Toxicity, Origin and Therapeutical Uses | Q26780441 | ||
| SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes | Q28118877 | ||
| NaN/Nav1.9: a sodium channel with unique properties | Q28215683 | ||
| Veratrum poisoning | Q28262283 | ||
| Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7 | Q28270611 | ||
| An SCN9A channelopathy causes congenital inability to experience pain | Q28278844 | ||
| International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels | Q28289125 | ||
| A de novo gain-of-function mutation in SCN11A causes loss of pain perception | Q28298515 | ||
| Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain | Q28542818 | ||
| RNA-Seq Analysis of Human Trigeminal and Dorsal Root Ganglia with a Focus on Chemoreceptors | Q28548290 | ||
| Colonic inflammation up‐regulates voltage‐gated sodium channels in bladder sensory neurons via activation of peripheral transient potential vanilloid 1 receptors | Q41968217 | ||
| Near-Perfect Synaptic Integration by Nav1.7 in Hypothalamic Neurons Regulates Body Weight | Q42057677 | ||
| The voltage-gated sodium channel Nav1.9 is required for inflammation-based urinary bladder dysfunction | Q42447844 | ||
| Regulation of voltage-gated sodium current by endogenous Src family kinases in cochlear spiral ganglion neurons in culture | Q42501501 | ||
| The phenotype of congenital insensitivity to pain due to the NaV1.9 variant p.L811P. | Q42611687 | ||
| Expression and localization of the Nav1.9 sodium channel in enteric neurons and in trigeminal sensory endings: implication for intestinal reflex function and orofacial pain | Q42614719 | ||
| Loss-of-function mutations in the Nav1.7 gene underlie congenital indifference to pain in multiple human populations | Q42618792 | ||
| Inhibition of NF-kappaB prevents mechanical allodynia induced by spinal ventral root transection and suppresses the re-expression of Nav1.3 in DRG neurons in vivo and in vitro. | Q42880746 | ||
| Enterochromaffin Cells Are Gut Chemosensors that Couple to Sensory Neural Pathways. | Q43666461 | ||
| The involvement of the tetrodotoxin-resistant sodium channel Na(v)1.8 (PN3/SNS) in a rat model of visceral pain. | Q43770582 | ||
| Experimental ulcers alter voltage-sensitive sodium currents in rat gastric sensory neurons | Q43879348 | ||
| Mild gastritis alters voltage-sensitive sodium currents in gastric sensory neurons in rats | Q43903212 | ||
| TNBS ileitis evokes hyperexcitability and changes in ionic membrane properties of nociceptive DRG neurons | Q43997191 | ||
| Characterization of a voltage-dependent Na(+) current in human esophageal smooth muscle | Q44133762 | ||
| Deficits in visceral pain and referred hyperalgesia in Nav1.8 (SNS/PN3)-null mice. | Q44156142 | ||
| TNFalpha is a key mediator of the pronociceptive effects of mucosal supernatant from human ulcerative colitis on colonic DRG neurons | Q44369078 | ||
| Sodium current in human intestinal interstitial cells of Cajal | Q44534176 | ||
| Ileitis modulates potassium and sodium currents in guinea pig dorsal root ganglia sensory neurons | Q44552166 | ||
| Gastric ulcers reduce A-type potassium currents in rat gastric sensory ganglion neurons | Q44606413 | ||
| Specialized functions of Nav1.5 and Nav1.9 channels in electrogenesis of myenteric neurons in intact mouse ganglia | Q44694803 | ||
| Two TTX-resistant Na+ currents in mouse colonic dorsal root ganglia neurons and their role in colitis-induced hyperexcitability | Q44942537 | ||
| Colonic inflammation increases Na+ currents in bladder sensory neurons | Q45166805 | ||
| Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy | Q45771999 | ||
| Neonatal maternal deprivation sensitizes voltage-gated sodium channel currents in colon-specific dorsal root ganglion neurons in rats | Q45772760 | ||
| Prostaglandin E(2) modulates TTX-R I(Na) in rat colonic sensory neurons | Q39403566 | ||
| Mechanosensitive Piezo Channels in the Gastrointestinal Tract | Q39448475 | ||
| Decreased Nav1.9 channel expression in Hirschsprung's disease | Q39686559 | ||
| Comparative study of the distribution of the alpha-subunits of voltage-gated sodium channels in normal and axotomized rat dorsal root ganglion neurons | Q39963179 | ||
| Distribution of the voltage-gated sodium channel Na(v)1.7 in the rat: expression in the autonomic and endocrine systems. | Q40089012 | ||
| Expression of the voltage-gated sodium channel NaV1.5 in the macrophage late endosome regulates endosomal acidification | Q40124031 | ||
| Localization of the type VI voltage-gated sodium channel protein in human CNS. | Q40908703 | ||
| Effects of Tetrodotoxin in Mouse Models of Visceral Pain. | Q41045838 | ||
| α-Conotoxin Vc1.1 inhibits human dorsal root ganglion neuroexcitability and mouse colonic nociception via GABAB receptors. | Q41160258 | ||
| The Domain II S4-S5 Linker in Nav1.9: A Missense Mutation Enhances Activation, Impairs Fast Inactivation, and Produces Human Painful Neuropathy. | Q41190621 | ||
| Sodium channel NaV1.9 mutations associated with insensitivity to pain dampen neuronal excitability | Q41661778 | ||
| Expression of Nav1.7 in DRG Neurons Extends from Peripheral Terminals in the Skin to Central Preterminal Branches and Terminals in the Dorsal Horn | Q41901986 | ||
| Selective spider toxins reveal a role for the Nav1.1 channel in mechanical pain | Q28594552 | ||
| Loss-of-Function of the Voltage-Gated Sodium Channel NaV1.5 (Channelopathies) in Patients With Irritable Bowel Syndrome | Q29544671 | ||
| Proteomics. Tissue-based map of the human proteome | Q29617248 | ||
| Experimental and computational evidence for an essential role of NaV1.6 in spike initiation at stretch-sensitive colorectal afferent endings | Q30646640 | ||
| Sodium current in human jejunal circular smooth muscle cells | Q30793397 | ||
| Multiple sodium channel isoforms mediate the pathological effects of Pacific ciguatoxin-1. | Q30839530 | ||
| SCN5A is expressed in human jejunal circular smooth muscle cells | Q30858816 | ||
| Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor | Q33390819 | ||
| Trinitrobenzenesulphonic acid colitis alters Na 1.8 channel expression in mouse dorsal root ganglia neurons | Q33411961 | ||
| Differential regulation of three sodium channel messenger RNAs in the rat central nervous system during development. | Q33608468 | ||
| Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia | Q33679483 | ||
| Tetrodotoxin for Moderate to Severe Cancer-Related Pain: A Multicentre, Randomized, Double-Blind, Placebo-Controlled, Parallel-Design Trial | Q33710457 | ||
| The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways | Q33871067 | ||
| Differential distribution of the tetrodotoxin-sensitive rPN4/NaCh6/Scn8a sodium channel in the nervous system | Q33895014 | ||
| Regulation of muscle sodium channel transcripts during development and in response to denervation | Q33960052 | ||
| Significant determinants of mouse pain behaviour. | Q34015630 | ||
| Contribution of Na(v)1.8 sodium channels to action potential electrogenesis in DRG neurons | Q34086296 | ||
| Voltage-gated sodium channels at 60: structure, function and pathophysiology | Q34265707 | ||
| Age-related gene expression analysis in enteric ganglia of human colon after laser microdissection | Q34342021 | ||
| Linaclotide inhibits colonic nociceptors and relieves abdominal pain via guanylate cyclase-C and extracellular cyclic guanosine 3',5'-monophosphate | Q34365070 | ||
| Multiple roles for NaV1.9 in the activation of visceral afferents by noxious inflammatory, mechanical, and human disease-derived stimuli | Q34451600 | ||
| First evidence of neosaxitoxin as a long-acting pain blocker in bladder pain syndrome | Q34457294 | ||
| Ion channelopathies in functional GI disorders | Q34537076 | ||
| Identification of the visceral pain pathway activated by noxious colorectal distension in mice. | Q34613124 | ||
| A-803467, a potent and selective Nav1.8 sodium channel blocker, attenuates neuropathic and inflammatory pain in the rat. | Q34625901 | ||
| Are Voltage-Gated Sodium Channels on the Dorsal Root Ganglion Involved in the Development of Neuropathic Pain? | Q34639336 | ||
| Neuroanatomy of visceral nociception: vagal and splanchnic afferent | Q34699772 | ||
| Colitis decreases mechanosensitive K2P channel expression and function in mouse colon sensory neurons | Q35087098 | ||
| Visceral pain: the ins and outs, the ups and downs | Q35738047 | ||
| Gastric hyperalgesia and changes in voltage gated sodium channel function in the rat | Q35790415 | ||
| Do the urinary bladder and large bowel interact, in sickness or in health?: ICI‐RS 2011 | Q35842916 | ||
| RGS proteins as targets in the treatment of intestinal inflammation and visceral pain: New insights and future perspectives | Q35906057 | ||
| Ranolazine inhibits voltage-gated mechanosensitive sodium channels in human colon circular smooth muscle cells. | Q36066445 | ||
| NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy | Q36229128 | ||
| Sodium channel NaV1.3 is important for enterochromaffin cell excitability and serotonin release | Q45866966 | ||
| Voltage-gated potassium channels in IB4-positive colonic sensory neurons mediate visceral hypersensitivity in the rat. | Q45985050 | ||
| Post-inflammatory colonic afferent sensitisation: different subtypes, different pathways and different time courses | Q46072922 | ||
| Activation of neurokinin 3 receptor increases Na(v)1.9 current in enteric neurons | Q46127921 | ||
| The Economic Costs of Pain in the United States | Q46218658 | ||
| Genetic effects on gene expression across human tissues | Q46285732 | ||
| Expression and Role of Voltage-Gated Sodium Channels in Human Dorsal Root Ganglion Neurons with Special Focus on Nav1.7, Species Differences, and Regulation by Paclitaxel | Q46380475 | ||
| Afferent hypersensitivity in a mouse model of post-inflammatory gut dysfunction: role of altered serotonin metabolism | Q46463734 | ||
| GTP up-regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9. | Q46838525 | ||
| Pathogenesis of abdominal pain in bowel obstruction: role of mechanical stress-induced upregulation of nerve growth factor in gut smooth muscle cells | Q47342967 | ||
| Activation of colo-rectal high-threshold afferent nerves by Interleukin-2 is tetrodotoxin-sensitive and upregulated in a mouse model of chronic visceral hypersensitivity. | Q47639141 | ||
| Two sodium channels contribute to the TTX-R sodium current in primary sensory neurons | Q47972903 | ||
| Na(v)1.7 and Na(v)1.3 are the only tetrodotoxin-sensitive sodium channels expressed by the adult guinea pig enteric nervous system | Q48090925 | ||
| Tetrodotoxin-resistant sodium channels Na(v)1.8/SNS and Na(v)1.9/NaN in afferent neurons innervating urinary bladder in control and spinal cord injured rats | Q48397699 | ||
| Immunolocalization of NaV1.2 channel subtypes in rat and cat brain and spinal cord with high affinity antibodies | Q48481503 | ||
| Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice | Q48680747 | ||
| Antinociceptive effects of tetrodotoxin (TTX) in rodents | Q48702148 | ||
| Intrarectal lidocaine is an effective treatment for abdominal pain associated with diarrhea-predominant irritable bowel syndrome | Q48797699 | ||
| Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs | Q48826387 | ||
| Nav1.1 is predominantly expressed in nodes of Ranvier and axon initial segments | Q49092166 | ||
| Peptide therapeutics from venom: Current status and potential. | Q49498134 | ||
| Citrobacter rodentium colitis evokes post-infectious hyperexcitability of mouse nociceptive colonic dorsal root ganglion neurons | Q50452510 | ||
| Sensitization of sodium channels by cystathionine β-synthetase activation in colon sensory neurons in adult rats with neonatal maternal deprivation | Q50852161 | ||
| Species dependent expression of intestinal smooth muscle mechanosensitive sodium channels | Q51081524 | ||
| Selective expression of a persistent tetrodotoxin-resistant Na+ current and NaV1.9 subunit in myenteric sensory neurons. | Q51300821 | ||
| Plasticity of Na+ Channels in Afferent Neurones Innervating Rat Urinary Bladder Following Spinal Cord Injury | Q51535275 | ||
| Ion channel gene expression in human lung, skin, and cord blood-derived mast cells | Q51676070 | ||
| Enhanced Excitability and Down-Regulated Voltage-Gated Potassium Channels in Colonic DRG Neurons from Neonatal Maternal Separation Rats | Q54389227 | ||
| Irritable bowel syndrome | Q56965677 | ||
| Sprouting of colonic afferent central terminals and increased spinal mitogen-activated protein kinase expression in a mouse model of chronic visceral hypersensitivity | Q57778656 | ||
| Selenoether oxytocin analogues have analgesic properties in a mouse model of chronic abdominal pain | Q58284514 | ||
| Lack of colonic inflammation-induced acute visceral hypersensitivity to colorectal distension in Nav1.9 knockout mice | Q60413857 | ||
| Splanchnic and pelvic mechanosensory afferents signal different qualities of colonic stimuli in mice | Q60730314 | ||
| Voltage-gated ion channel Nav1.7 innervation in patients with idiopathic rectal hypersensitivity and paroxysmal extreme pain disorder (familial rectal pain) | Q60731290 | ||
| Differential subcellular localization of the RI and RII Na+ channel subtypes in central neurons | Q69366003 | ||
| Kv7.2-7.5 voltage-gated potassium channel (KCNQ2-5) opener, retigabine, reduces capsaicin-induced visceral pain in mice | Q79447473 | ||
| The human macrophage sodium channel NaV1.5 regulates mycobacteria processing through organelle polarization and localized calcium oscillations | Q82548101 | ||
| Neonatal colonic inflammation sensitizes voltage-gated Na+channels via upregulation of cystathionine β-synthetase expression in rat primary sensory neurons | Q86225608 | ||
| Post-translational modifications of voltage-gated sodium channels in chronic pain syndromes | Q36249467 | ||
| A Novel Approach to High-Quality Postmortem Tissue Procurement: The GTEx Project | Q36362149 | ||
| Expression and function of the Scn5a-encoded voltage-gated sodium channel NaV 1.5 in the rat jejunum | Q36395633 | ||
| Colonic Hypersensitivity and Sensitization of Voltage-gated Sodium Channels in Primary Sensory Neurons in Rats with Diabetes | Q36426070 | ||
| Inflammatory mediators increase Nav1.9 current and excitability in nociceptors through a coincident detection mechanism | Q36471326 | ||
| Mechanism of sodium channel NaV1.9 potentiation by G-protein signaling | Q36568513 | ||
| P2Y Receptors Sensitize Mouse and Human Colonic Nociceptors | Q36610295 | ||
| Infrequent SCN9A mutations in congenital insensitivity to pain and erythromelalgia. | Q36674682 | ||
| The Nav1.9 channel regulates colonic motility in mice | Q36763929 | ||
| Sodium channel auxiliary subunits | Q36857924 | ||
| Type III sodium channel mRNA is expressed in embryonic but not adult spinal sensory neurons, and is reexpressed following axotomy | Q37019966 | ||
| Voltage sensor interaction site for selective small molecule inhibitors of voltage-gated sodium channels | Q37031862 | ||
| Voltage-gated sodium channel blockers as immunomodulators | Q37067359 | ||
| Sodium channel mutation in irritable bowel syndrome: evidence for an ion channelopathy | Q37100287 | ||
| Gain-of-function mutations in SCN11A cause familial episodic pain | Q37298152 | ||
| Ranolazine for chronic stable angina | Q37302094 | ||
| Potassium channels in peripheral pain pathways: expression, function and therapeutic potential | Q37362041 | ||
| Molecular basis of mechanosensitivity. | Q37580561 | ||
| Understanding and targeting centrally mediated visceral pain in inflammatory bowel disease | Q37617546 | ||
| Emerging tropical diseases in Australia. Part 2. Ciguatera fish poisoning | Q37811112 | ||
| Voltage-gated sodium channels: biophysics, pharmacology, and related channelopathies. | Q38026557 | ||
| Noncanonical roles of voltage-gated sodium channels | Q38153876 | ||
| Painful and painless channelopathies. | Q38210568 | ||
| Neuroplasticity and dysfunction after gastrointestinal inflammation | Q38227089 | ||
| No gain, no pain: NaV1.7 as an analgesic target | Q38238866 | ||
| Sodium channel genes in pain-related disorders: phenotype-genotype associations and recommendations for clinical use. | Q38260278 | ||
| Polymorphism in the SCN9A voltage-gated sodium channel gene associated with interstitial cystitis/bladder pain syndrome. | Q38400156 | ||
| NaV1.9: a sodium channel linked to human pain | Q38560993 | ||
| The voltage-gated sodium channel NaV 1.9 in visceral pain | Q38606237 | ||
| G Protein-Coupled Receptors: Dynamic Machines for Signaling Pain and Itch. | Q38641412 | ||
| Altered Ion Channel/Receptor Expression and Function in Extrinsic Sensory Neurons: The Cause of and Solution to Chronic Visceral Pain? | Q38886410 | ||
| Visceral and somatic pain modalities reveal NaV 1.7-independent visceral nociceptive pathways | Q39010857 | ||
| Breaking barriers to novel analgesic drug development | Q39360773 | ||
| P433 | issue | 5 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | sensory perception of pain | Q2734305 |
| P304 | page(s) | 785-807 | |
| P577 | publication date | 2018-02-06 | |
| P1433 | published in | Journal of Physiology | Q7743612 |
| P1476 | title | Voltage-gated sodium channels: (NaV )igating the field to determine their contribution to visceral nociception | |
| P478 | volume | 596 |
| Q60958347 | Mechanisms Underlying Overactive Bladder and Interstitial Cystitis/Painful Bladder Syndrome |
| Q89013977 | NaV1.1 inhibition can reduce visceral hypersensitivity |
| Q60046816 | Spinal Afferent Innervation of the Colon and Rectum |
| Q97520175 | Voltage-Gated K+/Na+ Channels and Scorpion Venom Toxins in Cancer |
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