human | Q5 |
P496 | ORCID iD | 0000-0003-1639-5003 |
P69 | educated at | University of British Columbia | Q391028 |
P108 | employer | University of British Columbia | Q391028 |
Peter MacCallum Cancer Centre | Q7175569 | ||
P734 | family name | Anglesio | Q99905808 |
Anglesio | Q99905808 | ||
Anglesio | Q99905808 | ||
P735 | given name | Michael | Q4927524 |
Michael | Q4927524 | ||
P106 | occupation | researcher | Q1650915 |
P21 | sex or gender | male | Q6581097 |
Q93025301 | A combination of the immunohistochemical markers CK7 and SATB2 is highly sensitive and specific for distinguishing primary ovarian mucinous tumors from colorectal and appendiceal metastases |
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Q35100056 | A functional proteogenomic analysis of endometrioid and clear cell carcinomas using reverse phase protein array and mutation analysis: protein expression is histotype-specific and loss of ARID1A/BAF250a is associated with AKT phosphorylation |
Q24610484 | ARID1A mutations in endometriosis-associated ovarian carcinomas |
Q38137231 | Accelerating type-specific ovarian carcinoma research: Calculator for Ovarian Subtype Prediction (COSP) is a reliable high-throughput tool for case review |
Q34233421 | Akt-mediated YB-1 phosphorylation activates translation of silent mRNA species |
Q53383644 | Are there any more ovarian tumor suppressor genes? A new perspective using ultra high-resolution copy number and loss of heterozygosity analysis. |
Q46748797 | Benign serous ovarian tumour: a redefining moment? |
Q53109067 | Cancer-associated somatic DICER1 hotspot mutations cause defective miRNA processing and reverse-strand expression bias to predominantly mature 3p strands through loss of 5p strand cleavage. |
Q37833199 | Clear cell carcinoma of the ovary: a report from the first Ovarian Clear Cell Symposium, June 24th, 2010. |
Q57287243 | Copy Number Aberrations in Benign Serous Ovarian Tumors: A Case for Reclassification? |
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Q42954255 | Correction: Type-Specific Cell Line Models for Type-Specific Ovarian Cancer Research. |
Q46756204 | Correction: Type-Specific Cell Line Models for Type-Specific Ovarian Cancer Research. |
Q39321636 | Cytokine gene expression signature in ovarian clear cell carcinoma. |
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Q96590073 | Development and validation of the gene-expression Predictor of high-grade-serous Ovarian carcinoma molecular subTYPE (PrOTYPE) |
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Q47610274 | Extrauterine high-grade serous carcinomas with bilateral adnexal involvement as the only two disease sites are clonal based on tp53 sequencing results: implications for biology, classification, and staging |
Q50848684 | Genomic classification of serous ovarian cancer with adjacent borderline differentiates RAS pathway and TP53-mutant tumors and identifies NRAS as an oncogenic driver |
Q40230451 | Genomic consequences of aberrant DNA repair mechanisms stratify ovarian cancer histotypes |
Q38205584 | Harnessing the potential of lipid-based nanomedicines for type-specific ovarian cancer treatments. |
Q33551425 | Hormone-receptor expression and ovarian cancer survival: an Ovarian Tumor Tissue Analysis consortium study |
Q39587868 | IL6-STAT3-HIF signaling and therapeutic response to the angiogenesis inhibitor sunitinib in ovarian clear cell cancer |
Q59102270 | Interfaces of Malignant and Immunologic Clonal Dynamics in Ovarian Cancer |
Q27851868 | LRP1B deletion in high-grade serous ovarian cancers is associated with acquired chemotherapy resistance to liposomal doxorubicin |
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Q36545843 | Molecular profiling of low grade serous ovarian tumours identifies novel candidate driver genes |
Q54289649 | Multifocal endometriotic lesions associated with cancer are clonal and carry a high mutation burden. |
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Q97533173 | Pathogenesis of bowel endometriosis |
Q37768784 | Profiling the cancer genome. |
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Q98158747 | Refined cut-off for TP53 immunohistochemistry improves prediction of TP53 mutation status in ovarian mucinous tumors: implications for outcome analyses |
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