| review article | Q7318358 |
| scholarly article | Q13442814 |
| P356 | DOI | 10.1016/S0027-5107(98)00209-7 |
| P698 | PubMed publication ID | 9852996 |
| P2093 | author name string | Stark AA | |
| P2860 | cites work | DNA polymerase delta is required for human mismatch repair in vitro | Q24313002 |
| The age distribution of cancer and a multi-stage theory of carcinogenesis | Q24564904 | ||
| A variant of DNA polymerase beta acts as a dominant negative mutant | Q24657672 | ||
| A genetic model for colorectal tumorigenesis | Q27860582 | ||
| The origin of mutants | Q28288915 | ||
| Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer | Q28294774 | ||
| MSH2 deficient mice are viable and susceptible to lymphoid tumours | Q28508486 | ||
| DHFR/MSH3 amplification in methotrexate-resistant cells alters the hMutSalpha/hMutSbeta ratio and reduces the efficiency of base-base mismatch repair | Q28610848 | ||
| cAMP-dependent SOS induction and mutagenesis in resting bacterial populations. | Q33719467 | ||
| Adaptive mutations in Escherichia coli as a model for the multiple mutational origins of tumors | Q34247386 | ||
| Preferential formation of benzo[a]pyrene adducts at lung cancer mutational hotspots in P53. | Q34399850 | ||
| The mutation rate and cancer | Q34410886 | ||
| Dominant negative mutator mutations in the mutS gene of Escherichia coli | Q34726651 | ||
| Mutator phenotypes in human colorectal carcinoma cell lines | Q35568305 | ||
| Proliferation of mutators in A cell population | Q35618973 | ||
| Selective pressures and ras activation in carcinogenesis | Q35703977 | ||
| How many mutations are required for tumorigenesis? Implications from human cancer data | Q36782475 | ||
| Carcinogen-induced mutations in the mouse c-Ha-ras gene provide evidence of multiple pathways for tumor progression | Q37671272 | ||
| Cell proliferation in carcinogenesis | Q37951208 | ||
| Dominant negative mutator mutations in the mutL gene of Escherichia coli | Q39716760 | ||
| Potential sources of multiple mutations in human cancers | Q40370562 | ||
| Hereditary Nonpolyposis Colorectal Cancer: the Syndrome, the Genes, and Historical Perspectives | Q40482404 | ||
| Multiple mutations in human cancers | Q41002717 | ||
| Conditional mutator phenotypes in hMSH2-deficient tumor cell lines | Q41091724 | ||
| DNA repair and colorectal cancer | Q41264767 | ||
| Cancer cells exhibit a mutator phenotype | Q41617067 | ||
| Comparison of ras activation during epidermal carcinogenesis in vitro and in vivo | Q41661819 | ||
| Application of quantitative stereology to the evaluation of phenotypically heterogeneous enzyme-altered foci in the rat liver | Q41890748 | ||
| Spontaneous Ha-ras gene activation in cultured primary murine keratinocytes: consequences of Ha-ras gene activation in malignant conversion and malignant progression | Q42800513 | ||
| Activation of ras oncogenes in chemically transformed BALB/MK-2 mouse keratinocytes | Q42800920 | ||
| Genomic instability in repeated sequences is an early somatic event in colorectal tumorigenesis that persists after transformation | Q46049171 | ||
| Multicopy single-stranded DNA of Escherichia coli enhances mutation and recombination frequencies by titrating MutS protein | Q50140322 | ||
| Role of mutator alleles in adaptive evolution. | Q54564129 | ||
| The Escherichia coli galK2 papillation assay: its specificity and application to seven newly isolated mutator strains. | Q54650910 | ||
| Defects in mismatch repair occur afterAPC mutations in the pathogenesis of sporadic colorectal tumours | Q57337208 | ||
| Somatic mutations in the hMSH2 gene in microsatellite unstable colorectal carcinomas | Q70972741 | ||
| Multicopy single-stranded DNAs with mismatched base pairs are mutagenic in Escherichia coli | Q72618648 | ||
| Genomic instability occurs in colorectal carcinomas but not in adenomas | Q72649470 | ||
| Observations on the formation of clones containing araB-lacZ cistron fusions | Q72815889 | ||
| Microsatellite instability in colorectal adenomas | Q73309470 | ||
| Sequential development of an angiogenic phenotype by human fibroblasts progressing to tumorigenicity | Q73309958 | ||
| Transient expression of a mutator phenotype in cancer cells | Q73713948 | ||
| Expression of hMSH2 and hMLH1 in colorectal carcinomas with microsatellite instability | Q74445140 | ||
| Mismatch repair deficiency leads to a unique mode of colorectal tumorigenesis characterized by intratumoral heterogeneity | Q74452713 | ||
| Microsatellite instability in human solid tumors | Q74546889 | ||
| P433 | issue | 2 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | microsatellite | Q265193 |
| carcinogenesis | Q1637543 | ||
| P304 | page(s) | 221-225 | |
| P577 | publication date | 1998-11-01 | |
| P1433 | published in | Mutation Research | Q6943732 |
| P1476 | title | Transient appearance of the mutator phenotype during carcinogenesis as a possible explanation for the lack of mini/microsatellite instability in many late stage tumors. | |
| P478 | volume | 421 |
| Q34781991 | DNA instability and human disease | cites work | P2860 |
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