| scholarly article | Q13442814 |
| P2093 | author name string | Ying Wu | |
| Xiaojing Wang | |||
| Luiz Belardinelli | |||
| Antao Luo | |||
| Jihua Ma | |||
| John C Shryock | |||
| Peihua Zhang | |||
| Chen Fu | |||
| Leilei Wang | |||
| Yejia Song | |||
| Zhenzhen Cao | |||
| P2860 | cites work | Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sites | Q24315218 |
| Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart | Q24323983 | ||
| Chronic intermittent hypoxia alters Ca2+ handling in rat cardiomyocytes by augmented Na+/Ca2+ exchange and ryanodine receptor activities in ischemia-reperfusion | Q28565998 | ||
| Resveratrol attenuates the Na(+)-dependent intracellular Ca(2+) overload by inhibiting H(2)O(2)-induced increase in late sodium current in ventricular myocytes | Q34525246 | ||
| Myocardial Na,K-ATPase and digoxin therapy in human heart failure. | Q35134810 | ||
| Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channels | Q35157593 | ||
| Digitalis: new actions for an old drug | Q36284056 | ||
| Phosphorylation of S1505 in the cardiac Na+ channel inactivation gate is required for modulation by protein kinase C. | Q36435813 | ||
| Modulation of late sodium current by Ca2+, calmodulin, and CaMKII in normal and failing dog cardiomyocytes: similarities and differences | Q36645745 | ||
| Endogenous and exogenous cardiac glycosides: their roles in hypertension, salt metabolism, and cell growth | Q36817975 | ||
| Late sodium current is a new therapeutic target to improve contractility and rhythm in failing heart | Q36954069 | ||
| Different roles of the cardiac Na+/Ca2+-exchanger in ouabain-induced inotropy, cell signaling, and hypertrophy | Q37175543 | ||
| The arrhythmogenic consequences of increasing late INa in the cardiomyocyte | Q37345048 | ||
| Sophocarpine attenuates the Na(+)-dependent Ca2(+) overload induced by Anemonia sulcata toxin-increased late sodium current in rabbit ventricular myocytes | Q39240103 | ||
| Mechanism of action of therapeutic levels of cardiac glycosides | Q40099875 | ||
| Phosphorylation-dependent regulation of cardiac Na+/Ca2+ exchanger via protein kinase C. | Q41192745 | ||
| Relation of sodium pump inhibition to positive inotropy at low concentrations of ouabain in rat heart muscle | Q41279879 | ||
| Sarcolemmal cation channels and exchangers modify the increase in intracellular calcium in cardiomyocytes on inhibiting Na+-K+-ATPase | Q42508830 | ||
| Regulation of the Na+/Ca2+ exchanger (NCX) in the murine embryonic heart | Q42511800 | ||
| Intracellular Na(+) concentration is elevated in heart failure but Na/K pump function is unchanged | Q44009242 | ||
| Diverse toxicity associated with cardiac Na+/K+ pump inhibition: evaluation of electrophysiological mechanisms. | Q44329151 | ||
| Ouabain increases sarcoplasmic reticulum calcium release in cardiac myocytes | Q44666460 | ||
| Increased late sodium current in myocytes from a canine heart failure model and from failing human heart. | Q45284438 | ||
| Milrinone inhibits hypoxia or hydrogen dioxide-induced persistent sodium current in ventricular myocytes. | Q45956540 | ||
| Ranolazine attenuates hypoxia- and hydrogen peroxide-induced increases in sodium channel late openings in ventricular myocytes | Q46809196 | ||
| Ranolazine attenuates the enhanced reverse Na⁺-Ca²⁺ exchange current via inhibiting hypoxia-increased late sodium current in ventricular myocytes | Q47907146 | ||
| Late sodium current inhibition alone with ranolazine is sufficient to reduce ischemia- and cardiac glycoside-induced calcium overload and contractile dysfunction mediated by reverse-mode sodium/calcium exchange | Q48139486 | ||
| Persistent sodium current and Na+/H+ exchange contributes to the augmentation of the reverse Na+/Ca2+ exchange during hypoxia or acute ischemia in ventricular myocytes | Q48196534 | ||
| Reducing the late sodium current improves cardiac function during sodium pump inhibition by ouabain | Q48286098 | ||
| Functional effects of protein kinase C activation on the human cardiac Na+ channel | Q48793689 | ||
| Repolarization abnormalities in cardiomyocytes of dogs with chronic heart failure: role of sustained inward current. | Q51461497 | ||
| Ionic mechanism of the effects of hydrogen peroxide in rat ventricular myocytes. | Q51543526 | ||
| Modulation of Na+ current inactivation by stimulation of protein kinase C in cardiac cells. | Q53964904 | ||
| High-affinity ouabain binding site and low-dose positive inotropic effect in rat myocardium | Q59073915 | ||
| P433 | issue | 4 | |
| P304 | page(s) | 399-409 | |
| P577 | publication date | 2015-03-10 | |
| P1433 | published in | Experimental Physiology | Q1091760 |
| P1476 | title | Protein kinase C and Ca(2+) -calmodulin-dependent protein kinase II mediate the enlarged reverse INCX induced by ouabain-increased late sodium current in rabbit ventricular myocytes. | |
| P478 | volume | 100 |
| Q37427347 | A novel mechanism for the treatment of angina, arrhythmias, and diastolic dysfunction: inhibition of late I(Na) using ranolazine |
| Q58773641 | Cardiotoxicity of Uremic Toxins: A Driver of Cardiorenal Syndrome |
| Q37688345 | Fibroblast growth factor 23 dysregulates late sodium current and calcium homeostasis with enhanced arrhythmogenesis in pulmonary vein cardiomyocytes |
| Q33734918 | Icariin, a Novel Blocker of Sodium and Calcium Channels, Eliminates Early and Delayed Afterdepolarizations, As Well As Triggered Activity, in Rabbit Cardiomyocytes |
| Q98196750 | Inhibition of the INa/K and the activation of peak INa contribute to the arrhythmogenic effects of aconitine and mesaconitine in guinea pigs |
| Q42579527 | Late I(Na) in the Heart: Physiology, Pathology, and Pathways |
| Q50061975 | Late sodium current associated cardiac electrophysiological and mechanical dysfunction |
| Q38532814 | Late sodium current: A mechanism for angina, heart failure, and arrhythmia |
| Q47109377 | Lithium and Tamoxifen Modulate Behavior and Protein Kinase C Activity in the Animal Model of Mania Induced by Ouabain |
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