| human | Q5 |
| P496 | ORCID iD | 0000-0002-0822-1233 |
| P1053 | ResearcherID | I-8376-2017 |
| P1153 | Scopus author ID | 6603154937 |
| P69 | educated at | National Autonomous University of Mexico | Q222738 |
| UAM Iztapalapa | Q6156345 | ||
| P108 | employer | Instituto Nacional de Cardiología | Q5917485 |
| P735 | given name | Sara | Q833345 |
| Sara | Q833345 | ||
| P106 | occupation | researcher | Q1650915 |
| P21 | sex or gender | female | Q6581072 |
| Q44236484 | Anti-mitochondrial therapy in human breast cancer multi-cellular spheroids |
| Q39477031 | Assessment of the low inhibitory specificity of oxamate, aminooxyacetate and dichloroacetate on cancer energy metabolism. |
| Q38827395 | Biochemistry and Physiology of Heavy Metal Resistance and Accumulation in Euglena. |
| Q37660632 | Bioenergetic pathways in tumor mitochondria as targets for cancer therapy and the importance of the ROS-induced apoptotic trigger |
| Q38208982 | Canonical and new generation anticancer drugs also target energy metabolism. |
| Q40394981 | Cas IIgly induces apoptosis in glioma C6 cells in vitro and in vivo through caspase-dependent and caspase-independent mechanisms. |
| Q39393795 | Casiopeina II-gly and bromo-pyruvate inhibition of tumor hexokinase, glycolysis, and oxidative phosphorylation. |
| Q83035266 | Control of cellular proliferation by modulation of oxidative phosphorylation in human and rodent fast-growing tumor cells |
| Q90573112 | Control of the NADPH supply and GSH recycling for oxidative stress management in hepatoma and liver mitochondria |
| Q38663844 | Control of the NADPH supply for oxidative stress handling in cancer cells |
| Q28574290 | Copper compound induces autophagy and apoptosis of glioma cells by reactive oxygen species and JNK activation |
| Q83204784 | Determining and understanding the control of glycolysis in fast-growth tumor cells. Flux control by an over-expressed but strongly product-inhibited hexokinase |
| Q51587575 | Dual regulation of energy metabolism by p53 in human cervix and breast cancer cells. |
| Q102068497 | Editorial: Metabolic Plasticity of Cancer |
| Q88628274 | Energy Metabolism Drugs Block Triple Negative Breast Metastatic Cancer Cell Phenotype |
| Q28288377 | Energy metabolism in tumor cells |
| Q40014579 | Energy metabolism transition in multi-cellular human tumor spheroids. |
| Q81360359 | Enhanced alternative oxidase and antioxidant enzymes under Cd(2+) stress in Euglena |
| Q61663159 | Erratum to: Inhibitors of Succinate: Quinone Reductase/Complex II Regulate Production of Mitochondrial Reactive Oxygen Species and Protect Normal Cells from Ischemic Damage but Induce Specific Cancer Cell Death |
| Q34398855 | GPI/AMF inhibition blocks the development of the metastatic phenotype of mature multi-cellular tumor spheroids. |
| Q92774155 | Gamma-glutamylcysteine synthetase and tryparedoxin 1 exert high control on the antioxidant system in Trypanosoma cruzi contributing to drug resistance and infectivity |
| Q41444067 | Glycoprotein Ib activation by thrombin stimulates the energy metabolism in human platelets. |
| Q37581909 | HIF-1alpha modulates energy metabolism in cancer cells by inducing over-expression of specific glycolytic isoforms. |
| Q47852092 | HPI/AMF inhibition halts the development of the aggressive phenotype of breast cancer stem cells |
| Q92545210 | Heart myxoma develops oncogenic and metastatic phenotype |
| Q38358175 | Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death. |
| Q51451285 | Hypoglycemia Enhances Epithelial-Mesenchymal Transition and Invasiveness, and Restrains the Warburg Phenotype, in Hypoxic HeLa Cell Cultures and Microspheroids. |
| Q57472123 | Hypoxia increases chemoresistance in human medulloblastoma DAOY cells via hypoxia‑inducible factor 1α‑mediated downregulation of the CYP2B6, CYP3A4 and CYP3A5 enzymes and inhibition of cell proliferation |
| Q51109970 | Identification of a metabolic and canonical biomarker signature in Mexican HR+/HER2-, triple positive and triple-negative breast cancer patients. |
| Q37277390 | Inhibition of Non-flux-Controlling Enzymes Deters Cancer Glycolysis by Accumulation of Regulatory Metabolites of Controlling Steps |
| Q37921677 | Inhibitors of succinate: quinone reductase/Complex II regulate production of mitochondrial reactive oxygen species and protect normal cells from ischemic damage but induce specific cancer cell death. |
| Q97874964 | Kinetic modeling of glucose central metabolism in hepatocytes and hepatoma cells |
| Q34997405 | Kinetics of transport and phosphorylation of glucose in cancer cells. |
| Q42512906 | Metabolic changes induced by cold stress in rat liver mitochondria. |
| Q37769788 | Metabolic control analysis indicates a change of strategy in the treatment of cancer |
| Q37217309 | Metabolic control analysis: a tool for designing strategies to manipulate metabolic pathways. |
| Q38864224 | Mitochondrial free fatty acid β-oxidation supports oxidative phosphorylation and proliferation in cancer cells. |
| Q34536966 | Mitochondrial targeting of vitamin E succinate enhances its pro-apoptotic and anti-cancer activity via mitochondrial complex II |
| Q82624228 | Modeling cancer glycolysis |
| Q51080469 | Modeling cancer glycolysis under hypoglycemia, and the role played by the differential expression of glycolytic isoforms. |
| Q39342751 | Molecular mechanism for the selective impairment of cancer mitochondrial function by a mitochondrially targeted vitamin E analogue |
| Q37890322 | Multi-biomarker pattern for tumor identification and prognosis |
| Q43572428 | Multisite control of the Crabtree effect in ascites hepatoma cells. |
| Q57114649 | Mutant p53 downregulates oxidative phosphorylation and upregulates glycolysis under normoxia and hypoxia in human cervix cancer cells |
| Q104493275 | Non-Steroidal Anti-Inflammatory Drugs Increase Cisplatin, Paclitaxel, and Doxorubicin Efficacy against Human Cervix Cancer Cells |
| Q57881658 | On the mechanism by which 6-ketocholestanol protects mitochondria against uncoupling-induced Ca2+ efflux |
| Q61192773 | On the properties of calcium-induced permeability transition in neonatal heart mitochondria |
| Q37889310 | Oxidative phosphorylation as a target to arrest malignant neoplasias. |
| Q42960308 | Oxidative phosphorylation is impaired by prolonged hypoxia in breast and possibly in cervix carcinoma |
| Q73693547 | Oxidative phosphorylation supported by an alternative respiratory pathway in mitochondria from Euglena |
| Q64067211 | Oxidized ATM protein kinase is a new signal transduction player that regulates glycolysis in CAFs as well as tumor growth and metastasis |
| Q39418406 | Phosphofructokinase type 1 kinetics, isoform expression, and gene polymorphisms in cancer cells |
| Q93200202 | Physiological Role of Glutamate Dehydrogenase in Cancer Cells |
| Q79526020 | Phytochelatin-cadmium-sulfide high-molecular-mass complexes of Euglena gracilis |
| Q61192846 | Post-conditioning Preserves Glycolytic ATP During Early Reperfusion: A survival Mechanism for the Reperfused Heart |
| Q39233625 | Reactive oxygen species are generated by the respiratory complex II--evidence for lack of contribution of the reverse electron flow in complex I. |
| Q92181482 | Repurposing drugs as pro-oxidant redox modifiers to eliminate cancer stem cells and improve the treatment of advanced stage cancers |
| Q92411834 | Resveratrol inhibits cancer cell proliferation by impairing oxidative phosphorylation and inducing oxidative stress |
| Q35869389 | Role of mitochondrial permeability transition pores in mitochondrial autophagy. |
| Q28576438 | Roles of mitophagy and the mitochondrial permeability transition in remodeling of cultured rat hepatocytes |
| Q24658163 | Selective degradation of mitochondria by mitophagy |
| Q73466313 | Substrate oxidation and ATP supply in AS-30D hepatoma cells |
| Q46119169 | Suppression of tumor growth in vivo by the mitocan alpha-tocopheryl succinate requires respiratory complex II. |
| Q37362329 | Targeting of cancer energy metabolism. |
| Q37971488 | The Warburg Hypothesis and the ATP Supply In Cancer Cells Is Oxidative Phosphorylation impaired in malignant neoplasias? |
| Q37486411 | The bioenergetics of cancer: is glycolysis the main ATP supplier in all tumor cells? |
| Q37703526 | The causes of cancer revisited: "mitochondrial malignancy" and ROS-induced oncogenic transformation - why mitochondria are targets for cancer therapy |
| Q33556360 | Tracker dyes to probe mitochondrial autophagy (mitophagy) in rat hepatocytes |
| Q90633242 | Transcriptional Regulation of Energy Metabolism in Cancer Cells |
| Q38595074 | Understanding the cancer cell phenotype beyond the limitations of current omics analyses |
| Q38186917 | Who controls the ATP supply in cancer cells? Biochemistry lessons to understand cancer energy metabolism |
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