human | Q5 |
P496 | ORCID iD | 0000-0002-8608-1352 |
P1153 | Scopus author ID | 7005371132 |
P69 | educated at | University of Virginia | Q213439 |
University of Michigan | Q230492 | ||
Maulana Azad Medical College | Q6792427 | ||
Virginia Commonwealth University | Q2303536 | ||
Eastern Virginia Medical School | Q5330559 | ||
P108 | employer | University of Virginia | Q213439 |
P106 | occupation | researcher | Q1650915 |
P21 | sex or gender | male | Q6581097 |
Q30436689 | A combination of ketamine and diazepam synergistically controls refractory status epilepticus induced by cholinergic stimulation |
Q30433508 | A comparison of three NMDA receptor antagonists in the treatment of prolonged status epilepticus |
Q49049294 | A gain-of-function mutation in the sodium channel gene Scn2a results in seizures and behavioral abnormalities |
Q30423646 | A mouse monoclonal antibody against the γ2 subunit of GABAA receptors |
Q49598739 | A novel therapeutic approach for treatment of catamenial epilepsy |
Q30408313 | A potassium leak channel silences hyperactive neurons and ameliorates status epilepticus |
Q30311832 | A presynaptic action of the neurosteroid pregnenolone sulfate on GABAergic synaptic transmission |
Q30436721 | Activity-dependent scaling of GABAergic synapse strength is regulated by brain-derived neurotrophic factor |
Q33696608 | Acute cellular alterations in the hippocampus after status epilepticus |
Q30444544 | Alterations in GABA(A) receptor mediated inhibition in adjacent dorsal midline thalamic nuclei in a rat model of chronic limbic epilepsy |
Q48163314 | Are myotonia and epilepsy linked by a chloride channel? |
Q27323026 | CaV3.2 calcium channels control NMDA receptor-mediated transmission: a new mechanism for absence epilepsy |
Q30418423 | Calcium-permeable AMPA receptors are expressed in a rodent model of status epilepticus |
Q30432854 | Central cholinesterase inhibition enhances glutamatergic synaptic transmission |
Q99585979 | Characterization of kindled VGAT-Cre mice as a new animal model of temporal lobe epilepsy |
Q28387562 | Characterization of status epilepticus induced by two organophosphates in rats |
Q30433141 | Characterization of the convulsant action of pregnenolone sulfate |
Q30435443 | Cultured Hippocampal Pyramidal Neurons Express Two Kinds of GABAA Receptors |
Q48904867 | Cyclic AMP-dependent protein kinase enhances hippocampal dentate granule cell GABAA receptor currents |
Q30310881 | Design, synthesis, and evaluation of analogues of 3,3,3-trifluoro-2-hydroxy-2-phenyl-propionamide as orally available general anesthetics |
Q30438602 | Development of gamma-aminobutyric acidergic synapses in cultured hippocampal neurons |
Q28365693 | Diminished allopregnanolone enhancement of GABA(A) receptor currents in a rat model of chronic temporal lobe epilepsy |
Q30436043 | Diminished neurosteroid sensitivity of synaptic inhibition and altered location of the alpha4 subunit of GABA(A) receptors in an animal model of epilepsy |
Q42152043 | Disordered migration of interneurons within focal cortical dysplasia |
Q30435142 | Distribution of alpha1, alpha4, gamma2, and delta subunits of GABAA receptors in hippocampal granule cells |
Q40467738 | Dormant Basket Cell Hypothesis Revisited em leader Again. |
Q46061966 | Electroencephalography and behavior patterns during experimental status epilepticus |
Q42183955 | Emerging role of pannexins in seizures and status epilepticus |
Q30432450 | Endogenous neurosteroid synthesis modulates seizure frequency |
Q30455061 | Engineering the synchronization of neuron action potentials using global time-delayed feedback stimulation. |
Q30252723 | Enhanced AMPA receptor-mediated neurotransmission on CA1 pyramidal neurons during status epilepticus |
Q30460435 | Evidence for a chronic loss of inhibition in the hippocampus after kindling: biochemical studies |
Q30460437 | Evidence for a chronic loss of inhibition in the hippocampus after kindling: electrophysiological studies |
Q30461268 | Evidence that repetitive seizures in the hippocampus cause a lasting reduction of GABAergic inhibition |
Q28378739 | Experimental status epilepticus alters gamma-aminobutyric acid type A receptor function in CA1 pyramidal neurons |
Q30311893 | Factors underlying bursting behavior in a network of cultured hippocampal neurons exposed to zero magnesium |
Q47149739 | Flupirtine and diazepam combination terminates established status epilepticus: results in three rodent models |
Q48238554 | Functional GABAA receptor heterogeneity of acutely dissociated hippocampal CA1 pyramidal cells |
Q30444648 | GABA(A) receptor internalization during seizures |
Q30411079 | GABAA receptor membrane insertion rates are specified by their subunit composition |
Q30441927 | GABAergic synaptic inhibition is reduced before seizure onset in a genetic model of cortical malformation. |
Q24605690 | GABAergic transmission in temporal lobe epilepsy: the role of neurosteroids |
Q45057687 | Gaining perspective on SUDEP: The new guideline |
Q42120307 | Galanin receptors modulate seizures |
Q30438665 | High-affinity, slowly desensitizing GABAA receptors mediate tonic inhibition in hippocampal dentate granule cells |
Q30306126 | Hippocampal neurons express GABA A receptor insensitive to diazepam in hyperexcitable conditions |
Q42952938 | Homeostatic plasticity hypothesis and mechanisms of neocortical epilepsies |
Q30435045 | Homeostatic regulation of synaptic excitability: tonic GABA(A) receptor currents replace I(h) in cortical pyramidal neurons of HCN1 knock-out mice |
Q30425997 | Homeostatic strengthening of inhibitory synapses is mediated by the accumulation of GABA(A) receptors |
Q30442074 | Hydroxyamide analogs of propofol exhibit state-dependent block of sodium channels in hippocampal neurons: implications for anticonvulsant activity |
Q30436691 | Impact of receptor changes on treatment of status epilepticus |
Q30405327 | Impact of transient acute hypoxia on the developing mouse EEG |
Q30278632 | Increased excitability and excitatory synaptic transmission during in vitro ischemia in the neonatal mouse hippocampus |
Q30310798 | Increased neurosteroid sensitivity of hippocampal GABAA receptors during postnatal development |
Q43184549 | Is epilepsy a disease of synaptic transmission? |
Q42131268 | Is mesial temporal sclerosis a necessary component of temporal lobe epilepsy? |
Q41785303 | Is the tyrosine kinase B receptor a target for preventing epilepsy? |
Q45057008 | Isocitrate dehydrogenase mutations: A biomarker for glioma-related excitability and seizures |
Q27016115 | Issues related to development of antiepileptogenic therapies |
Q30306363 | Ketamine controls prolonged status epilepticus |
Q45086872 | Loss of GABAA receptors during partial status epilepticus |
Q30408402 | Loss of cholecystokinin-containing terminals in temporal lobe epilepsy |
Q30461271 | Loss of inhibition precedes delayed spontaneous seizures in the hippocampus after tetanic electrical stimulation |
Q39033129 | Lowered vaccine dose for immunisation of calves against tropical theileriosis using Theileria annulata lymphoblasts |
Q30455624 | M-type potassium channels modulate Schaffer collateral-CA1 glutamatergic synaptic transmission |
Q57137825 | Mechanisms of status epilepticus: α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor hypothesis |
Q30412220 | N-methyl-D-aspartic acid receptor activation downregulates expression of δ subunit-containing GABAA receptors in cultured hippocampal neurons |
Q30453456 | NMDA receptor activation mediates the loss of GABAergic inhibition induced by recurrent seizures |
Q100671001 | Neocortical injury-induced status epilepticus |
Q72502645 | Neuronal Circuit Activity during Neonatal Hypoxic–Ischemic Seizures in Mice |
Q56383791 | Neurosteroid regulation of GABA receptors: A role in catamenial epilepsy |
Q30252495 | Neurosteroid-sensitive δ-GABAA receptors: A role in epileptogenesis? |
Q30434186 | Nitric oxide alters GABAergic synaptic transmission in cultured hippocampal neurons |
Q112584981 | Patterns of benzodiazepine underdosing in the Established Status Epilepticus Treatment Trial |
Q48935817 | Pharmacological properties of gamma-aminobutyric acidA receptors from acutely dissociated rat dentate granule cells |
Q33748593 | Pharmacological properties of recombinant and hippocampal dentate granule cell GABAA receptors. |
Q48692919 | Photothrombotic brain infarction results in seizure activity in aging Fischer 344 and Sprague Dawley rats |
Q48213958 | Physiological properties of GABAA receptors from acutely dissociated rat dentate granule cells |
Q48273202 | Postnatal development of hippocampal dentate granule cell gamma-aminobutyric acidA receptor pharmacological properties. |
Q30434201 | Prehospital Treatment of Status Epilepticus with Benzodiazepines Is Effective and Safe |
Q92234228 | Progesterone receptor activation regulates seizure susceptibility |
Q71813630 | Psychogenic elaboration of simple partial seizures |
Q33943268 | Rapid seizure-induced reduction of benzodiazepine and Zn2+ sensitivity of hippocampal dentate granule cell GABAA receptors |
Q27015717 | Receptor trafficking hypothesis revisited: plasticity of AMPA receptors during established status epilepticus |
Q30430081 | Receptors with low affinity for neurosteroids and GABA contribute to tonic inhibition of granule cells in epileptic animals |
Q30456822 | Recurrent spontaneous hippocampal seizures in the rat as a chronic sequela to limbic status epilepticus |
Q91530287 | Reduced neurosteroid potentiation of GABAA receptors in epilepsy and depolarized hippocampal neurons |
Q30460158 | Reduction of paired pulse inhibition in the CA1 region of the hippocampus by pilocarpine in naive and in amygdala-kindled rats |
Q40467287 | Responsiveness of Status Epilepticus to Treatment with Diazepan Decreases Rapidly as Seizure Duration Increases |
Q30312056 | Role of GABA(A) receptor-mediated inhibition in the pathogenesis of generalized seizures |
Q63976868 | Role of NMDA receptors in the pathophysiology and treatment of status epilepticus |
Q40467674 | Role of Neuronal Loss in the Pathogenesis of Recurrent Spontaneous Seizures. |
Q33899437 | Role of brain-derived neurotrophic factor in catamenial epilepsy |
Q45835693 | STEM-14CELLULAR RESTING TRANSMEMBRANE POTENTIAL INSTRUCTS DIFFERENTIATION STATE OF GBM STEM-LIKE CELLS. |
Q30436695 | Selective loss of dentate hilar interneurons contributes to reduced synaptic inhibition of granule cells in an electrical stimulation-based animal model of temporal lobe epilepsy |
Q30434034 | Slow intracellular accumulation of GABA(A) receptor delta subunit is modulated by brain-derived neurotrophic factor |
Q40466621 | Sodium Channel Mutations in GEFS(+) Produce Persistent Inward Current. |
Q30412864 | Somatostatin type-2 receptor activation inhibits glutamate release and prevents status epilepticus |
Q30303948 | Status epilepticus in epileptogenesis |
Q30436046 | Status epilepticus increases the intracellular accumulation of GABAA receptors |
Q58103312 | Structure-based Intensity Propagation for 3D Brain Reconstruction with Multilayer Section Microscopy |
Q30435939 | Subunit-specific trafficking of GABA(A) receptors during status epilepticus |
Q30434530 | Synaptic and extrasynaptic localization of brain-derived neurotrophic factor and the tyrosine kinase B receptor in cultured hippocampal neurons |
Q30300915 | Synchronization of action potentials during low-magnesium-induced bursting |
Q30424160 | TASK Channel Deletion Reduces Sensitivity to Local Anesthetic-induced Seizures |
Q30471019 | Temporal lobe epilepsy: studies in a rat model showing dormancy of GABAergic inhibitory interneurons. |
Q30274991 | The SAMUKeppra study in prehospital status epilepticus: lessons for future study |
Q95269680 | The association of patient weight and dose of fosphenytoin, levetiracetam, and valproic acid with treatment success in status epilepticus |
Q30412553 | The established status epilepticus trial 2013. |
Q30448893 | The impact of diazepam's discovery on the treatment and understanding of status epilepticus |
Q33712138 | Value of inpatient diagnostic CCTV-EEG monitoring in the elderly. |
Q91054784 | α-Amino-3-Hydroxy-5-Methyl-4-Isoxazolepropionic Acid Receptor Plasticity Sustains Severe, Fatal Status Epilepticus |
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