The CRISPR/Cas system in Neisseria meningitidis affects bacterial adhesion to human nasopharyngeal epithelial cells

scientific article published on 30 July 2018

The CRISPR/Cas system in Neisseria meningitidis affects bacterial adhesion to human nasopharyngeal epithelial cells is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1080/15476286.2018.1486660
P8608Fatcat IDrelease_he4ynaj7svdmdf5yvbdnsfarj4
P932PMC publication ID6546440
P698PubMed publication ID30059276

P50authorNadja HeidrichQ52422890
P2093author name stringJörg Vogel
Christoph Schoen
Antony Hagmann
Saskia Bauriedl
P2860cites workComparative genome biology of a serogroup B carriage and disease strain supports a polygenic nature of meningococcal virulenceQ39667409
The number of Neisseria meningitidis type IV pili determines host cell interaction.Q42075334
A novel link between Campylobacter jejuni bacteriophage defence, virulence and Guillain-Barré syndrome.Q52422645
Alternative roles for CRISPR/Cas systems in bacterial pathogenesisQ21089594
Gene Expression Omnibus: NCBI gene expression and hybridization array data repositoryQ27860523
NeMeSys: a biological resource for narrowing the gap between sequence and function in the human pathogen Neisseria meningitidisQ28749761
Evolution and classification of the CRISPR-Cas systemsQ29616645
The role of porins in neisserial pathogenesis and immunity.Q30164877
Laminin receptor initiates bacterial contact with the blood brain barrier in experimental meningitis modelsQ30487909
Cell invasion by Neisseria meningitidis requires a functional interplay between the focal adhesion kinase, Src and cortactinQ31071346
The primary transcriptome of Neisseria meningitidis and its interaction with the RNA chaperone Hfq.Q33741128
Meningococcal pneumonia: characterization and review of cases seen over the past 25 yearsQ33808314
Virulence evolution of the human pathogen Neisseria meningitidis by recombination in the core and accessory genomeQ33889347
A CRISPR-Cas system enhances envelope integrity mediating antibiotic resistance and inflammasome evasionQ34002469
An atlas of Hfq-bound transcripts reveals 3' UTRs as a genomic reservoir of regulatory small RNAsQ34033315
CRISPR-Cas systems: beyond adaptive immunityQ34040445
CRISPR-Cas immunity in prokaryotesQ34045076
Interactions of pathogenic neisseriae with epithelial cell membranesQ34059736
A CRISPR/Cas system mediates bacterial innate immune evasion and virulenceQ34339102
Comparative Genome Sequencing Reveals Within-Host Genetic Changes in Neisseria meningitidis during Invasive DiseaseQ36247891
Diversity, classification and evolution of CRISPR-Cas systemsQ36399437
Conquering the meningococcusQ36711456
Processing-independent CRISPR RNAs limit natural transformation in Neisseria meningitidisQ36959817
Degeneration of a CRISPR/Cas system and its regulatory target during the evolution of a pathogenQ37399515
Pathogenic neisseriae: surface modulation, pathogenesis and infection controlQ37415301
Meningococcal interactions with the hostQ37501041
Pathogenesis of meningococcemiaQ38111646
Neisseria genomics: current status and future perspectives.Q38673627
A virulence-associated filamentous bacteriophage of Neisseria meningitidis increases host-cell colonisation.Q38680833
P433issue4
P921main subjectmolecular biologyQ7202
Neisseria meningitidisQ154625
CRISPRQ412563
P304page(s)390-396
P577publication date2018-07-30
P1433published inRNA BiologyQ7277167
P1476titleThe CRISPR/Cas system in Neisseria meningitidis affects bacterial adhesion to human nasopharyngeal epithelial cells
P478volume16

Reverse relations

cites work (P2860)
Q90265541A Simple Criterion for Inferring CRISPR Array Direction
Q90148152CRISPR-Cas: more than ten years and still full of mysteries
Q93186310Cas9 Contributes to Group B Streptococcal Colonization and Disease
Q92663522PgFur participates differentially in expression of virulence factors in more virulent A7436 and less virulent ATCC 33277 Porphyromonas gingivalis strains
Q90053629The CRISPR-Cas systems were selectively inactivated during evolution of Bacillus cereus group for adaptation to diverse environments

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