| scholarly article | Q13442814 |
| P6179 | Dimensions Publication ID | 1022108170 |
| P356 | DOI | 10.1007/S11010-010-0631-2 |
| P953 | full work available at URL | http://link.springer.com/content/pdf/10.1007/s11010-010-0631-2.pdf |
| http://link.springer.com/article/10.1007/s11010-010-0631-2/fulltext.html | ||
| http://link.springer.com/content/pdf/10.1007/s11010-010-0631-2 | ||
| P698 | PubMed publication ID | 21069436 |
| P2093 | author name string | Biao Yan | |
| Men Kong | |||
| Yi-han Chen | |||
| P2860 | cites work | FIH-1: a novel protein that interacts with HIF-1alpha and VHL to mediate repression of HIF-1 transcriptional activity | Q24291819 |
| Intracellular localisation of human HIF-1 alpha hydroxylases: implications for oxygen sensing | Q24296765 | ||
| Humanin peptide suppresses apoptosis by interfering with Bax activation | Q24301403 | ||
| Jmjd6 catalyses lysyl-hydroxylation of U2AF65, a protein associated with RNA splicing | Q24310337 | ||
| Asparaginyl hydroxylation of the Notch ankyrin repeat domain by factor inhibiting hypoxia-inducible factor | Q24311481 | ||
| Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death | Q24312211 | ||
| Interaction with factor inhibiting HIF-1 defines an additional mode of cross-coupling between the Notch and hypoxia signaling pathways | Q24317627 | ||
| ASB4 is a hydroxylation substrate of FIH and promotes vascular differentiation via an oxygen-dependent mechanism | Q24320200 | ||
| Conformation of the Bax C-terminus regulates subcellular location and cell death | Q24534099 | ||
| Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis | Q24669892 | ||
| Movement of Bax from the cytosol to mitochondria during apoptosis | Q24677881 | ||
| Evidence That Two Enzyme-derived Histidine Ligands Are Sufficient for Iron Binding and Catalysis by Factor Inhibiting HIF (FIH) | Q27651089 | ||
| Science's STKE : signal transduction knowledge environment | Q27714885 | ||
| Disruption of dimerization and substrate phosphorylation inhibit factor inhibiting hypoxia-inducible factor (FIH) activity | Q28118969 | ||
| Protein kinase C epsilon signaling complexes include metabolism- and transcription/translation-related proteins: complimentary separation techniques with LC/MS/MS | Q28217272 | ||
| Induction of apoptosis by the Bcl-2 homologue Bak | Q28299871 | ||
| Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death | Q28363890 | ||
| Bax in murine thymus is a soluble monomeric protein that displays differential detergent-induced conformations | Q28506809 | ||
| The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues | Q28512628 | ||
| Bax can antagonize Bcl-XL during etoposide and cisplatin-induced cell death independently of its heterodimerization with Bcl-XL | Q28587967 | ||
| Oxygen sensing by HIF hydroxylases | Q29617806 | ||
| Ku70 suppresses the apoptotic translocation of Bax to mitochondria. | Q31136038 | ||
| Mass spectrometry and protein analysis | Q33239890 | ||
| E1B 19K blocks Bax oligomerization and tumor necrosis factor alpha-mediated apoptosis | Q33852843 | ||
| Peg3/Pw1 promotes p53-mediated apoptosis by inducing Bax translocation from cytosol to mitochondria | Q35373938 | ||
| The Bcl-2 family: roles in cell survival and oncogenesis. | Q35591695 | ||
| The hypoxia-inducible factor 2alpha N-terminal and C-terminal transactivation domains cooperate to promote renal tumorigenesis in vivo | Q35676249 | ||
| Constitutive expression of the machinery for programmed cell death | Q36236957 | ||
| E1B 19K inhibits Fas-mediated apoptosis through FADD-dependent sequestration of FLICE. | Q36264713 | ||
| Bax and Bak coalesce into novel mitochondria-associated clusters during apoptosis | Q36364894 | ||
| Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis | Q38326924 | ||
| The N-terminal end of Bax contains a mitochondrial-targeting signal. | Q38358682 | ||
| Genetic analysis of the role of the asparaginyl hydroxylase factor inhibiting hypoxia-inducible factor (FIH) in regulating hypoxia-inducible factor (HIF) transcriptional target genes [corrected]. | Q40527027 | ||
| Pharmacologic activation of p53 elicits Bax-dependent apoptosis in the absence of transcription | Q44687763 | ||
| Hierarchical regulation of mitochondrion-dependent apoptosis by BCL-2 subfamilies | Q79370601 | ||
| Cell-specific regulation of hypoxia-inducible factor (HIF)-1alpha and HIF-2alpha stabilization and transactivation in a graded oxygen environment | Q83947086 | ||
| P433 | issue | 1-2 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 1-9 | |
| P577 | publication date | 2010-11-11 | |
| P1433 | published in | Molecular and Cellular Biochemistry | Q1573176 |
| P1476 | title | Prevention of apoptosis by the interaction between FIH1 and Bax | |
| P478 | volume | 348 |
| Q48846055 | Escherichia coli induces apoptosis in human monocytic U937 cells through the Fas/FasL signaling pathway |
| Q41784472 | Expression of myogenes in longissimus dorsi muscle during prenatal development in commercial and local Piau pigs |
| Q33947033 | FIH-1-Mint3 axis does not control HIF-1 transcriptional activity in nucleus pulposus cells |
| Q90788856 | Hypermethylated gene ANKDD1A is a candidate tumor suppressor that interacts with FIH1 and decreases HIF1α stability to inhibit cell autophagy in the glioblastoma multiforme hypoxia microenvironment |
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