scholarly article | Q13442814 |
P356 | DOI | 10.1002/GLIA.21051 |
P698 | PubMed publication ID | 20645407 |
P2093 | author name string | Bradley J Kerr | |
Howard M Johnson | |||
Samuel David | |||
Jennifer L Berard | |||
P2860 | cites work | The comparative roles of suppressor of cytokine signaling-1 and -3 in the inhibition and desensitization of cytokine signaling | Q46941042 |
SOCS-1 suppresses TNF-alpha-induced apoptosis through the regulation of Jak activation | Q47406323 | ||
Myelin-laden macrophages are anti-inflammatory, consistent with foam cells in multiple sclerosis. | Q54626145 | ||
Failure of sensory neurons to express class I MHC is due to differential SOCS1 expression | Q62675334 | ||
Expression of ?suppressor of cytokine signalling? (SOCS) genes in the developing and adult mouse nervous system | Q62675369 | ||
Differential expression of suppressors of cytokine signaling-1 and -3 and related cytokines in central nervous system during remitting versus non-remitting forms of experimental autoimmune encephalomyelitis. | Q64948037 | ||
Anti-macrophage CR3 antibody blocks myelin phagocytosis by macrophages in vitro | Q68601076 | ||
Galectin-3/MAC-2 in experimental allergic encephalomyelitis | Q73320823 | ||
SOCS1 regulates interferon-gamma mediated sensory neuron survival | Q77322591 | ||
SOCS1/JAB is a negative regulator of LPS-induced macrophage activation | Q78518643 | ||
SOCS-1 participates in negative regulation of LPS responses | Q78518680 | ||
Loss of suppressor of cytokine signaling 1 in helper T cells leads to defective Th17 differentiation by enhancing antagonistic effects of IFN-gamma on STAT3 and Smads | Q80825258 | ||
Galectin-3/MAC-2, Ras and PI3K activate complement receptor-3 and scavenger receptor-AI/II mediated myelin phagocytosis in microglia | Q81613971 | ||
A family of cytokine-inducible inhibitors of signalling | Q24313168 | ||
A new protein containing an SH2 domain that inhibits JAK kinases | Q24313242 | ||
The IL-23/Th(17) axis: therapeutic targets for autoimmune inflammation | Q28266133 | ||
Structure and function of a new STAT-induced STAT inhibitor | Q28584775 | ||
Nitric oxide and macrophage function | Q29615327 | ||
Accelerated apoptosis of lymphocytes by augmented induction of Bax in SSI-1 (STAT-induced STAT inhibitor-1) deficient mice | Q31949951 | ||
Differential expression and potential role of SOCS1 and SOCS3 in Wallerian degeneration in injured peripheral nerve | Q33771100 | ||
Polycystic kidneys and chronic inflammatory lesions are the delayed consequences of loss of the suppressor of cytokine signaling-1 (SOCS-1). | Q33894724 | ||
Genetic models for CNS inflammation | Q34139624 | ||
Local nitric oxide production in viral and autoimmune diseases of the central nervous system | Q34209628 | ||
Suppressors of cytokine signaling and immunity | Q34279168 | ||
Uric acid, a peroxynitrite scavenger, inhibits CNS inflammation, blood-CNS barrier permeability changes, and tissue damage in a mouse model of multiple sclerosis. | Q34507928 | ||
Multiple sclerosis: deeper understanding of its pathogenesis reveals new targets for therapy. | Q34674896 | ||
Suppressors of cytokine signalling (SOCS) in the immune system | Q34716329 | ||
Transcription factors in the pathogenesis of autoimmunity | Q35724197 | ||
Regulation of signal transducer and activator of transcription and suppressor of cytokine-signaling gene expression in the brain of mice with astrocyte-targeted production of interleukin-12 or experimental autoimmune encephalomyelitis | Q35788671 | ||
SOCS: role in inflammation, allergy and homeostasis | Q35846396 | ||
Cytokines in multiple sclerosis: from bench to bedside | Q36113477 | ||
Elevated secretion of reactive nitrogen and oxygen intermediates by inflammatory leukocytes in hyperacute experimental autoimmune encephalomyelitis: enhancement by the soluble products of encephalitogenic T cells | Q36231527 | ||
Liver degeneration and lymphoid deficiencies in mice lacking suppressor of cytokine signaling-1. | Q36735213 | ||
Signals transducers and activators of transcription (STAT)-induced STAT inhibitor-1 (SSI-1)/suppressor of cytokine signaling-1 (SOCS-1) suppresses tumor necrosis factor alpha-induced cell death in fibroblasts | Q36975390 | ||
Putting out the fire: coordinated suppression of the innate and adaptive immune systems by SOCS1 and SOCS3 proteins | Q37257006 | ||
An interferon-gamma-activated site (GAS) is necessary for full expression of the mouse iNOS gene in response to interferon-gamma and lipopolysaccharide | Q38349380 | ||
Adenoviral expression of suppressor of cytokine signaling-1 reduces adenovirus vector-induced innate immune responses | Q40000317 | ||
Treatment of mice with the suppressor of cytokine signaling-1 mimetic peptide, tyrosine kinase inhibitor peptide, prevents development of the acute form of experimental allergic encephalomyelitis and induces stable remission in the chronic relapsing | Q40364719 | ||
A SOCS-1 peptide mimetic inhibits both constitutive and IL-6 induced activation of STAT3 in prostate cancer cells. | Q40462635 | ||
Characterization of a peptide inhibitor of Janus kinase 2 that mimics suppressor of cytokine signaling 1 function | Q40547454 | ||
Signalling events involved in interferon-gamma-inducible macrophage nitric oxide generation | Q40660108 | ||
Suppressor of cytokine signaling 1 inhibits cytokine induction of CD40 expression in macrophages | Q40708938 | ||
The IL-23/IL-17 axis in inflammation | Q42825302 | ||
Low responsiveness to IFN-gamma, after pretreatment of mouse macrophages with lipopolysaccharides, develops via diverse regulatory pathways | Q43900082 | ||
Peptidylprolyl isomerase A (PPIA) as a preferred internal control over GAPDH and beta-actin in quantitative RNA analyses | Q43965355 | ||
Regulation of the expression of inducible nitric oxide synthase | Q45089049 | ||
Nitric oxide synthase inhibitor, aminoguanidine, reduces inflammation and demyelination produced by Theiler's virus infection. | Q45757124 | ||
P433 | issue | 15 | |
P304 | page(s) | 1816-1826 | |
P577 | publication date | 2010-11-01 | |
P1433 | published in | Glia | Q15716658 |
P1476 | title | Differential expression of SOCS1 in macrophages in relapsing-remitting and chronic EAE and its role in disease severity | |
P478 | volume | 58 |
Q44596853 | A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis |
Q36131241 | Aging diminishes the resistance of AO rats to EAE: putative role of enhanced generation of GM-CSF Expressing CD4+ T cells in aged rats. |
Q59134975 | Connexin 30 Deficiency Attenuates Chronic but Not Acute Phases of Experimental Autoimmune Encephalomyelitis Through Induction of Neuroprotective Microglia |
Q35154391 | Developmental endothelial locus-1 is a homeostatic factor in the central nervous system limiting neuroinflammation and demyelination. |
Q35175890 | Inhibition of SOCS1-/- lethal autoinflammatory disease correlated to enhanced peripheral Foxp3+ regulatory T cell homeostasis |
Q55026303 | Intravenous C16 and angiopoietin-1 improve the efficacy of placenta-derived mesenchymal stem cell therapy for EAE. |
Q33988247 | Involvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis. |
Q38201907 | Macrophages: a double-edged sword in experimental autoimmune encephalomyelitis |
Q37325616 | Nogo receptor complex expression dynamics in the inflammatory foci of central nervous system experimental autoimmune demyelination. |
Q49543789 | Novel disease-modifying anti-rheumatic drug iguratimod suppresses chronic experimental autoimmune encephalomyelitis by down-regulating activation of macrophages/microglia through an NF-κB pathway. |
Q37670027 | Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases. |
Q37889633 | Repertoire of microglial and macrophage responses after spinal cord injury |
Q48413880 | Role of brain transmigrating neutrophils in depression-like behavior during systemic infection. |
Q38067258 | Role of the SOCS in monocytes/macrophages-related pathologies. Are we getting closer to a new pharmacological target? |
Q34645350 | The kinase inhibitory region of SOCS-1 is sufficient to inhibit T-helper 17 and other immune functions in experimental allergic encephalomyelitis |
Search more.