human | Q5 |
P496 | ORCID iD | 0000-0002-4001-2132 |
P69 | educated at | École Normale Supérieure de Lyon | Q10159 |
P108 | employer | École Normale Supérieure de Lyon | Q10159 |
French National Institute of Health and Medical Research | Q1474517 | ||
P735 | given name | Laetitia | Q12981877 |
Laetitia | Q12981877 | ||
P106 | occupation | researcher | Q1650915 |
Q24647958 | Inactivation of the UNC5C Netrin-1 receptor is associated with tumor progression in colorectal malignancies |
Q93264363 | Lack of muscle mTOR kinase activity causes early onset myopathy and compromises whole-body homeostasis |
Q27330378 | Muscle-specific Drp1 overexpression impairs skeletal muscle growth via translational attenuation. |
Q34345335 | Netrin-1 controls colorectal tumorigenesis by regulating apoptosis |
Q57191283 | The Proto-oncogene c-Kit Inhibits Tumor Growth by Behaving as a Dependence Receptor |
Q35575340 | The dependence receptors DCC and UNC5H as a link between neuronal guidance and survival |
Q24683182 | The netrin-1 receptors UNC5H are putative tumor suppressors controlling cell death commitment |
Q35566324 | Variants in the netrin-1 receptor UNC5C prevent apoptosis and increase risk of familial colorectal cancer |
Q53110486 | mTOR inactivation in myocardium from infant mice rapidly leads to dilated cardiomyopathy due to translation defects and p53/JNK-mediated apoptosis. |