Role of RANK-L as a potential inducer of ILC2-mediated type 2 inflammation in chronic rhinosinusitis with nasal polyps

scientific article published on 22 October 2019

Role of RANK-L as a potential inducer of ILC2-mediated type 2 inflammation in chronic rhinosinusitis with nasal polyps is …
instance of (P31):
scholarly articleQ13442814

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P356DOI10.1038/S41385-019-0215-8
P932PMC publication ID6917894
P698PubMed publication ID31641233

P2093author name stringPejman Soroosh
Anju T Peters
Tetsuo Himi
Whitney W Stevens
David B Conley
Robert C Kern
Atsushi Kato
Leslie C Grammer
Robert P Schleimer
Bruce K Tan
Kathryn E Hulse
Noriko Ogasawara
Ken-Ichi Takano
Kevin C Welch
Aiko I Klingler
Julie A Poposki
Stephanie S Smith
Joseph R Raviv
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ILC2s regulate adaptive Th2 cell functions via PD-L1 checkpoint control.Q40118341
Human innate lymphoid cellsQ40525663
Group 2 innate lymphoid cells are elevated and activated in chronic rhinosinusitis with nasal polyps.Q41506242
The chemokine receptor CXCR6 controls the functional topography of interleukin-22 producing intestinal innate lymphoid cellsQ41629384
The TNF-family cytokine TL1A promotes allergic immunopathology through group 2 innate lymphoid cells.Q41906298
Heterogeneous inflammatory patterns in chronic rhinosinusitis without nasal polyps in Chicago, Illinois.Q49769504
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The Tumor Necrosis Factor Superfamily Member RANKL Suppresses Effector Cytokine Production in Group 3 Innate Lymphoid CellsQ88954027
Epithelial activators of type 2 inflammation: Elevation of thymic stromal lymphopoietin, but not IL-25 or IL-33, in chronic rhinosinusitis with nasal polyps in Chicago, IllinoisQ89485067
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P433issue1
P921main subjectrhinosinusitisQ2148123
nasal polypsQ8055111
chronic rhinosinusitisQ55779961
P304page(s)86-95
P577publication date2019-10-22
P1433published inMucosal ImmunologyQ15817743
P1476titleRole of RANK-L as a potential inducer of ILC2-mediated type 2 inflammation in chronic rhinosinusitis with nasal polyps
P478volume13