scholarly article | Q13442814 |
P2093 | author name string | Pejman Soroosh | |
Anju T Peters | |||
Tetsuo Himi | |||
Whitney W Stevens | |||
David B Conley | |||
Robert C Kern | |||
Atsushi Kato | |||
Leslie C Grammer | |||
Robert P Schleimer | |||
Bruce K Tan | |||
Kathryn E Hulse | |||
Noriko Ogasawara | |||
Ken-Ichi Takano | |||
Kevin C Welch | |||
Aiko I Klingler | |||
Julie A Poposki | |||
Stephanie S Smith | |||
Joseph R Raviv | |||
P2860 | cites work | Prostaglandin D2 selectively induces chemotaxis in T helper type 2 cells, eosinophils, and basophils via seven-transmembrane receptor CRTH2 | Q24290855 |
TRANCE is a novel ligand of the tumor necrosis factor receptor family that activates c-Jun N-terminal kinase in T cells | Q24315251 | ||
The Interplay Between Monocytes/Macrophages and CD4(+) T Cell Subsets in Rheumatoid Arthritis | Q26774681 | ||
Crystal structure of the TRANCE/RANKL cytokine reveals determinants of receptor-ligand specificity | Q27635130 | ||
RANKL Employs Distinct Binding Modes to Engage RANK and the Osteoprotegerin Decoy Receptor | Q27674275 | ||
Bench to bedside: elucidation of the OPG-RANK-RANKL pathway and the development of denosumab | Q28265584 | ||
RANKL/RANK-beyond bones | Q28308042 | ||
Evidence of a role for B cell-activating factor of the TNF family in the pathogenesis of chronic rhinosinusitis with nasal polyps | Q33573926 | ||
RANK receptor oligomerisation in the regulation of NFκB signalling | Q33825207 | ||
Immunopathogenesis of Chronic Rhinosinusitis and Nasal Polyposis | Q33913202 | ||
Enhanced innate type 2 immune response in peripheral blood from patients with asthma | Q34111924 | ||
Glandular mast cells with distinct phenotype are highly elevated in chronic rhinosinusitis with nasal polyps | Q34270916 | ||
Biology of the RANKL-RANK-OPG System in Immunity, Bone, and Beyond | Q34366449 | ||
Prostaglandin D₂: a dominant mediator of aspirin-exacerbated respiratory disease | Q34892917 | ||
Increased expression of the chemokine CCL23 in eosinophilic chronic rhinosinusitis with nasal polyps | Q35085607 | ||
ICOS:ICOS-ligand interaction is required for type 2 innate lymphoid cell function, homeostasis, and induction of airway hyperreactivity. | Q35196224 | ||
Cytokine mRNA profiling identifies B cells as a major source of RANKL in rheumatoid arthritis | Q35249619 | ||
Reduced expression of antimicrobial PLUNC proteins in nasal polyp tissues of patients with chronic rhinosinusitis | Q36033394 | ||
TRANCE, a tumor necrosis factor family member critical for CD40 ligand-independent T helper cell activation | Q36368029 | ||
TRANCE (tumor necrosis factor [TNF]-related activation-induced cytokine), a new TNF family member predominantly expressed in T cells, is a dendritic cell-specific survival factor | Q36380989 | ||
Group 2 innate lymphoid cells license dendritic cells to potentiate memory TH2 cell responses | Q36389520 | ||
IL-25/IL-33-responsive TH2 cells characterize nasal polyps with a default TH17 signature in nasal mucosa | Q36857733 | ||
Chronic Rhinosinusitis with Nasal Polyps | Q37080775 | ||
Production of RANKL by Memory B Cells: A Link Between B Cells and Bone Erosion in Rheumatoid Arthritis | Q37115802 | ||
Thymic stromal lymphopoietin activity is increased in nasal polyps of patients with chronic rhinosinusitis | Q37138334 | ||
A role for IL-25 and IL-33-driven type-2 innate lymphoid cells in atopic dermatitis. | Q37397800 | ||
Prostaglandin D2 activates group 2 innate lymphoid cells through chemoattractant receptor-homologous molecule expressed on TH2 cells | Q37691114 | ||
TNF superfamily member TL1A elicits type 2 innate lymphoid cells at mucosal barriers | Q37721441 | ||
New knowledge on critical osteoclast formation and activation pathways from study of rare genetic diseases of osteoclasts: focus on the RANK/RANKL axis. | Q37748586 | ||
Immunopathology of chronic rhinosinusitis. | Q38403174 | ||
Group 2 innate lymphoid cells are recruited to the nasal mucosa in patients with aspirin-exacerbated respiratory disease. | Q38916857 | ||
Dexamethasone rapidly suppresses IL-33-stimulated mast cell function by blocking transcription factor activity. | Q39574726 | ||
ILC2s regulate adaptive Th2 cell functions via PD-L1 checkpoint control. | Q40118341 | ||
Human innate lymphoid cells | Q40525663 | ||
Group 2 innate lymphoid cells are elevated and activated in chronic rhinosinusitis with nasal polyps. | Q41506242 | ||
The chemokine receptor CXCR6 controls the functional topography of interleukin-22 producing intestinal innate lymphoid cells | Q41629384 | ||
The TNF-family cytokine TL1A promotes allergic immunopathology through group 2 innate lymphoid cells. | Q41906298 | ||
Heterogeneous inflammatory patterns in chronic rhinosinusitis without nasal polyps in Chicago, Illinois. | Q49769504 | ||
GITR co-signal in ILC2 controls allergic lung inflammation. | Q50086910 | ||
Tissue-Restricted Adaptive Type 2 Immunity Is Orchestrated by Expression of the Costimulatory Molecule OX40L on Group 2 Innate Lymphoid Cells. | Q55284399 | ||
Human IL-25- and IL-33-responsive type 2 innate lymphoid cells are defined by expression of CRTH2 and CD161 | Q84933191 | ||
In human basophils, IL-3 selectively induces RANKL expression that is modulated by IgER-dependent and IgER-independent stimuli | Q87457550 | ||
The Tumor Necrosis Factor Superfamily Member RANKL Suppresses Effector Cytokine Production in Group 3 Innate Lymphoid Cells | Q88954027 | ||
Epithelial activators of type 2 inflammation: Elevation of thymic stromal lymphopoietin, but not IL-25 or IL-33, in chronic rhinosinusitis with nasal polyps in Chicago, Illinois | Q89485067 | ||
TNF induces production of type 2 cytokines in human group 2 innate lymphoid cells | Q90130145 | ||
Group 2 Innate Lymphoid Cells in Airway Diseases | Q91975039 | ||
P433 | issue | 1 | |
P921 | main subject | rhinosinusitis | Q2148123 |
nasal polyps | Q8055111 | ||
chronic rhinosinusitis | Q55779961 | ||
P304 | page(s) | 86-95 | |
P577 | publication date | 2019-10-22 | |
P1433 | published in | Mucosal Immunology | Q15817743 |
P1476 | title | Role of RANK-L as a potential inducer of ILC2-mediated type 2 inflammation in chronic rhinosinusitis with nasal polyps | |
P478 | volume | 13 |