| human | Q5 |
| P496 | ORCID iD | 0000-0001-6359-5425 |
| P108 | employer | Temple University School of Medicine | Q4120361 |
| P735 | given name | ??? | Q17501985 |
| ??? | Q17501985 | ||
| P106 | occupation | researcher | Q1650915 |
| P21 | sex or gender | male | Q6581097 |
| Q47105229 | A Feline HFpEF Model with Pulmonary Hypertension and Compromised Pulmonary Function |
| Q37700677 | A Metric-Based System for Evaluating the Productivity of Preclinical Faculty at an Academic Medical Center in the Era of Clinical and Translational Science |
| Q36922076 | A Tension-Based Model Distinguishes Hypertrophic versus Dilated Cardiomyopathy |
| Q42550135 | A caveolae-targeted L-type Ca²+ channel antagonist inhibits hypertrophic signaling without reducing cardiac contractility |
| Q41850788 | A characterization and targeting of the infarct border zone in a swine model of myocardial infarction |
| Q97591629 | A low voltage activated Ca2+ current found in a subset of human ventricular myocytes |
| Q28588509 | A peptide encoded by a transcript annotated as long noncoding RNA enhances SERCA activity in muscle |
| Q89888187 | Abcg2-expressing side population cells contribute to cardiomyocyte renewal through fusion |
| Q75301226 | Abnormal frequency-dependent responses represent the pathophysiologic signature of contractile failure in human myocardium |
| Q91506703 | Acetylation of SERCA2a, Another Target for Heart Failure Treatment? |
| Q41112375 | Acute Catecholamine Exposure Causes Reversible Myocyte Injury Without Cardiac Regeneration. |
| Q35815095 | Acute aerobic exercise increases exogenously infused bone marrow cell retention in the heart |
| Q53573566 | Adolescent feline heart contains a population of small, proliferative ventricular myocytes with immature physiological properties. |
| Q37120202 | Adrenergic regulation of cardiac contractility does not involve phosphorylation of the cardiac ryanodine receptor at serine 2808 |
| Q62583214 | Alterations in Early Action Potential Repolarization Causes Localized Failure of Sarcoplasmic Reticulum Ca 2+ Release |
| Q51630929 | Altered myocardial Ca2+ cycling after left ventricular assist device support in the failing human heart. |
| Q38010863 | Animal models of heart failure: a scientific statement from the American Heart Association |
| Q57396465 | Are Resident c-Kit + Cardiac Stem Cells Really All That Are Needed to Mend a Broken Heart? |
| Q42316372 | Autologous c-Kit+ Mesenchymal Stem Cell Injections Provide Superior Therapeutic Benefit as Compared to c-Kit+ Cardiac-Derived Stem Cells in a Feline Model of Isoproterenol-Induced Cardiomyopathy |
| Q29568324 | Beta1-adrenergic receptors promote focal adhesion signaling downregulation and myocyte apoptosis in acute volume overload |
| Q36580410 | Blockade of NOX2 and STIM1 signaling limits lipopolysaccharide-induced vascular inflammation |
| Q33304156 | Bone marrow cells adopt the cardiomyogenic fate in vivo |
| Q30557136 | Bone-derived stem cells repair the heart after myocardial infarction through transdifferentiation and paracrine signaling mechanisms |
| Q78719610 | Ca influx via the Na/Ca exchanger maintains sarcoplasmic reticulum Ca content in failing human myocytes |
| Q42425047 | Ca(2+) influx through L-type Ca(2+) channels and transient receptor potential channels activates pathological hypertrophy signaling |
| Q37182590 | Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypes |
| Q50751761 | Ca2+ influx-induced sarcoplasmic reticulum Ca2+ overload causes mitochondrial-dependent apoptosis in ventricular myocytes. |
| Q35921523 | Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure |
| Q37396105 | CaMKII negatively regulates calcineurin-NFAT signaling in cardiac myocytes |
| Q51738765 | Calcineurin inhibition normalizes beta-adrenergic responsiveness in the spontaneously hypertensive rat. |
| Q42438635 | Calcium entry via Na/Ca exchange during the action potential directly contributes to contraction of failing human ventricular myocytes |
| Q42592376 | Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophy |
| Q78719614 | Calcium influx via I(NCX) is favored in failing human ventricular myocytes |
| Q89641851 | Cardiac Expression of Factor X Mediates Cardiac Hypertrophy and Fibrosis in Pressure Overload |
| Q41903724 | Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction |
| Q39828113 | Cardiac-restricted overexpression of the A(2A)-adenosine receptor in FVB mice transiently increases contractile performance and rescues the heart failure phenotype in mice overexpressing the A(1)-adenosine receptor |
| Q91943642 | Cardiometabolic Heart Failure and HFpEF: Still Chasing Unicorns |
| Q93079219 | Cardiomyocyte PKA Ablation Enhances Basal Contractility While Eliminates Cardiac β-Adrenergic Response Without Adverse Effects on the Heart |
| Q47951585 | Cardiomyocyte Regeneration: A Consensus Statement |
| Q36410356 | Caspase-1 mediates hyperlipidemia-weakened progenitor cell vessel repair |
| Q51062459 | Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart. |
| Q44324582 | Cellular basis of abnormal calcium transients of failing human ventricular myocytes |
| Q35013233 | Challenges facing early career academic cardiologists. |
| Q36629698 | Chronic β1-adrenergic blockade enhances myocardial β3-adrenergic coupling with nitric oxide-cGMP signaling in a canine model of chronic volume overload: new insight into mechanisms of cardiac benefit with selective β1-blocker therapy. |
| Q41150386 | Class I Histone Deacetylase Inhibition for the Treatment of Sustained Atrial Fibrillation |
| Q64274926 | Cortical Bone Derived Stem Cells for Cardiac Wound Healing |
| Q47638836 | Cortical Bone Stem Cell Therapy Preserves Cardiac Structure and Function After Myocardial Infarction |
| Q92811647 | Cortical bone-derived stem cell therapy reduces apoptosis after myocardial infarction |
| Q40053441 | Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice |
| Q50198230 | Dedifferentiation, Proliferation, and Redifferentiation of Adult Mammalian Cardiomyocytes After Ischemic Injury |
| Q58698076 | Diabetic Cardiomyopathy: Current and Future Therapies. Beyond Glycemic Control |
| Q78836226 | Differential effects of exercise training on skeletal muscle SERCA gene expression |
| Q92510142 | Differential microRNA-21 and microRNA-221 Upregulation in the Biventricular Failing Heart Reveals Distinct Stress Responses of Right Versus Left Ventricular Fibroblasts |
| Q37199523 | Does contractile Ca2+ control calcineurin-NFAT signaling and pathological hypertrophy in cardiac myocytes? |
| Q79161366 | Dynamic regulation of sodium/calcium exchange function in human heart failure |
| Q89149000 | Echocardiographic Strain Analysis for the Early Detection of Left Ventricular Systolic/Diastolic Dysfunction and Dyssynchrony in a Mouse Model of Physiological Aging |
| Q51778063 | Effects of forskolin on inotropic performance and phospholamban phosphorylation in exercise-trained hypertensive myocardium. |
| Q34048693 | Embryonic stem cell-derived cardiac myocytes are not ready for human trials |
| Q35786316 | Embryonic stem cell-derived exosomes promote endogenous repair mechanisms and enhance cardiac function following myocardial infarction |
| Q51807167 | Enhanced acidotic myocardial Ca2+ responsiveness with training in hypertension. |
| Q34092888 | Enhanced basal contractility but reduced excitation-contraction coupling efficiency and beta-adrenergic reserve of hearts with increased Cav1.2 activity |
| Q43717353 | Examining prehospital intubation for penetrating trauma in a swine hemorrhagic shock model |
| Q36365384 | Finding the rhythm of sudden cardiac death: new opportunities using induced pluripotent stem cell-derived cardiomyocytes |
| Q30487539 | G protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure |
| Q90668206 | GDF11 Decreases Pressure Overload-Induced Hypertrophy, but Can Cause Severe Cachexia and Premature Death |
| Q28267548 | GDF11 does not rescue aging-related pathological hypertrophy |
| Q34644069 | GRK5-mediated exacerbation of pathological cardiac hypertrophy involves facilitation of nuclear NFAT activity |
| Q78818609 | Gender differences in post-infarction hypertrophy in end-stage failing hearts |
| Q92503368 | HDAC inhibition improves cardiopulmonary function in a feline model of diastolic dysfunction |
| Q35774793 | Hyperhomocysteinemia suppresses bone marrow CD34+/VEGF receptor 2+ cells and inhibits progenitor cell mobilization and homing to injured vasculature-a role of β1-integrin in progenitor cell migration and adhesion |
| Q42552251 | Hyperphosphorylation of the cardiac ryanodine receptor at serine 2808 is not involved in cardiac dysfunction after myocardial infarction |
| Q99562795 | Identification and Comparison of Hyperglycemia-Induced Extracellular Vesicle Transcriptome in Different Mouse Stem Cells |
| Q42143116 | Imatinib activates pathological hypertrophy by altering myocyte calcium regulation |
| Q80534809 | Impaired contractile reserve in severe mitral valve regurgitation with a preserved ejection fraction |
| Q46557775 | Improved myocardial beta-adrenergic responsiveness and signaling with exercise training in hypertension |
| Q37120197 | Increased cardiac myocyte progenitors in failing human hearts |
| Q48045540 | Increasing T-type calcium channel activity by β-adrenergic stimulation contributes to β-adrenergic regulation of heart rates |
| Q42094321 | Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction |
| Q35612752 | Inhibition of PKCα/β with ruboxistaurin antagonizes heart failure in pigs after myocardial infarction injury |
| Q37078266 | Inhibition of angiotensin II Gq signaling augments beta-adrenergic receptor mediated effects in a renal artery stenosis model of high blood pressure |
| Q100943369 | Interaction of the Joining Region in Junctophilin-2 with the L-type Ca2+ Channel Is Pivotal for Cardiac Dyad Assembly and Intracellular Ca2+ Dynamics |
| Q62583212 | Intracellular sodium determines frequency-dependent alterations in contractility in hypertrophied feline ventricular myocytes |
| Q35844633 | Intracoronary Cytoprotective Gene Therapy: A Study of VEGF-B167 in a Pre-Clinical Animal Model of Dilated Cardiomyopathy |
| Q34520509 | Is Growth Differentiation Factor 11 a Realistic Therapeutic for Aging-Dependent Muscle Defects? |
| Q35080491 | Is depressed myocyte contractility centrally involved in heart failure? |
| Q43909394 | L-type Ca(2+) currents overlapping threshold Na(+) currents: could they be responsible for the "slip-mode" phenomenon in cardiac myocytes? |
| Q44145600 | L-type Ca2+ channel density and regulation are altered in failing human ventricular myocytes and recover after support with mechanical assist devices |
| Q34356130 | LETM1-dependent mitochondrial Ca2+ flux modulates cellular bioenergetics and proliferation. |
| Q37406309 | Left ventricular remodeling with exercise in hypertension |
| Q51740622 | Long-Term Caloric Restriction Improves Cardiac Function, Remodeling, Adrenergic Responsiveness, and Sympathetic Innervation in a Model of Postischemic Heart Failure. |
| Q96133440 | Loss of Protease-Activated Receptor 4 Prevents Inflammation Resolution and Predisposes the Heart to Cardiac Rupture after Myocardial Infarction |
| Q54565689 | Loss of beta1D-integrin function in human ischemic cardiomyopathy. |
| Q28116180 | MCUR1 Is a Scaffold Factor for the MCU Complex Function and Promotes Mitochondrial Bioenergetics |
| Q42487357 | Modulation of contractility by myocyte-derived arginase in normal and hypertrophied feline myocardium |
| Q35034545 | Modulation of contractility in failing human myocytes by reverse-mode Na/Ca exchange. |
| Q51836946 | Myocardial hypoperfusion/reperfusion tolerance with exercise training in hypertension. |
| Q44810009 | Myocyte nitric oxide synthase 2 contributes to blunted beta-adrenergic response in failing human hearts by decreasing Ca2+ transients |
| Q42162128 | Na(+)-Ca(2+) exchange current and submembrane [Ca(2+)] during the cardiac action potential |
| Q36463797 | Negative Regulation of miR-375 by Interleukin-10 Enhances Bone Marrow-Derived Progenitor Cell-Mediated Myocardial Repair and Function After Myocardial Infarction |
| Q37416128 | Negative inotropic effects of high-mobility group box 1 protein in isolated contracting cardiac myocytes |
| Q49666071 | Neonatal Transplantation Confers Maturation of PSC-Derived Cardiomyocytes Conducive to Modeling Cardiomyopathy |
| Q57176905 | New Myocyte Formation in the Adult Heart: Endogenous Sources and Therapeutic Implications |
| Q36719477 | Nuquantus: Machine learning software for the characterization and quantification of cell nuclei in complex immunofluorescent tissue images |
| Q27302343 | Obligatory role of neuronal nitric oxide synthase in the heart's antioxidant adaptation with exercise |
| Q58127111 | Outcomes Associated With a Strategy of Adjuvant Metolazone or High-Dose Loop Diuretics in Acute Decompensated Heart Failure: A Propensity Analysis |
| Q47107654 | Peptidyl-Prolyl Isomerase 1 Regulates Ca2+ Handling by Modulating Sarco(Endo)Plasmic Reticulum Calcium ATPase and Na2+/Ca2+ Exchanger 1 Protein Levels and Function |
| Q44495537 | Periostin and periostin-like factor in the human heart: possible therapeutic targets |
| Q45154764 | Pharmacological effects of ATI22-107 [2-(2-{2-[2-chloro-4-(6-oxo-1,4,5,6-tetrahydro-pyridazin-3-yl)-phenoxy]-acetylamino}-ethoxymethyl)-4-(2-chloro-phenyl)-6-methyl-1,4-dihydro-pyridine-3,5-dicarboxylic acid dimethyl ester)], a novel dual pharmacoph |
| Q44607648 | Phenotypic differences in transient outward K+ current of human and canine ventricular myocytes: insights into molecular composition of ventricular Ito. |
| Q83986834 | Phosphorylation of phospholamban at threonine-17 reduces cardiac adrenergic contractile responsiveness in chronic pressure overload-induced hypertrophy |
| Q46022665 | Pim-1 regulates cardiomyocyte survival downstream of Akt. |
| Q35088053 | Platelet endothelial cell adhesion molecule-1 mediates endothelial-cardiomyocyte communication and regulates cardiac function. |
| Q37386463 | Pleiotropic effects of neutrophils on myocyte apoptosis and left ventricular remodeling during early volume overload |
| Q104566780 | Post-surgery echocardiography can predict the amount of ischemia-reperfusion injury and the resultant scar size |
| Q64949499 | Prior β-blocker treatment decreases leukocyte responsiveness to injury. |
| Q37097008 | Prolyl hydroxylase domain protein 2 silencing enhances the survival and paracrine function of transplanted adipose-derived stem cells in infarcted myocardium |
| Q57396450 | Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction |
| Q37359189 | Protein kinase C{alpha}, but not PKC{beta} or PKC{gamma}, regulates contractility and heart failure susceptibility: implications for ruboxistaurin as a novel therapeutic approach |
| Q42524047 | Rate dependence of [Na+]i and contractility in nonfailing and failing human myocardium |
| Q42248990 | Reduced effects of BAY K 8644 on L-type Ca2+ current in failing human cardiac myocytes are related to abnormal adrenergic regulation |
| Q46954786 | Reduced sarcoplasmic reticulum Ca(2+) load mediates impaired contractile reserve in right ventricular pressure overload |
| Q62583213 | Regulated Overexpression of the A 1 -Adenosine Receptor in Mice Results in Adverse but Reversible Changes in Cardiac Morphology and Function |
| Q30300038 | Regulation of L-type calcium channel by phospholemman in cardiac myocytes |
| Q51757121 | Relative systolic dysfunction in female spontaneously hypertensive rat myocardium. |
| Q50926615 | Remodeling of repolarization and arrhythmia susceptibility in a myosin-binding protein C knockout mouse model. |
| Q41964256 | Repair of the injured adult heart involves new myocytes potentially derived from resident cardiac stem cells |
| Q35192591 | Requirement of FADD, NEMO, and BAX/BAK for aberrant mitochondrial function in tumor necrosis factor alpha-induced necrosis |
| Q34759446 | Research priorities in hypertrophic cardiomyopathy: report of a Working Group of the National Heart, Lung, and Blood Institute. |
| Q57396459 | Response to Torella et al |
| Q50954580 | Role of STIM1 (Stromal Interaction Molecule 1) in Hypertrophy-Related Contractile Dysfunction. |
| Q36118077 | SPG7 Is an Essential and Conserved Component of the Mitochondrial Permeability Transition Pore |
| Q36260333 | STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy |
| Q36141000 | Severe hyperhomocysteinemia promotes bone marrow-derived and resident inflammatory monocyte differentiation and atherosclerosis in LDLr/CBS-deficient mice |
| Q42502024 | Sex-based differences in myocardial contractile reserve |
| Q73158153 | Sodium and the heart: a hidden key factor in cardiac regulation |
| Q33681995 | Sorafenib cardiotoxicity increases mortality after myocardial infarction. |
| Q46613293 | Sprint training improves postischemic, left ventricular diastolic performance |
| Q84282694 | Stem cell therapy for heart failure |
| Q34013724 | Systems approach to understanding electromechanical activity in the human heart: a national heart, lung, and blood institute workshop summary. |
| Q37466162 | T-type Ca²⁺ channels regulate the exit of cardiac myocytes from the cell cycle after birth. |
| Q36615175 | Targeted STIM deletion impairs calcium homeostasis, NFAT activation, and growth of smooth muscle |
| Q39657199 | The Heart Failure Society of America in 2020: a vision for the future |
| Q41822328 | The Mitochondrial Calcium Uniporter Matches Energetic Supply with Cardiac Workload during Stress and Modulates Permeability Transition |
| Q42479642 | The Na+/Ca2+ exchanger/SR Ca2+ ATPase transport capacity regulates the contractility of normal and hypertrophied feline ventricular myocytes |
| Q33986863 | The gut hormone ghrelin partially reverses energy substrate metabolic alterations in the failing heart. |
| Q24680113 | The inotropic effect of cardioactive glycosides in ventricular myocytes requires Na+-Ca2+ exchanger function |
| Q30274561 | The mitochondrial Na+/Ca2+ exchanger is essential for Ca2+ homeostasis and viability |
| Q104790940 | Thomas L. Force, MD: 1951-2020: A Brilliant Physician-Scientist Gone Too Soon |
| Q92966673 | Transient Introduction of miR-294 in the Heart Promotes Cardiomyocyte Cell Cycle Reentry After Injury |
| Q34111821 | Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction |
| Q33540145 | Two-photon laser scanning microscopy of the transverse-axial tubule system in ventricular cardiomyocytes from failing and non-failing human hearts |
| Q99610827 | Uncoupling protein 2-mediated metabolic adaptations define cardiac cell function in the heart during transition from young to old age |
| Q47631599 | Unique Features of Cortical Bone Stem Cells Associated With Repair of the Injured Heart |
| Q41790233 | Validation of transcatheter left ventricular electromechanical mapping for assessment of cardiac function and targeted transendocardial injection in a porcine ischemia-reperfusion model. |
| Q46316576 | Voluntary wheel running and pacing-induced dysfunction in hypertension |
| Q35090046 | [Na+]i handling in the failing human heart |
| Q42690112 | alpha1G-dependent T-type Ca2+ current antagonizes cardiac hypertrophy through a NOS3-dependent mechanism in mice |
| Q33710767 | c-Cbl inhibition improves cardiac function and survival in response to myocardial ischemia |
| Q28581185 | c-Cbl ubiquitin ligase regulates focal adhesion protein turnover and myofibril degeneration induced by neutrophil protease cathepsin G |
| Q34004496 | c-Kit+ bone marrow stem cells differentiate into functional cardiac myocytes. |
| Q39403789 | microRNA in Cardiovascular Aging and Age-Related Cardiovascular Diseases |
| Q34092821 | {beta}1-Adrenergic receptor activation induces mouse cardiac myocyte death through both L-type calcium channel-dependent and -independent pathways |
| Q33680497 | β-Adrenergic receptor-mediated transactivation of epidermal growth factor receptor decreases cardiomyocyte apoptosis through differential subcellular activation of ERK1/2 and Akt. |
| Q38957870 | β-adrenergic receptor-mediated cardiac contractility is inhibited via vasopressin type 1A-receptor-dependent signaling |
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