scholarly article | Q13442814 |
P50 | author | Miklós Sahin-Tóth | Q38318675 |
Eszter Hegyi | Q90043970 | ||
P2093 | author name string | Andrea Geisz | |
Balázs Csaba Németh | |||
Zsanett Jancsó | |||
P2860 | cites work | Chymotrypsin C (caldecrin) promotes degradation of human cationic trypsin: identity with Rinderknecht's enzyme Y | Q24675338 |
Genetic Risk in Chronic Pancreatitis: The Trypsin-Dependent Pathway | Q33843764 | ||
Early trypsinogen activation in acute pancreatitis. | Q33953449 | ||
The epidemiology of pancreatitis and pancreatic cancer | Q34341477 | ||
Conditionally immortalized colonic epithelial cell line from a Ptk6 null mouse that polarizes and differentiates in vitro | Q34420694 | ||
Increased activation of hereditary pancreatitis-associated human cationic trypsinogen mutants in presence of chymotrypsin C | Q36016998 | ||
Why does pancreatic overstimulation cause pancreatitis? | Q36632168 | ||
Tighter Control by Chymotrypsin C (CTRC) Explains Lack of Association between Human Anionic Trypsinogen and Hereditary Pancreatitis | Q37065823 | ||
Autoactivation of mouse trypsinogens is regulated by chymotrypsin C via cleavage of the autolysis loop | Q37099760 | ||
Cerulein-induced chronic pancreatitis does not require intra-acinar activation of trypsinogen in mice | Q37591305 | ||
CLIC1 null mice demonstrate a role for CLIC1 in macrophage superoxide production and tissue injury | Q37699664 | ||
Chymotrypsin C (caldecrin) stimulates autoactivation of human cationic trypsinogen | Q38290039 | ||
Genome-wide association study identifies inversion in the CTRB1-CTRB2 locus to modify risk for alcoholic and non-alcoholic chronic pancreatitis | Q40103441 | ||
Clusterin and Pycr1 alterations associate with strain and model differences in susceptibility to experimental pancreatitis. | Q40414683 | ||
Role of cathepsin B in intracellular trypsinogen activation and the onset of acute pancreatitis | Q40735691 | ||
Intra-acinar trypsinogen activation mediates early stages of pancreatic injury but not inflammation in mice with acute pancreatitis | Q41628354 | ||
Relationship of strain-dependent susceptibility to experimentally induced acute pancreatitis with regulation of Prss1 and Spink3 expression | Q43161315 | ||
Variation feature of receptor binding sites of H1N1 influenza hemagglutinin in different hosts | Q44989044 | ||
Novel PRSS1 Mutation p.P17T Validates Pathogenic Relevance of CTRC-Mediated Processing of the Trypsinogen Activation Peptide in Chronic Pancreatitis. | Q50034038 | ||
Metamizol Relieves Pain Without Interfering With Cerulein-Induced Acute Pancreatitis in Mice. | Q52148725 | ||
A preclinical model of chronic pancreatitis driven by trypsinogen autoactivation | Q59806147 | ||
Cause-effect relationships between zymogen activation and other early events in secretagogue-induced acute pancreatitis | Q79856720 | ||
Human CPA1 mutation causes digestive enzyme misfolding and chronic pancreatitis in mice | Q90473602 | ||
Chymotrypsin Reduces the Severity of Secretagogue-Induced Pancreatitis in Mice | Q90750780 | ||
Elevated intracellular trypsin exacerbates acute pancreatitis and chronic pancreatitis in mice | Q92819652 | ||
P433 | issue | 14 | |
P304 | page(s) | e129717 | |
P577 | publication date | 2019-06-18 | |
P1433 | published in | JCI insight | Q27727187 |
P1476 | title | Natural single-nucleotide deletion in chymotrypsinogen C gene increases severity of secretagogue-induced pancreatitis in C57BL/6 mice | |
P478 | volume | 4 |
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