| scholarly article | Q13442814 |
| P50 | author | Xian-Guo Liu | Q70850602 |
| P2093 | author name string | Jing Xu | |
| Ting Xu | |||
| Ke Ma | |||
| Dai Li | |||
| Xiao-Long Zhang | |||
| Rui-Ping Pang | |||
| Man-Xiu Xie | |||
| Wei-An Zeng | |||
| P2860 | cites work | Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain | Q24562888 |
| Distinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neurons | Q24632552 | ||
| NF-kappaB, IkappaB, and IRAK control glutamate receptor density at the Drosophila NMJ | Q24658002 | ||
| SCN9A mutations in paroxysmal extreme pain disorder: allelic variants underlie distinct channel defects and phenotypes | Q28118877 | ||
| From genes to pain: Na v 1.7 and human pain disorders | Q28254063 | ||
| An SCN9A channelopathy causes congenital inability to experience pain | Q28278844 | ||
| International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels | Q28289125 | ||
| From calcium to NF-kappa B signaling pathways in neurons | Q30453037 | ||
| Loss-of-function mutations in sodium channel Nav1.7 cause anosmia. | Q30540365 | ||
| A-803467, a potent and selective Nav1.8 sodium channel blocker, attenuates neuropathic and inflammatory pain in the rat. | Q34625901 | ||
| Evidence that reactive oxygen species do not mediate NF-kappaB activation | Q35160731 | ||
| Tumor necrosis factor-α enhances voltage-gated Na⁺ currents in primary culture of mouse cortical neurons | Q35878205 | ||
| Physiological functions for brain NF-kappaB. | Q35996911 | ||
| Voltage sensor interaction site for selective small molecule inhibitors of voltage-gated sodium channels | Q37031862 | ||
| The IKK-NF-kappaB pathway: a source for novel molecular drug targets in pain therapy? | Q37193383 | ||
| Sodium channels in normal and pathological pain | Q37724336 | ||
| TNFR1-induced activation of the classical NF-κB pathway | Q37828175 | ||
| Regulation/modulation of sensory neuron sodium channels | Q38204928 | ||
| Synaptic NF-kappa B pathway in neuronal plasticity and memory | Q38214046 | ||
| ProTx-II, a selective inhibitor of NaV1.7 sodium channels, blocks action potential propagation in nociceptors. | Q38288024 | ||
| Significance of NF-κB as a pivotal therapeutic target in the neurodegenerative pathologies of Alzheimer's disease and multiple sclerosis | Q38344541 | ||
| Pain without nociceptors? Nav1.7-independent pain mechanisms. | Q38552593 | ||
| The proinflammatory cytokine tumor necrosis factor-α excites subfornical organ neurons | Q38715009 | ||
| Voltage-gated sodium channel Nav 1.7 promotes gastric cancer progression through MACC1-mediated upregulation of NHE1. | Q38751561 | ||
| Control of Neurotransmission by NaV1.7 in Human, Guinea Pig, and Mouse Airway Parasympathetic Nerves | Q38991080 | ||
| Functional expression of the voltage-gated Na⁺-channel Nav1.7 is necessary for EGF-mediated invasion in human non-small cell lung cancer cells | Q39103592 | ||
| Developmental switch of the expression of ion channels in human dendritic cells. | Q39800294 | ||
| Distribution of the voltage-gated sodium channel Na(v)1.7 in the rat: expression in the autonomic and endocrine systems. | Q40089012 | ||
| Inhibition of NF-kappaB prevents mechanical allodynia induced by spinal ventral root transection and suppresses the re-expression of Nav1.3 in DRG neurons in vivo and in vitro. | Q42880746 | ||
| TNF-α contributes to up-regulation of Nav1.3 and Nav1.8 in DRG neurons following motor fiber injury | Q42967059 | ||
| Acute topical application of tumor necrosis factor alpha evokes protein kinase A-dependent responses in rat sensory neurons | Q44121155 | ||
| DRG Voltage-gated Sodium Channel 1.7 is Up-regulated in Paclitaxel-Induced Neuropathy in Rats and in Humans with Neuropathic Pain. | Q47219652 | ||
| Up-regulation of NaV1.7 sodium channels expression by tumor necrosis factor-α in cultured bovine adrenal chromaffin cells and rat dorsal root ganglion neurons | Q47922241 | ||
| Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury | Q48139796 | ||
| Acute p38-mediated modulation of tetrodotoxin-resistant sodium channels in mouse sensory neurons by tumor necrosis factor-alpha. | Q48735844 | ||
| NF-κB, inflammation, immunity and cancer: coming of age. | Q49594691 | ||
| A reinterpretation of mammalian sodium channel gating based on single channel recording | Q59062717 | ||
| Targeting of NF-κB to Dendritic Spines Is Required for Synaptic Signaling and Spine Development | Q59754806 | ||
| Defining the Functional Role of Na1.7 in Human Nociception | Q64058142 | ||
| Increased sensitivity of sensory neurons to tumor necrosis factor alpha in rats with chronic compression of the lumbar ganglia | Q74695171 | ||
| Sensory stimuli induce nuclear translocation and phosphorylation of nuclear factor kappa B in primary sensory neurons of mice | Q84592112 | ||
| The role of TNF-alpha/NF-kappa B pathway on the up-regulation of voltage-gated sodium channel Nav1.7 in DRG neurons of rats with diabetic neuropathy | Q88011409 | ||
| P921 | main subject | protein-protein interaction | Q896177 |
| P304 | page(s) | 623-633 | |
| P577 | publication date | 2019-08-10 | |
| P1476 | title | Nuclear Factor-kappaB Gates Nav1.7 Channels in DRG Neurons via Protein-Protein Interaction | |
| P478 | volume | 19 |
| Q91928296 | Oral application of bulleyaconitine A attenuates morphine tolerance in neuropathic rats by inhibiting long-term potentiation at C-fiber synapses and protein kinase C gamma in spinal dorsal horn | cites work | P2860 |
Search more.