| scholarly article | Q13442814 |
| P50 | author | Aranzazu Villasante | Q43151931 |
| David G. Pisano | Q47823945 | ||
| Almudena R Ramiro | Q58330177 | ||
| Susana González López | Q71844387 | ||
| P2093 | author name string | Carlo Croce | |
| Lin He | |||
| Laura Belver | |||
| Virginia G de Yébenes | |||
| P2860 | cites work | Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2) | Q24290325 |
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| MicroRNA expression profiles classify human cancers | Q28032481 | ||
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| Tcl1 expression in chronic lymphocytic leukemia is regulated by miR-29 and miR-181 | Q28279673 | ||
| Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme | Q29547201 | ||
| Zebrafish MiR-430 promotes deadenylation and clearance of maternal mRNAs | Q29547837 | ||
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| Mechanisms of B-cell lymphoma pathogenesis | Q36085626 | ||
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| Immunoglobulin gene diversification | Q36312189 | ||
| Somatic hypermutation is limited by CRM1-dependent nuclear export of activation-induced deaminase | Q36399510 | ||
| MicroRNA expression and function in cancer | Q36637333 | ||
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| Life beyond cleavage: the case of Ago2 and hematopoiesis | Q36910691 | ||
| Oncogenic events triggered by AID, the adverse effect of antibody diversification | Q36931335 | ||
| Antibody class switching | Q41134290 | ||
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| AID is required for germinal center-derived lymphomagenesis | Q46861217 | ||
| MicroRNA Expression Abnormalities in Pancreatic Endocrine and Acinar Tumors Are Associated With Distinctive Pathologic Features and Clinical Behavior | Q57200299 | ||
| Switch recombination and germ-line transcription are division-regulated events in B lymphocytes | Q61853754 | ||
| P433 | issue | 10 | |
| P407 | language of work or name | English | Q1860 |
| P921 | main subject | Interleukin 4 | Q14865947 |
| Syndecan 1 | Q14912916 | ||
| Ig gamma-1 chain C region, membrane-bound form | Q21431336 | ||
| Activation-induced cytidine deaminase | Q21494793 | ||
| Protein tyrosine phosphatase, receptor type, C | Q21985631 | ||
| P304 | page(s) | 2199-2206 | |
| P577 | publication date | 2008-09-01 | |
| P1433 | published in | Journal of Experimental Medicine | Q3186912 |
| P1476 | title | miR-181b negatively regulates activation-induced cytidine deaminase in B cells | |
| P478 | volume | 205 |
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| Q37683558 | AIDing Chromatin and Transcription-Coupled Orchestration of Immunoglobulin Class-Switch Recombination. |
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| Q38500323 | Cholinesterase-Targeting microRNAs Identified in silico Affect Specific Biological Processes |
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| Q47640767 | Decreased production of class-switched antibodies in neonatal B cells is associated with increased expression of miR-181b |
| Q33484195 | Differential micro RNA expression in PBMC from multiple sclerosis patients |
| Q55208667 | Diversity in the Cow Ultralong CDR H3 Antibody Repertoire. |
| Q53577105 | Downregulated expression of miR-155, miR-17, and miR-181b, and upregulated expression of activation-induced cytidine deaminase and interferon-α in PBMCs from patients with SLE. |
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| Q37097751 | Epigenetics of the antibody response |
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| Q55109488 | Galectin-3 deficiency drives lupus-like disease by promoting spontaneous germinal centers formation via IFN-γ. |
| Q33392701 | Haploinsufficiency of activation-induced deaminase for antibody diversification and chromosome translocations both in vitro and in vivo |
| Q37776353 | Helicobacter pylori-induced activation-induced cytidine deaminase expression and carcinogenesis |
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| Q38062548 | Hormones and AID: balancing immunity and autoimmunity |
| Q82637413 | Human activated CD4+ T lymphocytes increase IL-2 expression by downregulating microRNA-181c |
| Q42867702 | Induction and Assessment of Class Switch Recombination in Purified Murine B Cells |
| Q40020386 | Loss of miR-182 affects B-cell extrafollicular antibody response |
| Q36157946 | Mechanisms of Yin Yang 1 in oncogenesis: the importance of indirect effects |
| Q37564257 | Memory B cells: effectors of long-lived immune responses. |
| Q48483217 | MiR-181b Antagonizes Atherosclerotic Plaque Vulnerability Through Modulating Macrophage Polarization by Directly Targeting Notch1. |
| Q42874375 | MiR-210 is induced by Oct-2, regulates B cells, and inhibits autoantibody production |
| Q33688205 | MicroRNA Activity in B Lymphocytes |
| Q33838816 | MicroRNA Regulation in Systemic Lupus Erythematosus Pathogenesis |
| Q35027849 | MicroRNA control of lymphocyte differentiation and function |
| Q51899002 | MicroRNA expression changes during zebrafish development induced by perfluorooctane sulfonate |
| Q33847322 | MicroRNA expression profiling identifies activated B cell status in chronic lymphocytic leukemia cells |
| Q37280366 | MicroRNA in autoimmunity and autoimmune diseases. |
| Q35175779 | MicroRNA regulation of molecular networks mapped by global microRNA, mRNA, and protein expression in activated T lymphocytes |
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| Q26824682 | MicroRNAs in B-cells: from normal differentiation to treatment of malignancies |
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| Q38599607 | MicroRNAs in Lymphoma: Regulatory Role and Biomarker Potential |
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| Q27010231 | MicroRNAs, Major Players in B Cells Homeostasis and Function |
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| Q38885490 | Noncoding RNAs in Cancer Immunology. |
| Q35637214 | Parp3 negatively regulates immunoglobulin class switch recombination. |
| Q37704005 | Preventing AID, a physiological mutator, from deleterious activation: regulation of the genomic instability that is associated with antibody diversity |
| Q35669089 | REG-γ associates with and modulates the abundance of nuclear activation-induced deaminase |
| Q36557058 | Regulation of AID, the B-cell genome mutator |
| Q26853622 | Regulation of Aicda expression and AID activity |
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| Q36939328 | Regulation of B-cell development and function by microRNAs. |
| Q34340626 | Regulation of activation-induced deaminase stability and antibody gene diversification by Hsp90. |
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| Q37419452 | Restricting activation-induced cytidine deaminase tumorigenic activity in B lymphocytes |
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| Q38646971 | Short-Circuiting Gene Regulatory Networks: Origins of B Cell Lymphoma |
| Q33732848 | Systemic delivery of microRNA-181b inhibits nuclear factor-κB activation, vascular inflammation, and atherosclerosis in apolipoprotein E-deficient mice |
| Q41921194 | The Clinical Application of MicroRNAs in Infectious Disease |
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| Q90705230 | The Pathogenic Role of Dysregulated Epigenetic Modifications in Autoimmune Diseases |
| Q53537247 | The clinical characteristics and prognostic significance of AID, miR-181b, and miR-155 expression in adult patients with de novo B-cell acute lymphoblastic leukemia. |
| Q41981500 | The mechanisms regulating the subcellular localization of AID. |
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| Q35307418 | The miR-155-PU.1 axis acts on Pax5 to enable efficient terminal B cell differentiation |
| Q36080716 | The microRNA-212/132 cluster regulates B cell development by targeting Sox4. |
| Q35723616 | The real culprit in systemic lupus erythematosus: abnormal epigenetic regulation |
| Q37729365 | The role of microRNAs in B-cell development and function |
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| Q48110072 | Transcription factor YY1 can control AID-mediated mutagenesis in mice |
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| Q39404034 | YY1 controls immunoglobulin class switch recombination and nuclear activation-induced deaminase levels |
| Q86941726 | [Potential relationship and clinical significance of miRNAs and Th17 related cytokines in patients with multiple myeloma] |
| Q53617978 | miR-148a promotes plasma cell differentiation and targets the germinal center transcription factors Mitf and Bach2. |
| Q90012891 | miR-181b inhibits chemoresistance in cisplatin-resistant H446 small cell lung cancer cells by targeting Bcl-2 |
| Q35220412 | miR-181b is a biomarker of disease progression in chronic lymphocytic leukemia |
| Q40340268 | miR-28 regulates the germinal center reaction and blocks tumor growth in preclinical models of non-Hodgkin lymphoma. |
| Q38096240 | miRNA and TMPRSS2-ERG do not mind their own business in prostate cancer cells |
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| Q39426666 | miRNAs in B Cell Development and Lymphomagenesis. |
| Q54421609 | microRNA-181b targets MLK2 in HL-60 cells. |
| Q37723987 | microRNAs: critical regulators in Th17 cells and players in diseases |
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