| scholarly article | Q13442814 |
| P819 | ADS bibcode | 2014PNAS..11110269H |
| P356 | DOI | 10.1073/PNAS.1410112111 |
| P932 | PMC publication ID | 4104897 |
| P698 | PubMed publication ID | 24982162 |
| P5875 | ResearchGate publication ID | 264633727 |
| P50 | author | Robin M Meyers | Q56421068 |
| Jiazhi Hu | Q61194625 | ||
| P2093 | author name string | Frederick W Alt | |
| Monica Gostissa | |||
| Suprawee Tepsuporn | |||
| P2860 | cites work | The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway | Q22065419 |
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| Breakpoint sites disclose the role of the V(D)J recombination machinery in the formation of T-cell receptor (TCR) and non-TCR associated aberrations in T-cell acute lymphoblastic leukemia | Q38125650 | ||
| Genome-wide translocation sequencing reveals mechanisms of chromosome breaks and rearrangements in B cells | Q38626707 | ||
| H2AX prevents DNA breaks from progressing to chromosome breaks and translocations | Q46904251 | ||
| Unrepaired DNA breaks in p53-deficient cells lead to oncogenic gene amplification subsequent to translocations. | Q53965557 | ||
| Telomere dysfunction and chromosome instability | Q24635882 | ||
| DNA repair protein Ku80 suppresses chromosomal aberrations and malignant transformation | Q28141630 | ||
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| The response to and repair of RAG-mediated DNA double-strand breaks | Q28256889 | ||
| ATM prevents the persistence and propagation of chromosome breaks in lymphocytes | Q33289263 | ||
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| Origin of chromosomal translocations in lymphoid cancer | Q33870612 | ||
| ATM-deficient thymic lymphoma is associated with aberrant tcrd rearrangement and gene amplification | Q33979941 | ||
| Mechanisms promoting translocations in editing and switching peripheral B cells | Q34008189 | ||
| Mechanisms that can promote peripheral B-cell lymphoma in ATM-deficient mice. | Q34143276 | ||
| Mechanisms of chromosomal translocations in B cell lymphomas | Q34405423 | ||
| ATM stabilizes DNA double-strand-break complexes during V(D)J recombination. | Q34541828 | ||
| Pathways that suppress programmed DNA breaks from progressing to chromosomal breaks and translocations | Q34560703 | ||
| Abnormal development of Purkinje cells and lymphocytes in Atm mutant mice | Q35109704 | ||
| Translocation-capture sequencing reveals the extent and nature of chromosomal rearrangements in B lymphocytes | Q35304991 | ||
| The BCL11B tumor suppressor is mutated across the major molecular subtypes of T-cell acute lymphoblastic leukemia | Q35483684 | ||
| Ataxia-telangiectasia: diagnosis and treatment | Q35599431 | ||
| H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximity | Q35623763 | ||
| Spatial organization of the mouse genome and its role in recurrent chromosomal translocations | Q35874015 | ||
| Role of genomic instability and p53 in AID-induced c-myc-Igh translocations. | Q36146840 | ||
| Evidence for replicative repair of DNA double-strand breaks leading to oncogenic translocation and gene amplification | Q36376595 | ||
| Myc translocations in B cell and plasma cell neoplasms | Q36525088 | ||
| Telomere-related genome instability in cancer. | Q36547726 | ||
| Chimeric IgH-TCRalpha/delta translocations in T lymphocytes mediated by RAG. | Q36654350 | ||
| Complementary functions of ATM and H2AX in development and suppression of genomic instability | Q36756926 | ||
| The impact of translocations and gene fusions on cancer causation | Q36760027 | ||
| Lymphocyte development: integration of DNA damage response signaling | Q36846366 | ||
| The role of mechanistic factors in promoting chromosomal translocations found in lymphoid and other cancers | Q37781516 | ||
| Mechanisms that promote and suppress chromosomal translocations in lymphocytes | Q37827139 | ||
| Understanding the limitations of radiation-induced cell cycle checkpoints | Q37869479 | ||
| Chromosome translocation, B cell lymphoma, and activation-induced cytidine deaminase | Q38043045 | ||
| Classical and alternative end-joining pathways for repair of lymphocyte-specific and general DNA double-strand breaks | Q38051860 | ||
| Mechanisms of programmed DNA lesions and genomic instability in the immune system. | Q38078491 | ||
| The ATM protein kinase: regulating the cellular response to genotoxic stress, and more | Q38088935 | ||
| P433 | issue | 28 | |
| P407 | language of work or name | English | Q1860 |
| P304 | page(s) | 10269-10274 | |
| P577 | publication date | 2014-06-30 | |
| P1433 | published in | Proceedings of the National Academy of Sciences of the United States of America | Q1146531 |
| P1476 | title | Developmental propagation of V(D)J recombination-associated DNA breaks and translocations in mature B cells via dicentric chromosomes | |
| P478 | volume | 111 |
| Q37052916 | A Damage-Independent Role for 53BP1 that Impacts Break Order and Igh Architecture during Class Switch Recombination |
| Q64096310 | At the intersection of DNA damage and immune responses |
| Q92542160 | Breakage-Fusion-Bridge Events Trigger Complex Genome Rearrangements and Amplifications in Developmentally Arrested T Cell Lymphomas |
| Q28253489 | Convergent transcription at intragenic super-enhancers targets AID-initiated genomic instability |
| Q51603184 | DNA Breaks and End Resection Measured Genome-wide by End Sequencing. |
| Q64389011 | DNA double-strand breaks as drivers of neural genomic change, function, and disease |
| Q35975147 | Detecting DNA double-stranded breaks in mammalian genomes by linear amplification-mediated high-throughput genome-wide translocation sequencing |
| Q35060760 | Genome-wide detection of DNA double-stranded breaks induced by engineered nucleases. |
| Q38961577 | Genomic profiling of Acute lymphoblastic leukemia in ataxia telangiectasia patients reveals tight link between ATM mutations and chromothripsis |
| Q34143276 | Mechanisms that can promote peripheral B-cell lymphoma in ATM-deficient mice. |
| Q37197994 | Orientation-specific RAG activity in chromosomal loop domains contributes to Tcrd V(D)J recombination during T cell development |
| Q38716411 | Phosphatidylinositol 3-kinase δ blockade increases genomic instability in B cells |
| Q36646380 | Transcription-associated processes cause DNA double-strand breaks and translocations in neural stem/progenitor cells. |
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