scholarly article | Q13442814 |
P50 | author | Edward G Lynn | Q57362755 |
Lionel Opie | Q61046058 | ||
P2093 | author name string | Lionel H. Opie | |
Sandrine Lecour | |||
Lydia Lacerda | |||
Joy McCarthy | |||
Michael N. Sack | |||
Shazia F. K. Mungly | |||
P2860 | cites work | Cardiology | Q15751978 |
Methods and achievements in experimental pathology | Q27712087 | ||
Ceramide generation by two distinct pathways in tumor necrosis factor alpha-induced cell death | Q28213775 | ||
A simplification of the protein assay method of Lowry et al. which is more generally applicable | Q29615220 | ||
J-aggregate formation of a carbocyanine as a quantitative fluorescent indicator of membrane potential | Q33539001 | ||
The mitochondrial production of reactive oxygen species: mechanisms and implications in human pathology | Q34109999 | ||
Activation of the protective Survivor Activating Factor Enhancement (SAFE) pathway against reperfusion injury: Does it go beyond the RISK pathway? | Q34974091 | ||
Mitochondrial metabolism of reactive oxygen species. | Q36088215 | ||
Combined sphingosine, S1P and ischemic postconditioning rescue the heart after protracted ischemia | Q36109455 | ||
Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer | Q36229684 | ||
The mitochondrial permeability transition pore and ischemia-reperfusion injury | Q37163574 | ||
Sphingosine kinase regulation and cardioprotection | Q37328806 | ||
Mitochondrial pathways for ROS formation and myocardial injury: the relevance of p66(Shc) and monoamine oxidase. | Q37401135 | ||
PKCepsilon activation augments cardiac mitochondrial respiratory post-anoxic reserve--a putative mechanism in PKCepsilon cardioprotection. | Q39268222 | ||
Enzymatic aspects of the cardiac muscle cell: mitochondria, sarcoplasmic reticulum and nonovalent cation active transport system | Q39886263 | ||
A mitochondrial pool of sphingomyelin is involved in TNFalpha-induced Bax translocation to mitochondria. | Q40498233 | ||
Ischemic and pharmacological preconditioning in Girardi cells and C2C12 myotubes induce mitochondrial uncoupling | Q40772249 | ||
Flow cytometric analysis of mitochondrial activity in situ: application to acetylceramide-induced mitochondrial swelling and apoptosis. | Q40993875 | ||
Cytotoxic activity of tumor necrosis factor is mediated by early damage of mitochondrial functions. Evidence for the involvement of mitochondrial radical generation | Q41635173 | ||
Tumor necrosis factor in the heart. | Q41733185 | ||
Different mechanisms of mitochondrial proton leak in ischaemia/reperfusion injury and preconditioning: implications for pathology and cardioprotection | Q41832408 | ||
Inhibition of target cell mitochondrial electron transfer by tumor necrosis factor | Q42028118 | ||
A PKC-beta inhibitor protects against cardiac microvascular ischemia reperfusion injury in diabetic rats | Q43203675 | ||
Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway | Q43295255 | ||
Rapid reactive oxygen species production by mitochondria in endothelial cells exposed to tumor necrosis factor-alpha is mediated by ceramide | Q43646549 | ||
Mitochondrial respiratory chain adjustment to cellular energy demand | Q43752287 | ||
Myocardial dysfunction with coronary microembolization: signal transduction through a sequence of nitric oxide, tumor necrosis factor-alpha, and sphingosine | Q43966261 | ||
Identification of a novel role for sphingolipid signaling in TNF alpha and ischemic preconditioning mediated cardioprotection | Q44022447 | ||
Classic ischemic but not pharmacologic preconditioning is abrogated following genetic ablation of the TNFalpha gene | Q44088296 | ||
Decreased ATP-synthesis ability of liver mitochondria in spontaneously hypertensive rats (SHR): role of calcium overload of the mitochondria | Q44257441 | ||
Delayed ischemic preconditioning activates nuclear-encoded electron-transfer-chain gene expression in parallel with enhanced postanoxic mitochondrial respiratory recovery | Q44995483 | ||
Sphingosine kinase activation mediates ischemic preconditioning in murine heart | Q45081177 | ||
Presence of connexin 43 in subsarcolemmal, but not in interfibrillar cardiomyocyte mitochondria. | Q46109567 | ||
Angiotensin II downregulates the fatty acid oxidation pathway in adult rat cardiomyocytes via release of tumour necrosis factor-alpha | Q46170790 | ||
Ischemic postconditioning in pigs: no causal role for RISK activation. | Q46225223 | ||
Ceramide-induced preconditioning involves reactive oxygen species | Q46722860 | ||
TNFalpha-induced cytoprotection requires the production of free radicals within mitochondria in C2C12 myotubes | Q46737977 | ||
Pharmacological preconditioning with tumor necrosis factor-alpha activates signal transducer and activator of transcription-3 at reperfusion without involving classic prosurvival kinases (Akt and extracellular signal-regulated kinase). | Q46847925 | ||
Protection of cardiac mitochondria by overexpression of MnSOD reduces diabetic cardiomyopathy | Q46966545 | ||
Uncoupling proteins 2 and 3 function in concert to augment tolerance to cardiac ischemia | Q47752894 | ||
Bidirectional role of tumor necrosis factor-alpha in coronary microembolization: progressive contractile dysfunction versus delayed protection against infarction. | Q51093781 | ||
Tumour necrosis factor-alpha uncouples respiration in isolated rat mitochondria. | Q51656671 | ||
Tumor necrosis factor-alpha and its receptors 1 and 2: Yin and Yang in myocardial infarction? | Q51789753 | ||
Tumor necrosis factor receptor 1 signaling resistance in the female myocardium during ischemia. | Q51797007 | ||
Reactive Oxygen Species Mediate Cellular Damage in Alzheimer Disease. | Q53250272 | ||
Tumor necrosis factor-alpha is released from the isolated heart undergoing ischemia and reperfusion. | Q54125180 | ||
Mitochondrial respiratory parameters in cardiac tissue: a novel method of assessment by using saponin-skinned fibers | Q69052166 | ||
Mitochondrial membrane potential in single living adult rat cardiac myocytes exposed to anoxia or metabolic inhibition | Q71750623 | ||
Expression and Functional Significance of Tumor Necrosis Factor Receptors in Human Myocardium | Q72034108 | ||
Characterization of a transient outward K+ current with inward rectification in canine ventricular myocytes | Q74408392 | ||
Tumour necrosis factor is trafficked to a mitochondrial tumour necrosis factor binding protein | Q74521421 | ||
Coronary microembolization: the role of TNF-alpha in contractile dysfunction | Q77548701 | ||
Tumor necrosis factor is delivered to mitochondria where a tumor necrosis factor-binding protein is localized | Q77769082 | ||
Ethnic and gender differences in ambulatory blood pressure trajectories: results from a 15-year longitudinal study in youth and young adults | Q79386617 | ||
Inhibition of mitochondrial permeability transition pore opening: the Holy Grail of cardioprotection | Q82471465 | ||
Ischemic preconditioning or p38 MAP kinase inhibition attenuates myocardial TNF alpha production and mitochondria damage in brief myocardial ischemia | Q82645102 | ||
P433 | issue | 6 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 751-762 | |
P577 | publication date | 2010-08-01 | |
P1433 | published in | Basic Research in Cardiology | Q2453360 |
P1476 | title | TNFα protects cardiac mitochondria independently of its cell surface receptors | |
P478 | volume | 105 |
Q36011190 | A novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection. |
Q35680234 | Central TNF inhibition results in attenuated neurohumoral excitation in heart failure: a role for superoxide and nitric oxide. |
Q42861824 | Ethanolamine is a novel STAT-3 dependent cardioprotective agent |
Q28073354 | From basic mechanisms to clinical applications in heart protection, new players in cardiovascular diseases and cardiac theranostics: meeting report from the third international symposium on "New frontiers in cardiovascular research" |
Q36895483 | High density lipoprotein/sphingosine-1-phosphate-induced cardioprotection: Role of STAT3 as part of the SAFE pathway |
Q33865316 | Impairment of pH gradient and membrane potential mediates redox dysfunction in the mitochondria of the post-ischemic heart. |
Q35835539 | Inflammation and cardiac outcome |
Q36355264 | Influence of tumour necrosis factor alpha on the outcome of ischaemic postconditioning in the presence of obesity and diabetes |
Q64936967 | Inhibition of CTRP9, a novel and cardiac-abundantly expressed cell survival molecule, by TNFα-initiated oxidative signaling contributes to exacerbated cardiac injury in diabetic mice. |
Q45329333 | Interaction between AT1 receptor and NF-κB in hypothalamic paraventricular nucleus contributes to oxidative stress and sympathoexcitation by modulating neurotransmitters in heart failure |
Q30399909 | Interaction between interleukin-1 beta and angiotensin II receptor 1 in hypothalamic paraventricular nucleus contributes to progression of heart failure |
Q46273075 | Interplay between SAFE and RISK pathways in sphingosine-1-phosphate-induced cardioprotection |
Q28072918 | Ischaemic conditioning and targeting reperfusion injury: a 30 year voyage of discovery |
Q35492308 | Mitochondria to nucleus translocation of AIF in mice lacking Hsp70 during ischemia/reperfusion |
Q41829826 | Myocardial infarction differentially alters sphingolipid levels in plasma, erythrocytes and platelets of the rat. |
Q35707188 | NF-κB in the paraventricular nucleus modulates neurotransmitters and contributes to sympathoexcitation in heart failure |
Q51872466 | Oxidative stress-induced formation of a positive-feedback loop for the sustained activation of p38 MAPK leading to the loss of cell division in cardiomyocytes soon after birth. |
Q35054906 | Paraventricular nucleus corticotrophin releasing hormone contributes to sympathoexcitation via interaction with neurotransmitters in heart failure |
Q48266257 | Remote intermittent ischemia before coronary artery bypass graft surgery: a strategy to reduce injury and inflammation? |
Q54390219 | STAT3α interacts with nuclear GSK3beta and cytoplasmic RISK pathway and stabilizes rhythm in the anoxic-reoxygenated embryonic heart. |
Q38078795 | Skeletal muscle mitochondrial dysfunction during chronic obstructive pulmonary disease: central actor and therapeutic target |
Q41239818 | TNFR2 Stimulation Promotes Mitochondrial Fusion via Stat3- and NF-kB-Dependent Activation of OPA1 Expression |
Q36102258 | The Effect of Lipopolysaccharide on Ischemic-Reperfusion Injury of Heart: A Double Hit Model of Myocardial Ischemia and Endotoxemia |
Q48238697 | The SAFE pathway for cardioprotection: is this a promising target? |
Q34809985 | The cardiokine story unfolds: ischemic stress-induced protein secretion in the heart |
Q45773481 | The effect of respiration buffer composition on mitochondrial metabolism and function |
Q38839545 | Tumor Necrosis Factor-α Promotes Phosphoinositide 3-Kinase Enhancer A and AMP-Activated Protein Kinase Interaction to Suppress Lipid Oxidation in Skeletal Muscle |
Q51688058 | Tumor necrosis factor alpha protects heart cultures against hypoxic damage via activation of PKA and phospholamban to prevent calcium overload. |
Q38901042 | Tumor necrosis factor alpha stimulates p62 accumulation and enhances proteasome activity independently of ROS. |
Q89985638 | Tumor necrosis factor receptor-2 signaling pathways promote survival of cancer stem-like CD133+ cells in clear cell renal carcinoma |
Q54953158 | Tumor necrosis factor-α and its role as a mediator in myocardial infarction: A brief review. |
Q37838085 | When are pro-inflammatory cytokines SAFE in heart failure? |
Search more.