scholarly article | Q13442814 |
P50 | author | Roisin F. Kelly | Q42861892 |
Lionel Opie | Q61046058 | ||
P2093 | author name string | Paul Thomas | |
Lionel H. Opie | |||
Sarin Somers | |||
Damian Hacking | |||
Sandrine Lecour | |||
Lydia Lacerda | |||
Kim T. Lamont | |||
P2860 | cites work | American Journal of Physiology | Q2160146 |
Overexpression of human copper, zinc-superoxide dismutase (SOD1) prevents postischemic injury | Q24323877 | ||
Sphingosine-1-phosphate as a mediator of high-density lipoprotein effects in cardiovascular protection | Q28235893 | ||
Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning | Q28771386 | ||
Modulation of anxiety through blockade of anandamide hydrolysis | Q29615902 | ||
Sphingosine-1-phosphate: an enigmatic signalling lipid | Q29620563 | ||
Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury | Q33195698 | ||
Preconditioning protects by inhibiting the mitochondrial permeability transition | Q33201290 | ||
An essential role of the JAK-STAT pathway in ischemic preconditioning | Q33929635 | ||
Translating novel strategies for cardioprotection: the Hatter Workshop Recommendations | Q34247485 | ||
New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway | Q34298555 | ||
The red wine hypothesis: from concepts to protective signalling molecules | Q34636347 | ||
Activation of the protective Survivor Activating Factor Enhancement (SAFE) pathway against reperfusion injury: Does it go beyond the RISK pathway? | Q34974091 | ||
Signal transducer and activator of transcription 3 is involved in the cardioprotective signalling pathway activated by insulin therapy at reperfusion | Q36099739 | ||
Genetic depletion of cardiac myocyte STAT-3 abolishes classical preconditioning | Q80440977 | ||
Cardioprotection by ischemic postconditioning is lost in aged and STAT3-deficient mice | Q81514899 | ||
Inhibition of mitochondrial permeability transition pore opening: the Holy Grail of cardioprotection | Q82471465 | ||
No risk, no ... cardioprotection? A critical perspective | Q84480416 | ||
The reperfusion injury salvage kinase pathway: a common target for both ischemic preconditioning and postconditioning. | Q36122804 | ||
TNFα protects cardiac mitochondria independently of its cell surface receptors | Q36148170 | ||
Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore | Q36148247 | ||
Structure and function of fatty acid amide hydrolase | Q36161171 | ||
Pharmacological profile of the selective FAAH inhibitor KDS-4103 (URB597). | Q36533477 | ||
Reperfusion injury salvage kinase signalling: taking a RISK for cardioprotection. | Q36836773 | ||
HDL and its sphingosine-1-phosphate content in cardioprotection | Q36842504 | ||
The myocardial JAK/STAT pathway: from protection to failure | Q37266843 | ||
Function of mitochondrial Stat3 in cellular respiration | Q37378567 | ||
TNFalpha in atherosclerosis, myocardial ischemia/reperfusion and heart failure | Q37771751 | ||
Effect of synthetic and natural phospholipids on N-acylphosphatidylethanolamine-hydrolyzing phospholipase D activity. | Q39806718 | ||
Anandamide protects from low serum-induced apoptosis via its degradation to ethanolamine | Q40182562 | ||
The JAK/STAT pathway is essential for opioid-induced cardioprotection: JAK2 as a mediator of STAT3, Akt, and GSK-3 beta | Q40308442 | ||
Ischemic and pharmacological preconditioning in Girardi cells and C2C12 myotubes induce mitochondrial uncoupling | Q40772249 | ||
Nano-liquid chromatography analysis of dansylated biogenic amines in wines | Q42164747 | ||
Anandamide reduces infarct size in rat isolated hearts subjected to ischaemia-reperfusion by a novel cannabinoid mechanism. | Q42844430 | ||
Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway | Q43295255 | ||
Role of STAT3 in ischemic preconditioning | Q43799529 | ||
Identification of a novel role for sphingolipid signaling in TNF alpha and ischemic preconditioning mediated cardioprotection | Q44022447 | ||
Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning? | Q44088292 | ||
Classic ischemic but not pharmacologic preconditioning is abrogated following genetic ablation of the TNFalpha gene | Q44088296 | ||
Ethanolamine and phosphoethanolamine inhibit mitochondrial function in vitro: implications for mitochondrial dysfunction hypothesis in depression and bipolar disorder | Q44742906 | ||
Reversible inhibitors of fatty acid amide hydrolase that promote analgesia: evidence for an unprecedented combination of potency and selectivity | Q44903331 | ||
Erythropoietin protects the infant heart against ischemia-reperfusion injury by triggering multiple signaling pathways | Q45196279 | ||
Free radicals trigger TNF alpha-induced cardioprotection | Q45203259 | ||
The cannabinoid CB1 receptor antagonist, rimonabant, protects against acute myocardial infarction. | Q46001116 | ||
TNFalpha is required to confer protection in an in vivo model of classical ischaemic preconditioning | Q46224947 | ||
Ischemic postconditioning in pigs: no causal role for RISK activation. | Q46225223 | ||
Sphingosine 1-phosphate S1P2 and S1P3 receptor-mediated Akt activation protects against in vivo myocardial ischemia-reperfusion injury | Q46503010 | ||
Mitochondrial permeability transition pore as a target for cardioprotection in the human heart | Q46551313 | ||
Sphingosine can pre- and post-condition heart and utilizes a different mechanism from sphingosine 1-phosphate | Q46639803 | ||
TNFalpha-induced cytoprotection requires the production of free radicals within mitochondria in C2C12 myotubes | Q46737977 | ||
The fatty-acid amide hydrolase inhibitor URB597 does not affect triacylglycerol hydrolysis in rat tissues | Q46772397 | ||
Pharmacological preconditioning with tumor necrosis factor-alpha activates signal transducer and activator of transcription-3 at reperfusion without involving classic prosurvival kinases (Akt and extracellular signal-regulated kinase). | Q46847925 | ||
Ischaemic preconditioning reduces infarct size following global ischaemia in the murine myocardium | Q47275705 | ||
The stimulatory effect of phosphatidylethanolamine on N-acylphosphatidylethanolamine-hydrolyzing phospholipase D (NAPE-PLD). | Q48094438 | ||
Plasmalogens in human serum positively correlate with high- density lipoprotein and decrease with aging. | Q51476537 | ||
Multiple protective pathways against reperfusion injury: a SAFE path without Aktion? | Q51752588 | ||
Changes of yolk biogenic amine concentrations during storage of shell hen eggs | Q58854766 | ||
Occurrence and evolution of amino acids during grape must cooking | Q59198719 | ||
Cardioprotection: nitric oxide, protein kinases, and mitochondria | Q79780264 | ||
High-density lipoproteins and their constituent, sphingosine-1-phosphate, directly protect the heart against ischemia/reperfusion injury in vivo via the S1P3 lysophospholipid receptor | Q80284338 | ||
P433 | issue | 6 | |
P407 | language of work or name | English | Q1860 |
P304 | page(s) | 763-770 | |
P577 | publication date | 2010-10-12 | |
P1433 | published in | Basic Research in Cardiology | Q2453360 |
P1476 | title | Ethanolamine is a novel STAT-3 dependent cardioprotective agent | |
P478 | volume | 105 |