scholarly article | Q13442814 |
P2093 | author name string | B Lamarca | |
P2860 | cites work | Eicosanoids in preeclampsia | Q28166550 |
Soluble endoglin contributes to the pathogenesis of preeclampsia | Q28244053 | ||
Endothelial NO synthase genotype and risk of preeclampsia: a multicenter case-control study | Q28281936 | ||
Cerebrovascular function in pregnancy and eclampsia | Q28305037 | ||
Role of endothelin in mediating soluble fms-like tyrosine kinase 1-induced hypertension in pregnant rats | Q28571017 | ||
Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
Changes in endothelial function precede the clinical disease in women in whom preeclampsia develops | Q33225347 | ||
Cerebral blood flow autoregulation and edema formation during pregnancy in anesthetized rats. | Q33268023 | ||
Effects of 17-hydroxyprogesterone on tumor necrosis factor-alpha-induced hypertension during pregnancy | Q33607465 | ||
Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
Estradiol enhances leukocyte binding to tumor necrosis factor (TNF)-stimulated endothelial cells via an increase in TNF-induced adhesion molecules E-selectin, intercellular adhesion molecule type 1, and vascular cell adhesion molecule type 1 | Q34117998 | ||
Summary of the NHLBI Working Group on Research on Hypertension During Pregnancy | Q34182166 | ||
Vitamin D effects on pregnancy and the placenta | Q34361116 | ||
Vitamin C and vitamin E in pregnant women at risk for pre-eclampsia (VIP trial): randomised placebo-controlled trial. | Q34514153 | ||
Vitamins C and E and the risks of preeclampsia and perinatal complications | Q34518082 | ||
Pathophysiology of preeclampsia: linking placental ischemia/hypoxia with microvascular dysfunction. | Q34702420 | ||
Induction of heme oxygenase 1 attenuates placental ischemia-induced hypertension | Q34904184 | ||
Hypertension in response to placental ischemia during pregnancy: role of B lymphocytes | Q35029700 | ||
CD4+ T-helper cells stimulated in response to placental ischemia mediate hypertension during pregnancy | Q35130793 | ||
Preeclampsia, a disease of the maternal endothelium: the role of antiangiogenic factors and implications for later cardiovascular disease | Q35143007 | ||
Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction | Q35576417 | ||
Heme Oxygenase-1 Attenuates Hypoxia-Induced sFlt-1 and Oxidative Stress in Placental Villi through Its Metabolic Products CO and Bilirubin | Q35615008 | ||
Potential roles of angiotensin receptor-activating autoantibody in the pathophysiology of preeclampsia | Q35681821 | ||
Oxidative stress and preeclampsia: rationale for antioxidant clinical trials | Q35869763 | ||
L-arginine supplementation abolishes the blood pressure and endothelin response to chronic increases in plasma sFlt-1 in pregnant rats | Q35950303 | ||
Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic rats | Q35994304 | ||
Role of the renin-angiotensin system in the pathogenesis of preeclampsia | Q36064478 | ||
Preeclampsia serum upregulates CD40/CD40L expression and induces apoptosis in human umbilical cord endothelial cells | Q36112505 | ||
Hypertension in response to CD4(+) T cells from reduced uterine perfusion pregnant rats is associated with activation of the endothelin-1 system. | Q36115279 | ||
Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation | Q36244980 | ||
Preeclampsia: recent insights | Q36288757 | ||
Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia | Q36355165 | ||
Angiotensin II type 1 receptor antibodies and increased angiotensin II sensitivity in pregnant rats | Q36683260 | ||
Immunoregulation in normal pregnancy and pre-eclampsia: an overview | Q36684096 | ||
Pathophysiology of hypertension in response to placental ischemia during pregnancy: a central role for endothelin? | Q37131819 | ||
First trimester vitamin D, vitamin D binding protein, and subsequent preeclampsia | Q37178546 | ||
Elevated asymmetric dimethylarginine concentrations precede clinical preeclampsia, but not pregnancies with small-for-gestational-age infants | Q37181052 | ||
Hypertension in response to autoantibodies to the angiotensin II type I receptor (AT1-AA) in pregnant rats: role of endothelin-1. | Q37447371 | ||
Endothelial dysfunction: a link among preeclampsia, recurrent pregnancy loss, and future cardiovascular events? | Q40268212 | ||
Preeclampsia: an endothelial cell disorder | Q41281924 | ||
Importance of prostaglandins in hypertension during reduced uteroplacental perfusion pressure | Q41284596 | ||
Placental cytokines and the pathogenesis of preeclampsia | Q41452017 | ||
Association of maternal endothelial dysfunction with preeclampsia | Q43553727 | ||
Reduced uterine perfusion pressure during pregnancy in the rat is associated with increases in arterial pressure and changes in renal nitric oxide. | Q43576536 | ||
Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction | Q43744794 | ||
Enhanced thromboxane synthesis during chronic reductions in uterine perfusion pressure in pregnant rats | Q44130345 | ||
Reduced endothelial NO-cGMP-mediated vascular relaxation and hypertension in IL-6-infused pregnant rats | Q44715072 | ||
L-arginine depletion in preeclampsia orients nitric oxide synthase toward oxidant species | Q44743355 | ||
Cytochrome P-450 inhibition attenuates hypertension induced by reductions in uterine perfusion pressure in pregnant rats | Q44752496 | ||
L-arginine attenuates hypertension in pregnant rats with reduced uterine perfusion pressure | Q44760481 | ||
TNF alpha concentrations and mRNA expression are increased in preeclamptic placentas | Q45102738 | ||
Maternal-placental interactions of oxidative stress and antioxidants in preeclampsia | Q45112360 | ||
Low-molecular-weight heparin lowers the recurrence rate of preeclampsia and restores the physiological vascular changes in angiotensin-converting enzyme DD women | Q45158743 | ||
S-nitrosoalbumin-mediated relaxation is enhanced by ascorbate and copper: effects in pregnancy and preeclampsia plasma | Q45166655 | ||
Lectin-like oxidized low-density lipoprotein 1 receptor in a reduced uteroplacental perfusion pressure rat model of preeclampsia | Q46324781 | ||
Systemic increase in the ratio between Foxp3+ and IL-17-producing CD4+ T cells in healthy pregnancy but not in preeclampsia | Q46332630 | ||
Circulating factors as markers and mediators of endothelial cell dysfunction in preeclampsia | Q46433836 | ||
System y+ arginine transport and NO production in peripheral blood mononuclear cells in pregnancy and preeclampsia | Q46847905 | ||
Pregnancy prevents hypertensive remodeling of cerebral arteries: a potential role in the development of eclampsia | Q46870281 | ||
Enhanced endothelin synthesis by endothelial cells exposed to sera from pregnant rats with decreased uterine perfusion | Q46877931 | ||
Women with preeclampsia have higher plasma endothelin levels than women with normal pregnancies | Q46878600 | ||
Moderate ascorbate deficiency increases myogenic tone of arteries from pregnant but not virgin ascorbate-dependent rats | Q46907903 | ||
Insulin resistance in preeclampsia | Q46919162 | ||
Implication of an AGT haplotype in a multigene association study with pregnancy hypertension | Q47364068 | ||
Preeclampsia is associated with lower percentages of regulatory T cells in maternal blood | Q47890265 | ||
Insulin resistance and alterations in angiogenesis: additive insults that may lead to preeclampsia | Q47945074 | ||
Decreased endothelium-dependent vascular relaxation during reduction of uterine perfusion pressure in pregnant rat. | Q50514586 | ||
Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of interleukin 6. | Q51139503 | ||
Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of tumor necrosis factor-alpha. | Q51359651 | ||
Lipoprotein subfraction changes in normal pregnancy: threshold effect of plasma triglyceride on appearance of small, dense low density lipoprotein. | Q51574204 | ||
Fatty acid pattern of esterified and free fatty acids in sera of women with normal and pre‐eclamptic pregnancy | Q51587356 | ||
Short- and long-term changes in plasma inflammatory markers associated with preeclampsia. | Q52087250 | ||
Relation between circulating angiotensin II type 1 receptor agonistic autoantibodies and soluble fms-like tyrosine kinase 1 in the pathogenesis of preeclampsia. | Q52933863 | ||
Agonistic autoantibodies to the AT1 receptor in a transgenic rat model of preeclampsia. | Q52940276 | ||
Soluble endoglin and other circulating antiangiogenic factors in preeclampsia. | Q53600812 | ||
Role of endothelin in mediating tumor necrosis factor-induced hypertension in pregnant rats. | Q54661322 | ||
Agonistic Angiotensin II Type 1 Receptor Autoantibodies in Postpartum Women With a History of Preeclampsia | Q56981931 | ||
Dysregulation of the Circulating and Tissue-Based Renin-Angiotensin System in Preeclampsia | Q56981959 | ||
In Preeclampsia, the Circulating Factors Capable of Altering In Vitro Endothelial Function Precede Clinical Disease | Q58035998 | ||
Changes in Endothelial Function Precede the Clinical Disease in Women in Whom Preeclampsia Develops | Q61830910 | ||
Cardiovascular Function in Mice During Normal Pregnancy and in the Absence of Endothelial NO Synthase | Q61964364 | ||
The effect of over-expression of sFlt-1 on blood pressure and the occurrence of other manifestations of preeclampsia in unrestrained conscious pregnant mice | Q64377179 | ||
Maternal plasma level of endothelin is increased in preeclampsia | Q67973452 | ||
Maternal and fetal serum nitric oxide (NO) concentrations in normal pregnancy, pre-eclampsia and eclampsia | Q73502640 | ||
Endothelin type a receptor blockade attenuates the hypertension in response to chronic reductions in uterine perfusion pressure | Q73567364 | ||
Nitric oxide production with preeclampsia | Q74601245 | ||
Plasma lipids and vascular dysfunction in preeclampsia | Q74757931 | ||
Plasma and 24-h NO(x) and cGMP during normal pregnancy and preeclampsia in women on a reduced NO(x) diet | Q78009520 | ||
Maternal fetal/placental interactions and abnormal pregnancy outcomes | Q79372322 | ||
Difference in neutrophil superoxide generation during pregnancy between preeclampsia and essential hypertension | Q80125849 | ||
Questionable role of the angiotensin II receptor subtype 1 autoantibody in the pathogenesis of preeclampsia | Q80336129 | ||
Generation of reactive oxygen species by neutrophils and endothelial cell injury in normal and preeclamptic pregnancies | Q81236313 | ||
Hypertension during pregnancy: a disorder begging for pathophysiological support | Q81397976 | ||
Angiotensin II type 1 receptor agonistic antibodies reflect fundamental alterations in the uteroplacental vasculature | Q81423834 | ||
Uterine artery Doppler and changes in endothelial function before clinical disease in preeclamptic women | Q82615541 | ||
P433 | issue | 4 | |
P921 | main subject | maternal health | Q6786626 |
arterial hypertension | Q41861 | ||
pre-eclampsia | Q61335 | ||
endothelium | Q111140 | ||
pathophysiology | Q1135939 | ||
P304 | page(s) | 309-320 | |
P577 | publication date | 2012-08-01 | |
P1433 | published in | Minerva Ginecologica | Q15752699 |
P1476 | title | Endothelial dysfunction. An important mediator in the pathophysiology of hypertension during pre-eclampsia | |
P478 | volume | 64 |
Q92180156 | A cross sectional study to assess the sFlt-1:PlGF ratio in pregnant women with and without preeclampsia |
Q53700889 | AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic Rats. |
Q38684330 | Dysregulation of the Fas/FasL system in an experimental animal model of HELLP syndrome. |
Q35547367 | Endocan, a putative endothelial cell marker, is elevated in preeclampsia, decreased in acute pyelonephritis, and unchanged in other obstetrical syndromes |
Q37512393 | Endothelin-1 down-regulates matrix metalloproteinase 14 and 15 expression in human first trimester trophoblasts via endothelin receptor type B. |
Q35800606 | Expression profile of C19MC microRNAs in placental tissue in pregnancy-related complications |
Q42679579 | Expression profile of C19MC microRNAs in placental tissue of patients with preterm prelabor rupture of membranes and spontaneous preterm birth. |
Q98613250 | Family history of chronic illness, preterm gestational age and smoking exposure before pregnancy increases the probability of preeclampsia in Omo district in southern Ethiopia: a case-control study |
Q35979122 | Heart Rate Variability and Autonomic Modulations in Preeclampsia |
Q34025660 | Hypertension, inflammation and T lymphocytes are increased in a rat model of HELLP syndrome |
Q88503866 | Impaired function of cerebral parenchymal arterioles in experimental preeclampsia |
Q55223615 | Int6/eIF3e Silencing Promotes Placenta Angiogenesis in a Rat Model of Pre-eclampsia. |
Q35690758 | Involvement of Visceral Adipose Tissue in Immunological Modulation of Inflammatory Cascade in Preeclampsia |
Q35883182 | Is the serum l-arginine level during early pregnancy a predictor of pregnancy-induced hypertension? |
Q91962935 | Natural killer cells contribute to mitochondrial dysfunction in response to placental ischemia in reduced uterine perfusion pressure rats |
Q64226376 | Placental CX3CL1 is Deregulated by Angiotensin II and Contributes to a Pro-Inflammatory Trophoblast-Monocyte Interaction |
Q57177277 | Preeclampsia and Related Cardiovascular Risk: Common Genetic Background |
Q36838343 | Preeclampsia is a biomarker for vascular disease in both mother and child: the need for a medical alert system |
Q26800272 | Preeclampsia: long-term consequences for vascular health |
Q41387285 | Red cell distribution width predicts incident dipstick albuminuria in Korean adults without chronic kidney disease |
Q50102668 | The profiles of soluble adhesion molecules in the "great obstetrical syndromes". |
Q51294484 | Vitamin D supplementation reduces some AT1-AA-induced downstream targets implicated in preeclampsia including hypertension. |
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