| scholarly article | Q13442814 |
| P50 | author | Kedra Wallace | Q56256861 |
| Babbette LaMarca | Q88459247 | ||
| P2093 | author name string | Marc R Parrish | |
| P2860 | cites work | Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 |
| Angiotensin receptor agonistic autoantibody is highly prevalent in preeclampsia: correlation with disease severity | Q33748680 | ||
| Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
| Recent Insights into the pathogenesis of pre-eclampsia | Q34682287 | ||
| Pathophysiology of preeclampsia: linking placental ischemia/hypoxia with microvascular dysfunction. | Q34702420 | ||
| Hypertension in response to placental ischemia during pregnancy: role of B lymphocytes | Q35029700 | ||
| Angiotensin II type 1 autoantibody induced hypertension during pregnancy is associated with renal endothelial dysfunction | Q35576417 | ||
| Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling | Q35681804 | ||
| Potential roles of angiotensin receptor-activating autoantibody in the pathophysiology of preeclampsia | Q35681821 | ||
| Oxidative stress and preeclampsia: rationale for antioxidant clinical trials | Q35869763 | ||
| Hypoxia and sFlt-1 in preeclampsia: the "chicken-and-egg" question | Q35920602 | ||
| Activating auto-antibodies against the AT1 receptor in preeclampsia | Q36010629 | ||
| Role of the renin-angiotensin system in the pathogenesis of preeclampsia | Q36064478 | ||
| B cells: a fundamental role in the pathogenesis of rheumatoid arthritis? | Q36106119 | ||
| Preeclampsia: a renal perspective | Q36120940 | ||
| Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation | Q36244980 | ||
| Circulating angiogenic factors in the pathogenesis and prediction of preeclampsia | Q36288766 | ||
| Agonistic antibodies directed at the angiotensin II, AT1 receptor in preeclampsia | Q36380482 | ||
| The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy | Q36407259 | ||
| Treatment of severe pemphigus with rituximab: report of 12 cases and a review of the literature | Q36914728 | ||
| Pathophysiology of hypertension during preeclampsia: linking placental ischemia with endothelial dysfunction | Q37022723 | ||
| Recent progress toward the understanding of the pathophysiology of hypertension during preeclampsia | Q37079628 | ||
| Autoantibodies to the angiotensin type I receptor in response to placental ischemia and tumor necrosis factor alpha in pregnant rats | Q37436219 | ||
| Hypertension in response to autoantibodies to the angiotensin II type I receptor (AT1-AA) in pregnant rats: role of endothelin-1. | Q37447371 | ||
| Role of reactive oxygen species in hypertension produced by reduced uterine perfusion in pregnant rats | Q37449704 | ||
| Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human trophoblast cells | Q44320558 | ||
| AT1 receptor agonistic antibodies from preeclamptic patients stimulate NADPH oxidase | Q44385661 | ||
| Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation | Q45069619 | ||
| Maternal-placental interactions of oxidative stress and antioxidants in preeclampsia | Q45112360 | ||
| Low-molecular-weight heparin lowers the recurrence rate of preeclampsia and restores the physiological vascular changes in angiotensin-converting enzyme DD women | Q45158743 | ||
| Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human mesangial cells and induce interleukin-6 and plasminogen activator inhibitor-1 secretion | Q46344658 | ||
| Enhanced endothelin synthesis by endothelial cells exposed to sera from pregnant rats with decreased uterine perfusion | Q46877931 | ||
| Implication of an AGT haplotype in a multigene association study with pregnancy hypertension | Q47364068 | ||
| Extra-placental expression of vascular endothelial growth factor receptor-1, (Flt-1) and soluble Flt-1 (sFlt-1), by peripheral blood mononuclear cells (PBMCs) in normotensive and preeclamptic pregnant women | Q47770771 | ||
| Endothelial dysfunction in preeclampsia | Q47879591 | ||
| Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of tumor necrosis factor-alpha. | Q51359651 | ||
| Agonistic Angiotensin II Type 1 Receptor Autoantibodies in Postpartum Women With a History of Preeclampsia | Q56981931 | ||
| Dysregulation of the Circulating and Tissue-Based Renin-Angiotensin System in Preeclampsia | Q56981959 | ||
| Epidemiology of preeclampsia and eclampsia in the United States, 1979-1986 | Q64128968 | ||
| AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor | Q73815485 | ||
| Severe persistent pemphigoid gestationis: long-term remission with rituximab | Q80439219 | ||
| Hypertension produced by reduced uterine perfusion in pregnant rats is associated with increased soluble fms-like tyrosine kinase-1 expression | Q81411178 | ||
| Angiotensin II type 1 receptor agonistic antibodies reflect fundamental alterations in the uteroplacental vasculature | Q81423834 | ||
| Reduced uterine perfusion pressure (RUPP) model for studying cardiovascular-renal dysfunction in response to placental ischemia | Q82705098 | ||
| Hypertension in response to AT1-AA: role of reactive oxygen species in pregnancy-induced hypertension | Q83799951 | ||
| P433 | issue | 3 | |
| P921 | main subject | pathophysiology | Q1135939 |
| pre-eclampsia | Q61335 | ||
| autoantibody | Q785022 | ||
| P304 | page(s) | 139-146 | |
| P577 | publication date | 2012-04-11 | |
| P1433 | published in | Gender Medicine | Q15816303 |
| P1476 | title | Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia | |
| P478 | volume | 9 |
| Q38126570 | A complicated role for the renin-angiotensin system during pregnancy: highlighting the importance of drug discovery |
| Q27027039 | Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyond |
| Q28534198 | Antibodies against AT1 receptors are associated with vascular endothelial and smooth muscle function impairment: protective effects of hydroxysafflor yellow A |
| Q28076193 | Antibody Subclass Repertoire and Graft Outcome Following Solid Organ Transplantation |
| Q36339273 | Blockade of CD40 ligand for intercellular communication reduces hypertension, placental oxidative stress, and AT1-AA in response to adoptive transfer of CD4+ T lymphocytes from RUPP rats |
| Q91564905 | Characterisation of the Selective Reduced Uteroplacental Perfusion (sRUPP) Model of Preeclampsia |
| Q26852668 | Elucidating immune mechanisms causing hypertension during pregnancy |
| Q37229246 | Endothelial dysfunction. An important mediator in the pathophysiology of hypertension during pre-eclampsia |
| Q34072217 | Exposure to AT1 receptor autoantibodies during pregnancy increases susceptibility of the maternal heart to postpartum ischemia-reperfusion injury in rats |
| Q55004295 | Maternal plasma fetuin-A concentration is lower in patients who subsequently developed preterm preeclampsia than in uncomplicated pregnancy: a longitudinal study. |
| Q26865332 | Pathophysiology of hypertension in pre-eclampsia: a lesson in integrative physiology |
| Q36838343 | Preeclampsia is a biomarker for vascular disease in both mother and child: the need for a medical alert system |
| Q48523077 | Preeclampsia: From Inflammation to Immunoregulation |
| Q33834419 | The role of inflammation in the pathology of preeclampsia |
| Q38116097 | Tumor necrosis factor-alpha, interleukin-6, and interleukin-10 levels are altered in preeclampsia: a systematic review and meta-analysis. |
Search more.