scholarly article | Q13442814 |
P50 | author | Susan M. Ramin | Q110252023 |
P2093 | author name string | Yang Xia | |
Rodney E Kellems | |||
Roxanna A Irani | |||
Sean C Blackwell | |||
Athar H Siddiqui | |||
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Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
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Latest advances in understanding preeclampsia | Q34425288 | ||
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AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor | Q73815485 | ||
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Explaining and predicting preeclampsia | Q80223255 | ||
Hypertension produced by reduced uterine perfusion in pregnant rats is associated with increased soluble fms-like tyrosine kinase-1 expression | Q81411178 | ||
Angiotensin II type 1 receptor agonistic antibodies reflect fundamental alterations in the uteroplacental vasculature | Q81423834 | ||
Pre-eclampsia | Q81446389 | ||
P433 | issue | 2 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | pre-eclampsia | Q61335 |
autoantibody | Q785022 | ||
maternal health | Q6786626 | ||
P304 | page(s) | 386-393 | |
P577 | publication date | 2009-12-07 | |
P1433 | published in | Hypertension | Q5958695 |
P1476 | title | Angiotensin receptor agonistic autoantibody is highly prevalent in preeclampsia: correlation with disease severity | |
P478 | volume | 55 |
Q37082096 | 1,25(OH)2D3 Induces Placental Vascular Smooth Muscle Cell Relaxation by Phosphorylation of Myosin Phosphatase Target Subunit 1Ser507: Potential Beneficial Effects of Vitamin D on Placental Vasculature in Humans |
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Q36355165 | Agonistic autoantibodies to the angiotensin II type I receptor cause pathophysiologic characteristics of preeclampsia |
Q90305559 | Angiotensin II Type 1 Receptor Autoantibodies in Primary Aldosteronism |
Q36683260 | Angiotensin II type 1 receptor antibodies and increased angiotensin II sensitivity in pregnant rats |
Q100408260 | Angiotensin II type 1 receptor antibodies in kidney transplantation: An evidence-based comprehensive review |
Q37353499 | Angiotensin II type 1 receptor autoantibody (AT1-AA)-mediated pregnancy hypertension |
Q27027039 | Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyond |
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Q38209381 | Clinical applications of biomarkers in preeclampsia |
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Q36836373 | Elevated Transglutaminase Activity Triggers Angiotensin Receptor Activating Autoantibody Production and Pathophysiology of Preeclampsia |
Q33740993 | Excess LIGHT contributes to placental impairment, increased secretion of vasoactive factors, hypertension, and proteinuria in preeclampsia |
Q34072217 | Exposure to AT1 receptor autoantibodies during pregnancy increases susceptibility of the maternal heart to postpartum ischemia-reperfusion injury in rats |
Q50893991 | Fetal sex and the circulating renin-angiotensin system during early gestation in women who later develop preeclampsia or gestational hypertension. |
Q38056887 | From preeclampsia to renal disease: a role of angiogenic factors and the renin-angiotensin aldosterone system? |
Q38151169 | Future therapies for pre-eclampsia: beyond treading water |
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Q36930454 | High viral load and elevated angiogenic markers associated with increased risk of preeclampsia among women initiating highly active antiretroviral therapy in pregnancy in the Mma Bana study, Botswana |
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Q37605722 | Increased risk of systemic lupus erythematosus in pregnancy-induced hypertension: A nationwide population-based retrospective cohort study |
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Q38303854 | Limited evidence for calcium supplementation in preeclampsia prevention: a meta-analysis and systematic review |
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Q89454188 | Lipoxin A4 suppresses angiotensin II type 1 receptor autoantibody in preeclampsia via modulating caspase-1 |
Q64897032 | Loss of placental growth factor ameliorates maternal hypertension and preeclampsia in mice. |
Q55004295 | Maternal plasma fetuin-A concentration is lower in patients who subsequently developed preterm preeclampsia than in uncomplicated pregnancy: a longitudinal study. |
Q46212939 | Maternal-Fetal rejection reactions are unconstrained in preeclamptic women |
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Q54901493 | Molecular Mechanisms of Preeclampsia. |
Q35175458 | Novel retro-inverso peptide inhibitor reverses angiotensin receptor autoantibody-induced hypertension in the rabbit |
Q90259719 | Placental Origins of Preeclampsia: Potential Therapeutic Targets |
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Q33591058 | The detrimental role of angiotensin receptor agonistic autoantibodies in intrauterine growth restriction seen in preeclampsia |
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Q38014802 | The pathophysiology of preeclampsia involves altered levels of angiogenic factors promoted by hypoxia and autoantibody-mediated mechanisms |
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Q37088970 | The role of immune activation in contributing to vascular dysfunction and the pathophysiology of hypertension during preeclampsia |
Q64233038 | Tissue Transglutaminase-Mediated AT1 Receptor Sensitization Underlies Pro-inflammatory Cytokine LIGHT-Induced Hypertension |
Q38493639 | Towards biomarker-based tests that can facilitate decisions about prevention and management of preeclampsia in low-resource settings |
Q37240779 | Transglutaminase is a Critical Link Between Inflammation and Hypertension |
Q44770951 | Treg cells are negatively correlated with increased memory B cells in pre-eclampsia while maintaining suppressive function on autologous B-cell proliferation |
Q38777852 | Understanding mechanisms of hypertension in systemic lupus erythematosus |
Q48025132 | Upregulation of cathepsin C expression contributes to endothelial chymase activation in preeclampsia |
Q36699839 | Vitamin D supplementation improves pathophysiology in a rat model of preeclampsia |
Q51294484 | Vitamin D supplementation reduces some AT1-AA-induced downstream targets implicated in preeclampsia including hypertension. |
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