review article | Q7318358 |
scholarly article | Q13442814 |
P356 | DOI | 10.3109/10641955.2015.1051227 |
P698 | PubMed publication ID | 26153629 |
P50 | author | Annemarie Hennessy | Q37837597 |
Angela Makris | Q76380588 | ||
Shikha Aggarwal | Q114573414 | ||
P2860 | cites work | A leading role for the immune system in the pathophysiology of preeclampsia | Q27003069 |
Soluble endoglin contributes to the pathogenesis of preeclampsia | Q28244053 | ||
Induction of neutrophil extracellular DNA lattices by placental microparticles and IL-8 and their presence in preeclampsia | Q28304349 | ||
Immunobiology of dendritic cells | Q29547465 | ||
Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
Maternal preeclampsia and neonatal outcomes | Q33395211 | ||
The detrimental role of angiotensin receptor agonistic autoantibodies in intrauterine growth restriction seen in preeclampsia | Q33591058 | ||
Angiotensin receptor agonistic autoantibody-mediated tumor necrosis factor-alpha induction contributes to increased soluble endoglin production in preeclampsia | Q33748650 | ||
Angiotensin receptor agonistic autoantibody is highly prevalent in preeclampsia: correlation with disease severity | Q33748680 | ||
Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
Pathogenesis and genetics of pre-eclampsia | Q33934445 | ||
Angiotensin receptor agonistic autoantibody-mediated soluble fms-like tyrosine kinase-1 induction contributes to impaired adrenal vasculature and decreased aldosterone production in preeclampsia | Q34040304 | ||
Recent Insights into the pathogenesis of pre-eclampsia | Q34682287 | ||
Cardiovascular sequelae of preeclampsia/eclampsia: a systematic review and meta-analyses | Q34897462 | ||
Hypertension in response to placental ischemia during pregnancy: role of B lymphocytes | Q35029700 | ||
Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling | Q35681804 | ||
Recombinant vascular endothelial growth factor 121 attenuates autoantibody-induced features of pre-eclampsia in pregnant mice | Q35683468 | ||
Autoantibody-mediated IL-6-dependent endothelin-1 elevation underlies pathogenesis in a mouse model of preeclampsia | Q35715914 | ||
Placental oxidative stress: from miscarriage to preeclampsia | Q35879133 | ||
Autoantibody-mediated angiotensin receptor activation contributes to preeclampsia through tumor necrosis factor-alpha signaling | Q36050771 | ||
IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy | Q36178540 | ||
Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation | Q36244980 | ||
Agonistic antibodies directed at the angiotensin II, AT1 receptor in preeclampsia | Q36380482 | ||
The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy | Q36407259 | ||
Immunoregulation in normal pregnancy and pre-eclampsia: an overview | Q36684096 | ||
Hypoxia and reoxygenation: a possible mechanism for placental oxidative stress in preeclampsia | Q36686796 | ||
The functional role of the renin-angiotensin system in pregnancy and preeclampsia | Q37039872 | ||
Protein composition of microparticles shed from human placenta during placental perfusion: Potential role in angiogenesis and fibrinolysis in preeclampsia | Q37146239 | ||
Autoantibodies to the angiotensin type I receptor in response to placental ischemia and tumor necrosis factor alpha in pregnant rats | Q37436219 | ||
Hypertension in response to autoantibodies to the angiotensin II type I receptor (AT1-AA) in pregnant rats: role of endothelin-1. | Q37447371 | ||
The pathophysiology of preeclampsia involves altered levels of angiogenic factors promoted by hypoxia and autoantibody-mediated mechanisms | Q38014802 | ||
The role of hypoxia and hypoxia-inducible factor-1alpha in preeclampsia pathogenesis | Q38049205 | ||
Vascular endothelial growth factor-A and aldosterone: relevance to normal pregnancy and preeclampsia | Q39185420 | ||
Regulation of placental growth by aldosterone and cortisol | Q39633468 | ||
Control of vascular responsiveness during human pregnancy | Q40122647 | ||
Sera from preeclamptic women specifically activate human umbilical vein endothelial cells in vitro: morphological and biochemical evidence | Q41098649 | ||
Preeclampsia: an endothelial cell disorder | Q41281924 | ||
Placental cytokines and the pathogenesis of preeclampsia | Q41452017 | ||
Lymphocyte intracellular free calcium concentration is increased in preeclampsia | Q42472176 | ||
Hypertension Induced by the Tyrosine Kinase Inhibitor Sunitinib Is Associated With Increased Circulating Endothelin-1 Levels | Q42928512 | ||
Study of plasma factors associated with neutrophil activation and lipid peroxidation in preeclampsia | Q43771322 | ||
Angiotensin type 1 receptor autoantibody from preeclamptic patients induces human fetoplacental vasoconstriction | Q43939628 | ||
Autoantibody against AT1 receptor from preeclamptic patients induces vasoconstriction through angiotensin receptor activation | Q44218760 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human trophoblast cells | Q44320558 | ||
AT1 receptor agonistic antibodies from preeclamptic patients stimulate NADPH oxidase | Q44385661 | ||
Agonistic autoantibodies directed against the angiotensin II AT1 receptor in patients with preeclampsia | Q44413491 | ||
Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation | Q45069619 | ||
Angiotensin II type 1-receptor activating antibodies in renal-allograft rejection | Q45259438 | ||
Increased intracellular free calcium and sensitivity to angiotensin II in platelets of preeclamptic women | Q46136176 | ||
Uteroplacental ischemia results in proteinuric hypertension and elevated sFLT-1. | Q46339760 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human mesangial cells and induce interleukin-6 and plasminogen activator inhibitor-1 secretion | Q46344658 | ||
Recognition of the long-term sequelae of eclampsia | Q50246480 | ||
Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of tumor necrosis factor-alpha. | Q51359651 | ||
Involvement of functional autoantibodies against vascular receptors in systemic sclerosis. | Q52900457 | ||
Immunology in hypertension, preeclampsia, and target-organ damage. | Q52909791 | ||
Role of endothelin in mediating tumor necrosis factor-induced hypertension in pregnant rats. | Q54661322 | ||
Prevalence of agonistic autoantibodies against the angiotensin II type 1 receptor and soluble fms-like tyrosine kinase 1 in a gestational age-matched case study. | Q54782424 | ||
Agonistic Angiotensin II Type 1 Receptor Autoantibodies in Postpartum Women With a History of Preeclampsia | Q56981931 | ||
P433 | issue | 3 | |
P921 | main subject | pre-eclampsia | Q61335 |
maternal health | Q6786626 | ||
P304 | page(s) | 369-382 | |
P577 | publication date | 2015-07-08 | |
P1433 | published in | Hypertension in Pregnancy | Q5958696 |
P1476 | title | Linking the old and new -- do angiotensin II type 1 receptor antibodies provide the missing link in the pathophysiology of preeclampsia? | |
P478 | volume | 34 |
Q39737548 | Anti-Angiotensin II Type 1 Receptor and Anti-Endothelial Cell Antibodies: A Cross-Sectional Analysis of Pathological Findings in Allograft Biopsies | cites work | P2860 |
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