scholarly article | Q13442814 |
P50 | author | Susan M. Ramin | Q110252023 |
P2093 | author name string | Yang Xia | |
Yong-Jian Geng | |||
Tiejuan Mi | |||
Yujin Zhang | |||
Jiaming Wen | |||
Rodney E Kellems | |||
Roxanna A Irani | |||
Sean C Blackwell | |||
Harnath Shelat | |||
Cissy Chenyi Zhou | |||
P2860 | cites work | Endoglin is a component of the transforming growth factor-beta receptor system in human endothelial cells | Q24337230 |
Identification of distinct epitopes of endoglin, an RGD-containing glycoprotein of endothelial cells, leukemic cells, and syncytiotrophoblasts | Q28186129 | ||
Soluble endoglin contributes to the pathogenesis of preeclampsia | Q28244053 | ||
Maternal plasma concentrations of the soluble tumor necrosis factor receptor 2 are increased prior to the diagnosis of preeclampsia | Q33821579 | ||
Pathogenesis and genetics of pre-eclampsia | Q33934445 | ||
Human tumour necrosis factor: physiological and pathological roles in placenta and endometrium | Q34280666 | ||
Latest advances in understanding preeclampsia | Q34425288 | ||
Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling | Q35681804 | ||
Angiotensin receptor agonistic autoantibodies induce pre-eclampsia in pregnant mice | Q35701236 | ||
Induction of placental heme oxygenase-1 is protective against TNFalpha-induced cytotoxicity and promotes vessel relaxation | Q35944003 | ||
Hypertension in response to chronic reductions in uterine perfusion in pregnant rats: effect of tumor necrosis factor-alpha blockade | Q37459545 | ||
Placental cytokines and the pathogenesis of preeclampsia | Q41452017 | ||
Endoglin expression is regulated by transcriptional cooperation between the hypoxia and transforming growth factor-beta pathways | Q42525897 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human trophoblast cells | Q44320558 | ||
AT1 receptor agonistic antibodies from preeclamptic patients stimulate NADPH oxidase | Q44385661 | ||
Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation | Q45069619 | ||
TNF alpha concentrations and mRNA expression are increased in preeclamptic placentas | Q45102738 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human mesangial cells and induce interleukin-6 and plasminogen activator inhibitor-1 secretion | Q46344658 | ||
End-tidal breath carbon monoxide measurements are lower in pregnant women with uterine contractions | Q48002106 | ||
Negative regulation of soluble Flt-1 and soluble endoglin release by heme oxygenase-1. | Q51037240 | ||
Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of tumor necrosis factor-alpha. | Q51359651 | ||
Interleukin-6, tumour necrosis factor and soluble tumour necrosis factor receptors in women with pre-eclampsia | Q52995596 | ||
Soluble endoglin and other circulating antiangiogenic factors in preeclampsia. | Q53600812 | ||
Expression of the placental cytokines tumor necrosis factor alpha, interleukin 1beta, and interleukin 10 is increased in preeclampsia. | Q53680742 | ||
Localization of endoglin, a transforming growth factor-beta binding protein, and of CD44 and integrins in placenta during the first trimester of pregnancy | Q71664957 | ||
Bioactive tumour necrosis factor alpha in pre-eclamptic patients with and without the HELLP syndrome | Q72422330 | ||
AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor | Q73815485 | ||
Expression of inflammatory cytokines in placentas from women with preeclampsia | Q73992120 | ||
Circulating levels of immunoreactive cytokines in women with preeclampsia | Q77384781 | ||
Tumor necrosis factor-alpha in the placenta is not elevated in pre-eclamptic patients despite its elevation in peripheral blood | Q81432957 | ||
TNF-receptor levels in preeclampsia--results of a longitudinal study in high-risk women | Q82158802 | ||
P433 | issue | 3 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | pre-eclampsia | Q61335 |
autoantibody | Q785022 | ||
P304 | page(s) | 436-444 | |
P577 | publication date | 2010-01-11 | |
P1433 | published in | Circulation | Q578091 |
P1476 | title | Angiotensin receptor agonistic autoantibody-mediated tumor necrosis factor-alpha induction contributes to increased soluble endoglin production in preeclampsia | |
P478 | volume | 121 |
Q34870775 | A2B adenosine receptor-mediated induction of IL-6 promotes CKD. |
Q38118625 | Activating autoantibodies and cardiovascular disease |
Q38082526 | Adaptive immune responses during pregnancy |
Q34714082 | Angiogenic factors and preeclampsia |
Q37353499 | Angiotensin II type 1 receptor autoantibody (AT1-AA)-mediated pregnancy hypertension |
Q27027039 | Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyond |
Q46300987 | Animal models of preeclampsia: translational failings and why. |
Q35715914 | Autoantibody-mediated IL-6-dependent endothelin-1 elevation underlies pathogenesis in a mouse model of preeclampsia |
Q34040357 | Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia |
Q45020552 | Differential regulation of TNF receptors in maternal leukocytes is associated with severe preterm preeclampsia |
Q36575572 | Elevated Endothelial Hypoxia-Inducible Factor-1α Contributes to Glomerular Injury and Promotes Hypertensive Chronic Kidney Disease |
Q34072217 | Exposure to AT1 receptor autoantibodies during pregnancy increases susceptibility of the maternal heart to postpartum ischemia-reperfusion injury in rats |
Q36351174 | Four Pathways Involving Innate Immunity in the Pathogenesis of Preeclampsia |
Q35552966 | Foxp3+-inducible regulatory T cells suppress endothelial activation and leukocyte recruitment |
Q38151169 | Future therapies for pre-eclampsia: beyond treading water |
Q48641988 | Interleukin 22 Promotes Blood Pressure Elevation and Endothelial Dysfunction in Angiotensin II-Treated Mice |
Q37346569 | Interleukin 6 underlies angiotensin II-induced hypertension and chronic renal damage |
Q38543831 | Linking the old and new -- do angiotensin II type 1 receptor antibodies provide the missing link in the pathophysiology of preeclampsia? |
Q35926413 | Loss of Akt activity increases circulating soluble endoglin release in preeclampsia: identification of inter-dependency between Akt-1 and heme oxygenase-1. |
Q37832037 | New insights into the etiology of preeclampsia: identification of key elusive factors for the vascular complications |
Q64926884 | Preeclampsia: Novel Mechanisms and Potential Therapeutic Approaches. |
Q36965155 | Preeclampsia: Updates in Pathogenesis, Definitions, and Guidelines |
Q36537709 | RAS in Pregnancy and Preeclampsia and Eclampsia |
Q39136791 | Recent Advances in Immunity and Hypertension |
Q34404445 | Recent insights into the pathophysiology of preeclampsia |
Q35707881 | Receptor-activating autoantibodies and disease: preeclampsia and beyond |
Q35683468 | Recombinant vascular endothelial growth factor 121 attenuates autoantibody-induced features of pre-eclampsia in pregnant mice |
Q35743816 | Renin angiotensin signaling in normal pregnancy and preeclampsia |
Q35591704 | The relevance of the Renin-Angiotensin system in the development of drugs to combat preeclampsia. |
Q26849333 | Therapeutic potential of statins and the induction of heme oxygenase-1 in preeclampsia |
Q38592484 | Thrombosis during pregnancy: Risks, prevention, and treatment for mother and fetus--harvesting the power of omic technology, biomarkers and in vitro or in vivo models to facilitate the treatment of thrombosis. |
Q37240779 | Transglutaminase is a Critical Link Between Inflammation and Hypertension |
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