scholarly article | Q13442814 |
P50 | author | Susan M. Ramin | Q110252023 |
P2093 | author name string | Hong Zhang | |
Yang Xia | |||
Tiejuan Mi | |||
Yujin Zhang | |||
M John Hicks | |||
Rodney E Kellems | |||
Roxanna A Irani | |||
Edwina J Popek | |||
Cissy C Zhou | |||
P2860 | cites work | Angiotensin receptors, autoimmunity, and preeclampsia | Q24605647 |
Soluble endoglin contributes to the pathogenesis of preeclampsia | Q28244053 | ||
Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
Pathogenesis and genetics of pre-eclampsia | Q33934445 | ||
Latest advances in understanding preeclampsia | Q34425288 | ||
Recent Insights into the pathogenesis of pre-eclampsia | Q34682287 | ||
Pathophysiology of preeclampsia: linking placental ischemia/hypoxia with microvascular dysfunction. | Q34702420 | ||
Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling | Q35681804 | ||
Potential roles of angiotensin receptor-activating autoantibody in the pathophysiology of preeclampsia | Q35681821 | ||
Angiotensin II induces soluble fms-Like tyrosine kinase-1 release via calcineurin signaling pathway in pregnancy | Q35699115 | ||
The uterine spiral arteries in human pregnancy: facts and controversies | Q36401771 | ||
The renin-angiotensin-aldosterone system in preeclampsia. A review | Q36453940 | ||
Inflammation and pre-eclampsia | Q36530771 | ||
Reduced uterine perfusion pressure during pregnancy in the rat is associated with increases in arterial pressure and changes in renal nitric oxide. | Q43576536 | ||
Pathophysiology of hypertension during preeclampsia linking placental ischemia with endothelial dysfunction | Q43744794 | ||
High-salt diet enhances vascular reactivity in pregnant rats with normal and reduced uterine perfusion pressure | Q43744797 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human trophoblast cells | Q44320558 | ||
AT1 receptor agonistic antibodies from preeclamptic patients stimulate NADPH oxidase | Q44385661 | ||
Agonistic autoantibodies directed against the angiotensin II AT1 receptor in patients with preeclampsia | Q44413491 | ||
Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation | Q45069619 | ||
Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human mesangial cells and induce interleukin-6 and plasminogen activator inhibitor-1 secretion | Q46344658 | ||
Fetal growth retardation in infants of multiparous and nulliparous women with preeclampsia | Q46665406 | ||
Enhanced endothelin synthesis by endothelial cells exposed to sera from pregnant rats with decreased uterine perfusion | Q46877931 | ||
Circulating angiogenic factors and the risk of preeclampsia | Q47217679 | ||
Hypertension produced by reductions in uterine perfusion in the pregnant rat: role of tumor necrosis factor-alpha. | Q51359651 | ||
Agonistic autoantibodies to the AT1 receptor in a transgenic rat model of preeclampsia. | Q52940276 | ||
The preeclampsia enigma and the renin-angiotensin system. | Q52972348 | ||
Soluble endoglin and other circulating antiangiogenic factors in preeclampsia. | Q53600812 | ||
Role of endothelin in mediating tumor necrosis factor-induced hypertension in pregnant rats. | Q54661322 | ||
Urinary Placental Growth Factor and Risk of Preeclampsia | Q56619498 | ||
Dysregulation of the Circulating and Tissue-Based Renin-Angiotensin System in Preeclampsia | Q56981959 | ||
Pre-eclampsia in second pregnancy | Q70057283 | ||
Hypertension induced in pregnant mice by placental renin and maternal angiotensinogen | Q71695937 | ||
Endothelin type a receptor blockade attenuates the hypertension in response to chronic reductions in uterine perfusion pressure | Q73567364 | ||
AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor | Q73815485 | ||
Hypertension produced by reduced uterine perfusion in pregnant rats is associated with increased soluble fms-like tyrosine kinase-1 expression | Q81411178 | ||
Angiotensin II type 1 receptor agonistic antibodies reflect fundamental alterations in the uteroplacental vasculature | Q81423834 | ||
P433 | issue | 8 | |
P407 | language of work or name | English | Q1860 |
P921 | main subject | pre-eclampsia | Q61335 |
autoantibody | Q785022 | ||
maternal health | Q6786626 | ||
P304 | page(s) | 855-862 | |
P577 | publication date | 2008-07-27 | |
P1433 | published in | Nature Medicine | Q1633234 |
P1476 | title | Angiotensin receptor agonistic autoantibodies induce pre-eclampsia in pregnant mice | |
P478 | volume | 14 |
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Q36050771 | Autoantibody-mediated angiotensin receptor activation contributes to preeclampsia through tumor necrosis factor-alpha signaling |
Q34040357 | Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia |
Q94311716 | Autoimmune disease: pre-eclampsia IgG produces a murine model of maternal morbidity |
Q36357099 | Biomarker development for presymptomatic molecular diagnosis of preeclampsia: feasible, useful or even unnecessary? |
Q37699015 | Characterization of antibody specificities associated with preeclampsia |
Q79803704 | Common reproductive disorders may have immunological basis |
Q30409861 | Complement activation is critical for placental ischemia-induced hypertension in the rat. |
Q51464431 | Complement component C1q as potential diagnostic but not predictive marker of preeclampsia. |
Q38220404 | Constitutive activity in the angiotensin II type 1 receptor: discovery and applications |
Q50131577 | Current model systems for the study of preeclampsia. |
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Q34544386 | Differential expression of microRNAs in decidua-derived mesenchymal stem cells from patients with pre-eclampsia |
Q34731047 | Disrupted balance of angiogenic and antiangiogenic signalings in preeclampsia |
Q35740222 | Domain coupling in GPCRs: the engine for induced conformational changes |
Q92235464 | Early Acute Microvascular Kidney Transplant Rejection in the Absence of Anti-HLA Antibodies Is Associated with Preformed IgG Antibodies against Diverse Glomerular Endothelial Cell Antigens |
Q39450324 | Effect on the production of soluble endoglin from human choriocarcinoma cells by preeclampsia sera |
Q33392329 | Effects of hypoxia-inducible factor-1alpha overexpression in pregnant mice: possible implications for preeclampsia and intrauterine growth restriction |
Q36575572 | Elevated Endothelial Hypoxia-Inducible Factor-1α Contributes to Glomerular Injury and Promotes Hypertensive Chronic Kidney Disease |
Q36836373 | Elevated Transglutaminase Activity Triggers Angiotensin Receptor Activating Autoantibody Production and Pathophysiology of Preeclampsia |
Q36575544 | Elevated placental adenosine signaling contributes to the pathogenesis of preeclampsia |
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Q33740993 | Excess LIGHT contributes to placental impairment, increased secretion of vasoactive factors, hypertension, and proteinuria in preeclampsia |
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