| scholarly article | Q13442814 |
| P2093 | author name string | Babbette LaMarca | |
| Denise C Cornelius | |||
| Mark W Cunningham | |||
| Ralf Dechend | |||
| Gerd Wallukat | |||
| Lorena Amaral | |||
| Nathan Usry | |||
| Nathan Campbell | |||
| Tarek Ibrahim | |||
| Venkata Ramana Vaka | |||
| Kristen McMaster | |||
| Shyanne McDuffy | |||
| P2860 | cites work | Changes of NK cells in preeclampsia | Q37987977 |
| Maternal autoantibodies from preeclamptic patients activate angiotensin receptors on human trophoblast cells | Q44320558 | ||
| Peripheral natural killer cell activity as a predictor of recurrent pregnancy loss: a large cohort study | Q44562312 | ||
| Death rates from ischemic heart disease in women with a history of hypertension in pregnancy | Q44845053 | ||
| Antibodies from preeclamptic patients stimulate increased intracellular Ca2+ mobilization through angiotensin receptor activation | Q45069619 | ||
| Placental mitochondria as a source of oxidative stress in pre-eclampsia | Q45115339 | ||
| Preeclampsia and the risk of end-stage renal disease | Q45748808 | ||
| Granzyme A induces caspase-independent mitochondrial damage, a required first step for apoptosis | Q46394719 | ||
| Angiotensin II receptor type 1 autoantibodies promote endothelial microparticles formation through activating p38 MAPK pathway | Q46922047 | ||
| The prognostic value of uNK cell count and histological dating in the mid-luteal phase of women with reproductive failure. | Q47214014 | ||
| Natural killer cells mediate pathophysiology in response to reduced uterine perfusion pressure | Q47637232 | ||
| Impairment of endothelial function in women with a history of preeclampsia: an indicator of cardiovascular risk. | Q51942463 | ||
| AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic Rats. | Q53700889 | ||
| Endometrial T, B, and NK cells in patients with recurrent spontaneous abortion. Altered profile and pregnancy outcome | Q71059494 | ||
| AT(1) receptor agonistic antibodies from preeclamptic patients cause vascular cells to express tissue factor | Q73815485 | ||
| Novel monoclonal antibodies against membrane structures that are preferentially expressed on IL-2-activated rat NK cells | Q74191520 | ||
| Changes in cardiac structure and function in rats immunized by angiotensin type 1 receptor peptides | Q82259476 | ||
| Women with a history of preeclampsia should be monitored for the onset and progression of chronic kidney disease | Q82570697 | ||
| Hypertension in response to AT1-AA: role of reactive oxygen species in pregnancy-induced hypertension | Q83799951 | ||
| Angiotensin II type I receptor agonistic autoantibody-induced apoptosis in neonatal rat cardiomyocytes is dependent on the generation of tumor necrosis factor-α | Q95445011 | ||
| Nitroso-redox balance and mitochondrial homeostasis are regulated by STOX1, a pre-eclampsia-associated gene | Q24294779 | ||
| How mitochondria produce reactive oxygen species | Q24643882 | ||
| Preeclampsia: long-term consequences for vascular health | Q26800272 | ||
| Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyond | Q27027039 | ||
| Bioactive factors in uteroplacental and systemic circulation link placental ischemia to generalized vascular dysfunction in hypertensive pregnancy and preeclampsia | Q28083815 | ||
| Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia | Q29615916 | ||
| Patients with preeclampsia develop agonistic autoantibodies against the angiotensin AT1 receptor | Q33848380 | ||
| Agonistic Autoantibodies to the Angiotensin II Type 1 Receptor Enhance Angiotensin II-Induced Renal Vascular Sensitivity and Reduce Renal Function During Pregnancy | Q34541831 | ||
| Preeclampsia and Stroke: Risks during and after Pregnancy | Q34557552 | ||
| Comparative proteomics analysis suggests that placental mitochondria are involved in the development of pre-eclampsia | Q34721353 | ||
| Novel retro-inverso peptide inhibitor reverses angiotensin receptor autoantibody-induced hypertension in the rabbit | Q35175458 | ||
| Granzyme B-induced mitochondrial ROS are required for apoptosis | Q35349538 | ||
| Autoantibody from women with preeclampsia induces soluble Fms-like tyrosine kinase-1 production via angiotensin type 1 receptor and calcineurin/nuclear factor of activated T-cells signaling | Q35681804 | ||
| Angiotensin receptor agonistic autoantibodies induce pre-eclampsia in pregnant mice | Q35701236 | ||
| Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic rats | Q35994304 | ||
| IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy | Q36178540 | ||
| A case-control study of maternal blood mitochondrial DNA copy number and preeclampsia risk | Q36282515 | ||
| The effect of immune factors, tumor necrosis factor-alpha, and agonistic autoantibodies to the angiotensin II type I receptor on soluble fms-like tyrosine-1 and soluble endoglin production in response to hypertension during pregnancy | Q36407259 | ||
| Risk factors for myocardial infarction in young women | Q36681069 | ||
| Angiotensin II type 1 receptor antibodies and increased angiotensin II sensitivity in pregnant rats | Q36683260 | ||
| Cardiovascular sequelae of toxaemia of pregnancy | Q36840471 | ||
| Regulatory T cells and regulatory natural killer (NK) cells play important roles in feto-maternal tolerance | Q36875737 | ||
| IL-10 supplementation increases Tregs and decreases hypertension in the RUPP rat model of preeclampsia | Q36997267 | ||
| Maternal blood mitochondrial DNA copy number and placental abruption risk: results from a preliminary study | Q37004984 | ||
| Identifying immune mechanisms mediating the hypertension during preeclampsia | Q37139722 | ||
| Angiotensin II type 1 receptor autoantibody (AT1-AA)-mediated pregnancy hypertension | Q37353499 | ||
| Endothelin-1, oxidative stress, and endogenous angiotensin II: mechanisms of angiotensin II type I receptor autoantibody-enhanced renal and blood pressure response during pregnancy | Q37353537 | ||
| Hypertension in response to autoantibodies to the angiotensin II type I receptor (AT1-AA) in pregnant rats: role of endothelin-1. | Q37447371 | ||
| Placental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia | Q37451544 | ||
| Evidence for participation of uterine natural killer cells in the mechanisms responsible for spontaneous preterm labor and delivery. | Q37483436 | ||
| The Endothelin Type A Receptor as a Potential Therapeutic Target in Preeclampsia | Q37729072 | ||
| Uterine and circulating natural killer cells and their roles in women with recurrent pregnancy loss, implantation failure and preeclampsia | Q37883647 | ||
| P304 | page(s) | 72-77 | |
| P577 | publication date | 2018-11-30 | |
| P1433 | published in | Pregnancy hypertension | Q27723626 |
| P1476 | title | Renal natural killer cell activation and mitochondrial oxidative stress; new mechanisms in AT1-AA mediated hypertensive pregnancy | |
| P478 | volume | 15 |
| Q90106114 | Letter to the Editor: Importance of B cells in response to placental ischemia | cites work | P2860 |
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